ライフサイエンスコーパス: HT

1 HT also attenuated the enhancement of the BP response in

2 HT decreased (p < 0.05) the antiproliferative activity o

3 HT pretreatment of sewage sludge had a weak effect on th

4 HT-DBP is a microscopy-based, single-cell resolution ass

5 HT-DBP requires only 24 hours of ex vivo culture, which

6 HT-induced indels occur disproportionately in nucleosome

7 tion experiments on FAP-transfected HT-1080 (HT-1080-FAP) or on mouse FAP-expressing (HEK-muFAP) and

8 ew effective procedures for reduction of T-2/HT-2 toxin levels is needed.

9 5-HT(2A)R agonist-induced head-twitch behavior was also au

10 5-HT(2A,) a G-protein coupled receptor, is widely expresse

11 5-HT(3) receptors are pentameric ligand-gated ion channels

12 5-HT(7) receptors in their inactive state associate with G

13 5-HT-ir neurons were also observed within the vocal motor

14 Serotonin 1A (5-HT(1A)) autoreceptors regulate brain-wide serotonin neur

15 1 (TAAR1) and 5-hydroxytryptamine type 1A (5-HT(1A)) receptors, may represent a new class of psychotr

16 854944) was to assess D(2)/D(3), 5-HT(1A), 5-HT(2A) and serotonin transporter (SERT) occupancies of b

17 e dependent manner, while binding to D(3), 5-HT(1A) receptors and SERT was not detectable with the ra

18 udy (NCT01854944) was to assess D(2)/D(3), 5-HT(1A), 5-HT(2A) and serotonin transporter (SERT) occupa

19 s of the homomeric 5-hydroxytryptamine 3A (5-HT(3A)) serotonin receptor for 15 to 20 mus to demonstra

20 at antagonizing serotonin (5-HT) type 3AB (5-HT(3AB)) receptors in brain areas involved in mood regul

21 hy volunteers were randomly allocated to a 5-HT depletion (N = 24), DA depletion (N = 24), or placebo

22 Compound 12 (CTW0415) was discovered as a 5-HT(2C)R PAM with improved pharmacokinetics and reduced o

23 timulation of beta-cells were blocked by a 5-HT(3)R antagonist and were enhanced by increasing seroto

24 erted more rapidly to baseline following a 5-HT-challenge.

25 We evaluated whether acute 5-HT(4)R activation in glutamatergic axon terminals arisin

26 In the absence of agonists, 5-HT(2A)R was primarily localized within intracellular com

27 ts, glutamate acting at AMPA receptors and 5-HT acting at 5-HT(2A) receptors.

28 r pathways regenerated into the graft, and 5-HT(+) neurons within graft and host brainstem neurons we

29 the 5-HT(2A) (5-hydroxytryptamine 2A) and 5-HT(1A) (5-hydroxytryptamine 1A) receptors.

30 ated in vivo binding to D(2) receptors and 5-HT(2A) receptors at steady state after 10 days of daily

31 the two receptors in 5-HT(2A)/mGlu(2) and 5-HT(2A)/mGlu(2)/Gqo5 cells.

32 A)-, mGlu(2)/Gqo5-, 5-HT(2A)/mGlu(2)-, and 5-HT(2A)/mGlu(2)/Gqo5-expressing HEK293 cells using a Ca(2

33 The inhibition of 5-HT(3A)Rs and 5-HT(3AB)Rs was non-use dependent and voltage independent,

34 of serotonin receptors, with 5-HT(2B) and 5-HT(7) expression restricted to M-CSF-primed monocyte-der

35 Na(V)1.8(-/-) impaired histamine and 5-HT-induced scratching while Na(V)1.9 was involved in itc

36 op of contractions induced by KCl, ACh and 5-HT.

37 Negligible occupancy (<5%) was observed at 5-HT(1A) and SERT at 4 mg/day.

38 cting at AMPA receptors and 5-HT acting at 5-HT(2A) receptors.

39 Behaviorally, 5-HT depleted subjects demonstrated impaired social reward

40 CD is required for the interaction between 5-HT(3A) and the chaperone protein resistance to inhibitor

41 sponses in 5-HT(2A)/mGlu(2) cells and both 5-HT- and Glu-induced responses in 5-HT(2A)/mGlu(2)/Gqo5 c

42 teric regulation of ion gating elements by 5-HT binding is indicative of a preactive state, which pro

43 ase was attenuated, suggesting blunted CeA 5-HT sensitivity, which partially recovered in protracted

44 and unravels the vulnerability of the CeA 5-HT system to chronic alcohol and protracted withdrawal.S

45 firing rate of DRN 5-HT neurons, cerebral 5-HT depletion, and optogenetic activation and silencing w

46 addition, we comprehensively characterized 5-HT-immunoreactive (-ir) innervation throughout the brain

47 vocal midbrain nuclei showed considerable 5-HT-ir innervation, as did thalamic and hindbrain auditor

48 pairment, but not increased frontal cortex 5-HT(2A)R density or psychedelic-induced head-twitch behav

49 It may thus be the case that decreased 5-HT levels contribute to social learning deficits in depr

50 These changes presumably lead to decreased 5-HT synthesis and/or increased 5-HT reuptake, thereby red

51 onal knockout male mice supports deficient 5-HT transmission underlying depression-related phenotypes

52 Expanding upon our recently described 5-HT(2C)R-positive allosteric modulators (PAMs) based on t

53 , in vivo recordings of firing rate of DRN 5-HT neurons, cerebral 5-HT depletion, and optogenetic act

54 sure and protracted withdrawal dysregulate 5-HT signaling in the CeA.

55 ) allosteric site to potentiate endogenous 5-HT tone may provide novel therapeutics to alleviate the

56 panel of pruritogens (C48/80, endothelin, 5-HT, chloroquine, histamine, lysophosphatidic acid, tryps

57 -HT) in the mammalian brain, and excessive 5-HT signaling is critically implicated in the etiology of

58 s, the major source of 5-HT, and expresses 5-HT receptor subtypes (e.g., 5-HT2C and 5-HT1A) criticall

59 to ZCNT 0.1 NCs as compared to others for 5-HT sensing studies.

60 ) using chitosan as cross-linked agent for 5-HT sensing studies; conducted through cyclic voltammetry

61 Here we report a novel role for 5-HT(2A) in cell-matrix adhesion.In HEK293 cells, which ar

62 demonstrate the existence of a functional 5-HT/5-HT(2B)/AhR axis in human macrophages and indicate t

63 eraction, we developed different MBP-fused 5-HT(3A)-ICD constructs by deleting large segments of its

64 characterized in 5-HT(2A)-, mGlu(2)/Gqo5-, 5-HT(2A)/mGlu(2)-, and 5-HT(2A)/mGlu(2)/Gqo5-expressing HE

65 bited 5-HT-induced currents in heteromeric 5-HT type 3AB receptors (5-HT(3AB)Rs) (IC(50) = 840 and 52

66 ligands targeting the putative heteromeric 5-HT(2A)/mGlu(2) receptor complex, based on the 5-HT(2A) a

67 fects that bupropion exerts on heteromeric 5-HT(3AB) receptors, in particular when constantly present

68 strate that bupropion inhibits heteromeric 5-HT(3AB)Rs as well as homomeric 5-HT(3A)Rs.

69 Serotonin turnover (ratio 5-HIAA/5-HT) was reduced in exposed females and males after 28 da

70 correlation between raphe and hippocampal 5-HT(1A) binding which was more pronounced in HV.

71 IFG and dmPFC correlated with hippocampal 5-HT(1A) binding.

72 teromeric 5-HT(3AB)Rs as well as homomeric 5-HT(3A)Rs.

73 hown to be an antagonist at homopentameric 5-HT type 3A receptors.

74 To date, however, no one has examined how 5-HT influences the dynamics of facial-emotion processing

75 Targeting the serotonin (5-HT) 5-HT(2C) receptor (5-HT(2C)R) allosteric site to potentiat

76 nstrate the existence of a functional 5-HT/5-HT(2B)/AhR axis in human macrophages and indicate that 5

77 the activation and modulation of the human 5-HT(3) type A receptor has been based only on macroscopic

78 only extend our knowledge about the human 5-HT(3)A molecular function but also provide novel insight

79 ding and unbinding of 5-hydroxytryptamine (5-HT) (i.e., serotonin) to the binding pocket located on t

80 5-hydroxytryptamine (5-HT) is equipped onto nanoparticles (NPs) loaded with pho

81 itters, estimation of 5-hydroxytryptamine (5-HT; Serotonin) release has proved to be challenging.

82 terobivalent ligands were characterized in 5-HT(2A)-, mGlu(2)/Gqo5-, 5-HT(2A)/mGlu(2)-, and 5-HT(2A)/

83 was observed between the two receptors in 5-HT(2A)/mGlu(2) and 5-HT(2A)/mGlu(2)/Gqo5 cells.

84 igands inhibited 5-HT-induced responses in 5-HT(2A)/mGlu(2) cells and both 5-HT- and Glu-induced resp

85 nd both 5-HT- and Glu-induced responses in 5-HT(2A)/mGlu(2)/Gqo5 cells.

86 decreased 5-HT synthesis and/or increased 5-HT reuptake, thereby reducing 5-HT transmission.

87 pathway specifically in EC cells to induce 5-HT secretion.

88 opion with 5-HT dose dependently inhibited 5-HT-induced currents in heteromeric 5-HT type 3AB recepto

89 ties, the seven bivalent ligands inhibited 5-HT-induced responses in 5-HT(2A)/mGlu(2) cells and both

90 ons in the superior raphe, an intermediate 5-HT-ir cell cluster, and an extensive inferior raphe popu

91 intake, genetically predisposed irregular 5-HT receptor density, or change in sensory bombardment ma

92 hat the 5-HT-mediated activation of AhR is 5-HT(2B) dependent because it is abrogated by the 5-HT(2B)

93 rotonin (5-HT) concentrations, and lowered 5-HT GC immunoreactivity.

94 In addition, three major 5-HT-ir cell groups were identified in the hypothalamus an

95 TSPAN5 can modulate the concentrations of 5-HT and kynurenine.

96 The MPO targeting property of 5-HT modified NPs is confirmed by noninvasive positron emi

97 fers new insights into the organization of 5-HT nuclei in teleosts and provides neuroanatomical evide

98 ies, we present an argument for the use of 5-HT(1A) agonists in the treatment of Dup15q epilepsy.

99 lts indicate that selective stimulation of 5-HT(1A) receptor in the mPFC exerts rapid and sustained a

100 increase in adhesion, while inhibition of 5-HT(2A) by antipsychotics, such as risperidone, olanzapin

101 The expression of 5-HT(2A)R also augmented intracellular localization of mGl

102 cellular compartments, and coexpression of 5-HT(2A)R with mGluR2 increased the intracellular distribu

103 ude longer than any previous simulation of 5-HT(3A), allow us to observe the dynamic binding and unbi

104 g enough to observe complete activation of 5-HT(3A), the allosteric regulation of ion gating elements

105 The inhibition of 5-HT(3A)Rs and 5-HT(3AB)Rs was non-use dependent and volta

106 Here we show that preassociation of 5-HT(7) serotonin receptors with G(s) heterotrimers is nec

107 al stress decreased the firing activity of 5-HT(DR->VTA) neurons.

108 that citalopram (which increases levels of 5-HT) caused sustained activation in key limbic regions du

109 dorsal raphe nucleus, the major source of 5-HT, and expresses 5-HT receptor subtypes (e.g., 5-HT2C a

110 be sensed by EC cells, inducing release of 5-HT.

111 results indicate that adiponectin acts on 5-HT neurons through AdipoR1 receptors to regulate depress

112 l secretagogues, drugs acting on opioid or 5-HT receptors, or minimally absorbed antibiotics (all of

113 y which it might be associated with plasma 5-HT concentrations and SSRI clinical response remained un

114 The presented 5-HT(1A) receptor-biased agonists, preferentially targetin

115 e-neuroendocrine axis in calibrating rapid 5-HT release for intestinal homeostasis.

116 expression and stimulation of human or rat 5-HT(2A) receptor by agonists such as serotonin or 2,5-dim

117 rontal cortex serotonin 5-HT(2A) receptor (5-HT(2A)R) density in the adult offspring, a phenotype pre

118 fects of specific serotoninergic receptor (5-HT(2A)R) stimulation with psilocybin in healthy humans.

119 dition to the serotonin 5-HT(2A) receptor (5-HT(2A)R), the dopamine D(2) receptor (D(2)R) is a key th

120 ng the serotonin (5-HT) 5-HT(2C) receptor (5-HT(2C)R) allosteric site to potentiate endogenous 5-HT t

121 The serotonin type 3 receptor (5-HT(3)) is a ligand-gated ion channel that converts the b

122 elated peptide, and the serotonin receptor 5-HT(3)R.

123 the class A serotonin 5-HT(2A) receptors (5-HT(2A)Rs) affected the localization and trafficking of c

124 ts in heteromeric 5-HT type 3AB receptors (5-HT(3AB)Rs) (IC(50) = 840 and 526 muM, respectively).

125 tion of serotonin (5-HT) type 4 receptors (5-HT(4)Rs) has been shown to have anxiolytic effects in a

126 increased 5-HT reuptake, thereby reducing 5-HT transmission.

127 2 to potentiate the effects of a selective 5-HT(2C)R agonist was established in a drug discrimination

128 f hemodynamics, we implanted serotonergic (5-HT(+)) neuron-enriched embryonic raphe nucleus-derived n

129 , acetylcholine (ACh 10 muM) or serotonin (5-HT 10 muM).

130 Targeting the serotonin (5-HT) 5-HT(2C) receptor (5-HT(2C)R) allosteric site to pot

131 AN5 were associated with plasma serotonin (5-HT) concentrations which were themselves associated with

132 one, markedly reduced placental serotonin (5-HT) concentrations, and lowered 5-HT GC immunoreactivity

133 ficantly associated with plasma serotonin (5-HT) concentrations, which were themselves associated wit

134 with altered dopamine (DA) and serotonin (5-HT) functioning, the current study aimed to elucidate th

135 amine (DA), uric acid (UA), and serotonin (5-HT) in 0.1 M PBS (pH = 7.4).

136 phe nucleus, the main source of serotonin (5-HT) in the mammalian brain, and excessive 5-HT signaling

137 binding of the neurotransmitter serotonin (5-HT) into a transient cation current that mediates fast e

138 Serotonin (5-HT) is a modulator of neural circuitry underlying motor

139 droxylase 2 (TPH2), a marker of serotonin (5-HT) neurons.

140 anslate environmental cues into serotonin (5-HT) production, contributing to intestinal physiology.

141 Serotonin (5-HT) represents a quintessential neuromodulator, having b

142 Herein, the efficient serotonin (5-HT) sensing studies have been conducted using the (ZnO N

143 dies indicate that antagonizing serotonin (5-HT) type 3AB (5-HT(3AB)) receptors in brain areas involv

144 Activation of serotonin (5-HT) type 4 receptors (5-HT(4)Rs) has been shown to have

145 igh affinity and selectivity for serotonin 5-HT(1A) receptors were obtained and tested in four functi

146 up-regulation of frontal cortex serotonin 5-HT(2A) receptor (5-HT(2A)R) density in the adult offspri

147 In addition to the serotonin 5-HT(2A) receptor (5-HT(2A)R), the dopamine D(2) receptor

148 Here, we showed that the class A serotonin 5-HT(2A) receptors (5-HT(2A)Rs) affected the localization

149 ees of conformational change in the setron-5-HT(3A)R structures, throughout the channel and particula

150 Significant 5-HT-ir innervation was found in components of the vocal p

151 Since 5-HT(2A) is associated with many disorders such as dementi

152 amphetamine challenge to evaluate synaptic 5-HT changes in the living human brain.

153 ransmembrane domain (TMD) from synergistic 5-HT binding.

154 modeling-based fMRI analyses revealed that 5-HT depletion altered social reward prediction signals in

155 These results indicate that 5-HT depletion impairs learning from social rewards, on bo

156 Moreover, we found that 5-HT dose dependently upregulates the expression of AhR ta

157 xis in human macrophages and indicate that 5-HT potentiates the activity of a transcription factor (A

158 Additionally, we have shown that 5-HT(3A)-ICD fused to maltose-binding protein (MBP) direct

159 -chemical properties in correlation to the 5-HT sensing activity.

160 hts into modulation of CeA activity by the 5-HT system and unravels the vulnerability of the CeA 5-HT

161 sults show that intra-mPFC infusion of the 5-HT(1A) receptor agonist 8-OH-DPAT induces rapid and long

162 in the mPFC, and provide evidence for the 5-HT(1A) receptor as a target for the treatment of MDD.

163 Finally, selective stimulation of the 5-HT(1A) receptor increased levels of synaptic proteins an

164 th spatial gene expression patterns of the 5-HT(2A) (5-hydroxytryptamine 2A) and 5-HT(1A) (5-hydroxyt

165 ynthesized monovalent ligands retained the 5-HT(2A) antagonist and mGlu(2) ago-PAM functionalities, t

166 2A)/mGlu(2) receptor complex, based on the 5-HT(2A) antagonist MDL-100,907 and the mGlu(2) ago-PAM JN

167 not understood, although activation of the 5-HT(2A) serotonin receptor (HTR2A) is key.

168 ) dependent because it is abrogated by the 5-HT(2B)-specific antagonist SB204741.

169 sing a simplified model of the pore of the 5-HT(3) receptor (5HT3R) which restrains the backbone stru

170 eterminant for the interaction between the 5-HT(3A)-ICD and RIC-3.

171 entify a 24-amino-acid-long segment of the 5-HT(3A)-ICD as a molecular determinant for the interactio

172 Previously, we have demonstrated that the 5-HT(3A)-ICD is required for the interaction between 5-HT(

173 itoneally) or intra-mPFC infusion with the 5-HT(4)R agonist, RS67333 (0.5 mug/side), were examined in

174 n of AhR target genes in M-MO and that the 5-HT-mediated activation of AhR is 5-HT(2B) dependent beca

175 In dependent rats, this 5-HT-induced increase of GABA release was attenuated, sugg

176 es exhibit higher sensing activity towards 5-HT in accordance to its higher surface area, lower parti

177 .9 was involved in itch signalling towards 5-HT, C48/80 and SLIGRL.

178 lation in the DR that projects to the VTA (5-HT(DR->VTA) neurons).

179 lating expression of genes associated with 5-HT biosynthesis and metabolism.

180 tion of bupropion or hydroxybupropion with 5-HT dose dependently inhibited 5-HT-induced currents in h

181 he expression of serotonin receptors, with 5-HT(2B) and 5-HT(7) expression restricted to M-CSF-primed

182 oxins were deoxynivalenol-3-glucoside (63%), HT-2 toxin (15%), and tenuazonic acid (13%).

183 e were observed on planar scintigraphy for a HT-1080-FAP-xenotransplanted mouse.

184 Here we demonstrate the use of a HT platform based on desorption electrospray ionization

185 In addition, HT attenuated the upregulation of the P2X(3) and increas

186 pressures measured in the first 12 mo after HT.

187 ated that pregnancy should be avoided in all HT recipients, and only 43% (n=52) reported that their c

188 d to HT (123.8 cells/mm(2)) (p = 0.0003) and HT+V (97.5 cells/mm(2)) compared to HT (p = 0.012).

189 sule for HT+M compared to HT (p = 0.001) and HT+V versus HT (p = 0.006).

190 ivalenol, fumonisin B1, zearalanone, T-2 and HT-2 toxin) and emerging mycotoxins (enniatins, beauveri

191 nk P = .018) at 60 months in the A+I arm and HT arm, respectively.

192 Under AT and HT, the lipidome was dominated by phosphatidylcholine (P

193 no temporal relationship between bSUVmax and HT.

194 onship between baseline SUVmax (bSUVmax) and HT risk was assessed using cutoff values for SUVmax >10

195 In the presence of MS, DM, and HT, the presence of RSL was at a significantly higher le

196 aled six genetic loci associated with HN and HT above the Bonferroni multiple-test threshold.

197 ion of BMSC-induced hormone independence and HT resistance in anchorage-independent cells revealed di

198 ne on positron emission tomography (PET) and HT risk.

199 o- to threefold higher at HT than at ST, and HT-growth causes an ~19- to 23-fold increase in indel fr

200 de linking of the microelectrode surface and HT-2 toxin antigen binding fragment of antibody (anti-HT

201 is approximately two- to threefold higher at HT than at ST, and HT-growth causes an ~19- to 23-fold i

202 Discordant metabolite effects between HT regimens may partially mediate the differences in CHD

203 xp(B)=1.30, 95% CI 1.10 to 1.56, p<0.01) but HT did not associate with any of the outcomes.

204 Twelve metabolites had discordant effects by HT type and were associated with incident CHD in the WHI

205 Effective compounds identified by HT-DBP induced tumor regression in genetically engineere

206 ropathology, which showed neuroprotection by HT throughout the brain, particularly in females.

207 coelastic moduli and mass of adherent colon (HT-29) and breast cancer (MCF-7) cells from the interpha

208 rative effects on cervical (HeLa) and colon (HT-29) cancer cells.

209 In addition, compounds HT C1, C2, C4 and C6 inhibited endothelial cell migratio

210 adult mice with reduced DAT expression (DAT-HT) were hypersensitive to short active (SA; 19:5 L:D) p

211 We then tested DAT-HT mice and WT littermates in psychiatry-relevant behavi

212 present hydroxytyrosol synthetic derivative HT C6 as a new antiangiogenic compound and as a good can

213 Here, we develop HT-recruit, a pooled assay where protein libraries are r

214 ypic variations and generalizes to different HT image assays.

215 f six synthetic hydroxytyrosyl alkyl ethers (HT C1, C2, C4, C6, C8 and C12).

216 le benefit in rebiopsy of lesions to exclude HT based on SUVmax alone before initiating therapy in pa

217 SUVmax > 10, with 3.3% of these experiencing HT.

218 ll subjects <21 years old who received first HT at Boston Children's Hospital during 1986-2015 with a

219 ejections occurred within 3 months following HT.

220 formal policy regarding pregnancy following HT.

221 rsus HT (p = 0.036) and internal capsule for HT+M compared to HT (p = 0.001) and HT+V versus HT (p =

222 ized protection was seen in white matter for HT+M versus HT (p = 0.036) and internal capsule for HT+M

223 ng approaches for classifying artifacts from HT image data.

224 lets were randomized to: (i) Cooling 1-13 h (HT; n = 6); (ii) HT+ 2.5% ethanol vehicle (HT+V; n = 7);

225 a type of tannin acyl-hydrolase hydrolyzing HTs, CT monomer gallates and depsides - has been reporte

226 Hydroxytyrosol (HT), which is a polyphenol with a high antioxidant power

227 vel, type 2 diabetes mellitus, hypertension (HT), and metabolic syndrome (MS) were investigated.

228 tional study including grade 1 hypertensive (HT) and normotensive (NT) subjects.

229 zed to: (i) Cooling 1-13 h (HT; n = 6); (ii) HT+ 2.5% ethanol vehicle (HT+V; n = 7); (iii) HT + Melat

230 T+ 2.5% ethanol vehicle (HT+V; n = 7); (iii) HT + Melatonin (HT+M; n = 7).

231 xchangeable phosphate were higher at 48 h in HT+M versus HT (p = 0.036, p = 0.049 respectively).

232 verall TUNEL positive cells were observed in HT+M (47.1 cells/mm(2)) compared to HT (123.8 cells/mm(2

233 ve regulators of innate immune signaling, in HT-29 intestinal epithelial cells challenged with TNF-al

234 terplays with discriminant bacterial taxa in HT and NT subjects highlight the potential role of speci

235 in 2018, these were HT and OF in Lubbock (LU-HT, LU-OF), Overton (OV-HT, OV-OF), Uvalde (UV-HT, UV-OF

236 vehicle (HT+V; n = 7); (iii) HT + Melatonin (HT+M; n = 7).

237 T-2 and its major metabolite HT-2 toxin are naturally occurring contaminants in cerea

238 ent DOX release profiles in response to mild HT.

239 led release profile in combination with mild HT, which correlated well with their cytotoxicity studie

240 Most HT-induced indels are 1-2 bp in size and particularly af

241 ucleosome-free regions, suggesting that much HT-induced mutational damage occurs during cell-cycle ph

242 Furthermore, the antiangiogenic activity of HT C6 was confirmed in vivo in the chick chorioallantoic

243 nmental biomarkers in metastatic biopsies of HT-resistant patients can help refine treatment approach

244 munoenzymatic biosensor for the detection of HT-2 mycotoxin based on carbodiimide linking of the micr

245 ncluding pilot data, examining the effect of HT in the Vannucci model.

246 hanisms leading to the beneficial effects of HT.

247 1:1 ratio to receive three cycles of A+I or HT.

248 8%]) were randomly assigned to either A+I or HT.

249 nd OF in Lubbock (LU-HT, LU-OF), Overton (OV-HT, OV-OF), Uvalde (UV-HT, UV-OF), and Weslaco (WE-HT, W

250 in yeast is a promising tool to overproduce HT at the expense of endogenous tyrosol through central

251 an rejection on protocol biopsy in pediatric HT recipients.

252 ed a retrospective chart review of pediatric HT recipients who had undergone HT between January 2010

253 provides an alternative method of performing HT label-free assays.

254 spital during 1986-2015 with at least 1 post-HT coronary angiogram.

255 riod and decreased to a stable level by post-HT day 90.

256 iac pressures are elevated in the early post-HT period and decrease to levels typical of the native h

257 urements were elevated in the immediate post-HT period and decreased to a stable level by post-HT day

258 MBs, not only during the first 6 months post-HT (1.6% vs. 33.3%, P < .0001), but more so during the 6

259 modality for rejection beyond 6 months post-HT.

260 des and practices surrounding pregnancy post-HT.

261 Neither bSUVmax nor bSUVrange predicted HT in GALLIUM, suggesting that there may be little benef

262 abies immunoglobulin (HRIG; Imogam(R) Rabies-HT), in a ratio of 1:1.

263 ogaea L.) anther lipidome under heat stress (HT) will aid in understanding the mechanisms of heat tol

264 ation, growth at stressful high temperature (HT; 29 degrees C) is highly mutagenic, increasing the mu

265 rker of baseline renal dysfunction, and that HT recipients who develop proteinuria after conversion t

266 We demonstrated that HT attenuates the enhancement of the BP response induced

267 cancer patients, our data demonstrated that HT-DBP could be used to generate personalized pharmacoty

268 The HT regimens were as follows: trabectedin in HG-MLPS; gem

269 The HT-2 toxin and anti-HT2 (10) Fab reaction was reported b

270 the range of intracardiac pressures in these HT patients is important for the clinical interpretation

271 In this HT-LC-HRMS approach, raw spectra of the peaks eluting fr

272 Image-based high throughput (HT) screening provides a rich source of information on d

273 Thus, HT-DBP appears to be an ex vivo functional method with s

274 erved in HT+M (47.1 cells/mm(2)) compared to HT (123.8 cells/mm(2)) (p = 0.0003) and HT+V (97.5 cells

275 6) and internal capsule for HT+M compared to HT (p = 0.001) and HT+V versus HT (p = 0.006).

276 003) and HT+V (97.5 cells/mm(2)) compared to HT (p = 0.012).

277 d competition experiments on FAP-transfected HT-1080 (HT-1080-FAP) or on mouse FAP-expressing (HEK-mu

278 ma (FL) undergo histological transformation (HT).

279 ic assessment in pediatric heart transplant (HT) patients, expected intracardiac pressure measurement

280 We report 2 cases of heart transplant (HT) recipients with COVID-19.

281 lopathy (CAV) in pediatric heart transplant (HT) recipients.

282 Pregnancy after heart transplantation (HT) is a concern for many female recipients.

283 was performed to examine if heat treatment (HT) has beneficial effects on the exaggerated exercise p

284 Testing environments were high tunnel (HT) and open-field (OF) in Uvalde in 2017 while in 2018,

285 ts 18:2 fatty acids to 18:3) decreased under HT for the heat-tolerant genotype SPT 06-07 but not for

286 of pediatric HT recipients who had undergone HT between January 2010 and December 2015 at Lucile Pack

287 (1.4%) patients with bSUVmax > 20 underwent HT.

288 , LU-OF), Overton (OV-HT, OV-OF), Uvalde (UV-HT, UV-OF), and Weslaco (WE-HT, WE-OF).

289 (HT; n = 6); (ii) HT+ 2.5% ethanol vehicle (HT+V; n = 7); (iii) HT + Melatonin (HT+M; n = 7).

290 ion was seen in white matter for HT+M versus HT (p = 0.036) and internal capsule for HT+M compared to

291 phosphate were higher at 48 h in HT+M versus HT (p = 0.036, p = 0.049 respectively).

292 M compared to HT (p = 0.001) and HT+V versus HT (p = 0.006).

293 -OF), Uvalde (UV-HT, UV-OF), and Weslaco (WE-HT, WE-OF).

294 in Uvalde in 2017 while in 2018, these were HT and OF in Lubbock (LU-HT, LU-OF), Overton (OV-HT, OV-

295 he differences in CHD risk between the 2 WHI-HT trials.

296 ssion status were 24.4% (WHI-OS), 25.7% (WHI-HT), and 44.7% (NHSII-MBS).

297 In the WHI-HT trials (Women's Health Initiative Hormone Therapy), t

298 ed among 1295 WHI Hormone Therapy Trial (WHI-HT) participants.

299 While HT significantly reduced BS-binding efficiency despite n

300 The first is a 59-year-old female with HT in 2012 who presented on March 20, 2020 with fever, h