ライフサイエンスコーパス: His bundle

1 distance between the aortic annulus and the His bundle.

2 summit, and rarely the LVOT septum near the His bundle.

3 ines, the tricuspid annulus, and the AV node-His bundle.

4 bundle (PAVB) and distal AV bundle (DAVB) or His bundle.

5 left side of the septum in proximity to the His-bundle.

6 The percentage of patients undergoing His bundle ablation was greatest in the > or =80-year-ol

7 nd 9+/-1 ms, respectively), without altering His bundle activation time.

8 and in AV nodal conduction delay (atrium-to-His bundle, AH, interval) in guinea pig isolated hearts

9 and the eustachian ridge (ER), and from the His bundle and coronary sinus catheters.

10 this Review, we summarize the anatomy of the His bundle and early clinical observations, implantation

11 ricular pacing was performed adjacent to the His bundle and proximal right bundle branch (HB-RB), ini

12 vation time relative to the QRS onset at the His bundle and successful ablation sites than the LV sum

13 Catheters were targeted to the right atrium, His bundle, and right ventricle of 10 mongrel dogs (23 t

14 successfully positioned at the right atrial, His bundle, and right ventricular target sites of all an

15 in the AV node, penetrated the septum as the His bundle, and then divided into left and right bundle

16 ate depolarizations in the atrioventricular (His)-bundle associated with lethal ventricular arrhythmi

17 n increase in S-A interval without change in His bundle-atrial interval or atrial activation sequence

18 the sinoatrial node, atrioventricular node, His bundle, bundle branches, and Purkinje fibers, were a

19 n, atrial electrograms recorded close to the His bundle changed from near to far field potentials whe

20 The stimulus to His bundle conduction times (SH) at both sites (SH(P) an

21 ronary sinus, atrial activation close to the His bundle could be advanced by late extrastimuli delive

22 In 14 patients, the His bundle could be reliably stimulated.

23 A His bundle deflection was present in the blocked beats i

24 Catheter ablation was not attempted because His bundle deflections were recorded from this site duri

25 ests that fast and slow wavefronts reach the His bundle differently, producing functional longitudina

26 d with the use of whole-cell patch-clamp and His bundle electrogram techniques, respectively.

27 The ventricular deflection in the His bundle electrogram was significantly later relative

28 ng artifacts and inconsistent inscription of His bundle electrograms.

29 on procedure of an intramural focus near the His bundle failed in 2 of 7 patients.

30 ioventricular septum was mapped via EAM, and His bundle (HB) electrograms, selective, and nonselectiv

31 is study aimed to evaluate if intraoperative His bundle (HB) mapping during complex biventricular CHD

32 and hemodynamic effects of LVs with BiV and His bundle (HB) pacing in CRT patients.

33 he acute hemodynamic effects of simultaneous His-bundle (HIS) and left ventricular (LV) pacing.

34 icular summit in 4, and LVOT septum near the His bundle in 1.

35 tients with CCB was at the level of the left His bundle in 72% and in the proximal left bundle branch

36 x (PHC) will always perturb AVRT because the His bundle is obligatory to the circuit.

37 During permanent nonselective His bundle (ns-HB) pacing, it is crucial to confirm HB c

38 termine whether the FP or the SP reaches the His bundle on a beat-by-beat basis.

39 of recording the activation sequence of the His bundle or right bundle branch (RB) for diagnoses of

40 His bundle pacing (HBP) may also improve clinical outcom

41 His bundle pacing (HBP) remains technically challenging

42 His bundle pacing (HBP) results in physiological ventric

43 ate between selective (S), nonselective (NS) His bundle pacing (HBP), and right ventricular septal ca

44 ither cardiac biventricular pacing (BiVP) or His bundle pacing (HisBP) may prevent adverse structural

45 Conventional permanent His Bundle pacing (PHBP) can be challenging and associat

46 ed to investigate a practical alternative to His bundle pacing after atrioventricular (AV) junctional

47 while in effect permitting the equivalent of His bundle pacing after AV junctional ablation.

48 e increased our understanding of the role of His bundle pacing and left bundle branch area pacing to

49 Left bundle branch pacing and His bundle pacing are novel interventions that imitate t

50 lock), which was most amenable to corrective His bundle pacing by recruitment of latent Purkinje fibe

51 His bundle pacing corrected wide QRS in 54% of all patie

52 The His bundle pacing intervention resulted in the most dram

53 His bundle pacing is preferred after AV junctional ablat

54 onal challenge is programming the system, as His bundle pacing may have specific configurations and r

55 have seen further insights into the role of His bundle pacing on improving outcomes.

56 es (CIEDs): left bundle branch pacing versus His bundle pacing versus biventricular pacing and conven

57 Response to His bundle pacing was assessed in patients with and with

58 Although permanent His bundle pacing was first reported almost 2 decades ag

59 elds similar improvement in contractility as His bundle pacing whilst being more easy to execute.44 I

60 rnative techniques such as multisite pacing, His bundle pacing, and endocardial left ventricular paci

61 de practical recommendations for programming His bundle pacing, to deliver optimal therapy and ensure

62 han surface ECG criteria for correction with His bundle pacing.

63 tivation demonstrated correction of QRS with His bundle pacing.

64 Direct His-bundle pacing (DHBP) produces synchronous ventricula

65 Permanent His-bundle pacing has emerged as the leading candidate f

66 common reason for performing LBBP was a high His-Bundle pacing threshold (n=23) at implant.

67 tricular pacing, conduction system pacing by His-bundle pacing, left bundle branch pacing, left septa

68 ble pacing options, with particular focus on His-bundle pacing.

69 ar pacing, left ventricular (LV) pacing, and His-bundle pacing.

70 cardial layers in conditions of AV block and His-bundle pacing.

71 attern, the retrograde H-RB (right bundle to His bundle) pattern, and the chevron H-RB pattern (simul

72 's triangle with a decapolar catheter in the His bundle position, a multipolar catheter in the corona

73 ecorded from the anterolateral right atrium, His bundle position, and coronary sinus.

74 gs were obtained from the high right atrium, His bundle, posterior triangle of Koch, and coronary sin

75 and bipolar recordings were made at the HRA, His bundle, posterior triangle of Koch, and CS.

76 , regardless of the presence or absence of a His bundle potential in blocked beats, 2:1 block during

77 node reentrant tachycardia, the absence of a His bundle potential in the blocked beats has been consi

78 ormed at an anterior site (A) just above the His bundle recording site and at a posterior atrial site

79 an atrial premature beat (APB) given during His bundle refractoriness followed by resetting of an an

80 actor T-box 18 (TBX18) was injected into the His bundle region in either of 2 experimental protocols:

81 very of AAV-TBX18 (but not AAV-GFP) into the His bundle region in pigs increased heart rate in a clin

82 early activation was always recorded in the His bundle region, regardless of the location of the VA

83 , at which point TBX18 was injected into the His bundle region.

84 TC-ADAC (5 microM) prolonged the stimulus to His bundle (SH) interval by 2.1-fold; this response coul

85 enosine receptor binding) on the stimulus-to-His bundle (SH) interval, a measure of AV nodal conducti

86 trial tissue surrounding the AV node and the His bundle was isolated using sequential radiofrequency

87 HEs recorded from the superior margin of the His bundle were of greater amplitude during basic beats

88 HEs were mapped along the His bundle with roving surface electrodes, along with re