ライフサイエンスコーパス: Helicobacter pylori

1 s associated with tetracycline resistance in Helicobacter pylori.

2 rgy and infection with the gastric bacterium Helicobacter pylori.

3 ainst SK from Mycobacterium tuberculosis and Helicobacter pylori.

4 s has been questioned since the discovery of helicobacter pylori.

5 bacteria such as the human gastric pathogen Helicobacter pylori.

6 dy the epithelial response to infection with Helicobacter pylori.

7 main protein from the human gastric pathogen Helicobacter pylori.

8 uman papilloma virus, hepatitis C virus, and Helicobacter pylori.

9 was described as a chemotaxis attractant for Helicobacter pylori.

10 ence factor secreted by the gastric pathogen Helicobacter pylori.

11 uman papilloma virus, hepatitis C virus, and Helicobacter pylori.

12 se is infection by a gram-negative bacterium Helicobacter pylori.

13 lation is infected with the stomach pathogen Helicobacter pylori.

14 h functional dyspepsia who test positive for Helicobacter pylori.

15 ings behind the regulation of a type I TA in Helicobacter pylori.

16 Helicobacter pylori, a 2 x 1 mum spiral-shaped bacterium

17 rophage EGFR signaling during infection with Helicobacter pylori, a bacterial pathogen that causes pe

18 The genome of the gastric pathogen Helicobacter pylori, a Gram-negative epsilonproteobacter

19 Although all individuals were infected with Helicobacter pylori, abnormal expression of alpha(1,2)fu

20 The Helicobacter pylori adhesin BabA binds mucosal ABO/Le(b)

21 l. (2016) perform structural analyses of the Helicobacter pylori adhesin BabA to determine how the ba

22 Combining these enzymes with Helicobacter pylori alpha1-3-fucosyltransferase, we deve

23 Several important human pathogens, such as Helicobacter pylori and Campylobacter jejuni, have escap

24 ator of M1 macrophage activation during both Helicobacter pylori and Citrobacter rodentium infection.

25 ce were infected with Helicobacter felis and Helicobacter pylori and compared with noninfected animal

26 Pse on their surface polysaccharides (e.g., Helicobacter pylori and Enterobacter cloacae), revealing

27 One example is Helicobacter pylori and gastric cancer.

28 Clinically, Helicobacter pylori and GC-associated UHMK1 mutation ind

29 pathogens is being recognized, for example, Helicobacter pylori and human papillomavirus in the case

30 oid cells also reduced the T(H)1 response to Helicobacter pylori and impaired immune control of the b

31 Colonization of the human stomach by Helicobacter pylori and its role in causing gastric canc

32 study, OM-NPs are made with the membrane of Helicobacter pylori and shown to bind with gastric epith

33 -d-cholesteryl glucopyranoside (alphaCAG) of Helicobacter pylori and the corresponding galactose anal

34 Although Helicobacter pylori and use of non-steroidal anti-inflam

35 on Caulobacter crescentus, Vibrio cholerae, Helicobacter pylori, and Campylobacter jejuni, organisms

36 cquired infections, clarithromycin-resistant Helicobacter pylori, and fluoroquinolone-resistant Campy

37 ng for celiac disease, autoimmune gastritis, Helicobacter pylori, and hereditary forms of IDA is reco

38 entury, and shortly thereafter the bacterium Helicobacter pylori, and later Fusobacterium nucleatum,

39 forces do, however, appear to play a role in Helicobacter pylori, and some individual genes in all gr

40 Helicobacter pylori antibiotic resistance is widespread

41 enocarcinoma and seropositivity to different Helicobacter pylori antigens using multiplex serology ha

42 Helicobacter pylori arginase, a bimetallic enzyme, is cr

43 g a highly prevalent gram-negative pathogen, Helicobacter pylori, as a model organism.

44 as being involved in the pathophysiology of Helicobacter pylori-associated diseases, the role of oth

45 The pathogenesis of Helicobacter pylori-associated gastric cancer is depende

46 n chronic inflammatory conditions, including Helicobacter pylori-associated gastritis, where its prod

47 that shares similarities to the etiology of Helicobacter pylori-associated intestinal-type gastric a

48 of cancer death worldwide, is infection with Helicobacter pylori bacterial strains that inject cytoto

49 Expression of the Helicobacter pylori blood group antigen binding adhesin

50 ce of this coincides with the eradication of Helicobacter pylori, both of which might alter the oesop

51 e in the evolution of the bacterial pathogen Helicobacter pylori, but its dynamics remain incompletel

52 tantly related species Bacillus subtilis and Helicobacter pylori, but its role in bacterial chemotaxi

53 essary for efficient stomach colonization by Helicobacter pylori, but the molecular mechanisms for ge

54 mach biopsies, and validated the presence of Helicobacter pylori by quantitative PCR.

55 The Helicobacter pylori Cag T4SS translocates CagA, a bacter

56 Heterogeneity at the Helicobacter pylori cagA gene promoter region has been l

57 Helicobacter pylori CagA is a secreted effector protein

58 ation of particular polymorphisms within the Helicobacter pylori CagL hypervariable motif (CagLHM) in

59 ave recently found that the gastric pathogen Helicobacter pylori can activate gastric stem cells and

60 Lifelong infection of the gastric mucosa by Helicobacter pylori can lead to peptic ulcers and gastri

61 : Despite inducing an inflammatory response, Helicobacter pylori can persist in the gastric mucosa fo

62 ecific distribution of risk factors (such as Helicobacter pylori) can vary by race and ethnicity and

63 tly associated with sex, breast-feeding, and Helicobacter pylori carriership.

64 We report on the protonation state of Helicobacter pylori catalase compound II.

65 epithelial cells to the bacterial carcinogen Helicobacter pylori causes DNA double strand breaks.

66 Helicobacter pylori causes numerous alterations in gastr

67 ution crystal structure of dCACHE LBD of the Helicobacter pylori chemoreceptor TlpC.

68 Helicobacter pylori coinfection in human immunodeficienc

69 Chronic Helicobacter pylori colonization in animal models often

70 Helicobacter pylori colonization may affect the mucosal

71 Helicobacter pylori colonizes the gastric mucosa and sec

72 Helicobacter pylori colonizes the human stomach and cont

73 Helicobacter pylori colonizes the human stomach and incr

74 Helicobacter pylori colonizes the stomach in about half

75 Helicobacter pylori colonizes the stomach of around 50%

76 Helicobacter pylori confers protection against the anaph

77 We surveyed national Helicobacter pylori diagnostic testing practices and dia

78 Helicobacter pylori differentially binds SPEM glands in

79 The human pathogen Helicobacter pylori displays extensive genetic diversity

80 We found that Helicobacter pylori DNA can be detected in human stool s

81 The Helicobacter pylori energy sensor TlpD determines tactic

82 Helicobacter pylori eradication and endoscopic surveilla

83 Helicobacter pylori eradication rates in Portugal are de

84 Helicobacter pylori eradication therapy to prevent gastr

85 Rifabutin-based triple therapy (RHB-105) for Helicobacter pylori eradication: a double-blind, randomi

86 to the HobA-interacting surface of DnaA from Helicobacter pylori even though HobA is an activator of

87 Helicobacter pylori exhibits a high level of intraspecie

88 hich is solely a GTPase, the V. cholerae and Helicobacter pylori FeoB proteins have both GTPase and A

89 s lumazine synthase (LuS) and the 24-subunit Helicobacter pylori ferritin.

90 Interestingly, Helicobacter pylori flagellin triggered robust Nlrc4 pho

91 s homologs from Agrobacterium, Brucella, and Helicobacter pylori form heterodimers.

92 he gastric tissues of patients infected with Helicobacter pylori from gastritis to precancerous intes

93 Non Helicobacter pylori gastric Helicobacters (NHPGHs) are a

94 umulating evidence indicates that persistent Helicobacter pylori gastric infection influences immune

95 antified the expression of a large number of Helicobacter pylori genes and found high expression of g

96 Helicobacter pylori genetic diversity is known to be inf

97 reened, for the first time to our knowledge, Helicobacter pylori GML-associated strains for their cap

98 Helicobacter pylori GroES (HpGroES), a potent immunogen,

99 In patients with chronic Helicobacter pylori (H pylori) infection, parietal and c

100 stent evidence about the association between Helicobacter pylori (H. pylori) and dental diseases with

101 Empiric triple treatments for Helicobacter pylori (H. pylori) are increasingly unsucce

102 We investigated whether Helicobacter pylori (H. pylori) CagA contributes to the

103 e risk of gastric cancer (GC) declines after Helicobacter pylori (H. pylori) eradication and long-ter

104 Helicobacter pylori (H. pylori) eradication using standa

105 er mortality due to therapy resistance, with Helicobacter pylori (H. pylori) infection being a major

106 Helicobacter pylori (H. pylori) infection is a major cau

107 Helicobacter pylori (H. pylori) infection is considered

108 Helicobacter pylori (H. pylori) infection is strongly as

109 The role Helicobacter Pylori (H. pylori) infection plays in the a

110 rictions to endoscopy and high prevalence of Helicobacter pylori (H. pylori) infection.

111 ammation (gastritis) triggered by persistent Helicobacter pylori (H. pylori) infection.

112 Helicobacter pylori (H. pylori) is a species of bacteria

113 Helicobacter pylori (H. pylori) is the strongest identif

114 Helicobacter pylori (H. pylori) is the strongest known r

115 Helicobacter pylori (H. pylori) is the strongest known r

116 cal studies indicated that colonization with Helicobacter pylori (H. pylori) may affect body mass ind

117 Helicobacter pylori (H. pylori) secretes the chaperone a

118 -proliferation was reported in patients with Helicobacter pylori (H. pylori)-infected gastric mucosa

119 ve rapid urease test may be reused to detect Helicobacter pylori (H. pylori).

120 r's disease, primary open-angle glaucoma and Helicobacter pylori (H.pylori) infection in all possible

121 Helicobacter pylori (H.pylori) infections are prevalent

122 f of the world's population is infected with Helicobacter pylori (H.pylori), a bacterium shown to be

123 helical shape of the human stomach pathogen Helicobacter pylori has been suggested to provide mechan

124 The emergence of drug resistance in Helicobacter pylori has resulted in a greater need for s

125 Helicobacter pylori (HP) and Epstein-Barr virus (EBV) ha

126 Co-infection between Helicobacter pylori (Hp) and groups of periodontal patho

127 Helicobacter pylori (HP) colonizes the human stomach and

128 long-term complete remission after frontline Helicobacter pylori (HP) eradication (HPE).

129 We evaluated the effect of Helicobacter pylori (HP) eradication on p53, cyclin D1 e

130 Helicobacter pylori (HP) infection is present in about 5

131 Helicobacter pylori (Hp) infection is the main cause of

132 It is known that the prevalence of Helicobacter pylori (Hp) infection, a risk factor for ga

133 Helicobacter pylori (Hp) secrete VacA, a diffusible pore

134 Helicobacter pylori (Hp) strains that carry the cag type

135 bination of serum pepsinogens(PGs), IgG anti-Helicobacter pylori (HpAb), and osteopontin (OPN) can be

136 alpha-Carbonic anhydrase of Helicobacter pylori (HpalphaCA) plays an important role

137 acterized the binding of parS and Spo0J from Helicobacter pylori (HpSpo0J) and solved the crystal str

138 , in some cases such as the gastric pathogen Helicobacter pylori, HtrA is secreted where it cleaves t

139 -sensitive populations of the human pathogen Helicobacter pylori in an environment without antibiotic

140 We detected Helicobacter pylori in only the upper GI tract.

141 evalence of primary antibiotic resistance in Helicobacter pylori in the Asia-Pacific region.

142 for the survival of the pathogenic bacteria Helicobacter pylori in the fluctuating pH of the human s

143 ions, particularly infections resulting from Helicobacter pylori in the gastric tract.

144 Half of all humans harbor Helicobacter pylori in their stomachs.

145 he polymorphic CagA toxin is associated with Helicobacter pylori-induced disease.

146 Helicobacter pylori-induced gastritis is the strongest r

147 Helicobacter pylori-induced miR-135b-5p promotes cisplat

148 the Gram-negative, microaerophilic bacterium Helicobacter pylori induces an inflammatory response and

149 Helicobacter pylori induces strong inflammatory response

150 Helicobacter pylori induces the antiapoptotic protein my

151 A total of 200 Helicobacter pylori infected patients were retrospective

152 , gastric immunopathology was accelerated in Helicobacter pylori-infected Gkn2 knockout mice and was

153 proposed that the accompanying microbiota in Helicobacter pylori-infected individuals might affect di

154 ive protein (aOR: 0.82; 95% CI: 0.73, 0.92), Helicobacter pylori infection (aOR: 0.11; 95% CI: 0.05,

155 wheat allergy (WA), coeliac disease (CD) and Helicobacter pylori infection (HP).

156 y, small intestinal bacterial overgrowth and Helicobacter pylori infection affect motor fluctuations

157 icting data regarding an association between Helicobacter pylori infection and iron deficiency anemia

158 We examined the association between Helicobacter pylori infection and the immune response fo

159 Both the prevalence of Helicobacter pylori infection and the incidence of gastr

160 ing clarithromycin as a model antibiotic and Helicobacter pylori infection as a model disease.

161 Using Helicobacter pylori infection as a model for chronic muc

162 Helicobacter pylori infection causes gastric cancer, the

163 cing identified more IM patients with active Helicobacter pylori infection compared with histopatholo

164 Helicobacter pylori infection contributes to the develop

165 ence from developed countries indicates that Helicobacter pylori infection correlates with a reduced

166 Eradication of Helicobacter pylori infection has been reported to reduc

167 The role of Helicobacter pylori infection in activating or favouring

168 5 Ser-536 phosphorylation also occurs during Helicobacter pylori infection in macrophages and gastric

169 Gastric injury, such as from Helicobacter pylori infection in patients with chronic a

170 h may indicate an ethiopathogenesis role for Helicobacter pylori infection in this disease.

171 Helicobacter pylori infection induces a number of pro-in

172 Helicobacter pylori infection induces chronic gastric in

173 Persistent Helicobacter pylori infection induces chronic inflammati

174 Helicobacter pylori infection is a major risk factor for

175 Helicobacter pylori infection is a proven carcinogen for

176 Helicobacter pylori infection is associated with colorec

177 Helicobacter pylori infection is characterized by chroni

178 Among other risk factors, Helicobacter pylori infection is considered the main dri

179 Helicobacter pylori infection is implicated in the aetio

180 Helicobacter pylori infection is increasingly difficult

181 Helicobacter pylori infection is marked by a vast preval

182 Helicobacter pylori infection is the main risk factor fo

183 Helicobacter pylori infection is the main risk factor fo

184 Helicobacter pylori infection is the major cause of gast

185 Helicobacter pylori infection is the most important risk

186 Helicobacter pylori infection not only induces gastric i

187 Helicobacter pylori infection triggers chronic inflammat

188 Chronic Helicobacter pylori infection triggers neoplastic transf

189 Helicobacter pylori infection was associated with decrea

190 amed stomachs (from TxA23 mice and mice with Helicobacter pylori infection) identified more metaplast

191 Risk factors for the condition include Helicobacter pylori infection, age, high salt intake, an

192 egulation of iNOS has been observed in human Helicobacter pylori infection, but the cellular sources

193 In human Helicobacter pylori infection, mucosal PC express induci

194 ncer databases, which do not include data on Helicobacter pylori infection, the most well-known risk

195 tric submucosal vessels in a murine model of Helicobacter pylori infection.

196 Chronic gastritis was induced in B6 mice by Helicobacter pylori infection.

197 ct the gastric epithelium, especially during Helicobacter pylori infection.

198 f non-steroidal anti-inflammatory drugs, and Helicobacter pylori infection.

199 ctiveness of standard therapies to eradicate Helicobacter pylori infection.

200 ication rates of standard triple therapy for Helicobacter pylori infections have decreased in recent

201 Although consensus supports eradication of Helicobacter pylori infections, antimicrobial resistance

202 Helicobacter pylori infects the human stomach and causes

203 Helicobacter pylori inhabits the gastric mucosa where it

204 The gastric pathogen Helicobacter pylori interacts intimately with the gastri

205 Furthermore, we quantify the invasion of Helicobacter pylori into the glands of the mouse stomach

206 Helicobacter pylori is a bacterial pathogen associated w

207 The bacterial pathogen Helicobacter pylori is a constituent of the normal gastr

208 Helicobacter pylori is a Gram-negative bacterium that co

209 Helicobacter pylori is a Gram-negative bacterium that in

210 Helicobacter pylori is a gram-negative bacterium that pe

211 Helicobacter pylori is a human-specific pathogen that ch

212 The gastric pathogen Helicobacter pylori is a major cause of acute chronic ga

213 Helicobacter pylori is a pathogen that chronically colon

214 Helicobacter pylori is a spiral-shaped Gram-negative bac

215 Helicobacter pylori is a successful pathogen of the huma

216 Helicobacter pylori is accounted as the most etiologic a

217 Helicobacter pylori is an important human pathogen assoc

218 ytotoxin associated gene A (CagA) protein of Helicobacter pylori is associated with increased virulen

219 Infection with Helicobacter pylori is associated with severe digestive

220 Helicobacter pylori is one of the most common bacterial

221 The bacterium Helicobacter pylori is one of the most common infectious

222 The stomach bacterium Helicobacter pylori is one of the most prevalent human p

223 BACKGROUND & AIMS: Helicobacter pylori is remarkable for its genetic variat

224 Infection with the gram-negative bacterium Helicobacter pylori is the most prevalent chronic bacter

225 Infection by Helicobacter pylori is the primary cause of gastric aden

226 Helicobacter pylori is the principal cause of gastric ca

227 The bacteria Helicobacter pylori is the single leading carcinogenic i

228 Helicobacter pylori is the strongest risk factor for gas

229 Helicobacter pylori is the strongest risk factor for gas

230 Helicobacter pylori is the strongest risk factor for the

231 distribution of shared gene pools for global Helicobacter pylori isolates.

232 nic atrophic gastritis due to infection with Helicobacter pylori; it might be a precursor to intestin

233 Helicobacter pylori lacks a gene encoding a homologue of

234 ks (UC), vitamin D deficiency (IBD), and non-Helicobacter pylori-like enterohepatic Helicobacter spec

235 isseria meningitidis, Neisseria gonorrhoeae, Helicobacter pylori, Moraxella catarrhalis, and Streptoc

236 tructure of a soluble variant of full-length Helicobacter pylori MotB in which the plug helix was eng

237 In a Helicobacter pylori mouse infection model, PMN infiltrat

238 The Helicobacter pylori MTAN (HpMTAN) hydrolyzes 6-amino-6-d

239 The gastric pathogen Helicobacter pylori must combat chronic acid and oxidati

240 results in more efficient immune control of Helicobacter pylori, Mycobacterium bovis bacillus Calmet

241 Hypergastrinemia occurred more frequently in Helicobacter pylori negative patients and of these 20/22

242 was induced by infection of mice with either Helicobacter pylori or Helicobacter felis.

243 e human gastrointestinal microbiota, such as Helicobacter pylori, other members of the neonatal intes

244 The Helicobacter pylori phase variable gene modH, typified b

245 omavirus, herpes virus) and bacterial (e.g., Helicobacter pylori, pneumonia) diseases, and autoimmune

246 link is provided by the association between Helicobacter pylori-positive gastritis and gastric MALT

247 re commonly used as a first-line therapy for Helicobacter pylori-positive patients; however, resistan

248 The bacterium Helicobacter pylori possess only one OASS (hp0107) gene

249 The gastric pathogen Helicobacter pylori possesses a highly active urease to

250 X-ray crystal structures of the Helicobacter pylori protein CagL revealed that RGD motif

251 mologs from Campylobacter coli (R.CcoLI) and Helicobacter pylori (R.HpyAXII) and demonstrated their D

252 Infection with the Gram-negative bacterium Helicobacter pylori remains the most important modifiabl

253 The gastric microbe Helicobacter pylori represents an ancestral constituent

254 The gastric pathogen Helicobacter pylori requires a noncanonical cytosolic ch

255 The pathogen Helicobacter pylori requires two nickel-containing enzym

256 s, including norovirus, Campylobacter fetus, Helicobacter pylori, Salmonella enterica, and Giardia la

257 Helicobacter pylori secretes a pore-forming VacA toxin t

258 , P = .13); 20.0% of the cohort had positive Helicobacter pylori serology (16 vs 2 in the CG, P = .00

259 Helicobacter pylori seropositivity was defined as those

260 Helicobacter pylori specifically colonizes the human gas

261 Helicobacter pylori status was tested using a stool anti

262 ogical and molecular features including age, Helicobacter pylori status, tumor site, patient survival

263 Infection of the stomach with Helicobacter pylori stimulates increased secretion of Rs

264 Helicobacter pylori stimulates or inhibits depending upo

265 Colonization of the human stomach with Helicobacter pylori strains containing the cag pathogeni

266 Most Helicobacter pylori strains express the BabA adhesin, wh

267 Infection with CagA+ Helicobacter pylori strains is linked to an increased ri

268 Carriage of Helicobacter pylori strains producing more active (s1/i1

269 and gastric cancer are caused most often by Helicobacter pylori strains that harbor the cag pathogen

270 Helicobacter pylori strains that harbor the oncoprotein

271 nd showed that it plays an important role in Helicobacter pylori stress tolerance and survival in the

272 erated against the O chain polysaccharide of Helicobacter pylori that contains polymeric Le(x) struct

273 Helicobacter pylori, the causative agent of gastric canc

274 Helicobacter pylori, the dominant member of the human ga

275 Helicobacter pylori, the main cause of peptic ulcer dise

276 Gram-negative bacteria, and in particular of Helicobacter pylori, the mechanisms underlying the handl

277 dies reported an association of the bacteria Helicobacter pylori, the primary cause of gastric cancer

278 cronutrients modulate gene expression within Helicobacter pylori, the strongest identified risk facto

279 tic distribution of human pathogens, such as Helicobacter pylori, thereby demonstrating the potential

280 Chemotaxis is important for Helicobacter pylori to colonize the stomach.

281 bA adhesin mediates high-affinity binding of Helicobacter pylori to the ABO blood group antigen-glyco

282 Adherence of Helicobacter pylori to the gastric epithelial cell line

283 Adherence of Helicobacter pylori to the gastric epithelial cell line

284 Adhesion of Helicobacter pylori to the gastric mucosa is a necessary

285 Family history of gastric cancer and Helicobacter pylori treatment.

286 Helicobacter pylori type IV secretion system injects the

287 The gastric pathogen Helicobacter pylori undergoes host-mediated oxidant stre

288 The gastric cancer-causing pathogen Helicobacter pylori up-regulates spermine oxidase (SMOX)

289 as inhibitors of Sporosarcina pasteurii and Helicobacter pylori ureases.

290 Helicobacter pylori uses MTAN in its unusual menaquinone

291 The human pathogen Helicobacter pylori uses the host receptor alpha5beta1 i

292 The gastric pathogen Helicobacter pylori uses the thioredoxin system to maint

293 Helicobacter pylori VacA is a secreted pore-forming toxi

294 The Helicobacter pylori virulence gene, cagA, and active for

295 of galectin-3 (Gal3) in gastric infection by Helicobacter pylori We first demonstrated that Gal3 was

296 In the gastric pathogen Helicobacter pylori we identify six distinct subtypes, o

297 ed from the chromosome of the human pathogen Helicobacter pylori We show that expression of the AapA1

298 oteins in the bacterial chemotaxis system of Helicobacter pylori, which requires two nonredundant cou

299 s at the interface of the bacterial pathogen Helicobacter pylori with its host.

300 we present two high-resolution structures of Helicobacter pylori XerH with its recombination site DNA