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1 cumulative incidence function curve with the Kaplan-Meier curve.
2 Cumulative TB risk was estimated with Kaplan-Meier curves.
3 Survival was assessed using Kaplan-Meier curves.
4 imes to event outcomes were summarized using Kaplan-Meier curves.
5 nadjusted observed survival was inspected by Kaplan-Meier curves.
6 groups are compared using log-rank test and Kaplan-Meier curves.
7 and the prognostic value was determined with Kaplan-Meier curves.
8 dysfunction and failure) were compared using Kaplan-Meier curves.
9 elative CBV and time to progression by using Kaplan-Meier curves.
10 nd pouch retention rates were analyzed using Kaplan-Meier curves.
11 had and did not have PTNB was compared using Kaplan-Meier curves.
12 iers versus wild types was examined by using Kaplan-Meier curves.
13 was compared among white and Hispanics using Kaplan-Meier curves.
14 umulative incidence of advanced neoplasia by Kaplan-Meier curves.
15 fully describes the actual survival based on Kaplan-Meier curves.
16 luation of which was by log rank analysis of Kaplan-Meier curves.
17 ome left ventricular ejection fraction using Kaplan-Meier curves.
18 Mortality over time was expressed with Kaplan-Meier curves.
19 n the HM3 and HVAD cohorts was compared with Kaplan-Meier curves.
20 using Fine and Gray cumulative incidence and Kaplan-Meier curves.
21 pring age was determined using meta-analytic Kaplan-Meier curves.
22 alysis, univariate analysis or directly from Kaplan-Meier curves.
23 ere analyzed using Cox-regression models and Kaplan-Meier curves.
24 summary statistics extracted from individual Kaplan-Meier curves.
25 were generated based on data extracted from Kaplan-Meier curves.
26 ulative 5-year mortality were estimated with Kaplan-Meier curves.
27 nitial 48 hours after hospital arrival using Kaplan-Meier curves.
28 to MRI response category were assessed using Kaplan-Meier curves.
29 imized follow-up intervals were derived from Kaplan-Meier curves.
30 rgery was calculated for illustration of the Kaplan-Meier curves.
31 Survival was analyzed using Kaplan-Meier curves.
32 ll survival time and cancer recurrence using Kaplan-Meier curves.
33 Survival was described using Kaplan-Meier curves.
34 Survival was analyzed with Kaplan-Meier curves.
35 ormed by descriptive methods and survival by Kaplan-Meier curves.
36 hted Cox proportional hazards regression and Kaplan-Meier curves.
37 breast cancer diagnosis was plotted by using Kaplan-Meier curves.
38 d using C statistics, calibration plots, and Kaplan-Meier curves.
39 g-term allograft survival was compared using Kaplan-Meier curves.
42 lysis to determine adenovirus incidence, and Kaplan-Meier curve analysis to determine the timing of e
48 d prognostic factors were assessed using the Kaplan-Meier curve and Cox proportional hazard model.
50 and overall survival (OS) was assessed with Kaplan-Meier curves and a corresponding log-rank test fo
55 We analysed cumulative rupture rates with Kaplan-Meier curves and assessed predictors with Cox pro
56 by stratified univariate log-rank test with Kaplan-Meier curves and by multivariate Cox proportional
58 rtality for each tertile was determined with Kaplan-Meier curves and compared by the modified Peto-Pe
60 urvival free from an AE was calculated using Kaplan-Meier curves and Cox hazard ratios were derived.
61 accine-targeted type; and 3) construction of Kaplan-Meier curves and Cox models to evaluate sequentia
63 ospital all-cause mortality was evaluated by Kaplan-Meier curves and Cox proportional hazard modeling
65 ther secondary end points were examined with Kaplan-Meier curves and Cox proportional hazard models.
66 tcome of all-cause mortality with unadjusted Kaplan-Meier curves and Cox proportional hazard models.
67 ree from adverse events was calculated using Kaplan-Meier curves and Cox proportional hazard ratios w
74 e and associated factors were assessed using Kaplan-Meier curves and Cox proportional hazards models,
76 erapy/CCRT PET/CT imaging was examined using Kaplan-Meier curves and Cox proportional hazards models.
77 Statistical analysis was performed using Kaplan-Meier curves and Cox proportional hazards ratios.
79 lity of treatment weighting (IPTW) -adjusted Kaplan-Meier curves and Cox proportional hazards regress
80 ity and morbidity events were analyzed using Kaplan-Meier curves and Cox proportional hazards regress
81 Disease-free survival was examined using Kaplan-Meier curves and Cox proportional hazards regress
82 lity of treatment weighting (IPTW) -adjusted Kaplan-Meier curves and Cox regression analyses were use
100 OS and updated PFS data are presented using Kaplan-Meier curves and log-rank tests stratified for ho
119 te of ACM or first CVH were plotted by using Kaplan-Meier curves and summarized with a stratified Cox
129 , using Cox proportional hazards regression, Kaplan-Meier curves, and calculation of Harrell's c inde
130 , prevalence- and bias-adjusted kappa value, Kaplan-Meier curves, and Cox proportional hazard models.
131 ver operating characteristic (ROC) analysis, Kaplan-Meier curves, and Cox proportional hazard regress
132 mes were assessed using frequency of events, Kaplan-Meier curves, and Cox proportional hazards regres
138 with that of an inverse probability-weighted Kaplan-Meier curve applied after treating bacteremia as
139 The area under the curve of a conventional Kaplan-Meier curve applied to the observed data was comp
142 rable outcome derived from the time-to-event Kaplan-Meier curve at 10 years was 0.64 (95% CI 0.58-0.6
148 sing a Cox hazards model, the log-rank test, Kaplan-Meier curves, competing-risks regression, and con
149 with all-cause mortality were studied using Kaplan-Meier curves, Cox proportional hazards regression
151 sion of TZP or carbapenems was assessed with Kaplan-Meier curves, Cox-regression model, and estimatio
154 9; logistic odds for events 0.44, p = 0.02); Kaplan-Meier curves demonstrated significant differences
156 ox proportional hazards regression model and Kaplan-Meier curves determined whether black race affect
159 o, 2.27 [95% CI, 1.84-2.82], P=6.3x10(-14)), Kaplan-Meier curves differed significantly between HFrEF
160 Amputation-free survival rate assessed with Kaplan-Meier curves differed through 12 months: 59% (41
161 g both the Cox proportional hazard model and Kaplan-Meier curves each show that the proposed method f
166 ty was estimated based on the area under the Kaplan-Meier curve for symptomatic grade 2 or greater fa
169 CCQ Overall Summary scores was assessed with Kaplan-Meier curves for death and all-cause hospitalizat
176 n model for recurrent time-to-event data and Kaplan-Meier curves for time to antibody negativity were
179 rvival (TFS) was defined as the area between Kaplan-Meier curves for two conventional time-to-event e
184 al analysis demonstrates a divergence of the Kaplan-Meier curves in favor of patients in whom APBF wa
185 Estimated crude 10-year mortality based on Kaplan-Meier curves in mothers of infants with NAS was 5
186 e extracted from the text of articles or the Kaplan-Meier curves independently by investigators who w
190 immunohistochemistry were investigated using Kaplan-Meier curves, log rank tests, and Cox regression
192 sion-free survival (PFS) were compared using Kaplan-Meier curves, log-rank tests and Cox models.
193 database performed between 1991 and 2003 by Kaplan-Meier curves, log-rank tests, and Cox proportiona
195 redictors of survival were analyzed with the Kaplan-Meier curve method (log-rank test) and multivaria
199 r uniformity at a coarse scale value of 2.5, Kaplan-Meier curves of the proportion of patients withou
204 We estimated time to first pregnancy using Kaplan-Meier curves; pregnancy and HIV incidence were es
207 : 0.60; 95% CI: 0.45-0.80; P = 0.0005), with Kaplan-Meier curves separating at month 3 and continuing
231 t important finding of the study was that in Kaplan-Meier curves stratified by mean dose, longer PFS
237 Survival rates computed from stage-specific Kaplan-Meier curves (time to melanoma-specific death) we
239 eveloped Cox proportional hazards models and Kaplan-Meier curves to compare women who underwent oopho
240 f IGF-1 and VEGF with overall survival (OS), Kaplan-Meier curves to estimate OS, and recursive partit
243 SGLT-2i compared with DPP-4i and GLP-1RA and Kaplan-Meier curves to visualize fracture risk over time
251 bility of VT/VF: two-year risk of VT/VF from Kaplan-Meier curves was 40% in highest quartile versus 2
252 Valve survival analysis (Cox regression and Kaplan-Meier curves) was used to study the natural progr
267 ty for Medical Oncology meetings' libraries, Kaplan-Meier curves were extracted from phase 3 clinical
275 We found that survival estimates from the Kaplan-Meier curves were largely congruent with those of
284 grouped using propensity score methods, and Kaplan-Meier curves were used to compare time to measles
295 method demonstrated similar c-statistics and Kaplan-Meier curves when used in survival analyses.
297 e-bleeding free survival was evaluated using Kaplan-Meier curves with log rank test, whilst predictor
299 atients was reconstructed from the published Kaplan-Meier curves with the aid of a computer vision pr
300 Individual patient data were extracted from Kaplan-Meier curves with WebPlotDigitizer version 5 and