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1                                              K(m) values for NnOMT1 and NnOMT5 were 20 and 13 mum for
2                                              K(mem/w) values were consistent with the literature data
3                                              K-PPn was originally discovered in budding yeast (Saccha
4                                              K-Ras must interact primarily with the plasma membrane (
5                                              K[7(*)] is not long-term stable in a THF/c-hexane soluti
6 halpy of vaporization of n-eicosane at 359.0 K was calculated to be Delta(vap)H = 91.27 +/- 0.28 kJ/m
7 anglement and no magnetic ordering down to 0 K.
8 r effective temperatures between 2,600-9,000 K.
9 n be lifted by exchange interactions below 1 K.
10 les, a detection limit of 75 +/- 12 mg L(-1) K(+), and an adequate reproducibility and repeatability.
11 are a linear range from 250 to 4000 mg L(-1) K(+), covering all the concentrations expected in must a
12 tice thermal conductivity of about 4 W m(-1) K(-1) at room temperature, one order of magnitude smalle
13  diameter, reaching a value of 8 um(2) s(-1) K(-1) for a 10 um polystyrene bead.
14 nly span a factor of 4 (i.e., 0.1-0.4 Wm(-1) K(-1) ).
15 y, and low thermal conductivity (0.24 W m(-1)K(-1)) based on a process of spatially selective deligni
16 hermal conductivities of less than 1 W.m(-1).K(-1) at room temperature.
17  constant with coherent tunneling down to 10 K, indicating the carbon paint does not add spurious the
18 ron by X-rays at cryogenic temperatures (100 K).
19       Our calculations up to 11 GPa and 1000 K indicate a higher concentration of bicarbonates in wat
20 ie temperature ([Formula: see text]) of 6-13 K.
21          Switching behavior persists from 15 K up to room temperature.
22 her than previously reported values (ca. 150 K), reaching a maximum T(c) of 166 K at 157 GPa.
23  (ca. 150 K), reaching a maximum T(c) of 166 K at 157 GPa.
24 nt and is found to vary between 300 and 1800 K nm for a wide range of particle compositions.
25 m flame temperature (T(max,c) ~ 1791 to 1857 K) and the highest soot luminosity region temperature (T
26 out pairing of the 5f(2)-electrons down to 2 K.
27 end the rate constant evaluation down to 200 K.
28  propane dominates at lower temperature (240 K) due to the persistence of isobutanal on the surface a
29  that reaches 76.6% and 61.6% at 273 and 298 K and 1 bar, respectively.
30 igher) molar CO(2) versus N(2) uptake at 298 K, except the 19-fold CO(2) uptake for CTH-12 containing
31 yhedral boron-based substituent (e.g., -BF(3)K, -B(OH)(2)).
32  Seebeck coefficient of 200 muV K(-1) at 300 K.
33 80 K) and the cold photodetector (T(D) ~ 300 K) from ~ 500 nm down to ~ 100 nm.
34 ure and 6 orders of magnitude from 10 to 300 K.
35     Glucose isomerization rates (per Ti, 373 K) are undetectable when Ti sites are confined within me
36  most potent ligands [(+/-)-47, and (+/-)-38 K(i) = 10.20 and 23.6 nM, respectively] into their two e
37 etic ordering in the temperature range 5-395 K.
38  of water vapor is not observable at 200-400 K.
39 over a very broad temperature range (145-415 K) with a photoluminescence quantum yield (PLQY) of at l
40 hylation was measured using the Illumina 450 K platform.
41 ion-doped Cr-BST film is demonstrated at 2.5 K, and the spin Hall angle drastically decreases to 0.3-
42 ng transitions in samples from each, above 5 K.
43 on a sodium chloride bilayer on Cu(111) at 5 K, and imaged by high-resolution atomic force microscopy
44 d with a total of ~550,000 SNPs (Illumina 50 K SNP Chip and RNA-seq).
45  transitions were observed from 1.8 K to 523 K.
46 7) residues of the Walker A motif (-GPAGTG(6)K(7)S-) were found to be critical to the PI5P-binding ab
47  DMPC liposome collisions with K(3)Fe(CN)(6)/K(4)Fe(CN)(6) as the encapsulated aqueous redox probe.
48  high TE figure of merit (zT) of ~1.9 at 710 K.
49 nt (0.5 mu(B) /Co) and Curie temperature (75 K), values larger than previously reported for any monol
50 ral phase transitions were observed from 1.8 K to 523 K.
51 )R-G279S(7.44) was enhanced by about 2 and 8 K when compared with wild-type A(1)-receptor and A(1)R-Y
52 ng the gap between a hot-emitter (T(E) ~ 880 K) and the cold photodetector (T(D) ~ 300 K) from ~ 500
53 ve anti-folate activity, was found to have a K(i) of 34 muM, well below peak plasma diflunisal levels
54 found that it binds the scFv antibody with a K(D) (equilibrium dissociation constant) of 46 nM.
55 ilide corrected both congenital and acquired K(v)11.1 trafficking defects, resulting in functional K(
56 cific knockout (KO) of the calcium-activated K+ channel SK2 (L7-SK2) show intact vestibulo-ocular ref
57            Previously, we showed that active K-Ras4B dimerizes in silico and in vitro through two maj
58                                    The adult K/BxN transgenic mouse develops spontaneous autoimmune a
59 ed variants with varying binding affinities (K(D) as low as 216 pM), co-crystallized it with the rece
60 nal Amplification (gamma), Binding affinity (K(d)), Receptor activation Efficacy (epsilon), and const
61 inds RNA with an unexpectedly high affinity (K(D) ~ 100 pm).
62  that bind TAR with low micromolar affinity (K(D) values ranging from 3.6 to 22 mum).
63  associated with reduced selectivity against K(+) We conclude that optimization for ammonium transpor
64                   Bis(2-ethylhexyl)ammonium (K(50) 29 uM), triisopentylammonium (K(50) 196 uM) and di
65 re, by combining femtosecond Fe K(alpha) and K(beta) X-ray emission spectroscopy (XES) with Fe K-edge
66 d spin-sensitive femtosecond Fe K(alpha) and K(beta) X-ray emission spectroscopy at an X-ray free-ele
67 racteristic curve (AUC) for ADC, D(app), and K(app) to discriminate malignant from benign lesions was
68 al agreement (kappa = 0.81) between C.D. and K.P.L. in independently assigning categories of definiti
69 cation sites by MS after Lys-C digestion and K-epsilonGG-peptide enrichment.
70 The distance between the anion framework and K(+) resembles a frustrated Lewis pair-like structure, w
71                                    H(+)- and K(+)-selective electrodes thus prepared exhibit highly s
72 luteolin-hexuronide, salvianolic acids I and K.
73 nal and longitudinal comparisons of K(i) and K(i) change found significant effects of Parkinson disea
74 tivity filter (SF) disrupts inactivation and K(+)-selective transport in hERG1, leading to arrhythmog
75 lity, common-ion effect, pK(a), pH(max), and K(sp) values of three model compounds in a fast and low
76 derivatives exhibit selectivity to Na(+) and K(+) ions within detection ranges of 0-100 and 0-50 mmol
77 gh morphologically similar, K. ocellatus and K. hermaphroditus had remarkably different evolutionary
78 salt treatment after 120 days without P- and K-fertilizer addition.
79 ed ion channels, including Na(V), Ca(V), and K(V) channels.
80 someric aromatic peptide amphiphiles (APAs), K(S)C'EK(S) and C'EK(S)K(S) (K(S) = S-aroylthiooxime-mod
81  stochastic fluctuation in abundances around K, but higher temporal variation in beta within herds.
82   Striatal dopamine activity was assessed as K(i)(cer) value using [(18)F]DOPA PET before and 6 weeks
83                     Both PpMyoXI and AtMyoXI-K interact with PpRabE14, and the interaction is specifi
84 almost 40 years ago when one of the authors (K.J.H.) published an organized system to quantify the ac
85 ent studies have demonstrated that bacterial K(+) channels have integral roles in electrical signalin
86 m those of WT vessels, suggesting that basal K(ATP) channel activity in LSM is not an essential compo
87 lations between P50 and psi(min) and between K(s) and Hv show signs of deeper evolutionary integratio
88 Results: A linear relation was found between K(i) and TBR, with a square of Pearson correlation of 0.
89 h a nanobody fusion-protein disclosing bound K(+) ions.
90 odel of non-small cell lung cancer driven by K-Ras G12D and p53 deficiency, G6PD knockout did not blo
91 AFS) spectroscopy measurements at the carbon K edge on thin molecular films in the laboratory.
92 acuolar polyP could nonphysiologically cause K-PPn of nuclear and cytosolic targets.
93 o the results from cancerous cervical cells, K(Ca)3.1-dependent H33258 uptake was rarely observed in
94      We examined the contribution of central K(ATP) channels to glucose effectiveness.
95 A2, which encodes voltage-gated K(+) channel K(V) 1.2.
96          Two-pore-domain potassium channels (K(2P)) are the major determinants of the background pota
97 blocker of voltage-gated potassium channels (K(V)1 family) clinically approved for the symptomatic tr
98 d Ca(2+) oscillations, and the patch-clamped K(ATP) channel opened more frequently when glucose was h
99 ithelia, the [K(+) ] in the synaptic cleft ([K(+) ](c) ) contributes to setting the hair cell and aff
100 area is selection of the number of clusters (K).
101 (D(app)), and apparent kurtosis coefficient (K(app)) between malignant and benign lesions were assess
102  high octanol-air partitioning coefficients (K(OA)) are likely to have a greater potential to undergo
103 bon source, pointing to S. elongatus-E. coli K-12 as the most active community.
104 lator activity was with ginsenoside-compound K (CK), containing a monosaccharide (glucose) attached a
105 nctional intermediate- and small-conductance K(+) (IK and SK) channels and endothelial nitric oxide s
106 d with the previously reported Ni congener, [K(2.2.2-cryptand)][(tBu) LNi(S)] ((tBu) L={(2,6-(i) Pr(2
107 ding stem P13, with a dissociation constant (K(D) ) of ~700 pm Analyses with mutated RNA constructs,
108  3) binds to tubulin [dissociation constant (K(d)) 0.4 +/- 0.1 muM] and inhibits tubulin polymerizati
109 de matrix lowered the dissociation constant (K(D)) by two orders of magnitude compared to the linear
110 toward glucose with a dissociation constant (K(d)) of 3 x 10(-8) M whereas the MIPs without AuNPs cou
111 r affinity than P2K1 (dissociation constant [K (d)] = 44.47 +/- 15.73 nm).
112                       The binding constants (K(b)) of the para-substituted phenols with the surfactan
113 utations, 1:1 binding equilibrium constants (K(1:1)) with a UV-vis active sensor, (31)P NMR shifts up
114                             In addition, CPE-K OECTs operate in the accumulation mode, which allows f
115 hown that the conjugated polyelectrolyte CPE-K functions as a conductive matrix to electronically con
116 nding of the N(2) unit in the same crystal, [K(crypt)](2){[(R(2)N)(3)Gd](2)[mu-eta(x):eta(x)-N(2)]} (
117 22 elements (As, Ba, Be, Bi, Cd, Co, Cr, Cu, K, Mn, Mo, Na, Ni, P, Pb, Th, Tl, Sb, U, V, Y and Zn) in
118  first converts 3D images to key-value data (K-V).
119 ively active form of acetylcholine-dependent K(+) current (I(KACh)), called I(KH); this is an importa
120 s in different growth stages under different K levels were observed to clarify the mechanism regulati
121                        Therefore, disrupting K-Ras PM interaction is a tractable approach to block on
122 -EM reveals that DNAs transported into E-S/E-K compartments are 'clamped' in a sub-compartment create
123               Concentrations of 16 elements (K, Na, Mg, Ca, Fe, Zn, Hg, Se, As, Cu, Cd, Mn, Ni, Cr, P
124 x of K(+) through G(BK) can rapidly elevate [K(+) ](c) , which speeds the activation and slows the in
125  a single well were used to determine enzyme K(m) and V(max) values.
126 ed to the linear peptide aptamer, estimating K(D) as 10.1 nM, which is the lowest concentration repor
127  affinity on the corresponding (S)-eutomers (K(i) = 6.30 and 11.10 nM, respectively).
128 experiments, whereby the binding of external K(+) impedes the forward translocation of the blocker, p
129 stability of carbonate-based electrolytes, F K-edge to study the electrolyte salt and binder stabilit
130            Here, by combining femtosecond Fe K(alpha) and K(beta) X-ray emission spectroscopy (XES) w
131 g element- and spin-sensitive femtosecond Fe K(alpha) and K(beta) X-ray emission spectroscopy at an X
132 a) X-ray emission spectroscopy (XES) with Fe K-edge X-ray absorption near-edge structure (XANES), we
133 further determination of Al, Ca, Cr, Cu, Fe, K, Mn, Mo and Ni in rice samples by ICP OES.
134 imary T4, right colon), biological features (K/N-RAS status), and response to chemotherapy (Response
135  of this distinction was probed via fluorine K-edge X-ray absorption spectroscopy.
136 entially interesting electronic behavior for K(3)Ir(2)O(6) is supported by electronic structure calcu
137 s revealed a critical and selective role for K(v)1 channel inactivation in synaptic facilitation of e
138     The average migration times and %RSD for K(+), Na(+), and Li(+) were measured to be 22.04 s (1.59
139 1 in arterial smooth muscle cells is to form K(+) channels in the sarcolemma.
140 trafficking defects, resulting in functional K(v)11.1 current.
141                                 Furthermore, K(+) -uptake-channel activities were reduced in cpk3/5/6
142 ivation of a second potassium conductance, G(K(LV)) .
143 ariant in KCNA2, which encodes voltage-gated K(+) channel K(V) 1.2.
144                                Voltage-gated K(+) channels function in macromolecular complexes with
145 duced splicing events are regulated by hnRNP K, a host protein required for efficient splicing of the
146 cription-repressive complex containing hnRNP-K/L proteins and show that knockdown of these factors st
147 ate consists of a 3D Zn-Sb framework hosting K(+) ions inside polyhedral cages, some of which are rem
148 f serogroups (B, C1, C2 to C3, D1, E1, G, I, K, N, O, and Q); however, Salmonella enterica serovar Ty
149                   The most active, a Co(III)/K(I) complex, shows a turnover frequency of 800 h(-1) at
150 on the crucial roles of OsJAZ9 for improving K deficiency tolerance in rice by altering JA levels and
151          The allosteric coupling constant in K-type allosteric systems is defined as a ratio of the b
152 rkH is a bacterial ion channel implicated in K(+) uptake and pH regulation.
153 ase in sound spectrum levels and increase in K. brevis cell concentrations also coincided with decrea
154 nderscore the role of the alpha-interface in K-Ras4B homodimerization and the beta-surface in effecto
155  novel GES-5-encoding plasmid was present in K. oxytoca, Escherichia coli, and Enterobacter cloacae i
156  mOmega cm, T is the absolute temperature in K, S is the Seebeck coefficient, and k(B) /e = 86.3 uV K
157 escheduling of guard cell starch turnover in K. fedtschenkoi compared with that observed in Arabidops
158 ee nitric oxide and nitrosothiols (k (inact)/K(I) >= 5 m(-1) s(-1)), which is the first report of Sir
159 ambridge, United Kingdom, in 2015 to isolate K. pneumoniae from stool, blood, and the environment.
160 etes, inhibit pancreatic ATP-sensitive K(+) (K(ATP) ) channels to increase insulin release.
161                                        KCNJ2 K(+) channel expression in peripheral blood mononuclear
162 showed varied effects on transport kinetics (K(m) and V(max)) and substrate specificity.
163 ism designed for PLA2 inhibition (denoted "L&K-NP").
164 tered with a lethal dose of venomous PLA2, L&K-NPs also inhibit hemolysis and confer a significant su
165 re K(a, capsaicin) = 3.5206 x 10(-16) mol/L, K(a, allicin) = 5.0227 x 10(-15) mol/L, K(a, sanshool) =
166 l/L, K(a, allicin) = 5.0227 x 10(-15) mol/L, K(a, sanshool) = 1.7832 x 10(-15) mol/L.
167 on between Psi(leaf) at 50% loss of K(leaf) (K(leaf) P(50) ) and maximum K(leaf) (K(leaf-max) ) acros
168 (leaf) (K(leaf) P(50) ) and maximum K(leaf) (K(leaf-max) ) across species.
169  in the upper airways and intestine to limit K. pneumoniae colonization within these niches.
170 ss is slower, leading to 98% Tc removal (log K(d) = 4.5 +/- 0.1) after 35 days.
171          Tsivgoulis G, Katsanos AH, Malhotra K, et al.
172 loss of K(leaf) (K(leaf) P(50) ) and maximum K(leaf) (K(leaf-max) ) across species.
173 depended on SAP in the autoantibody-mediated K/BxN model, organized insulitis and diabetes onset were
174                   Recent advanced EPR and Mn K-edge X-ray spectroscopy studies converge upon the HO m
175 re linear IMS based on the absolute mobility K at moderate normalized electric field E/N and field as
176 0.5 kcal/mol; DeltaS*= -24.3 +/- 1.7 cal/mol.K) were measured using Eyring analysis, implying a highl
177 ate to high affinity (human/guinea pig/mouse K(d): 24/28/94 nM).
178 ed so far (E(max) = 118%, EC(50) = 0.24 muM, K(D) = 19.6 nM; inactive at autotaxin and LPA(2-6) recep
179 1) and a high Seebeck coefficient of 200 muV K(-1) at 300 K.
180  L amino acid transporter activity and Na(+) K(+) -ATPase activity using sarcolemmal membranes isolat
181 34 to -0.59 A F(-1) and a decrease in Na(+) ,K(+) -ATPase current from 1.09 A F(-1) to 0.54 A F(-1) d
182 e carrier family 9 member A6 (SLC9A6)/(Na(+),K(+))/H(+) exchanger 6 (NHE6) gene that cause Christians
183                                        Na(+)-K(+) pump current, I(p), was measured in voltage-clamped
184            Stimulation of the membrane Na(+)-K(+) pump should lower Na(+) concentrations, and the bet
185 1 shows that it is a highly selective, Na(+)/K(+)-conducting channel and, in contrast to known cation
186 evels of intracellular Ca(2+) uptake and Na, K-ATPase mRNA were determined in the cultured epithelial
187 2) RuX(6) (X=Cl or Br), MA(2) MRuX(6) (M=Na, K or Ag; X=Cl or Br) and MA(3) Ru(2) X(9) (X=Br) based u
188       The dendrite-free sodium-potassium (Na-K) liquid alloy composed of two alkali metals is one of
189          Mutations of the ion pump alpha2-Na/K ATPase cause familial hemiplegic migraine, but the mec
190   Here, we show that mice in which alpha2-Na/K ATPase is conditionally deleted in astrocytes display
191 and metabolomic analyses show that alpha2-Na/K ATPase loss alters metabolic gene expression with cons
192 raine, but the mechanisms by which alpha2-Na/K ATPase mutations lead to the migraine phenotype remain
193  mechanism regulated by astrocytic alpha2-Na/K ATPase that triggers episodic motor paralysis in mice.
194 ees were more likely to have extramural (NIH K-award) versus intramural (KL2) or other career develop
195 ticles of the electrode, such as the C and O K-edges to track the stability of carbonate-based electr
196 Kpi contributes positively to the ability of K. pneumoniae to form biofilms and adhere to different h
197 ); NaHS plus glibenclamide, an antagonist of K(ATP) opening (NaHS Glib), and Glib alone (Glib).
198  to delimit the present distribution area of K.
199 in chloroplast morphology, likely because of K redistribution from vacuole to chloroplast.
200  the Crassulaceaean Acid Metabolism (CAM) of K.
201 to the PM with a dissociation coefficient of K(d) ~16 muM and Hill coefficient of n ~2.
202 ss-sectional and longitudinal comparisons of K(i) and K(i) change found significant effects of Parkin
203 y was used to measure diffusion constants of K(+), Cu(2+), and Cl(-) diffusing through loblolly pine
204                         Genetic depletion of K(v)beta1 blocked all broadening of the AP(syn) during h
205                                    Efflux of K(+) through G(BK) can rapidly elevate [K(+) ](c) , whic
206 form IMS (FAIMS) relying on the evolution of K at high E/N causing strong ion heating.
207                                The family of K(+)-dependent Na(+)/Ca(2+)-exchangers, NCKX, are import
208 ing mechanism in voltage-dependent gating of K(V)7.1 as triggered by VSD activations to the intermedi
209                               The genomes of K pneumoniae carrying bla(NDM) and bla(KPC) were sequenc
210 te appears to arise from the inefficiency of K(+) in stabilizing an active (i.e. conductive) SF confo
211 cv Spence was the high steady-state level of K(+) in cv HI10.
212 correlation between Psi(leaf) at 50% loss of K(leaf) (K(leaf) P(50) ) and maximum K(leaf) (K(leaf-max
213 rescence, and FRET quenching, and a range of K(d) values were determined.
214 methodological approach allows separation of K(+)-induced G-quadruplex formation and subsequent refol
215 creased the extracellular space and speed of K(+) redistribution.
216                      And what about those of K-Ras4B versus N-Ras?
217 pproaches have advanced our understanding of K. pneumoniae taxonomy, ecology and evolution as well as
218 terized the effects of these two peptides on K. pneumoniae, along with their physical interactions wi
219 n is a tractable approach to block oncogenic K-Ras activity.
220 (0)-catalyst, transmetallation of the Na- or K-enolate generated in situ, and subsequent reductive el
221                             We review oxygen K-edge X-ray absorption spectra of both molecules and so
222 cluding those not identified by the common P K-edge XANES.
223                        Enzymatic parameters (K(M) and V(max)), residual activity, effect of bentonite
224 gical resemblance with one of its parentals (K. daigremontiana).
225 on of the diamagnetic BNB-doped 1H-phenalene K[7H].
226 ed to the nucleolus by a low-complexity poly-K region within its IDR.
227                                   Potassium (K) metal anodes suffer from a challenging problem of den
228 ) current (~33%), reduced outward potassium (K(+)) currents (~30%), and increased sodium/calcium exch
229 activation of inwardly rectifying potassium (K(IR) ) channels underlies vasodilatation with elevated
230                   Thus, precisely predicting K(v) spatially has remained conceptual, experimental, an
231 hat these genes encode proteins that protect K. pneumoniae against neutrophil-related effector functi
232 leaved by extracellularly applied proteinase K (PK), an N-terminal truncation up to amino acid residu
233 number of identified proteins for proteinase K by 731%.
234 dified protocol that replaces the proteinase K digestion applied in FiT-seq with extended heating at
235  made it no longer susceptible to proteinase K-mediated cleavage.
236 We compared BIN-Lasso with SNP-Lasso and Q + K-LMM in a simulation experiment, and showed that the ne
237 lyproline tracts is in the micromolar range (K (diss) ~ 17 and ~ 31 muM), but binding of a second mol
238 cause by overexpressing the inward rectifier K channel Kir2.1 in stabilizer cells.
239 ediated by inhibition of inwardly rectifying K(+) (Kir) channels.
240 on via inhibition of the inwardly rectifying K(+) channels.
241 ion of pathogenicity in carbapenem-resistant K. pneumoniae, resulting in the repeated convergence of
242                               Using Ripley's K to quantify clustering, we found that FLAPS configurat
243 iphiles (APAs), K(S)C'EK(S) and C'EK(S)K(S) (K(S) = S-aroylthiooxime-modified lysine, C' = citrulline
244 e amphiphiles (APAs), K(S)C'EK(S) and C'EK(S)K(S) (K(S) = S-aroylthiooxime-modified lysine, C' = citr
245 rises the analysis of 20 elements (Mg, P, S, K, Ca, V, Cr, Mn, Fe, Co, Cu, Zn, Se, Br, Rb, Sr, Mo, I,
246 alenyl 7 (BMes, NMe), the radical-anion salt K[7(*)] was generated through chemical reduction with K
247                 In Focus: Seress, G, Sandor, K, Evans, KL, Liker, A.
248 r diabetes, inhibit pancreatic ATP-sensitive K(+) (K(ATP) ) channels to increase insulin release.
249 MC ATP level was not necessary for Pro-sigma(K) cleavage by SpoIVFB, based on analysis of mutants tha
250 cate that, although morphologically similar, K. ocellatus and K. hermaphroditus had remarkably differ
251 mutations in KCNT1, the gene encoding Slack (K(Na)1.1) channels, result in epilepsy of infancy with m
252        We also found that herds with smaller K had less stochastic fluctuation in abundances around K
253                   When the number of studies K is large, the 3K - 1 possible differential expression
254      We identify binding sites for substrate K(+) and Cl(-) ions, demonstrate the importance of key c
255 stablishing the reversible potassium sulfide K(2) S(n) phase sequence, the parasitic polysulfide shut
256           The "masked" terminal Zn sulfide, [K(2.2.2-cryptand)][(Me) LZn(S)] (2) ((Me) L={(2,6-(i) Pr
257       Our findings demonstrate that systemic K(ATP) channel inhibition reduces V O(2) max and critica
258 sequences present in two high-affinity TBPs (K(D) values of 4.2 +/- 0.3 and 3.0 +/- 0.3 nm).
259 y binding of InsP(8) to the XPR1 N-terminus (K (d) = 180 nM) was demonstrated by isothermal titration
260 rldwide with a Bayesian model and found that K(d) 490, a measurement positively related to turbidity,
261         Epidemiological studies suggest that K. pneumoniae host-to-host transmission requires close c
262                                          The K(2P) channel subunits TRESK and TREK-2 provide the pred
263 ow that serine phosphorylation abolishes the K(+)-dependence of ATP hydrolysis and blocks the catalyt
264 , they showed a reduction in the size of the K(+) current and non-linear capacitance, a readout of pr
265                        Across all soils, the K(d) values of all short-chain PFASs (<=5 -CF2- moieties
266 , while allowing water greater access to the K(+) conduction pathway.
267                   Divergent responses to the K-channel antagonist, kappaM-conopeptide RIIIJ (RIIIJ),
268 estibular semicircular canal epithelia, the [K(+) ] in the synaptic cleft ([K(+) ](c) ) contributes t
269 adiofluorinated analog of 4AP, also binds to K(V)1 channels and can be used as a PET tracer for the d
270                    Here, fracture toughness (K(Ic)) of a representative glass, namely ZIF-62 glass (Z
271 ibution of host and bacterial factors toward K. pneumoniae dissemination.
272                    P2RY1 activation triggers K(+) efflux and depolarization of hair cells, as well as
273 mmonium (K(50) 29 uM), triisopentylammonium (K(50) 196 uM) and dioctanoylammonium showed a low Hill s
274 F, Barrett toric calculator, or Barrett True-K formulas were not used.
275 rmation and decomposition of P12(1) /c1-type K(2) CO(3) at the efficient carbon-based catalyst.
276  (18)F-FDG PET/CT scans were selected from U.K. and Dutch studies on DLBCL to provide a balance betwe
277 atchment scale in the River Thames system (U.K.) and explored their sources and environmental fate.
278 the management of PCB contamination in the U.K. and reinforce the need to prevent PCBs entering the m
279                              Here, utilizing K(V)7.1's unique two open states, we report a two-stage
280 e Seebeck coefficient, and k(B) /e = 86.3 uV K(-1) .
281 ancer in patients with confirmed BRAF(V600E)/K mutations.
282 y value (K-mean), highest keratometry value (K-max), thinnest point, anterior segment optical coheren
283 ometric astigmatism, mean keratometry value (K-mean), highest keratometry value (K-max), thinnest poi
284                                     Vascular K(ATP) channel function (topical glibenclamide superfuse
285                                      Vitamin K activates both hepatic coagulation factors and extrahe
286 0 on OAC (direct anticoagulants: 26, vitamin K antagonists: 4), with no differences in baseline-proce
287 KAs and high-dose vitamin K2 improve vitamin K status in patients on hemodialysis, but have no signif
288 gnancy, and anticoagulation (LMWH or vitamin K antagonists [VKAs]) should be continued until 6 weeks
289 alysis, with 80 allocated to receive vitamin K and 79 to receive placebo.
290 had less bleeding with apixaban than vitamin K antagonist (VKA) and with placebo than aspirin.
291 12 inhibitor) versus triple therapy (vitamin K antagonist plus aspirin and P2Y12 inhibitor) in patien
292 ticoagulants (heparin or switched to vitamin K antagonist).
293 o Evaluate the Safety of Apixaban vs Vitamin K Antagonist and Aspirin vs Aspirin Placebo in Patients
294 ili pepper, garlic and mountain pepper, were K(a, capsaicin) = 3.5206 x 10(-16) mol/L, K(a, allicin)
295 o single redox DMPC liposome collisions with K(3)Fe(CN)(6)/K(4)Fe(CN)(6) as the encapsulated aqueous
296 significantly and positively correlated with K(OA), but declined with increasing relative humidity.
297  parameters after thresholding the data with K(i) < 5.3 x 10(-3) mL/cm(3)/min.
298  along with their physical interactions with K. pneumoniae capsule.
299 as generated through chemical reduction with K metal and characterized by EPR spectroscopy.
300 ite the presence of dofetilide (1 muM) in WT K(v)11.1 cells.

 
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