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1 endent on transmission by ticks in the genus Ixodes.
4 solated from the ticks Ixodes scapularis and Ixodes affinis, the cotton rat (Sigmodon hispidus), and
5 ets obtained from public databases, both for Ixodes and non-Ixodes hard tick species, using soft tick
6 64 records of ticks of the genera Amblyomma, Ixodes, and Haemaphysalis and their hosts in the Neotrop
7 lyomma maculatum, 383 Ixodes scapularis, two Ixodes brunneus, and 35 Dermacentor variabilis) were tes
8 ected on B. burgdorferi in unfed or engorged Ixodes dammini (also known as I. scapularis) ticks, furt
9 tory birds in the long-distance dispersal of Ixodes dammini ticks and in the spread of Lyme disease,
10 of nine mice infected by Ixodes scapularis (Ixodes dammini) tick inoculation of an isolate from Nant
11 Wisconsin and Minnesota, Ixodes scapularis (Ixodes dammini) ticks are the vector of three microorgan
13 Most of the strains isolated from the tick Ixodes dentatus in Missouri and Georgia belonged to the
17 se peptides were identified in the available Ixodes genome and expressed sequence tag (EST) database.
20 burgdorferi, is transmitted by ticks of the Ixodes genus and, if untreated, can cause significant mo
21 ts confirm previous studies showing that the Ixodes genus is monophyletic and clarify the relationshi
23 om public databases, both for Ixodes and non-Ixodes hard tick species, using soft ticks as an outgrou
24 ted of a single species, the nidiculous tick Ixodes hexagonus (prevalence = 24.3%; mean intensity = 7
28 ting Borrelia sp. nov. isolated from nymphal Ixodes minor collected in South Carolina showed their cl
29 wo enzootic tick vectors, Ixodes affinis and Ixodes minor, rarely bite humans but are more important
32 pathogenic Ehrlichia bacteria isolated from Ixodes ovatus (IOE) reveal, however, that this paradigm
33 y related virulent ehrlichiae transmitted by Ixodes ovatus (IOE) ticks given intraperitoneally or int
34 rlichiosis caused by Ehrlichia bacteria from Ixodes ovatus (IOE) to determine whether OMP immunizatio
35 ng infection with a strain of Ehrlichia from Ixodes ovatus (IOE) were evaluated using a model that cl
36 lowing lethal infection with highly virulent Ixodes ovatus ehrlichia (IOE), an obligate intracellular
37 uris, which causes persistent infection, and Ixodes ovatus Ehrlichia (IOE), which is either acutely l
39 wild-type (WT) mice with virulent Ehrlichia (Ixodes ovatus Ehrlichia [IOE]) results in CD8+ T-cell-me
40 t mice cleared a primary low-dose (nonfatal) Ixodes ovatus ehrlichia infection, a secondary low-dose
42 in vitro coculture of memory CD8 T cells and Ixodes ovatus ehrlichia-infected peritoneal exudate cell
46 sely related to E. chaffeensis isolated from Ixodes ovatus ticks in Japan, causes fatal infection of
47 duced actin phosphorylation was dependent on Ixodes p21-activated kinase (IPAK1)-mediated signaling.
52 ation in the continuous part of the range of Ixodes pacificus reinforces recent recognition of the di
58 he response of the western blacklegged tick (Ixodes pacificus), the primary Lyme disease vector in we
64 ssaged Ixodes scapularis cell line ISE18 and Ixodes ricinus cell lines IRE/CTVM19 and IRE/CTVM20 had
68 he midgut transcriptome composition in adult Ixodes ricinus females during early and late phase of en
72 m induces the phosphorylation of actin in an Ixodes ricinus tick cell line and Ixodes scapularis tick
74 ed to silencing of virus replication in live Ixodes ricinus ticks and abolished virus neurotropism in
76 conducted on an arthropod species (the tick Ixodes ricinus) on which de novo sequencing was performe
77 protist Trichomonas vaginalis, the hard tick Ixodes ricinus, and the flatworm Schistosoma mansoni.
79 ocytophilum infection on an enclosed area of Ixodes ricinus-infested pasture in North Wales, United K
80 ent (WI-1) and nine of nine mice infected by Ixodes scapularis (Ixodes dammini) tick inoculation of a
82 ft genome for the Rickettsia endosymbiont of Ixodes scapularis (REIS), a symbiont of the deer tick ve
83 es of subjects who presented with a definite Ixodes scapularis (Say) tick bite were measured to deter
84 ribe the 2.1 Gbp nuclear genome of the tick, Ixodes scapularis (Say), which vectors pathogens that ca
87 mine whether impaired TLR signaling enhances Ixodes scapularis acquisition of B. burgdorferi, we fed
88 quitination machinery is present in the tick Ixodes scapularis and demonstrate that the E3 ubiquitin
89 st southern strains, isolated from the ticks Ixodes scapularis and Ixodes affinis, the cotton rat (Si
92 ivary protein 20 (Salp20) is a member of the Ixodes scapularis anti-complement protein-like family of
93 A. phagocytophilum induces ticks to express Ixodes scapularis antifreeze glycoprotein (iafgp), which
94 omma americanum, Dermacentor variabilis, and Ixodes scapularis are among the principal tick species a
95 rved protein that was discovered recently in Ixodes scapularis as a tick protective antigen and has a
101 Additionally, the recent sequencing of the Ixodes scapularis genome and characterization of tick im
102 Out of three encoding genes representing Ixodes scapularis genome paralogs, IrCD1 is the most dis
105 es (PMN) treated with the saliva of the tick Ixodes scapularis have reduced expression of beta(2) int
106 immune deficiency (IMD) pathway of the tick Ixodes scapularis How XIAP activates the IMD pathway in
107 nce of the miRNA response of the tick vector Ixodes scapularis in response to Anaplasma phagocytophil
114 ly, we show that a dae gene in the deer tick Ixodes scapularis limits proliferation of Borrelia burgd
116 etes to ticks after capillary inoculation of Ixodes scapularis nymphs and the subsequent spirochetal
117 spC expression by B. burgdorferi in infected Ixodes scapularis nymphs as they fed on uninfected mice
118 lp lipoproteins in mice after challenge with Ixodes scapularis nymphs harboring B. burgdorferi 297.
119 protected from infection when infested with Ixodes scapularis nymphs harboring virulent B. burgdorfe
121 a tick bite and challenged 16 weeks later by Ixodes scapularis nymphs infected with either the same o
123 nd challenged either 12 or 16 weeks later by Ixodes scapularis nymphs infected with the same agent.
124 were infested four times with pathogen-free Ixodes scapularis nymphs prior to infestation with nymph
130 Previous studies have demonstrated that both Ixodes scapularis saliva and Borrelia burgdorferi antige
132 now describe a feeding-inducible protein in Ixodes scapularis saliva, Salp15, that inhibits CD4(+) T
137 A. phagocytophilum induces expression of the Ixodes scapularis salp16 gene in the arthropod salivary
138 phalitis following a documented bite from an Ixodes scapularis tick and the erythema migrans rash ass
139 ear whether antimicrobial treatment after an Ixodes scapularis tick bite will prevent Lyme disease.
140 nine components to estimate the frequency of Ixodes scapularis tick bites and the resulting incidence
141 agated continuously for over 500 days in the Ixodes scapularis tick cell line IDE8 by using the Garde
143 iftly upregulated when spirochetes leave the Ixodes scapularis tick gut, migrate to the salivary glan
144 agent of human granulocytic anaplasmosis, in Ixodes scapularis tick salivary glands, to detect protei
145 me disease is transmitted by the bite of the Ixodes scapularis tick, which can also transmit Anaplasm
147 infected mice (a reservoir model) to nymphal Ixodes scapularis ticks (a biological vector) and subseq
148 aled that both uninfected larval and nymphal Ixodes scapularis ticks acquired B. burgdorferi as early
149 The mutant cells were able to survive within Ixodes scapularis ticks after a blood meal from naive mi
151 isingly, sigma54 mutants were able to infect Ixodes scapularis ticks and be maintained for at least 2
152 is an intracellular pathogen transmitted by Ixodes scapularis ticks and causing human granulocytic a
153 were identified from among 99 isolates from Ixodes scapularis ticks and from white-footed mice (Pero
154 valence of Borrelia burgdorferi infection in Ixodes scapularis ticks and Peromyscus sp. mice captured
155 The assay was tested on field-collected Ixodes scapularis ticks and shown to have 100% concordan
156 urgdorferi bbk32 and bbk50 expression within Ixodes scapularis ticks and the murine host, and the eff
157 ssible from inoculated C.B-17 mice to larval Ixodes scapularis ticks and, subsequently, from infected
160 s amplified from template DNA extracted from Ixodes scapularis ticks collected in Rhode Island and fr
161 hether the salivary gland extract (SGE) from Ixodes scapularis ticks facilitates the transmission and
162 d to show that spirochetes could be found in Ixodes scapularis ticks feeding on 4 of 10 antibiotic-tr
165 o 2 strains of DTV that had been detected in Ixodes scapularis ticks from Massachusetts in 1996 and i
169 . phagocytophilum strain HZ in SCID mice and Ixodes scapularis ticks infected with strain NTN-1.
172 In this paper we explore the contribution of Ixodes scapularis ticks to the pathogenicity of Borrelia
177 gene expression within the guts of engorging Ixodes scapularis ticks was examined by use of different
180 burgdorferi produce OspA in the gut of unfed Ixodes scapularis ticks, and many spirochetes repress Os
181 the level of thiamin and its derivatives in Ixodes scapularis ticks, the enzootic vector of Bb, is e
182 ctin in an Ixodes ricinus tick cell line and Ixodes scapularis ticks, to alter the ratio of monomeric
192 ized Lyme disease focus with an abundance of Ixodes scapularis vector ticks and the first documentati
194 tect remnants of blood in blacklegged ticks (Ixodes scapularis) and correctly determine the vertebrat
195 , infecting local populations of deer ticks (Ixodes scapularis) and to test the fit to a neutral IAM.
196 B. burgdorferi in nymphal blacklegged ticks (Ixodes scapularis) collected at the sites the following
197 discover that a toxin in blacklegged ticks (Ixodes scapularis) horizontally acquired from bacteria-c
199 ain 297 naturally colonized their arthropod (Ixodes scapularis) vector, were maintained in ticks thro
200 relia burgdorferi) and its main tick vector (Ixodes scapularis) was studied concurrently and comparat
201 ds to the gut of the intermediate tick host (Ixodes scapularis), and that this interaction is mediate
202 s aegypti and Culex quinquefasciatus), tick (Ixodes scapularis), body louse (Pediculus humanus), kiss
205 atural antibodies when its arthropod vector, Ixodes scapularis, begins feeding on a mammalian host.
206 and that the typical vector of Lyme disease, Ixodes scapularis, can acquire the spirochetes from infe
207 ber 1994 through December 1995 from the tick Ixodes scapularis, collected from vegetation, and from t
208 nhanced by the presence of B. burgdorferi in Ixodes scapularis, first indicating that spirochaetes mi
209 va of the hard tick and Lyme disease vector, Ixodes scapularis, has a repertoire of compounds that co
210 ding the role that nymphal and female ticks, Ixodes scapularis, have in the epidemiology of Lyme dise
211 rtant than the human biting "bridge" vector, Ixodes scapularis, in maintaining the enzootic spirochet
215 eeking larvae uninfected with the spirochete Ixodes scapularis, thereby perpetuating the agent throug
216 omma americanum, 76 Amblyomma maculatum, 383 Ixodes scapularis, two Ixodes brunneus, and 35 Dermacent
217 e to the range expansion of the tick vector, Ixodes scapularis, upon which the causative agent, Borre
218 itor (TFPI), Ixolaris, from the ixodid tick, Ixodes scapularis, which has 10 cysteines, and a thrombi
220 a burgdorferi, colonizes the gut of the tick Ixodes scapularis, which transmits the pathogen to verte
235 novel neurotransmitter signaling pathway in Ixodes SG, and suggests its role in water uptake by type
238 human anaplasmosis, are both transmitted by Ixodes sp. ticks and may occasionally coinfect a host.
240 nfects mostly neutrophils, it transmitted by Ixodes species ticks, and occurs mostly in the upper mid
247 , is maintained in an enzootic cycle between Ixodes spinipalpis ticks and Neotoma mexicana rats (27).
248 DNA sequencing showed the similarity between Ixodes spp. cell lines at different passages, their kary
251 ffered from 2n = 28 chromosomes for parental Ixodes spp. ticks, and both increase and decrease in chr
252 philum is a bacterium that is transmitted by Ixodes spp. ticks, in which it resides in salivary gland
253 urgdorferi sensu lato (s.l.), transmitted by Ixodes spp. ticks, is the causative agent of Lyme diseas
256 wave morphology and borrelial DNA in endemic Ixodes stilesi ticks collected in Chile from environment
259 cits (HDs) at the CP scale in raccoon ticks (Ixodes texanus; n=718) collected from raccoons (Procyon
260 ve isolates are (partially) protected by the Ixodes Tick Salivary Lectin Pathway Inhibitor (TSLPI) pr
261 ughout the Northern Hemisphere, and the same Ixodes tick species transmitting the etiologic agents of
263 he Lyme disease pathogen, cycles between its Ixodes tick vector and vertebrate hosts, adapting to vas
264 of longevity of some reservoir hosts and the Ixodes tick vectors' life cycle, long-term studies are r
267 gression through the enzootic cycle using an Ixodes tick/C3H-HeJ mouse model and tick immersion feedi
269 nking H14 stem varies between six species of Ixodes ticks (Acari: Ixodidae) where only 33 invariant s
270 transmitted to vertebrate hosts via infected Ixodes ticks and are the etiologic agents of Lyme diseas
277 ient B. burgdorferi survive and replicate in Ixodes ticks but are attenuated for infection and dissem
284 miyamotoi is a relapsing fever spirochete in Ixodes ticks that has been recently identified as a huma
287 elia burgdorferi, is transmitted by bites of Ixodes ticks to mammalian reservoir hosts and humans.
288 Amblyomma, Dermacentor, Rhipicephalus, and Ixodes ticks were reported as the most predominant ectop
289 ochetal bacterium that depends on arthropod (Ixodes ticks) and mammalian (rodent) hosts for its persi
290 zootic cycle, transiting between its vector, Ixodes ticks, and a diverse range of vertebrate hosts.
292 rochetes are able to survive normally in the Ixodes ticks, mice fed upon by the DeltacheY2-infected t
294 rstanding of the epidemiology and ecology of ixodes ticks, the established vector for transmission of
296 isease is transmitted by the bite of certain Ixodes ticks, which can also transmit Anaplasma phagocyt