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1 nstration of human T-lymphocyte responses to Hantaan virus.
2 previous laboratory-acquired infections with Hantaan virus.
3 us (RVFV), La Crosse virus, Andes virus, and Hantaan virus, all of which can cause severe disease in
4 erous orthohantaviruses, including Andes and Hantaan virus and are also susceptible to live hantaviru
6 combinant metastable helical nucleocapsid of Hantaan virus (Hantaviridae family, Bunyavirales order)
9 en antiviral activity and GTP repression for Hantaan virus (HTNV) and evidence for RBV's ability to p
10 nfected by pathogenic Andes virus (ANDV) and Hantaan virus (HTNV) and nonpathogenic Tula virus (TULV)
11 causing Andes virus (ANDV) and HFRS causing Hantaan virus (HTNV) are inhibited by alpha(v)beta(3) in
12 eveloped to gather whole genome sequences of Hantaan virus (HTNV) from HFRS patients and rodent hosts
13 n constant, K(d), for the interaction of the Hantaan virus (HTNV) N protein and its genomic S segment
14 d region (amino acids 175 to 217) within the Hantaan virus (HTNV) N protein that interacts with a hig
17 ilarity) for their abilities to inhibit SNV, Hantaan virus (HTNV), and Prospect Hill virus (PHV) infe
18 ontrast to pathogenic NY-1 virus (NY-1V) and Hantaan virus (HTNV), nonpathogenic Prospect Hill virus
20 Gn glycoprotein ectodomain from the Asiatic Hantaan virus (HTNV), the most prevalent pathogenic hant
21 il 1978 that they were found to be caused by Hantaan virus (HTNV), the most prevalent pathogenic hant
22 indings indicate that Andes virus (ANDV)- or Hantaan virus (HTNV)-infected endothelial cells specific
23 vitro, ribavirin acts as a lethal mutagen in Hantaan virus (HTNV)-infected Vero E6 cells, resulting i
26 in will inhibit the production of infectious Hantaan virus (HTNV); however, its mechanism of action i
27 c hantaviruses [New York-1 virus (NY-1V) and Hantaan virus (HTNV)] bind immobilized beta3 polypeptide
28 d with HPS [New York-1 virus (NY-1V)], HFRS [Hantaan virus (HTNV)], or by a hantavirus not associated
29 0), were used to examine the pathogenesis of Hantaan virus infection in a mouse model and identify vi
31 -cell responses were directed against either Hantaan virus nucleocapsid (N) or G1 protein, and these
33 isolated from these two donors included both Hantaan virus-specific CTLs and hantavirus cross-reactiv
37 cts against the Old World hantavirus species Hantaan virus when administered pre- or post-exposure.