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1 ecies of bacteria: Neisseria gonorrhoeae and Haemophilus ducreyi.
2 Human subjects were infected with Haemophilus ducreyi.
3 Human volunteers were challenged with Haemophilus ducreyi.
4 approximately 17 kDa in soluble extracts of Haemophilus ducreyi.
5 tules of humans experimentally infected with Haemophilus ducreyi.
6 receptor for hemoglobin (Hb) acquisition of Haemophilus ducreyi.
7 of whom were receiving antiretrovirals) with Haemophilus ducreyi.
8 The etiologic agent of chancroid is Haemophilus ducreyi.
9 ogs of the losA and losB genes described for Haemophilus ducreyi.
10 haride (LOS) sialyltransferase gene, lst, of Haemophilus ducreyi.
11 of Yersinia, UspA2 of Moraxella, and DsrA of Haemophilus ducreyi.
12 wn to be responsible for NAD independence in Haemophilus ducreyi.
13 n that is expressed by all tested strains of Haemophilus ducreyi.
15 harides of the sexually transmitted pathogen Haemophilus ducreyi 35000 have been previously found to
17 xtremely large open reading frames (ORFs) in Haemophilus ducreyi 35000, lspA1 and lspA2, each of whic
19 into a defined beta(1-4) glucosyltransferase Haemophilus ducreyi 35000glu- mutant in trans, and monoc
28 tion (PCR) assay, 56 (39%) were positive for Haemophilus ducreyi, 44 (31%) for herpes simplex virus,
29 lso strong homology with the Cdt proteins of Haemophilus ducreyi (87-91%), but only partial homology
32 ovar Typhimurium, Neisseria gonorrhoeae, and Haemophilus ducreyi and determined interaction of each o
33 from Escherichia coli, Salmonella enterica, Haemophilus ducreyi and Neisseria gonorrhoeae, together
38 ients with chancroid contain antibodies to a Haemophilus ducreyi antigen with an approximate molecula
42 cause skin infections in the Pacific islands-Haemophilus ducreyi-as causes of skin ulceration in a ya
43 ed immunosorbent assay-based assay to assess Haemophilus ducreyi binding to extracellular matrix (ECM
44 examined sites experimentally infected with Haemophilus ducreyi by immunohistochemistry and flow cyt
56 amino acid substitutions were mapped in the Haemophilus ducreyi Cdt crystal structure providing some
74 lytica, Actinobacillus pleuropneumoniae, and Haemophilus ducreyi form a lineage distinct from other P
76 for Treponema pallidum (TP), and by PCR for Haemophilus ducreyi (HD), herpes simplex virus (HSV) and
77 s of CDTs from Escherichia coli (Ec-CDT) and Haemophilus ducreyi (Hd-CDT), which share limited amino
78 nucleotide sequence of pNAD1, a plasmid from Haemophilus ducreyi identified on the basis of its abili
80 a sialyltransferase deficient lst mutant of Haemophilus ducreyi, implicating its role in sialylation
82 of the surface lipooligosaccharide (LOS) of Haemophilus ducreyi in the pathogenesis of chancroid, Tn
83 n about the virulence mechanisms employed by Haemophilus ducreyi in the production of genital ulcers.
86 y the role of macrophage polarization during Haemophilus ducreyi infection, we analyzed a panel of ma
95 The major outer membrane protein (MOMP) of Haemophilus ducreyi is an OmpA homolog that migrates on
97 aride (LOS) present in the outer membrane of Haemophilus ducreyi is likely a virulence factor for thi
99 volunteers, the cutaneous immune response to Haemophilus ducreyi is orchestrated by serum, polymorpho
110 id, a sexually transmitted disease caused by Haemophilus ducreyi, is one of the most common genital u
115 uman volunteers inoculated at two sites with Haemophilus ducreyi, outcomes for a subject were not ind
121 re used to determine the genetic defect of a Haemophilus ducreyi pyocin-resistant lipooligosaccharide
122 s indicated that experimental infection with Haemophilus ducreyi recruits CD4 cells to the skin at th
125 the crystal structure of the holotoxin from Haemophilus ducreyi reveals that CDT consists of an enzy
127 ) (CMP-NeuAc synthetase) was isolated from a Haemophilus ducreyi strain 35000 cell lysate and partial
128 ighly conserved outer membrane proteins from Haemophilus ducreyi strain 35000 were cloned, overexpres
131 e have initiated studies of the enzymes from Haemophilus ducreyi (the infectious agent of chancroid)
132 lls and contain very low numbers of cells of Haemophilus ducreyi, the bacterial agent of chancroid.
150 major causes of genital ulcer disease (GUD), Haemophilus ducreyi, Treponema pallidum, and herpes simp
151 ex polymerase chain reaction assay to detect Haemophilus ducreyi, Treponema pallidum, and herpes simp
152 polymerase chain reaction (M-PCR) assay for Haemophilus ducreyi, Treponema pallidum, and herpes simp
153 multaneous amplification of DNA targets from Haemophilus ducreyi, Treponema pallidum, and herpes simp
154 eviously, we showed that serum resistance in Haemophilus ducreyi type strain 35000HP required express
156 charide [LOS] EIA) that detect antibodies to Haemophilus ducreyi was evaluated with serum specimens o
159 coli and construction of isogenic mutants of Haemophilus ducreyi, we showed that the hemoglobin recep
162 crystal structure of the cytosolic domain of Haemophilus ducreyi YhcB shows a unique tetrameric a-hel