ライフサイエンスコーパス: IMT

1 IMT for the common and internal carotid arteries was det

2 IMT was measured through bilateral carotid ultrasound.

3 Twenty-five patients required 1 IMT and 4 needed 2 drugs.

4 d NSAIDs in 144 (36.7%), SAIDs in 29 (7.4%), IMT in 149 (38.0%), BRMs in 56 (14.3%), and none (N = 14

5 ars after the end of DCCT but did not affect IMT progression thereafter (6-12 years).

6 All IMT patients survived while all osteosarcoma patients di

7 High IMT was defined as an IMT >/=90th percentile according to age-, sex-, race-, a

8 ow a simple electrical circuit which uses an IMT device can exploit SR in engineering applications.

9 ions between the FFGRS, fasting glucose, and IMT.

10 T and coronary artery disease in the IMT and IMT-Progression as Predictors of Vascular Events (IMPROV

11 VE (Carotid Intima Media Thickness [IMT] and IMT-Progression as Predictors of Vascular Events in a Hi

12 determined for the herbicides, IMP, IMZ and IMT, respectively, were greatly superior when compared w

13 whole carotid tree (IMT(mean), IMT(max), and IMT(mean-max)), were analyzed.

14 there was no correlation between DI-RISK and IMT (r = -0.10, P = .215).

15 o association was observed between sCD93 and IMT, sCD93 levels were significantly lower in subjects w

16 e molecular link between the NMD pathway and IMTs has implications for the diagnosis and treatment of

17 had a lower risk of increased carotid artery IMT (0.66[0.50-0.88]) in compared with those with persis

18 BP had increased risk of high carotid artery IMT (relative risk [95% confidence interval]) 1.82[1.47-

19 these population-based data, carotid artery IMT and carotid plaques had a weak relationship to the i

20 rable to estimation of common carotid artery IMT in imaging cardiovascular risk in PE.

21 en with 6 internal and common carotid artery IMT phenotypes using an additive measured genotype model

22 Carotid artery IMT was evaluated by ultrasound.

23 Carotid artery IMT was measured in the left common carotid artery.

24 stimate the individual common carotid artery IMTs in 55 women at PE diagnosis and in 64 women with no

25 Plaque was defined as IMT >1.5 mm in any segment.

26 Baseline IMT (hazard ratio, 1.18 per 10% increment; 95% confidenc

27 ology (i.e., IMT>or=0.9 mm) is needed before IMT consistently relates to poor neuropsychological test

28 her adherence to this dietary pattern before IMT affects injury susceptibility during training remain

29 ch in calcium, potassium, and protein before IMT is positively associated with bone indexes in young

30 IV-infected participants had CCA-IMT and BIF-IMT values that were similar to or lower than those in H

31 n was associated with higher CCA-IMT and BIF-IMT values.

32 ion assessed associations of CCA-IMT and BIF-IMT with HIV infection and cardiovascular disease risk f

33 CCA-IMT) and carotid artery bifurcation (BIF-IMT) between 2010 and 2013.

34 ith a FRS = 22.6% (cohort average), and both IMT(mean-max) and ICCAD above the median, had a 6.5% ris

35 % risk for those with the same FRS, and both IMT(mean-max) and ICCAD below the median.

36 uch more robust than the 24-hour systolic BP-IMT relationship (r = 0.16, P = 0.04).

37 linical and surgical history was taken and C-IMT was measured using B-mode ultrasonography Aplio XG (

38 nternal c-IMT and CAC scores (P < 0.05 for c-IMT and CAC).

39 associated with higher common and internal c-IMT and CAC scores (P < 0.05 for c-IMT and CAC).

40 duals presented with higher right and left c-IMT (p = 0.005 and p = 0.002, respectively), average 24-

41 tudy showed significant difference in mean C-IMT between two groups (p value < 0.001) when correlated

42 Mean C-IMT in the BAC(+) group was 0.86 +/- 0.21 mm vs. 0.71 +/

43 All measures of C-IMT and the interadventitia common carotid artery diamet

44 A risk stratification strategy based on C-IMT and ICCAD as an adjunct to FRFs is a rational approa

45 easures of carotid intima-media thickness (C-IMT) as predictors of cardiovascular events (CVEs), and

46 position, carotid intimal-media thickness (c-IMT), ambulatory blood pressure monitoring (BP), fasting

47 ally significant positive correlation with C-IMT was observed.

48 mmography is independently associated with C-IMT.

49 Carotid IMT as well as MR imaging remodeling index, lipid core,

50 Carotid IMT was assessed by ultrasonography at baseline and 12 a

51 Carotid IMT was measured at 3 locations (common carotid artery,

52 he genetic correlation between the 2 carotid IMT arterial segments was 0.51.

53 ith HeFH who were >/=6 years of age, carotid IMT was significantly greater at baseline compared with

54 fidence interval [CI]: 0.3, 0.7) and carotid IMT (mean difference, 37 mum; 95% CI: 25, 49) were highe

55 th total and regional aortic PWV and carotid IMT while adjusting for several possible confounding fac

56 with aortic pulse wave velocity and carotid IMT.

57 alysis included 14 studies assessing carotid IMT and 7 assessing brachial artery FMD%.

58 ive effect of the A allele on common carotid IMT in women only (women: beta=-0.0047, P=1.63 x 10(-4);

59 nt showed stronger effects on common carotid IMT in women, raising questions about the mechanism of t

60 Common carotid IMT progression from EDIC years 1 to 6 was 0.019 mm less

61 orphisms for association with common carotid IMT was undertaken in 5 independent European cohorts (to

62 For carotid IMT and FMD% values, we computed a pooled estimate of th

63 and Complications Trial (DCCT), had carotid IMT measurements at EDIC years 1, 6, and 12.

64 increase in the odds of having high carotid IMT, respectively, after adjusting for conventional risk

65 any effect on the 40% difference in carotid IMT between Axl genotypes.

66 was used to evaluate differences in carotid IMT between children with HeFH and the unaffected siblin

67 As a result, no difference in carotid IMT could be detected between the 2 groups after 2 years

68 2 years of follow-up, the change in carotid IMT was 0.0054 mm/y (95% confidence interval, 0.0030-0.0

69 owed that SSc patients had increased carotid IMT (summary mean difference 0.11 mm, 95% confidence int

70 cantly less progression of increased carotid IMT in children with HeFH than untreated unaffected sibl

71 In addition, the mean internal carotid IMT levels were higher in subjects with plasma PK levels

72 The internal carotid IMT was 0.77 and 0.81 mm in the periodontal disease and

73 lasma PK and progression of internal carotid IMT.

74 The end-of-study difference in mean carotid IMT between children with HeFH and unaffected siblings a

75 so associated with an increased mean carotid IMT of 15 mum (95% CI: 0, 29) but not after additional a

76 with non-BM-derived cells) mediates carotid IMT.

77 esents the first large-scale GWAS of carotid IMT in a non-European population and identified several

78 progression and increased levels of carotid IMT in type 1 diabetes.

79 ifference and explored predictors of carotid IMT using random-effects meta-regression.

80 n in BM cells contributes to <30% of carotid IMT.

81 -year treatment with rosuvastatin on carotid IMT in children with HeFH.

82 differences in the Endo-PAT index or carotid IMT or stiffness.

83 At baseline, mean+/-SD carotid IMT was significantly greater for the 197 children with

84 It is unclear whether the carotid IMT is a risk indicator of processes affecting Bruch's m

85 Steatosis was associated with carotid IMT and CAC, but not with FP, independent of age, diabet

86 us on chromosome 16, associated with carotid IMT and coronary artery disease in the IMT and IMT-Progr

87 ctivation is associated with both CAC and CC IMT in otherwise healthy individuals, consistent with th

88 n carotid artery intimal media thickness (CC IMT) in European-Americans [memory: beta = 0.02 (0.006,

89 fied the association between the GRS and CCA IMT (p for interaction=0.001).

90 The associations between the GRS and CCA IMT were stronger in participants with systolic blood pr

91 en genetic predisposition to obesity and CCA IMT.

92 as significantly associated with greater CCA IMT (p<0.001) after adjustment for age and gender.

93 of the GRS relating to 0.028 mm greater CCA IMT, p for trend<0.001) than those with SBP<120 mmHg and

94 The primary outcome was mean CCA IMT, measured at baseline and 12 mo, with B-mode ultraso

95 At 12 mo, CCA IMT regressed (mean +/- SD: -0.01 +/- 0.04 mm; P < 0.001

96 lled type 1 and type 2 diabetes may slow CCA IMT progression.

97 ent of the GRS relating to 0.001 smaller CCA IMT, p for trend=0.930).

98 n carotid artery intima media thickness (CCA IMT) progression, compared with a control group continui

99 y included 428 young Chinese adults with CCA IMT measured using a high-resolution B-mode tomographic

100 t far wall of the common carotid artery (CCA-IMT) and carotid artery bifurcation (BIF-IMT) between 20

101 thickness of the common carotid artery (CCA-IMT), pulse wave velocity (PWV), augmentation index, blo

102 ociations with incident CAD and baseline CCA-IMT were analyzed by using Cox regression and ANCOVA, re

103 in women, 757 to 790 microm in men), but CCA-IMT progression did not differ by HIV serostatus, either

104 ough the 1-y intervention did not change CCA-IMT or BP, clinically relevant improvements in arterial

105 ntervention did not significantly change CCA-IMT, augmentation index, or BP, but pulse pressure varia

106 In the included group, CCA-IMT was significantly correlated with snoring sound ener

107 75 years), HIV-infected participants had CCA-IMT and BIF-IMT values that were similar to or lower tha

108 HIV infection was associated with higher CCA-IMT and BIF-IMT values.

109 were also not associated with increased CCA-IMT.

110 usted rate of progression in the maximal CCA-IMT compared with nonusers (14 mum/year versus 22 mum/ye

111 Among the 158 RA patients, the maximal CCA-IMT increased in 82% (median 16 mum/year; P < 0.001) and

112 ted average yearly change in the maximal CCA-IMT was significantly greater in patients with earlier R

113 Unadjusted mean CCA-IMT increased (725 to 752 microm in women, 757 to 790 mi

114 rospectively enrolled for measuring mean CCA-IMT with B-mode ultrasonography, body mass index, metabo

115 near regression assessed associations of CCA-IMT and BIF-IMT with HIV infection and cardiovascular di

116 n carotid artery intima-media thickness (CCA-IMT) and new focal carotid artery plaque formation (IMT

117 n carotid artery intima-media thickness (CCA-IMT) were available for 846 men.

118 n carotid artery intima-media thickness (CCA-IMT) with snoring sounds in OSA patients.

119 d, controlling for clinical characteristics, IMT reader, and imaging device.

120 th progression of both internal and combined IMT (Wilks Lambda P value of 0.005).

121 and internal carotid arteries, and composite IMT variables considering the whole carotid tree (IMT(me

122 performed significantly worse than composite IMTs that incorporated plaques (p < 0.001).

123 In conclusion, IMT is a rare lung tumor in adults and may simulate mali

124 The use of concurrent IMT did not show a significant influence on DRT.

125 differences regarding the use of concurrent IMT.

126 g charge-spin coupling drives the concurrent IMT and AFM-to-FM transition, which fosters the near roo

127 n, we suggest that the phragmoplast contains IMTs and highly dynamic noninterdigitating MTs, which wo

128 6 weeks (late IMT) and with historical data (IMT added when uveitis uncontrolled).

129 arrier generation leading to a doping driven IMT.

130 strated that a threshold of pathology (i.e., IMT>or=0.9 mm) is needed before IMT consistently relates

131 ly show that a purely non-thermal electrical IMT can occur in both materials.

132 In the elderly, clinically elevated IMT is related to diminished attention-executive functio

133 d with bone indexes in young adults entering IMT.

134 tibia BMC and strength in recruits entering IMT.

135 o the neck or chest also had greater femoral IMT.

136 nt regimen had increased carotid and femoral IMTs and higher t-PA and PAI-I levels, indicating vascul

137 years) after treatment, carotid and femoral IMTs in CCSs were not different from those of controls.

138 de significant discovery P=6.75 x 10(-7) for IMT(max); replication P=7.24x10(-6) for common cIMT; adj

139 sed genome-wide association study (GWAS) for IMT to identify polymorphisms influencing IMT and to det

140 d new focal carotid artery plaque formation (IMT >1.5 mm) over median 7 years.

141 Greater IMT was associated with presence of cardiovascular risk

142 Similarly, G1 CHC patients had a greater IMT compared with control patients (1.04 +/- 0.21 versus

143 icrom/y (95% CI 2.6 to 7.4 microm/y) greater IMT progressions among persons in the same metropolitan

144 High IMT was defined as an IMT >/=90th percentile according t

145 ith MetS are at increased risk of adult high IMT and T2DM, these data indicate that the resolution of

146 ce, carotid artery plaque presence, and high IMT for incident CVD events.

147 95% confidence interval: 2.4 to 4.9) of high IMT and 12.2 times the risk (95% confidence interval: 6.

148 and follow-up and examined for risk of high IMT and T2DM.

149 HD more than carotid plaque presence or high IMT.

150 Higher IMT in midlife was associated with an increased hazard r

151 IGT individuals also demonstrated higher IMT in right and left carotid arteries (P = 0.017 and P

152 /- 4.6 years at baseline, 60% women), higher IMT in midlife was associated with development of small

153 16 mum/year; P < 0.001) and the maximal ICA-IMT increased in 70% (median 25 mum/year; P < 0.001).

154 For the maximal ICA-IMT, cumulative prednisone exposure was associated with

155 resulting from the combination of FRFs+ICCAD+IMT(mean-max) was +12.1% (p < 0.01).

156 zapyr (IMP), imazapic (IMZ) and imazethapyr (IMT) with determination by HPLC-PAD (High performance li

157 The difference in IMT predicted by a 1 SD increment in the FGGRS (0.0048 m

158 There were no significant differences in IMT between groups.

159 Differences in IMT progression between DCCT intensive and conventional

160 f IMT, explaining most of the differences in IMT progression between DCCT treatment groups.

161 the GRS was related to 0.023 mm increment in IMT.

162 IgE class switching events were observed in IMT samples, consistent with NIK upregulation in these t

163 rgets of the NMD pathway were upregulated in IMT samples, indicating that the UPF1 mutations led to r

164 CCSs treated with localized RT had increased IMT outside the primary irradiation field.

165 concentrations are associated with increased IMT progression and that greater reductions in PM2.5 are

166 ctive on the temperature- and doping-induced IMT process.

167 although it may be that these loci influence IMT through nonglucose pathways.

168 or IMT to identify polymorphisms influencing IMT and to determine if distinct carotid artery segments

169 IMT was given before vs after 6 weeks (late IMT) and with historical data (IMT added when uveitis un

170 festations of three adult patients with lung IMT.

171 mm for mean IMT and 1.25 vs 1.21 mm for max IMT; P < .05), while PWV did not differ between groups (

172 mm for mean IMT and 1.25 vs 1.21 mm for max IMT; p<0.05), while PWV did not differ between groups (p

173 The average of 8 maximal IMT measurements (IMT(mean-max)), alone or combined with

174 Similar associations were found for maximum IMT.

175 The presence of at least 1 plaque (maximum IMT >1.5 mm) performed significantly worse than composit

176 Mean IMT was associated with the 10-year incidence of pure ge

177 Mean IMT>/=75th percentile (for age, sex, and race) alone did

178 n-events better than the common carotid mean IMT (net reclassification improvement [NRI]: +11.6% and

179 r H (CFH) genotypes, and other factors, mean IMT was associated with the 10-year incidence of early A

180 than HIV- children (1.05 vs 1.02 mm for mean IMT and 1.25 vs 1.21 mm for max IMT; P < .05), while PWV

181 o HIV- children (1.05 mm vs 1.02 mm for mean IMT and 1.25 vs 1.21 mm for max IMT; p<0.05), while PWV

182 The mean IMT of the LCCA tended to differ across the KD subgroups

183 gnificantly associated (P = 0.009) with mean IMT.

184 nsidering the whole carotid tree (IMT(mean), IMT(max), and IMT(mean-max)), were analyzed.

185 The average of 8 maximal IMT measurements (IMT(mean-max)), alone or combined with ICCAD, classified

186 Median IMT was slightly thicker in PHIVs compared to HIV- child

187 Median IMT was slightly thicker in PHIVs than HIV- children (1.

188 nce of early AMD (odds ratio [OR] per 0.1 mm IMT, 1.11; 95% confidence interval [CI], 1.00-1.21; P =

189 = 0.02) but not exudative AMD (OR per 0.1 mm IMT, 1.14; CI, 0.97-1.34; P = 0.11).

190 -1.21; P = 0.03) and late AMD (OR per 0.1 mm IMT, 1.27; CI, 1.10-1.47; P = 0.001).

191 ce of pure geographic atrophy (OR per 0.1 mm IMT, 1.31; CI, 1.05-1.64; P = 0.02) but not exudative AM

192 to the incidence of late AMD (OR per 0.1 mm IMT, 2.79 for 4-6 sites vs. none; CI, 1.06-7.37; P = 0.0

193 electively cross-linked interdigitating MTs (IMTs) to allow antiparallel MTs to be closely engaged in

194 nsitive variations in the thermal nanodomain IMT behaviour, this suggests that the IMT is highly susc

195 atients who had a current or past history of IMT or who were in remission.

196 lood pressure were significant predictors of IMT progression.

197 were strongly associated with progression of IMT, explaining most of the differences in IMT progressi

198 e (ALK) fusion genes in approximately 50% of IMTs and the role of ALK inhibition in the treatment of

199 mmune infiltration that is characteristic of IMTs.

200 Although the presence of IMTs was not essential for vesicle trafficking, they wer

201 Often IMT is measured as the average of these 2 arteries; yet,

202 was no correlation between length of time on IMT and the risk of histoplasmosis.

203 tronic mechanism dominating the photoinduced IMT, but also highlight the difficulty to deduce microsc

204 Predictive values of carotid plaque, IMT, and CAC presence were compared using Cox proportion

205 dnisolone 1 mg/kg daily with slow taper plus IMT within 3 months.

206 s with metastatic or inoperable ALK-positive IMT received crizotinib orally twice daily.

207 bout the molecular pathways that precipitate IMT formation.

208 A decay (NMD) pathway, in 13 of 15 pulmonary IMT samples.

209 The mean left and right IMT were 0.754 (0.210) mm and 0.751 (0.187) mm, respecti

210 Intensive treatment slowed IMT progression for 6 years after the end of DCCT but di

211 eline PM2.5 were also associated with slowed IMT progression (-2.8 microm/y [95% CI -1.6 to -3.9 micr

212 er reductions in PM2.5 are related to slower IMT progression.

213 ctive AA genotype was associated with slower IMT progression in women (P=0.04) but not in men.

214 Segment-specific IMT measurements of common carotid, bifurcation, and int

215 comprised of patients who received systemic IMT for ocular inflammation but did not develop histopla

216 The IMT and presence of plaque were assessed using B-mode ul

217 Fundamentally, the IMT in VO2 can be triggered on femtosecond timescale to

218 rotid IMT and coronary artery disease in the IMT and IMT-Progression as Predictors of Vascular Events

219 ng techniques, we simultaneously monitor the IMT in VO2 and the change of plasmons on gold infrared n

220 We confirm that the IMT actually occurs concomitantly with the FM transition

221 In this study, we find that the IMT behavior of NbO2 follows the field-induced nucleatio

222 domain IMT behaviour, this suggests that the IMT is highly susceptible to local changes in, for examp

223 atients who opted to have PPV to treat their IMT type 2 and FTMH compared with those who did not unde

224 Concurrent classic immunomodulatory therapy (IMT) was given to 251 patients.

225 iation of systemic immunomodulatory therapy (IMT).

226 ive therapy drugs (immunomodulatory therapy [IMT]), or biologic response modifiers (BRMs) was assesse

227 consumption associated with the non-thermal IMT is extremely low, rivaling that of state-of-the-art

228 chanisms of carotid intima-media thickening (IMT) are largely unknown.

229 , hsCRP, and zonulin correlated with thicker IMT in PHIV (P <= .05).

230 o, hsCRP and zonulin correlated with thicker IMT in PHIV (p<=0.05).

231 ] plaques defined as intima-media thickness (IMT) > 1.5 mm), coronary computed tomography scan (sever

232 Intima-media thickness (IMT) and carotid plaques, defined as focal thickening of

233 ommon carotid artery intima-media thickness (IMT) and pulse wave velocity (PWV) were evaluated in 101

234 ommon carotid artery intima-media thickness (IMT) and pulse-wave velocity (PWV) were evaluated in 101

235 T was related to the intima-media thickness (IMT) and to atherosclerotic plaque in carotid arteries i

236 od on carotid artery intima-media thickness (IMT) and type 2 diabetes mellitus (T2DM).

237 ommon carotid artery intima-media thickness (IMT) during or after PE has not indicated any increased

238 ls on carotid artery intima-media thickness (IMT) in adulthood.

239 Carotid intima-media thickness (IMT) is a marker of subclinical atherosclerosis that can

240 nd increased carotid intima-media thickness (IMT) may indicate elevated cardiovascular disease (CVD)

241 Intima-media thickness (IMT) of the common and internal carotid arteries is an e

242 in the mean maximal intima-media thickness (IMT) of the common carotid artery (CCA) and the internal

243 tude Testing index), intima-media thickness (IMT) of the right common carotid artery (RCCA) and the l

244 Carotid intima-media thickness (IMT) was acquired and measured by trained research nurse

245 hy subjects; carotid intima media thickness (IMT) was assessed as a marker of systemic vascular disea

246 Carotid intima-media thickness (IMT) was assessed by ultrasound.

247 Carotid plaques and intima media thickness (IMT) were measured at baseline (1991-1994).

248 and internal carotid intima-media thickness (IMT) were measured by B-mode ultrasonography in EDIC yea

249 carotid artery (CCA) intima-media thickness (IMT), a measure of subclinical atherosclerosis.

250 s plaque and carotid intima-media thickness (IMT), computed tomography, magnetic resonance imaging, f

251 id- and femoral-wall intima-media thickness (IMT), flow-mediated vasodilatation of the brachial arter

252 ommon carotid artery intima-media thickness (IMT).

253 by increased carotid intima-media thickness (IMT).

254 egulation or carotid intima-media thickness (IMT).

255 progression of the intima-medial thickness (IMT) of the common carotid artery, as an indicator of at

256 the IMPROVE (Carotid Intima Media Thickness [IMT] and IMT-Progression as Predictors of Vascular Event

257 of atherosclerosis (intima-media thickness [IMT] by echo-color Doppler) in a large, inclusive survey

258 = 0.23, P = 0.002) were directly related to IMT, and these associations were much more robust than t

259 However, no relationship to IMT was seen.

260 h associated systemic disease may respond to IMT or BRMs.

261 ts with necrotizing scleritis may respond to IMT, mainly alkylating agents.

262 ge daytime BP and clinic B were unrelated to IMT.

263 s elevated during initial military training (IMT), particularly in lower-extremity bones such as the

264 cally induced insulator-to-metal transition (IMT) characteristic.

265 tching of VO2 insulator-to-metal transition (IMT) locally on the scale of 15 nm or less and control o

266 The insulator-metal transition (IMT) of vanadium dioxide (VO2) has remained a long-stand

267 rs before the insulator-to-metal transition (IMT), which is still controversial.

268 hich triggers an insulator-metal transition (IMT).

269 hold noise of an insulator-metal-transition (IMT) material can enable SR.

270 ariables considering the whole carotid tree (IMT(mean), IMT(max), and IMT(mean-max)), were analyzed.

271 Inflammatory myofibroblastic tumor (IMT) is a distinctive mesenchymal neoplasm characterized

272 Inflammatory myofibroblastic tumor (IMT), also known as inflammatory pseudotumor, is a benig

273 n = 6), inflammatory myofibroblastic tumors (IMT; n = 6), and pleuropulmonary blastoma (n = 5).

274 Inflammatory myofibroblastic tumors (IMTs) are characterized by myofibroblast proliferation a

275 REVIEW: Inflammatory myofibroblastic tumors (IMTs) are indolent mesenchymal neoplasms associated with

276 CL) and inflammatory myofibroblastic tumors (IMTs).

277 The timescales of the ultrafast IMT vary from 40+/-8 fs, that is, shorter than a suggest

278 This phenomenon is key for understanding IMT physics and developing novel memory elements and bra

279 relapsed ALCL and metastatic or unresectable IMT highlight the importance of the ALK pathway in these

280 The NRI for US IMT in addition to traditional risk factors was not sign

281 Results were compared when IMT was given before vs after 6 weeks (late IMT) and wit

282 nder the curve, approximately 0.95), whereas IMT was not (area under the curve, 0.49).

283 measured by zonulin remained associated with IMT (beta = 0.03 and 0.02, respectively; P <= .03).

284 measured by zonulin remained associated with IMT (beta=0.03 and 0.02 respectively, p<=0.03).

285 h renal and splenic RIs were associated with IMT (renal RI: r = 0.19, P = .022; splenic RI: r = 0.23,

286 BP metrics are independently associated with IMT.

287 o maintained an independent association with IMT (beta = 0.14, P = 0.04).

288 ping chronic recurrent uveitis compared with IMT given as clinically indicated.

289 sease, renal and splenic RIs correlated with IMT (renal RI: r = 0.33, P < .001; splenic RI: r = 0.30,

290 High-dose corticosteroids with IMT within 3 months resulted in improved visual outcomes

291 Eyes with IMT initiated within 6 weeks had better visual outcome t

292 he significant association of the FGGRS with IMT suggests a possible causal association of elevated f

293 significant associations were not found with IMT progression without adjustment for metropolitan area

294 ate (eGFR; r = -0.19, P = .001) but not with IMT (r = 0.08, P = .174).

295 long-term partial response in a patient with IMT carrying an ALK translocation but not in a patient w

296 The overall response rate for patients with IMT (treated at 100, 165, and 280 mg/m(2)/dose) was 86%.

297 eatment option for a subset of patients with IMT and pulmonary adenocarcinoma.

298 s, respectively, and in 43% of patients with IMT.

299 CL280, and 36% (five of 14) of patients with IMT.

300 Successful treatment with IMT was associated with diffuse or nodular scleritis wit