ライフサイエンスコーパス: HS

1 HS appears to be a primary abnormality in the pilosebace

2 HS diet significantly increased the gene expression of p

3 HS has profound negative effects on patients' quality of

4 HS in DSW appeared to lower starch synthesis causing pro

5 HS is a ubiquitous glycosaminoglycan located on the cell

6 HS is characterised by extensive neuronal loss in differ

7 HS is reported to negatively affect the bi-directional c

8 HS oligosaccharides may represent a potential new therap

9 HS production around the lesion allows Sonic hedgehog (S

10 HS was defined as new onset altered mental status after

11 HS-AFM data reveal that the CNTPs undergo diffusive moti

12 HS-AFM experiments, supported by molecular dynamics simu

13 HS-SPME coupled with in-situ derivatization was more str

14 HS-SPME-MS-enose turned out to be a fast, cost-effective

15 y as much as 13.2% when forming MoSSe/MoS(2) HS as compared to the pristine MoS(2) counterparts.

16 There were 29 HS and 79 non-HS recipients included.

17 During Heinrich Stadial 3 (HS 3) we reconstruct more open and dry conditions, compa

18 This study applies a HS-SPME-MS-enose in combination with chemometrics to obt

19 ed cCMVI screening among neonates who fail a HS could be a reasonable, cost-effective strategy to ide

20 inhibiting hexasaccharides identified from a HS oligosaccharide library screen demonstrates that the

21 lecule affecting conformational changes on a HS oligosaccharide.

22 ed anti-TNF therapy, respectively, achieving HS clinical response after 12 weeks of treatment.

23 data from 2009 to 2017 to study 39 907 adult HS (cPRA >= 80%) waitlisted candidates and 19 003 recipi

24 However, these changes affected HS recipients differently based on cPRA.

25 e 12-56-fold and 3-7-fold higher 1-2 h after HS for HSPA1B transgenes and endogenous genes, respectiv

26 of DDKT and KPD increased over time for all HS candidates (adjusted subhazard ratio [aSHR] Era 3 vs

27 s 1 and 2 (TNFR1 and TNFR2), CD44, or alpha2-HS glycoprotein (AHSG), all of which are involved in the

28 ver, rejection-free survival was lower among HS recipients converted within the first-year posttransp

29 conversion to belatacept over 5 years among HS and non-HS recipients.

30 An HS-SPME-GC-MS method was optimized for their quantitatio

31 ortex and medulla in SS(Nox4-/-) rats fed an HS diet.

32 , differed between IBS patients (n = 17) and HS (n = 20).

33 eas, Q(VA) was higher throughout both EX and HS (EX: Delta16 +/- 21% vs. HS: Delta16 +/- 23%, time ef

34 The Q(ECA) increased throughout both EX and HS (EX: Delta42 +/- 58% vs. HS: Delta53 +/- 28%, time ef

35 The Q(ICA) was unchanged with EX and HS interventions (P = 0.665), consistent with the unchan

36 ncreased levels of DII:VENUS, IAA18:GUS, and HS::AXR3-NT:GUS were also observed in tni, perhaps due t

37 ZIKV NS1 induced shedding of HA and HS and altered expression of CD44 and lymphatic endothel

38 argeted methods, i.e. (1)H NMR, LC-HRMS, and HS-SPME/MS-eNose, combined with chemometrics, were used

39 ssed high spin (HS, S = 2), and mixed LS and HS states.

40 of SIBO was similar between IBS patients and HS.

41 gioedema, pemphigus, bullous pemphigoid, and HS.

42 were significantly different between PF and HS animals (P < 0.05).

43 was measured using the sensory technique and HS-SPME-GC/MS analyses.

44 ion in both schizophrenia patients (WIN) and HS (WIN and QuickSIN), as well as auditory frequency mod

45 In both CPs and HSs, the incidence of large slow waves within individual

46 groups, with similar values in patients and HSs.

47 tterns and show that synthetic anticoagulant HS oligosaccharides limit liver ischemia reperfusion inj

48 HRMS, while the flavor was mapped via aroma (HS-SPME-GC-MS) and generic descriptive analysis (trained

49 over 5 years follow-up were compared between HS and non-HLA-sensitized (non-HS) recipients using the

50 The new DTI features can distinguish between HS and FTD subgroups with an accuracy of 76%.

51 iogeochemistry to study interactions between HSs and N cycling.

52 assess the classification power for binary (HS vs. FTD), and multiclass (HS and FTD subtypes) classi

53 Volatile compounds were analysed by HS-SPME GC-MS; an expert panel performed sensory analysi

54 f wort and beer flavour-related compounds by HS-SPME followed by GC-MS quantification, no generalized

55 s in milk after LPS treatment was delayed by HS.

56 87.5%; 95% CI, 46.6-99.7) were identified by HS.

57 abolomics revealed that citrate increased by HS, whereas choline, phosphocholine, N-acetylcarbohydrat

58 Here, carefully characterized HS (n = 20) and schizophrenia patients (n = 22) were tes

59 lized and cheap sources viz: Horse chestnut (HS), Water chestnut (WS) and Lotus stem (LS) by using mi

60 lation rodent model which displays classical HS damage similar to that found in patients with MTLE.

61 s the release of viral particles by cleaving HS.

62 ns against known values for 36 fully defined HS saccharide structures (from di- to decasaccharides) p

63 array and SPR assays of structurally defined HS oligosaccharides show that a rare 3-O-sulfation (3-O-

64 dual column/dual flame ionization detector (HS-GC-FID/FID), a technique routinely used in forensic t

65 who received empiric therapy did not develop HS.

66 3 patients developed HS a median of 7 days post-transplant; 2 died of HS.

67 P was relatively unchanged, forming diffuse, HS-deficient deposits in both the Prnp180Q/196Q and WT m

68 , the effect of fiber-sample distance during HS-SPME in pre-equilibrium conditions.

69 T(skin) and T(core) increased during HS (P < 0.01) but were not different between conditions

70 W(skin) was elevated in HIGH vs. LOW during HS (60 min: by + 0.06 +/- 0.07 a.u., P <= 0.04).

71 clearly impacts the results obtained during HS-SPME when conditions are such that no equilibrium is

72 ore, to improve the method robustness during HS-SPME studies, we suggest specifying the fiber penetra

73 y 1 month of age, MPS IIIB dogs had elevated HS levels in brain and cerebrospinal fluid.

74 -) selective receptors based on the enhanced HS(-) selectivity over other competing anions in this sy

75 less prions that interact with extracellular HS.

76 gnificant improvement of 1.2% (p < 0.05) for HS, though for keloids this was 0.4% (p = 0.26).

77 RTI-102, a strong partial agonist for HS alpha4beta2 receptors, was effectively an antagonist

78 om a preference of the aryl C-H HB donor for HS(-) in this host scaffold.

79 of an aryl C-H hydrogen bond (HB) donor for HS(-) over other HCh(-) and halides.

80 ighlight the importance of C-H HB donors for HS(-) recognition.

81 ors are established aetiological factors for HS.

82 ikelihood of DDKT, KPD, and non-KPD LDKT for HS candidates (Era 1: January 1, 2009-December 31, 2011;

83 s, suggesting an electrostatic mechanism for HS binding.

84 The therapeutic options for HS comprise antibiotic treatment, neutralization of tumo

85 We showed that hair follicle cells from HS patients had an increased number of proliferating pro

86 ir follicle stem cells (HFSCs) isolated from HS patients and more precisely the outer root sheath cel

87 round state is an electronic relaxation from HS to LS taking place in ~30 ps.

88 ding the informational content of functional HS motifs which is crucial for their future biomedical e

89 During the second period, one group (HS) was housed at 30 degrees C and fed ad libitum.

90 ollowed by subsequent decrease in headspace (HS) intensities over the course of analysis.

91 interlayer coupling in vdW heterostructures (HSs) by utilizing an emerging 2D material, Janus transit

92 ice were fed a high-fat and high-sucrose (HF/HS) diet or a control diet for 12 weeks to investigate i

93 the value of CC mice in combination with HF/HS diet-induced alterations as an approach to study the

94 In Hs3st1(-/-) (HS 3-O-sulfotransferase-1 knockout) cells, reduced 3-O-S

95 sion had a significant reduction of 29.6% in HS and 22.7% in Keloids.

96 ed milk lactose, with more marked changes in HS goats.

97 min/1.73 m/y [0.03 to 0.53]) but declined in HS recipients (-0.44 mL/min/1.73 m/y [-0.85 to -0.03]).

98 Butyrate absorption decreased in HS animals (P < 0.05) but increased in PF animals (P < 0

99 mma+ NK cell% were significantly elevated in HS versus NC sera in Allo-CFC-1 (10 +/- 3% versus 2 +/-

100 levated body temperature due to heat load in HS goats.

101 owever, rejection-free survival was lower in HS recipients converted to belatacept within the first-y

102 urtailed sexual reproduction of E. maxima in HS chickens.

103 we developed a bioassay to remove nitrite in HS samples using the denitrifying bacterium Pseudomonas

104 isposition and skin inflammation observed in HS.

105 a restricted replication of the parasite in HS chickens evidenced by significantly reduced oocyst sh

106 l to reliably assess mechanistic pathways in HS.

107 the distribution of O(2) unloading rates in HS RBCs identified a subpopulation of spherocytes with g

108 s are such that no equilibrium is reached in HS.

109 direction for future mechanistic studies in HS, including more comprehensive inflammatory endotype p

110 stigating the efficacy of such treatments in HS.

111 S-sham tACS facilitated single-pulse MEPs in HSs, but not in patients.

112 tably reconstituted with frataxin, indicated HS-1-associated protein X-1 (HAX-1) as the most signific

113 identified a highly potent IRAK-4 inhibitor (HS-243).

114 Sequencing (SIMMS(2)) method in which intact HS saccharides are dissociated in an ion mobility mass s

115 a kinome-wide screen of 468 protein kinases, HS-243 had exquisite selectivity toward both IRAK-1 (IC(

116 n was seen for FreeSurfer in the AD and left-HS datasets, and for MAPER in the right-HS dataset.

117 end in a preclinical porcine model of lethal HS, despite decreased OCC from substantial volume-expans

118 LPS induced by high salt concentration (LLPS-HS), and compare it to electrostatically driven LLPS rep

119 We show that LLPS-HS promotes tau protein dehydration, undergoes maturatio

120 d controls and determined risk factors for M-HS in PWH.

121 The prevalence of M-HS was lower in PWH compared with uninfected controls (8

122 e of moderate-to-severe hepatic steatosis (M-HS) in PWH with human immunodeficiency virus (HIV)-uninf

123 ed by headspace solid-phase microextraction (HS-SPME) and analysed by GC-FID.

124 Headspace solid-phase microextraction (HS-SPME) was used in order to verified linalool enantiom

125 Head space solid phase microextraction (HS-SPME) with a 65 um divinylbenzene/polydimethylsiloxan

126 mpact headspace solid-phase microextraction (HS-SPME), it is important to reach the highest level of

127 ) and headspace solid-phase microextraction (HS-SPME).

128 ave used high-speed atomic force microscopy (HS-AFM) and all-atom molecular dynamics (MD) simulations

129 we used high-speed atomic force microscopy (HS-AFM) and kinetic modeling which allowed us to determi

130 , we use high-speed atomic force microscopy (HS-AFM), fluorescence recovery after photobleaching (FRA

131 -time by high-speed atomic force microscopy (HS-AFM).

132 h KAS increased DDKT likelihood for the most HS candidates, it also decreased the use of non-KPD LDKT

133 with western blots and compared between MTLE-HS patients who were showing (n = 06) or not showing (n

134 aken together with previous findings in MTLE-HS patients with IF who were evaluated by stereotactic i

135 lepsy related to hippocampus sclerosis (MTLE-HS) have seizures with fear, which is called ictal fear

136 wer for binary (HS vs. FTD), and multiclass (HS and FTD subtypes) classification problems.

137 cells, but rescues the normal cells (namely, HS-5 cells) by trafficking the BTZ into lysosomes.

138 l (log-rank P = 0.17; at 5 y, HS: 78.5%, non-HS: 89.8%) in the 2 groups.

139 s (log-rank P = 0.03; at 5 y, HS: 55.6%; non-HS: 75.0%).

140 l (log-rank P = 0.75; at 5 y, HS: 85.7%, non-HS: 83.7%) or graft survival (log-rank P = 0.17; at 5 y,

141 There were 29 HS and 79 non-HS recipients included.

142 val (log-rank P = 0.30; at 5 y, HS: 82%; non-HS: 84.6%); however, rejection-free survival was lower a

143 to belatacept over 5 years among HS and non-HS recipients.

144 on rate slope improved postconversion in non-HS (0.28 mL/min/1.73 m/y [0.03 to 0.53]) but declined in

145 pared between HS and non-HLA-sensitized (non-HS) recipients using the Kaplan-Meier product-limit meth

146 he first-year posttransplant compared to non-HS recipients (log-rank P = 0.03; at 5 y, HS: 55.6%; non

147 .0% of GP, 60.5%-92.0% of AV, 73.0%-95.0% of HS pairs.

148 ual relative data; the parent sex in 100% of HS and 99.6% of GP pairs; and it completes this analysis

149 s for 93.5% of GP, 97.7% of AV, and 92.2% of HS pairs that have sufficient mutual relative data; the

150 is likewise essential for the activation of HS-responsive genes.

151 , there is an enormous need for awareness of HS, understanding of its pathogenesis and novel treatmen

152 HS removal, it also facilitates cleavage of HS-containing ectodomains of syndecan-1, thereby further

153 The comorbidities of HS include metabolic and cardiovascular disorders, which

154 n in the distal nephron in the conditions of HS- and hyperglycemia-induced kidney injury.

155 The coordination of HS(-) to d-block elements produces a unique class of sub

156 ed therapy to reduce risk for development of HS, but future confirmatory studies are needed.

157 median of 7 days post-transplant; 2 died of HS.

158 there is a significant detrimental effect of HS on the pathogenesis of E. maxima infection in broiler

159 This mechanistic analysis of the etiology of HS in the HFSC compartment establishes a formal link bet

160 was altered in ORSCs from hair follicles of HS patients, leading to activation of the ATR/CHK1 pathw

161 ld be investigated in the next generation of HS(-) selective receptors based on the enhanced HS(-) se

162 ch binding are suggested to be inhibition of HS cleavage in MDA-MB-231 triple-negative breast cancer

163 e-1 knockout) cells, reduced 3-O-S levels of HS diminished both cell surface binding and internalizat

164 the volatile profiles identified by means of HS-SPME-GC-MS analysis, significantly differed in terms

165 e) was lower in HIGH vs. LOW at 40-50 min of HS (P <= 0.01).

166 A model of HS pathogenesis is proposed, integrating the role of com

167 sanguineous, LVR solution in acute models of HS through mechanisms targeting cell swelling-induced mi

168 (CCD) was conducted for the optimization of HS-SPME conditions. Under optimal conditions, a good lin

169 The estimated prevalence of HS is ~1% in most studied countries.

170 ntly found to affect the redox properties of HS both in sorbed and dissolved state.

171 es show that a rare 3-O-sulfation (3-O-S) of HS significantly enhances tau binding.

172 ccepting capacity (EAC), and redox state) of HS.

173 .7% and 28.2% reduction in scar thickness of HS and Keloids, respectively.

174 In this study, a variety of HS oligosaccharides with different chain lengths and N-

175 oxic nitrogen (N) cycling to the function of HSs as a redox battery, however, remains poorly understo

176 In addition, there is one HS site at the vertex of each q3 and i3 spike (the "axia

177 vestigate the biological significance of OPG-HS interaction in bone remodeling, we created OPG knock-

178 ; 15 to 20 degrees C; 40 to 45% humidity) or HS (n = 4; 35 degrees C day, 28 degrees C night; 40% hum

179 odel, we altered the structure of osteoblast HS genetically to make it incapable of binding to OPG.

180 discharge events as documented during other HS.

181 "Peripheral" HS binding sites were found to be associated with the ba

182 Pooled HS and 4 of 5 IVIg commercial preparations contained ant

183 pretreated with anti-HLA antibody positive (HS) or negative (NC) sera to measure IFNgamma+ NK cell%.

184 Posttraumatic HS is a major cause of preventable death.

185 ogether with a number of previously prepared HS oligosaccharides, were printed as a glycan microarray

186 to protection and regeneration by producing HS.

187 seventh protein currently known to recognize HS 3-O-sulfation.

188 ng us to determine how heparanase recognizes HS substrate and selects a favorable cleavage site.

189 the antiresorption activity of OPG requires HS.

190 LLTS reversed HS diet-induced changes at both these sites.

191 left-HS datasets, and for MAPER in the right-HS dataset.

192 In a cell culture, the addition of a 3-O-S HS 12-mer reduced both tau cell surface binding and cell

193 The acceptance of H(2)S/HS(-) as important biological signaling compounds during

194 fed with either low salt (LS) or high salt (HS) diet for a period of 6 weeks, followed by sham or ac

195 speed atomic force microscopy line-scanning (HS-AFM-LS) combined with automated state assignment and

196 volving 21 patients with hypertrophic scars (HS) (n = 9) and keloids (n = 12) resulting from various

197 Hippocampal sclerosis (HS) is a common histopathological abnormality found in p

198 effectiveness of newborn hearing screening (HS) in identifying neonates with CMV-related HL.

199 d protracted activation of high sensitivity (HS) alpha4- and alpha2-containing receptors with the sto

200 Prioritization of highly sensitized (HS) candidates under the kidney allocation system (KAS)

201 cineurin inhibitors (CNI) in HLA-sensitized (HS) kidney transplant recipients has not been establishe

202 Undiluted pooled human serum (HS) and five different commercial preparations of IVIg w

203 examine the binding selectivities of several HS-binding proteins.

204 rmekimab in patients with moderate-to-severe HS who are naive to or have failed prior anti-TNF therap

205 eutic option for treating moderate-to-severe HS.

206 onal activation occurring during heat shock (HS) induction is associated with the generation of DSBs,

207 sp70 genes turn on 2-4 min after heat shock (HS), irrespective of their distance to speckles.

208 g major trauma, burns, or hemorrhagic shock (HS).

209 AV) pairs, and 75.5%-98.5% of half-siblings (HS) pairs compared to PADRE's rates of 38.5%-76.0% of GP

210 action gas chromatography-mass spectrometry (HS-SPME-GC-MS) method for the quantification of 3-monoch

211 d with gas chromatography-mass spectrometry (HS-SPME-GC-MS) was used to analyze the volatile compound

212 action-Gas Chromatography-Mass Spectrometry (HS-SPME-GC-MS).

213 wed by gas chromatography mass spectrometry (HS-SPME/GC-MS).

214 ed tortuosity) and hereditary spherocytosis (HS; expanded pathlength).

215 (LS) hexacoordinated haem to the high spin (HS, S = 2) pentacoordinated domed deoxy-myoglobin (deoxy

216 n (LS, S = 0), thermally accessed high spin (HS, S = 2), and mixed LS and HS states.

217 o trap both the low-spin (LS) and high-spin (HS) states of the iron(II) Hofmann spin crossover framew

218 ction in broiler chickens under heat stress (HS) and mRNA expression of host cytokines that might aff

219 e Indian wheat cv. PBW343 under heat stress (HS) at 42 degrees C for 2 h using RNA-sequencing (RNA-se

220 Follicular cells respond to heat stress (HS) by activating the expression of heat shock family pr

221 GH), followed by 60 min passive heat stress (HS) where the water temperature in the suit was incremen

222 compared to see the effects of heat stress (HS).

223 e (LPS) could be conditioned by heat stress (HS).

224 max) and (ii) 75 min of passive heat stress (HS; 49 degrees C water-perfused suit) to match the exerc

225 rmation processing in both healthy subjects (HS) and patients with schizophrenia.

226 6 subjects with FTD and 84 healthy subjects (HS) were included in the study.

227 nts (OFF condition) and 16 healthy subjects (HSs) underwent iTBS during gamma-tACS (iTBS-gamma tACS)

228 ole of stress-related EVs against subsequent HS was assessed.

229 has induced adaptive response to subsequent HS.

230 ent cells and make them robust to subsequent HS.

231 Natural organic matter and humic substances (HS) in soils and sediments participate in numerous bioge

232 Humic substances (HSs) are important electron acceptors and donors in soil

233 an antibody that recognizes heparan sulfate (HS) chains attached to multiple HSPGs without diminishin

234 s in its ability to exploit heparan sulfate (HS) for attachment to cells.

235 Heparan sulfate (HS) is a sulfated glycosaminoglycan abundant on the cell

236 eparanase cleaves polymeric heparan sulfate (HS) molecules into smaller oligosaccharides, allowing fo

237 evident regulatory roles of heparan sulfate (HS) saccharides in numerous biological processes, defini

238 OPG is to bind cell surface heparan sulfate (HS), but the in vivo evidence was lacking.

239 n expression and release of heparan sulfate (HS), hyaluronic acid (HA), and sialic acid on human trop

240 ll-signaling events through heparan sulfate (HS)-protein interactions and are associated with the ons

241 ed cell signalling molecule heparan sulfate (HS).

242 eased levels of nondegraded heparan sulfate (HS).

243 d in ADAM10-cleaved PrP and heparan sulfate (HS).

244 tau binding to cell surface heparan sulfate (HS).

245 ss Ndst1, a key enzyme for heparan sulfates (HS) synthesis.

246 d animal models of hidradenitis suppurativa (HS) display issues with fidelity and validity to human d

247 e dysregulation in hidradenitis suppurativa (HS) has yet to be elucidated, recent studies identified

248 Hidradenitis suppurativa (HS) is a chronic, relapsing, inflammatory skin disease.

249 n the treatment of hidradenitis suppurativa (HS).

250 apeutic targets in hidradenitis suppurativa (HS).

251 Hidradenitis suppurativa (HS; also designated as acne inversa) is a chronic inflam

252 een linked to fatal hyperammonemia syndrome (HS) in lung transplant recipients.

253 Here, we synthesized HS oligosaccharides with defined sulfation patterns and

254 Using a small targeted HS library, we demonstrate that an oligosaccharide that

255 es that this rare 3-O-sulfation enhances tau-HS binding and likely the transcellular spread of tau, p

256 However, the structural determinants for tau-HS interaction are not well understood.

257 Of the 91 (0.8%) newborns who failed the HS, 24 (26.4%) were confirmed with HL, including 7 (29.2

258 Finally, palaeoecological data from the HS 3 interval unveiled an internal variability suggestin

259 Motion trajectories extracted from the HS-AFM movies indicate that CNTPs exhibit diffusion coef

260 beta2 receptors were similar to those of the HS alpha4beta2 receptors.

261 es C and pair-fed to match the intake of the HS group.

262 Another newborn with cCMVI passed the HS but was confirmed with HL at age 21 days.

263 Mechanistically, we propose that the HS immobilizes secreted OPG at the surface of osteoblast

264 the change from the LS (S = 1/2) MbNO to the HS haem occurs in ~800 fs, and that it proceeds via an i

265 ryo-EM) structure of EEEV complexed with the HS analog heparin.

266 confirmed by a quantitative analysis through HS-SPME-GC-MS.

267 dened the transplant modalities available to HS candidates.

268 The mutated OPG is incapable of binding to HS but binds RANKL normally.

269 Vs generated from granulosa cells exposed to HS has the potential to shuttle bioactive molecules to r

270 sults showed that granulosa cells exposed to HS increased the accumulation of ROS, total oxidized pro

271 NAs of bovine granulosa cells in response to HS.

272 either kept at 37 degrees C or subjected to HS (42 degrees C).

273 his asymmetry was greater in CPs relative to HSs.

274 microbial therapy in preventing and treating HS.

275 previously found to be effective in treating HS, was evaluated using a subcutaneous formulation in pa

276 eported skin microbiota shifts in unaffected HS skinfolds as well.

277 tter photosynthate energy partitioning under HS with a significant reduction in the non-photochemical

278 ated and 17 transcripts were repressed under HS conditions.

279 te a direct correlation with tolerance under HS conditions.

280 Using HS-243 and takinib, we evaluated the consequences of cyt

281 volatile compound profile was examined using HS-SPME-GC-MS.

282 ifically, chocolate aroma was profiled using HS/SPME-GC-MS for three different time and temperature c

283 al, 67 volatiles in PPIs were identified via HS-SPME-GC-MS/O.

284 ition to directly enabling viral release via HS removal, it also facilitates cleavage of HS-containin

285 hout both EX and HS (EX: Delta16 +/- 21% vs. HS: Delta16 +/- 23%, time effect: P = 0.006) with no bet

286 hout both EX and HS (EX: Delta42 +/- 58% vs. HS: Delta53 +/- 28%, time effect: P < 0.001; condition e

287 n T(c) (EX: Delta0.75 +/- 0.33 degrees C vs. HS: Delta0.77 +/- 0.33 degrees C, P = 0.855).

288 ding to the ambient temperature (i.e. TN vs. HS).

289 a spp in lung transplant was associated with HS.

290 were incubated with Farage B (FB) cells with HS or NC sera to measure 7-AAD+ lysed FB cell%.

291 Interaction of heparanase with HS chains is regulated by specific substrate sulfation s

292 Both surgical injury with HS and counterregulatory hormone (epinephrine) infusion

293 Patients with HS (n = 42) were divided into groups A and B based on wh

294 a subcutaneous formulation in patients with HS naive to or having failed anti-TNF therapy.

295 al complement abnormalities in patients with HS.

296 were analyzed with GC-FID and volatiles with HS-SPME-GC-MS and GC-O.

297 on-HS recipients (log-rank P = 0.03; at 5 y, HS: 55.6%; non-HS: 75.0%).

298 r graft survival (log-rank P = 0.17; at 5 y, HS: 78.5%, non-HS: 89.8%) in the 2 groups.

299 on-free survival (log-rank P = 0.30; at 5 y, HS: 82%; non-HS: 84.6%); however, rejection-free surviva

300 patient survival (log-rank P = 0.75; at 5 y, HS: 85.7%, non-HS: 83.7%) or graft survival (log-rank P