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1 array to identify antigens in the context of Johne's disease.
2 ined from a cow exhibiting clinical signs of Johne's disease.
3 human tuberculosis, bovine tuberculosis and Johne's disease.
4 is strains to understand the epidemiology of Johne's disease.
5 w generations of diagnostic tests for bovine Johne's disease.
6 e a major impact on improved diagnostics for Johne's disease.
7 tered with strains derived from animals with Johne's disease.
8 symptoms similar to those found in clinical Johne's disease.
9 jor agents in bovine respiratory disease and Johne's disease.
10 es are pivotal events in the pathogenesis of Johne's disease.
11 tion is a sensitive indicator of subclinical Johne's disease.
12 commercially available diagnostic tests for Johne's disease.
13 non-infected cattle and cattle sensitized to Johne's disease.
14 urther testing as vaccine candidates against Johne's disease.
15 tuberculosis (MAP) is the causative agent of Johne's Disease, a chronic granulomatous enteritis of ru
16 nto the milk and feces of cows with advanced Johne's disease, allowing the transmission of M. avium s
17 . paratuberculosis is the causative agent of Johne's Disease, an economically important intestinal ai
18 cterium avium subsp. paratuberculosis causes Johne's disease, an enteric infection in cattle and othe
19 mental evidence defining MAP as the cause of Johne's disease and the issues and controversies surroun
23 d by sera from 100% of animals with advanced Johne's disease (clinical stage) (12 cattle, 2 goats, an
24 tectable early in the course of experimental Johne's disease, even preceding the development of speci
26 . paratuberculosis is the causative agent of Johne's disease in animals and has been hypothesized to
27 cterium avium subsp. paratuberculosis causes Johne's disease in cattle and is a serious problem for t
30 f immunoassays specific for the diagnosis of Johne's disease in cattle requires antigens specific to
33 cterium avium subsp. paratuberculosis causes Johne's disease in ruminants, a chronic enteric disease
35 levels (>1.5-fold; P < 0.05) in tissues from Johne's disease-infected animals relative to noninfected
36 avium subsp. paratuberculosis, the agent of Johne's disease, infects ruminant hosts by translocation
45 effort has been directed toward controlling Johne's disease (JD), a chronic granulomatous intestinal
51 or the diagnosis of clinical and subclinical Johne's disease (paratuberculosis) compared to bacteriol
54 ne expression profiles of ileal tissues from Johne's disease-positive (n = 6), and Johne's disease-ne
55 n increased protein expression in tissues of Johne's disease-positive cattle relative to tissues from
56 could result in cells within the lesions of Johne's disease-positive cattle that are highly resistan
57 F1 (27.5-fold) gene expression in tissues of Johne's disease-positive cows relative to tissues from c
58 cells (PBMCs) from clinical and subclinical Johne's disease-positive Holstein cows (n = 2 per group)
59 Development of models to mimic aspects of Johne's disease remains an elusive goal because of the c
60 ammatory bowel diseases (IBD) and veterinary Johne's disease suggests that Mycobacterium avium subsp.
63 all, novel insights into the early stages of Johne's disease were obtained, and a practical model of
64 c wasting disease termed paratuberculosis or Johne's disease, whereas M. avium subsp. avium causes on