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1 and no cases following live attenuated 2009 H1N1 vaccine.
2 cords on more than 2.6 million doses of 2009 H1N1 vaccine.
3 en <10 years of age required 2 doses of 2009 H1N1 vaccine.
4 accine than against the homotypic seasonal A(H1N1) vaccine.
5 l, 39 726 infants were prenatally exposed to H1N1 vaccine (13 845 during the first trimester) and 29
8 few studies examine the immune responses to H1N1 vaccine among children and adults of various body m
9 e no significant differences in responses to H1N1 vaccine among children or adults of various BMI fol
11 o those against the heterovariant seasonal A(H1N1) vaccine and were similar between young and elderly
12 monovalent 2009 H1N1 influenza vaccine (2009 H1N1 vaccine) and seasonal trivalent inactivated influen
13 re immunized with commercial monovalent 2009 H1N1 vaccine, and antigen-specific antibody responses an
14 These changes allowed direct delivery of an H1N1 vaccine antigen and a diclofenac nanotherapeutic in
17 We administered 1 or 2 doses of pandemic H1N1 vaccine (CA/7 ca), a seasonal trivalent inactivated
18 tantly, vaccination of pigs with a ty/04 att-H1N1 vaccine candidate resulted in sterilizing immunity
20 responses and protectiveness induced by 2009 H1N1 vaccine could be extremely compromised in diet-indu
21 /34, A/Texas/36/91, or A/New Caledonia/20/99 H1N1 vaccines displayed partial protection from lethal c
22 ing exposed and unexposed to the influenza A(H1N1) vaccine during pregnancy were matched 1:4 on prope
25 rate that vaccination with the 2009 pandemic H1N1 vaccine elicits 1918 virus cross-protective antibod
26 CR) serum antibodies that recognize both the H1N1 (vaccine-H1) and H3N2 (vaccine-H3) components of an
27 potential adverse effects of the pandemic A(H1N1) vaccine have supported that influenza A(H1N1) vacc
28 lammatory occlusive retinal vasculitis, post-H1N1 vaccine, hypertensive retinopathy, migraine disorde
31 led, observer-blind clinical trial of a 2009 H1N1 vaccine in 1313 young (age, 18-64 years) and older
32 the long-term antibody response to the 2009-H1N1 vaccine in solid organ transplant recipients (SOTR)
33 that of 2 doses of nonadjuvanted influenza A(H1N1) vaccine in children 6 months to <10 years of age i
34 ratio following receipt of inactivated 2009 H1N1 vaccine (incidence rate ratio = 2.50, 95% confidenc
35 le 7.5-mug dose of a monovalent unadjuvanted H1N1 vaccine induced protective HI antibody levels in ad
37 mained whether the inactivated 2009 pandemic H1N1 vaccine, like the infection, could also induce thes
38 estern state reported receiving the pandemic H1N1 vaccine, physicians and nurses differed significant
39 ng for a diagnosis of asthma, 1 dose of 2009 H1N1 vaccine provided 69% protection (95% confidence int
40 1 or 2 doses of AS03-adjuvanted influenza A(H1N1) vaccine relative to that of 2 doses of nonadjuvant
41 and cellular phenotypes play in influenza A(H1N1) vaccine responsiveness in HIV-infected subjects re
44 rization motifs and NA domains from a recent H1N1 vaccine strain and compared for enzymatic propertie
46 cine (fluvirin: 18 mug of haemagglutinin per H1N1 vaccine strain, 17 mug of haemagglutinin per H3N2 v
49 similar to that of the A/New Caledonia/20/99(H1N1) vaccine strain both antigenically and genetically,
50 ponses against the 2009 pandemic influenza A(H1N1) vaccine strain were significantly enhanced, compar
53 ranasal immunization with PS-GAMP-adjuvanted H1N1 vaccine, strong cross-protection was elicited again
54 e response to a single dose of pandemic 2009 H1N1 vaccine suggests that a large segment of the popula
57 controlled trial of a monovalent influenza A H1N1 vaccine to establish the safety and immunogenicity
62 ol studies demonstrated that monovalent 2009 H1N1 vaccines were effective in preventing infection wit