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1 Little evidence suggests a link with Kaposi sarcoma-associated herpesvirus.
2 KSbcl-2, from human herpesvirus 8 (HHV8) or Kaposi sarcoma-associated herpesvirus.
3 uch as HIV, Epstein-Barr virus, malaria, and Kaposi sarcoma-associated herpesvirus.
4 plasm of lymphatic endothelium infected with Kaposi sarcoma-associated herpesvirus.
5 -like inhibitory protein (FLIP) protein from Kaposi sarcoma-associated herpesvirus activates the NF-k
6 hundred percent of PELs carry the genome of Kaposi sarcoma-associated herpesvirus and a majority are
7 virus (n = 2310), Merkel cell polyomavirus, Kaposi sarcoma-associated herpesvirus, and human T-cell
8 The oncogenic gamma-herpesviruses EBV and Kaposi sarcoma-associated herpesvirus are ubiquitous hum
9 an gammaherpesviruses Epstein-Barr virus and Kaposi Sarcoma-associated herpesvirus both contain a gly
10 avity-based lymphoma cells were positive for Kaposi sarcoma-associated herpesvirus by PCR and were ne
14 alysis of lymphoma DNA showed high levels of Kaposi sarcoma-associated herpesvirus (> 40 to 80 genome
15 other viruses such as Epstein-Barr virus and Kaposi sarcoma-associated herpesvirus have led to the ge
16 their use as models for human infection with Kaposi sarcoma-associated herpesvirus (human herpesvirus
17 This family includes Epstein-Barr virus and Kaposi sarcoma-associated herpesvirus, human pathogens a
23 ly that the open reading frame 45 (ORF45) of Kaposi sarcoma-associated herpesvirus interacts with p90
24 sion of the viral interleukin-6 homolog from Kaposi sarcoma-associated herpesvirus is also sufficient
26 interleukin-6 (vIL-6), a virokine encoded by Kaposi sarcoma- associated herpesvirus, is an issue of c
31 n 6 (IL-6) is an important growth factor for Kaposi sarcoma- associated herpesvirus (KSHV)-associated
33 invariably associated with infection by the Kaposi sarcoma-associated herpesvirus (KSHV or HHV8).
35 xpression and viral latency is important for Kaposi sarcoma-associated herpesvirus (KSHV) and human i
36 que rhadinovirus (RRV) is closely related to Kaposi sarcoma-associated herpesvirus (KSHV) and is asso
37 Kaposi sarcoma is caused by infection of Kaposi sarcoma-associated herpesvirus (KSHV) and is char
38 A-binding domains (DBDs), LANA homologs from Kaposi sarcoma-associated herpesvirus (KSHV) and MHV68 e
39 ion that is transferable to MHV68.IMPORTANCE Kaposi sarcoma-associated herpesvirus (KSHV) and murine
40 s subfamily of gammaherpesviruses, including Kaposi sarcoma-associated herpesvirus (KSHV) and murine
41 maherpesviruses Epstein-Barr virus (EBV) and Kaposi sarcoma-associated herpesvirus (KSHV) are associa
42 In Sub-Saharan Africa, where HIV and the Kaposi sarcoma-associated herpesvirus (KSHV) are endemic
46 T-cell immunity is important for controlling Kaposi sarcoma-associated herpesvirus (KSHV) diseases su
50 n tumor viruses Epstein-Barr virus (EBV) and Kaposi sarcoma-associated herpesvirus (KSHV) establish p
51 nces were all approximately equidistant from Kaposi sarcoma-associated herpesvirus (KSHV) gB sequence
56 , a multifunctional protein expressed by the Kaposi sarcoma-associated herpesvirus (KSHV) in latently
61 me studies have inferred that an epidemic of Kaposi sarcoma-associated herpesvirus (KSHV) infection i
64 e of the JCI, Ganem and colleagues show that Kaposi sarcoma-associated herpesvirus (KSHV) inhibits CD
72 ically, the thymidine kinase (TK) encoded by Kaposi sarcoma-associated herpesvirus (KSHV) is an extre
90 ng of genes believed to be essential for the Kaposi sarcoma-associated herpesvirus (KSHV) latent life
91 newly recognized gamma herpesvirus known as Kaposi sarcoma-associated herpesvirus (KSHV) or human he
95 show that the lytic replication cycle of the Kaposi sarcoma-associated herpesvirus (KSHV) promotes do
97 falciparum (Pf) malaria infection had higher Kaposi sarcoma-associated herpesvirus (KSHV) viral load,
98 In a previous study, it was shown that the Kaposi sarcoma-associated herpesvirus (KSHV) was specifi
99 at treatment of cells latently infected with Kaposi sarcoma-associated herpesvirus (KSHV) with glycyr
104 associated with epigenetic modifications of Kaposi sarcoma-associated herpesvirus (KSHV), an oncogen
105 Human herpesvirus 8 (HHV-8), also termed Kaposi sarcoma-associated herpesvirus (KSHV), encodes th
108 ncoviruses include Epstein-Barr virus (EBV), Kaposi sarcoma-associated herpesvirus (KSHV), hepatitis
109 egalovirus (HCMV), Epstein-Barr virus (EBV), Kaposi Sarcoma-associated Herpesvirus (KSHV), Hepatitis
113 erpesvirus (rhadinovirus) closely related to Kaposi sarcoma-associated herpesvirus (KSHV), the human
114 rtant role in early childhood infection with Kaposi sarcoma-associated herpesvirus (KSHV), the matern
115 hereas the incidence of Hodgkin lymphoma and Kaposi sarcoma-associated herpesvirus (KSHV)-associated
116 virus (HIV) type 1-infected subjects without Kaposi sarcoma-associated herpesvirus (KSHV)-associated
118 We recently identified polymorphisms in Kaposi sarcoma-associated herpesvirus (KSHV)-encoded mic
121 pregnancy affects transplacental transfer of Kaposi sarcoma-associated herpesvirus (KSHV)-specific an
134 ) to infection by a novel human herpesvirus (Kaposi sarcoma-associated herpesvirus [KSHV]) is one of
135 tiated blood vascular endothelial cells with Kaposi sarcoma-associated herpesvirus leads to their lym
136 o the sustained activation of ERK and RSK in Kaposi sarcoma-associated herpesvirus lytic replication.
138 lity complex class I in cells expressing the Kaposi sarcoma-associated herpesvirus protein K3, is una
139 n gamma-2 herpesvirus closely related to the Kaposi sarcoma-associated herpesvirus, replicates lytica
141 and endothelial cells latently infected with Kaposi sarcoma-associated herpesvirus, suggesting that a
142 V4), cytomegalovirus (HHV5), HHV6, HHV7, and Kaposi sarcoma-associated herpesvirus (termed KSV or HHV