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1  into recombinant A/Vietnam/1203/04 (VN1203) H5N1 influenza virus.
2 ckens (B(2)/B(2)) protected them from lethal H5N1 influenza virus.
3  protected mice from a lethal challenge with H5N1 influenza virus.
4 ted with a lethal dose of A/Vietnam/1203/04 (H5N1) influenza virus.
5 ibited the N1 NAs of highly pathogenic avian H5N1 influenza viruses.
6 for a few persistent host markers than avian H5N1 influenza viruses.
7 otential as live attenuated vaccines against H5N1 influenza viruses.
8 1) against antigenically distinct strains of H5N1 influenza viruses.
9  duck has become the "Trojan horse" of Asian H5N1 influenza viruses.
10 ogens and stimulate cross-protection against H5N1 influenza viruses.
11 uation and spread of these highly pathogenic H5N1 influenza viruses.
12 d create a situation for the perpetuation of H5N1 influenza viruses.
13  we developed two antibodies that neutralize H5N1 influenza viruses.
14 se the transmissibility of highly pathogenic H5N1 influenza viruses.
15 ed with 10-100 MLD50 of H1N1, H3N1, H3N2 and H5N1 influenza viruses.
16 le in the pathogenicity of highly pathogenic H5N1 influenza viruses.
17 caused by the highly pathogenic avian (HPAI) H5N1 influenza viruses.
18 and risk assessment of currently circulating H5N1 influenza viruses.
19 ion of the lethally infected mice by H1N1 or H5N1 influenza viruses.
20 mic virus as well as heterosubtypic H3N2 and H5N1 influenza viruses.
21 ent neutralizing antibody responses to avian H5N1 influenza viruses.
22 ng the pandemic potential of recent Egyptian H5N1 influenza viruses.
23 enuated vaccine strains based on recombinant H5N1 influenza virus A/Viet Nam/1203/04 was generated.
24   In this report, the natural history of the H5N1 influenza virus A/Vietnam/1203/04 influenza infecti
25 ponse to human infections caused by an avian H5N1 influenza virus, A/Hong Kong/213/03.
26 he pandemic potential of widely disseminated H5N1 influenza viruses, a ferret contact model using exp
27 apable of protecting from infection with the H5N1 influenza viruses actively circulating in dairy cat
28                      A sustained outbreak of H5N1 influenza virus among wild fowl and domestic livest
29 e dose of an antigenically variant strain of H5N1 influenza virus and could be a useful strategy for
30 tively replicate in macrophages is unique to H5N1 influenza viruses and may contribute to their incre
31 H5 HA receptor-binding site that neutralized H5N1 influenza viruses and pseudoviruses carrying the HA
32 e immunity against the currently circulating H5N1 influenza viruses and that this protective immunity
33 eriodic outbreaks of highly pathogenic avian H5N1 influenza viruses and the current H1N1 pandemic hig
34                    The high pathogenicity of H5N1 influenza viruses and their capacity for transmissi
35 specificities of two highly efficacious anti-H5N1 influenza virus antibodies into a bispecific FcDART
36 results show that the PA and PB1 genes of HP H5N1 influenza viruses are associated with lethality in
37                                        Avian H5N1 influenza viruses are circulating widely in cattle
38                      Highly pathogenic avian H5N1 influenza viruses are now widespread in poultry in
39                      We also show that human H5N1 influenza viruses are significantly more likely to
40 , clinical, and laboratory data suggest that H5N1 influenza viruses are transmitted through and predo
41                          The spread of avian H5N1 influenza viruses around the globe has become a wor
42      This study is the first to characterize H5N1 influenza viruses as the only subtype of influenza
43 ot be as effective against highly pathogenic H5N1 influenza viruses as they are against less pathogen
44                   To determine whether avian H5N1 influenza viruses associated with human infections
45 ed complete protection against a spectrum of H5N1 influenza viruses at a single low dose.
46  of malaria falciparum (LSA-NRC) or HA1 from H5N1 influenza virus ('avian flu'), the system selected
47                                           If H5N1 influenza viruses become transmissible among humans
48 e substantial efforts to control and contain H5N1 influenza viruses, bird flu viruses continue to spr
49  earlier survived the lethal challenge of an H5N1 influenza virus, but infected birds shed H5N1 influ
50 n subject demonstrates that vaccination with H5N1 influenza virus can elicit B cells expressing stem
51 in late 2002, outbreaks of highly pathogenic H5N1 influenza virus caused deaths among wild migratory
52 tected ferrets against lethal heterosubtypic H5N1 influenza virus challenge despite the absence of de
53 otection against a lethal A/Duck/Laos/25/06 (H5N1) influenza virus challenge, with no evidence of mor
54 ction against heterosubtypic H1N1, H3N2, and H5N1 influenza virus challenges.
55        Testing approved antivirals against A(H5N1) influenza viruses circulating in peridomestic spec
56 r of human infections with highly pathogenic H5N1 influenza viruses continues to rise, these viruses
57 ted that exposure of the digestive system to H5N1 influenza viruses could initiate infection either t
58                                        Avian H5N1 influenza viruses currently circulating in southeas
59 H274Y NA mutation affects the fitness of two H5N1 influenza viruses differently.
60 us to ask whether waterfowl are resistant to H5N1 influenza virus disease and whether they can still
61 highlights the exceedingly low prevalence of H5N1 influenza virus during the 2024-2025 respiratory se
62      Currently circulating highly pathogenic H5N1 influenza viruses encode an NS1 protein with the ES
63  on the contribution of HA acid stability to H5N1 influenza virus fitness and transmissibility in mam
64    The data suggest that adaptation of avian H5N1 influenza virus for infection in mammals is support
65         The isolation of a highly pathogenic H5N1 influenza virus from poultry indicates that such vi
66                          The transmission of H5N1 influenza viruses from birds to humans poses a sign
67 hallenge with mouse-adapted EBOV or selected H5N1 influenza viruses from clades 0, 1, and 2.
68                      Animals challenged with H5N1 influenza virus greater than 3 months after recover
69 n IAVs, recent isolates of highly pathogenic H5N1 influenza viruses had a high proportion of PB1-F2 t
70 s of oseltamivir-resistant highly pathogenic H5N1 influenza viruses has important clinical implicatio
71      Although the pathogenic mechanism(s) of H5N1 influenza viruses has not been fully elucidated, it
72 ction of humans with highly pathogenic avian H5N1 influenza viruses has suggested viral mutation as o
73 uman infections with avian and bovine origin H5N1 influenza viruses have been documented, raising con
74 uch mechanisms and virulent determinants for H5N1 influenza viruses have not been fully elucidated.
75                      Highly pathogenic avian H5N1 influenza viruses have sporadically transmitted to
76 rus-like particles (VLP) (M8-VLP) expressing H5N1 influenza virus hemagglutinin (HA) and neuraminidas
77 e backbone of DNA vaccine vectors expressing H5N1 influenza virus hemagglutinin (HA).
78 HA protein influence the fitness of an avian H5N1 influenza virus in a mammalian model, we infected C
79             Recurrent outbreaks of the avian H5N1 influenza virus in Asia represent a constant global
80       We show that inhalation of aerosolized H5N1 influenza virus in cynomolgus macaques results in f
81 fluences the properties of highly pathogenic H5N1 influenza virus in mammalian hosts.
82  influenza virus did not cross-react with an H5N1 influenza virus in neutralization or hemagglutinati
83 acheal inoculation of a liquid suspension of H5N1 influenza virus in nonhuman primates likely results
84 d with recent, widely circulating strains of H5N1 influenza virus in poultry, the recurring introduct
85 tation was found to enhance the growth of an H5N1 influenza virus in the mammalian upper respiratory
86 5 respiratory season.IMPORTANCEThe spread of H5N1 influenza virus in the United States has led to the
87 5N1 influenza virus, but infected birds shed H5N1 influenza virus in their feces.
88 vestigated serologic evidence of exposure to H5N1 influenza virus in Vietnamese pigs in 2004.
89 lay critical roles in the virulence of avian H5N1 influenza viruses in a mammalian host in vitro and
90               Outbreaks of highly pathogenic H5N1 influenza viruses in avian species began in Asia an
91                  The widespread incidence of H5N1 influenza viruses in bird populations poses risks t
92 read distribution of highly pathogenic avian H5N1 influenza viruses in domesticated and wild birds co
93 otein plays a key role in the propagation of H5N1 influenza viruses in ducks and may be a novel molec
94                    Despite the prevalence of H5N1 influenza viruses in global avian populations, comp
95 findings indicate that the human and poultry H5N1 influenza viruses in Hong Kong in 1997 were reassor
96   Since the reemergence of highly pathogenic H5N1 influenza viruses in humans in 2003, these viruses
97 plain the mechanism of the high virulence of H5N1 influenza viruses in humans.
98 temic replication and pathogenicity of these H5N1 influenza viruses in mice.
99 rotected chickens from lethal infection with H5N1 influenza viruses in the Hong Kong markets in 1997
100 he likely determinants of virulence of human H5N1 influenza viruses in this model.
101  current great genetic diversity in H9N2 and H5N1 influenza viruses in this region.
102                        The 2004 outbreaks of H5N1 influenza viruses in Vietnam and Thailand were high
103 ntinuing evolution of highly pathogenic (HP) H5N1 influenza viruses in wild birds with transmission t
104 e spread of highly pathogenic clade 2.3.4.4b H5N1 influenza viruses in wild birds, there have been nu
105 ng lethal infection with highly pathogenic A(H5N1) influenza virus in mice.
106                             We conclude that H5N1 influenza virus-induced pathology is affected by a
107 tes, and administration of STAg 2 days after H5N1 influenza virus infection enhanced survival, lowere
108 asma gondii prior to highly pathogenic avian H5N1 influenza virus infection led to decreased lung vir
109         Patients receiving NA inhibitors for H5N1 influenza virus infection should be closely monitor
110 enous IFN-gamma alone enhanced survival from H5N1 influenza virus infection, although not to the same
111                            Following H1N1 or H5N1 influenza virus infection, ferrets were found to re
112 atory cytokines are markedly elevated during H5N1 influenza virus infection, the "cytokine storm" is
113 TNF-alpha may contribute to morbidity during H5N1 influenza virus infection, while IL-1 may be import
114                               However, avian H5N1 influenza virus infections can result in prolonged
115 he protective immune response against severe H5N1 influenza virus infections even when a single dose
116 y differs from human highly pathogenic avian H5N1 influenza virus infections, there is a similar rapi
117 a viruses in protecting chickens from lethal H5N1 influenza virus infections.
118               The A/VN/1203/04 strain of the H5N1 influenza virus is capable of infecting the CNS of
119           Control of highly pathogenic avian H5N1 influenza viruses is a major public-health concern.
120 olution, from a highly pathogenic Vietnamese H5N1 influenza virus, is more related to the 1918 and ot
121 type, we cloned the human A/Vietnam/1203/04 (H5N1) influenza virus isolate that is highly pathogenic
122           Here, we propose that the PA of an H5N1 influenza virus isolated from a human in Vietnam (A
123 report that the polymerase PB2 protein of an H5N1 influenza virus isolated from a human in Vietnam (A
124 ared to those of a highly pathogenic chicken H5N1 influenza virus isolated from Hong Kong in April 19
125 ulated mallards with antigenically different H5N1 influenza viruses isolated between 1997 and 2003.
126 cell-mediated immune responses against avian H5N1 influenza viruses isolated from people.
127 ra tested, 8 (0.25%) were positive for avian H5N1 influenza viruses isolated in 2004 by virus neutral
128 oculated juvenile mallards with 23 different H5N1 influenza viruses isolated in Asia between 2003 and
129                     Despite harboring lethal H5N1 influenza viruses, most chickens in the Hong Kong p
130              After intranasal challenge with H5N1 influenza virus, naive ferrets rapidly succumbed to
131                  A/Goose/Guangdong/1/96-like H5N1 influenza viruses now circulating in southeastern C
132       There were no cross-reactions with non-H5N1 influenza viruses or other avian viruses.
133                               Since the 1997 H5N1 influenza virus outbreak in humans and poultry in H
134                                  In 1997, an H5N1 influenza virus outbreak occurred in chickens in Ho
135 ong poultry and wild birds in the face of an H5N1 influenza virus outbreak.
136 ntification of amino-acid mutations in avian H5N1 influenza virus polymerase complexes that confer in
137                                              H5N1 influenza viruses pose a pandemic threat but have n
138                                      Avian A/H5N1 influenza viruses pose a pandemic threat.
139                                      Avian A(H5N1) influenza virus poses an elevated zoonotic threat
140                      Highly pathogenic avian H5N1 influenza viruses preferentially infect alveolar ty
141                      Highly pathogenic avian H5N1 influenza viruses remain a pandemic threat.
142  the digestive tract is not the main site of H5N1 influenza virus replication in ducks and that the f
143    The threat posed by the highly pathogenic H5N1 influenza virus requires public health authorities
144                      Highly pathogenic avian H5N1 influenza viruses sometimes transmit to humans and
145 ith the highly pathogenic A/Vietnam/1203/04 (H5N1) influenza virus strain; the vaccines encoded influ
146 y a subset of highly pathogenic avian (HPAI) H5N1 influenza virus strains could productively replicat
147 09 H1N1 and 1918), and potentially pandemic (H5N1) influenza virus strains.
148 l genes of Qa/HK/G1/97 virus to those of the H5N1 influenza viruses suggests that the quail virus may
149 searchers announced that they had created an H5N1 influenza virus that was transmissible between ferr
150                            The origin of the H5N1 influenza viruses that killed six of eighteen infec
151 recent emergence of highly pathogenic avian (H5N1) influenza viruses, their epizootic and panzootic n
152 n of DNA target sequences derived from avian H5N1 influenza virus to gold surface-attached single-str
153  is essential for the adaptation of pandemic H5N1 influenza virus to humans.
154 onal transmission of highly pathogenic avian H5N1 influenza viruses to humans causes severe pneumonia
155      Multiple cases of transmission of avian H5N1 influenza viruses to humans illustrate the urgent n
156                The factors that enable avian H5N1 influenza viruses to replicate in humans are not co
157                               In 1997, avian H5N1 influenza virus transmitted from chickens to humans
158                                              H5N1 influenza viruses transmitted from poultry to human
159                                          The H5N1 influenza viruses transmitted to humans in 1997 wer
160                     Here we show that lethal H5N1 influenza viruses, unlike other human, avian and sw
161           In 1997, a highly pathogenic avian H5N1 influenza virus was transmitted directly from live
162 oadly specific against antigenically drifted H5N1 influenza viruses, we developed two neutralizing mo
163 late the acid stability of the HA protein of H5N1 influenza viruses, we performed a mutational analys
164 or by aerosols with a human (H3N2) or avian (H5N1) influenza virus, we demonstrate that aerosol inocu
165                                          The H5N1 influenza virus, which killed humans and poultry in
166                                              H5N1 influenza viruses, which cause disease in humans, h
167 54074/2015 (H1N1), and A/Viet Nam/1203/2004 (H5N1) influenza viruses, with NA-neutralizing activity a
168 n fully inactivated pandemic H1N1 and bovine H5N1 influenza viruses yet preserved hemagglutinin (HA)

 
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