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1  specifically enriched in genes regulated by Kruppel-like factors.
2                             Here we identify Kruppel-like factor 1 (KLF-1) as a mediator of a cytopro
3                                          The Kruppel-like factor 1 (KLF1) and KLF2 positively regulat
4 l-like factor (EKLF), which is also known as Kruppel-like factor 1 (KLF1), play a coordinating role i
5                                              Kruppel-like factor 1 (KLF1/EKLF) is a transcription fac
6 ns in the key erythroid transcription factor Kruppel-like factor 1 in patients who presented with sev
7                                              Kruppel-like factor 1(KLF1) is a hematopoietic-specific
8 aused by mutations in a trans-acting factor (Kruppel-like factor 1) and reveal an important pathway r
9 rs GATA1 (GATA1 binding protein 1) and KLF1 (Kruppel-like factor 1) cause benign and disease phenotyp
10                  They find that mutations in Kruppel-like factor-1 (KLF1) are significantly more prev
11 ssense mutation in the second zinc finger of Kruppel-like factor-1 (KLF1) leads to degenerate DNA-bin
12           The circadian transcription factor Kruppel like factor 10 (KLF10) has been involved in live
13 t findings have supported a primary role for Kruppel-like factor 10 (KLF10) as an important transcrip
14 , NAB2 (Ngfi-A-binding protein-2) and KLF10 (Kruppel-like factor-10) that, when individually silenced
15 levels of a MAO A-transcriptional activator, Kruppel-like factor 11 (KLF11 , also recognized as trans
16                              The function of Kruppel-like factor 11 (KLF11) in the regulation of meta
17 volutionarily conserved metabolic regulator, Kruppel-like factor 11 (KLF11), as a novel browning tran
18 s-induced MAO A and the transcription factor Kruppel-like factor 11 (KLF11, also called TIEG2, a memb
19 e (ImmGen) Consortium studies, we identified kruppel-like factor 12 (Klf12), encoding a novel transcr
20 viously showed that the transcription factor Kruppel-like factor 13 (KLF13) controls the late (3-5 da
21   Here we show that the transcription factor Kruppel-like factor 13 (KLF13) has a critical role in th
22 gene locus encoding the transcription factor Kruppel-like factor 14 (KLF14) are strongly associated w
23        Here we report that disruption of the Kruppel-like factor 14 (KLF14) gene in mice causes centr
24                                            A Kruppel-like factor 15 (dKlf15) loss-of-function strateg
25       Although previous studies have defined Kruppel-like factor 15 (KLF15) as a transcriptional repr
26                 In this study, we identified Kruppel-like factor 15 (KLF15) as an essential regulator
27         In the present study, we report that Kruppel-like factor 15 (KLF15) is a novel mediator of b-
28            Although the transcription factor Kruppel-like factor 15 (KLF15) is an important regulator
29 y induced the ergogenic transcription factor Kruppel-like factor 15 (Klf15) while decreasing Fbxo32.
30  with known CREB target genes, we identified Kruppel-like factor 15 (KLF15), a kidney-enriched nuclea
31                We previously determined that Kruppel-like factor 15 (KLF15), a kidney-enriched zinc f
32                    Here, we demonstrate that Kruppel-like factor 15 (KLF15), a liver-enriched transcr
33 ined that dual transcriptional regulation of Kruppel-like factor 15 (Klf15), by both the transforming
34 we established that PR-mediated induction of Kruppel-like factor 15 (KLF15), which can bind to GC-ric
35 the control of a clock-dependent oscillator, kruppel-like factor 15 (Klf15).
36 rt and neural crest derivatives expressed 2, Kruppel-like factor 15, and cyclin G1 and the subsequent
37 dexamethasone-induced transcription factors, Kruppel-like factor 15, Slug, and SPDEF, stimulated the
38                TWEAK represses the levels of Kruppel-like factor 15; myocyte enhancer factor 2, and p
39                         Here, we report that Kruppel-like factor 17 (Klf17), like Grhl3, acts downstr
40 y up-regulation of its transcription factor, Kruppel-like factor 2 (KLF-2).
41 othelial nitric oxide synthase (P< .005) and Kruppel-like factor 2 (KLF2) (P< .005).
42 cific deficiency of the transcription factor Kruppel-like factor 2 (KLF2) accelerates atherosclerosis
43 in expression of the atheroprotective factor Kruppel-like factor 2 (KLF2) and cytokines were also stu
44                                              Kruppel-like factor 2 (KLF2) and endothelial nitric oxid
45   The expression of the transcription factor Kruppel-like factor 2 (KLF2) and its vasoprotective targ
46 fects of flow-activated transcription factor Kruppel-like factor 2 (KLF2) are compromised in PAH.
47                    Here, we identify myeloid Kruppel-like factor 2 (KLF2) as an essential regulator o
48 icoagulant protein C system, is regulated by Kruppel-like factor 2 (KLF2) at the transcriptional leve
49 ndent upon the vascular transcription factor Kruppel-like factor 2 (KLF2) binding near the transcript
50      We now report that transcription factor Kruppel-like factor 2 (KLF2) controls naive Treg migrati
51                     The transcription factor Kruppel-like factor 2 (KLF2) controls the emigration of
52 es are regulated by the transcription factor Kruppel-like factor 2 (KLF2) in conventional alphabeta T
53 eases expression of the transcription factor Kruppel-like factor 2 (KLF2) in multiple cell types.
54 ion of NF-kappaB and increased expression of Kruppel-like factor 2 (KLF2) in RAW 264.7 cells.
55 flammatory role for the transcription factor Kruppel-like factor 2 (KLF2) in regulating activation of
56                     The transcription factor Kruppel-like factor 2 (KLF2) is a critical anti-inflamma
57 l expansion and survival, we now report that Kruppel-like factor 2 (KLF2) is a key transcription fact
58 though mechanosensitive transcription factor Kruppel-like factor 2 (KLF2) is a major regulator of end
59                                              Kruppel-like factor 2 (KLF2) is a positive transcription
60                                              Kruppel-like factor 2 (KLF2) is a potent regulator of my
61                                              Kruppel-like Factor 2 (KLF2) is a shear stress-sensitive
62                                              Kruppel-like factor 2 (KLF2) is a transcription factor t
63                     The transcription factor Kruppel-like factor 2 (KLF2) is critical for normal traf
64 eroprotective flow, the transcription factor Kruppel-like factor 2 (KLF2) is crucial for maintaining
65      We now report that transcription factor Kruppel-like factor 2 (KLF2) is necessary for the genera
66                  In this study, we show that Kruppel-like factor 2 (Klf2) is phosphorylated by Erk2 a
67                     The transcription factor Kruppel-like factor 2 (KLF2) is required for the quiesce
68 surface S1P-R1 expression, as well S1PR1 and Kruppel-like factor 2 (KLF2) transcripts, were significa
69                     The transcription factor Kruppel-like factor 2 (KLF2) was proposed to regulate ge
70 gulates other stress-sensitive genes such as Kruppel-like factor 2 (Klf2), endothelial nitric oxide s
71 l cell homeostasis as shown by activation of Kruppel-like factor 2 (KLF2), increased endothelial nitr
72 ons, which was associated with modulation of Kruppel-like factor 2 (KLF2), Kruppel-like factor 4 (KLF
73 shear stress-sensitive transcription factor, Kruppel-like Factor 2 (KLF2), mediates increased CBC sen
74 to facilitate a physical interaction between Kruppel-like factor 2 (KLF2), the major regulator of she
75 s the expression of the transcription factor Kruppel-like factor 2 (KLF2).
76  critical upstream transcriptional activator Kruppel-like factor 2 (KLF2).
77 h the regulation of the transcription factor Kruppel-like factor 2 (Klf2).
78 n of the vasoprotective transcription factor Kruppel-like Factor 2 (KLF2).
79 he adipogenesis inhibitors Pref-1, Sox9, and Kruppel-like factor 2 (KLF2).
80 ession is regulated by shear stress (SS) via Kruppel-like factor 2 (KLF2).
81  cell lung cancer through down-regulation of Kruppel-like factor 2 and p21, contrary to tumor suppres
82  This activation is instrumental in inducing Kruppel-like factor 2 and several immediate early genes
83 elial anti-inflammatory transcription factor Kruppel-like factor 2 as well as endothelial nitric oxid
84                    Aberrant up-regulation of Kruppel-like factor 2 by CNOT3 depletion leads to impair
85                                   S1P-R1 and Kruppel-like factor 2 expression were monitored after S1
86                                              Kruppel-like factor 2 expression, a flow sensitive trans
87                  Upregulation of endocardial Kruppel-like factor 2 in Adamts19 knockout mice precedes
88                       Importantly, restoring Kruppel-like factor 2 in IRF2BP2-deficient macrophages a
89 s present within a repressing complex on the Kruppel-like factor 2 promoter.
90      In addition, we identified the mRNA for Kruppel-like factor 2 transcription factor, an inducer o
91 f the anti-inflammatory transcription factor Kruppel-like factor 2 was markedly reduced.
92 065], P=1.73E-03) and had lower IRF2BP2 (and Kruppel-like factor 2) protein levels in peripheral bloo
93 Furthermore, we found that the expression of Kruppel-like factor 2, a key anti-inflammatory transcrip
94 f the sphingosine 1-phosphate 1 receptor and Kruppel-like factor 2, both of which regulate thymic and
95 as a transcriptional corepressor to regulate Kruppel-like factor 2-dependent gene expression.
96 is required for MEF2-dependent activation of Kruppel-like factor 2.
97 ancer factor 2 (MEF2) transcriptional target Kruppel-like factor 2.
98 PRCP(gt/gt) aortas express reduced levels of Kruppel-like factors 2 and 4, thrombomodulin, and eNOS m
99 onsequent detrimental overexpression of KLF (Kruppel-like factor) 2 and KLF4, recapitulating the hall
100 n of the zinc finger transcriptional factor, Kruppel-like factor-2 (KLF2) in tumor endothelial cells.
101 regulating NF-E2-related factor-2 (Nrf2) and Kruppel-like factor-2 (KLF2), two transcription factors
102 ed antiinflammatory tissue expression of von Kruppel-like Factor-2.
103 ow that the zinc finger transcription factor Kruppel-like factor 3 (KLF3) directly represses galectin
104                                              Kruppel-like factor 3 (KLF3) is a transcriptional regula
105 well-characterized transcriptional repressor Kruppel-like factor 3 (KLF3), which, in turn, directly r
106                   Constitutive expression of Kruppel-like factor 3 (KLF3, BKLF) increases marginal zo
107 tracellular domain) transactivated the mouse Kruppel-like factor 4 (Klf) promoter and that Klf4 direc
108                   Previously, we showed that Kruppel-like factor 4 (KLF4) activates certain quiescent
109 istically, we found that miR-135b-5p targets Kruppel-like factor 4 (KLF4) and binds to its 3' UTR, le
110     Here we report our studies that identify Kruppel-like factor 4 (KLF4) as a critical regulator of
111 in and TF footprint analysis which unearthed Kruppel-like Factor 4 (KLF4) as a potential key regulato
112 ed that the zinc finger transcription factor Kruppel-like factor 4 (KLF4) can promote RAS-ERK signali
113 thickness and PU mouse models, we found that Kruppel-like factor 4 (KLF4) deficiency resulted in decr
114 Here, we explore the mechanisms that loss of Kruppel-like factor 4 (KLF4) exacerbates oncogenic TGF-b
115                                    Decreased Kruppel-like factor 4 (KLF4) expression has been observe
116 omplex long-range enhancer cluster governing Kruppel-like factor 4 (Klf4) expression in naive pluripo
117  we have shown that the transcription factor Kruppel-like factor 4 (KLF4) governs mitochondrial bioge
118                     The transcription factor Kruppel-like factor 4 (KLF4) has the ability, along with
119 s death-associated protein kinase (DAPK) and Kruppel-like factor 4 (KLF4) in CRC cells, resulting in
120 to overexpress the transcriptional regulator Kruppel-like factor 4 (Klf4) in esophageal epithelia of
121 ole for the zinc finger transcription factor Kruppel-like factor 4 (KLF4) in the modulation of the in
122                           Here, we show that Kruppel-like factor 4 (KLF4) is a central regulator of s
123                            We found that the Kruppel-like factor 4 (Klf4) is a potent activator of Ag
124                                              Kruppel-like factor 4 (Klf4) is a putative gastric tumor
125                                              Kruppel-like factor 4 (KLF4) is a transcription factor a
126                                              Kruppel-like factor 4 (Klf4) is a transcription factor i
127                                          The Kruppel-like factor 4 (KLF4) is a transcriptional regula
128                                              Kruppel-like factor 4 (KLF4) is a zinc finger protein hi
129                                  Endothelial Kruppel-Like Factor 4 (KLF4) is an important anti-inflam
130                     The transcription factor Kruppel-like factor 4 (KLF4) is an important regulator o
131                         Transcription factor Kruppel-like factor 4 (Klf4) is essential for somatic ce
132                                              Kruppel-like factor 4 (KLF4) is expressed in neural stem
133                                              Kruppel-like factor 4 (KLF4) is highly expressed in more
134                                              Kruppel-like factor 4 (KLF4) is involved in self-renewal
135         Here, we have provided evidence that Kruppel-like factor 4 (Klf4) is required in IRF4-express
136                   Last, we demonstrated that Kruppel-like factor 4 (Klf4) mediates Hh-dependent TAM M
137                                    Effect of Kruppel-like factor 4 (Klf4) on Slurp1 promoter was eval
138                         Here, we report that Kruppel-like factor 4 (KLF4) promotes disease progressio
139         SMC-specific conditional knockout of Kruppel-like factor 4 (Klf4) resulted in reduced numbers
140 ophage lineage identity transcription factor Kruppel-like factor 4 (KLF4) to be inhibited during LC d
141        Although the biological importance of Kruppel-like factor 4 (KLF4) transcription factor in the
142 d that gemfibrozil induced the activation of Kruppel-like factor 4 (KLF4) via the PI 3-kinase-AKT pat
143 ng cascade involving sustained expression of Kruppel-like factor 4 (KLF4), a regulator of stem cell m
144  We demonstrate that miR-29 directly targets Kruppel-like factor 4 (KLF4), a transcription factor req
145                                              Kruppel-like factor 4 (KLF4), a transcription factor tha
146                                              Kruppel-like factor 4 (KLF4), a transcription factor, pl
147                                              Kruppel-like factor 4 (KLF4), a zinc finger-containing t
148                                              Kruppel-like factor 4 (KLF4), a zinc finger-containing t
149 date the underlying mechanism, we focused on Kruppel-like factor 4 (KLF4), and decoupled its mCpG- an
150  modulation of Kruppel-like factor 2 (KLF2), Kruppel-like factor 4 (KLF4), and suppressor of cytokine
151  many human transcription factors, including Kruppel-like factor 4 (KLF4), as sequence-specific DNA m
152        The zinc finger transcription factor, Kruppel-like factor 4 (KLF4), is expressed in the post-m
153                      AMPK activation targets Kruppel-like factor 4 (KLF4), known to govern progenitor
154                         Transcription factor Kruppel-like factor 4 (Klf4), one of the factors directi
155        The zinc finger transcription factor, Kruppel-like factor 4 (KLF4), regulates numerous biologi
156 polarization involves induction of STAT6 and Kruppel-like factor 4 (KLF4), which induce each other an
157 s, a pathological mechanism also governed by Kruppel-like factor 4 (KLF4).
158                                              Kruppel-like factor 4 (KLF4, GKLF) is a zinc-finger tran
159                                              Kruppel-like factor 4 (Klf; previously known a gut-enric
160 man gastric tumors had reduced expression of Kruppel-like factor 4 and increased expression of FoxM1
161 in keratinocytes via the combined actions of Kruppel-like factor 4 and NF-kappaB transcription factor
162 arget of rapamycin pathway inhibits ELF4 and Kruppel-like factor 4 expression downstream of ERK and P
163 xpression of the E74-like factor (ELF) 4 and Kruppel-like factor 4 genes to release naive CD8(+) T ce
164                                Expression of Kruppel-like factor 4 suppressed transcription of FoxM1.
165  of key VSMC differentiation genes; notably, Kruppel-like factor 4 was found to be a direct target of
166 ssion of transcription factors such as KLF4 (Kruppel-like factor 4) and EGR1 (early-growth-response 1
167 CIN (fascin actin-bundling protein 1), KLF4 (Kruppel-like factor 4), Yes1 (YES proto-oncogene 1), and
168 n factor A) and dramatically represses KLF4 (Kruppel-like factor 4).
169 ophage polarization, including STAT3, STAT6, Kruppel-like factor 4, and peroxisome proliferator-activ
170 eased expression of the transcription factor Kruppel-like factor 4.
171 ntrol of expression of transcription factors Kruppel-like factors 4 or 5 (Klf4 or Klf5) which in turn
172                                              Kruppel-like factor-4 (KLF4) is known to inhibit activat
173 or-1alpha (HIF-1alpha, encoded by HIF1A) and Kruppel-like factor-4 (KLF4)-dependent.
174 n by repressing a transcriptional activator, Kruppel-like factor-4 (KLF4).
175 eated mice also showed reduced expression of Kruppel-like factor-4.
176                                              Kruppel-like factor 5 (Klf5) encodes a zinc-finger trans
177 ociated with increased ERK1/2 activation and Kruppel-like factor 5 (KLF5) expression in IEC.
178                     The transcription factor Kruppel-like Factor 5 (KLF5) has been shown to mediate c
179 tigated the role of the transcription factor Kruppel-like factor 5 (KLF5) in ADM and KRAS-mediated fo
180                                              Kruppel-like factor 5 (KLF5) is a pro-proliferative tran
181                                              Kruppel-like factor 5 (Klf5) is a transcription factor e
182                                              Kruppel-like factor 5 (KLF5) is a transcription factor t
183                                              Kruppel-like factor 5 (KLF5) is a zinc finger transcript
184                                          The Kruppel-like factor 5 (KLF5) is a zinc-finger transcript
185                                              Kruppel-like factor 5 (KLF5) is an effector of LPA-induc
186         The zinc finger transcription factor Kruppel-like factor 5 (KLF5) is regulated posttranslatio
187                                              Kruppel-like factor 5 (KLF5) is transcription factor tha
188 ow that the zinc finger transcription factor Kruppel-like factor 5 (KLF5) transactivates NOTCH1 in th
189 are primarily regulated by ovarian hormones, Kruppel-like factor 5 (KLF5), a zinc finger-containing t
190 ced age-related disorders, it is unclear how Kruppel-like factor 5 (Klf5), an essential transcription
191 s, Friend leukaemia integration 1 (Fli1) and Kruppel-like factor 5 (KLF5).
192                                              Kruppel-like factor 5 binds directly to the promoter of
193 rian cancer cells was mediated by C3a and is Kruppel-like factor 5 dependent.
194        In vivo-inducible gain-of-function of Kruppel-like factor 5 in haematopoietic stem cells incre
195         Altogether, these data indicate that Kruppel-like factor 5 is indispensable for adhesion, hom
196                                              Kruppel-like factor 5 regulates pluripotent stem cell se
197                            Here we show that Kruppel-like factor 5-deficient haematopoietic stem cell
198 d with increased cardiac expression of KLF5 (Kruppel-like factor-5) and PPARalpha (peroxisome prolife
199                          Here we report that Kruppel like factor 6 (KLF6), a transcription factor of
200 onal network involving the tumor suppressors Kruppel-like factor 6 (KLF6) and forkhead box O1 (FOXO1)
201          In the current study, we identified Kruppel-like factor 6 (KLF6) as a critical transcription
202  programmed cell death protein 4 (PDCD4) and Kruppel-like factor 6 (KLF6) as critical regulators and
203                                              Kruppel-like factor 6 (Klf6) belongs to a family of zinc
204                     Here, we discovered that Kruppel-like factor 6 (KLF6) governs macrophage function
205 of the transcription factor/tumor suppressor Kruppel-like factor 6 (KLF6) has been described in prost
206                                              Kruppel-like factor 6 (KLF6) is a transcription factor a
207 etic activation of the tumor suppressor gene Kruppel-like factor 6 (KLF6) plays a role in ERMA-induce
208      The polymorphism, KLF6-IVS1-27A, in the Kruppel-like factor 6 (KLF6) transcription factor gene e
209 estingly, a key factor in tissue remodeling, Kruppel-like factor 6 (KLF6) translocates to the cell nu
210                                              Kruppel-like factor 6 (KLF6) was identified as a key tra
211                                 Among these, Kruppel-like factor 6 (KLF6) was reduced in DDLPS, with
212  domain of LCoR and the transcription factor Kruppel-like factor 6 (KLF6), a putative tumor suppresso
213                          Here, we identified Kruppel-like factor 6 (KLF6), a zinc finger domain trans
214                                              Kruppel-like factor 6 (KLF6), a zinc finger transcriptio
215                         Transcription factor Kruppel-like factor 6 (KLF6)-myeloid-specific conditiona
216                                              Kruppel-like factor 6 (Klf6; copeb in zebrafish) is a zi
217 manner dependent on the transcription factor Kruppel-like factor 6 , suggesting that this pathway und
218                                              Kruppel-like factor 6 and miR-223 signaling axis regulat
219 onjunction with the tumor suppressor protein Kruppel-like factor 6 functioning as an AhR binding part
220 nding protein 4, GATA-binding protein 6, and Kruppel-like factor 6.
221 d, in part, by the transcriptional activator Kruppel-like factor-6 (Klf6).
222                      Increased expression of Kruppel-like factor 7 (KLF7) is an independent predictor
223   Here we show that the transcription factor Kruppel-like factor 7 (KLF7) is overexpressed in PDACs,
224 nown coactivator of the transcription factor Kruppel-like factor 7 (KLF7), which regulates genes requ
225                  We have recently identified Kruppel-like factor 8 (KLF8) as a critical inducer of EM
226                                              Kruppel-like factor 8 (KLF8) is a transcriptional factor
227         We have previously demonstrated that Kruppel-like factor 8 (KLF8) participates in oncogenic t
228                                              Kruppel-like factor 8 (KLF8) regulates critical gene tra
229                                              Kruppel-like factor 8 (KLF8) regulates critical gene tra
230 -Myc, Cyclin D1, Bcl-xL, Survivin, VEGF, and Kruppel-like factor 8, which is identified as a Stat3 ta
231 eogenesis utilizing the transcription factor Kruppel-like factor 9 (KLF9) in physiology and disease s
232               However, some genes, including Kruppel-like factor 9 (KLF9), a putative transcriptional
233 41 transcription factors including circadian Kruppel-like factor 9 (KLF9), and ~260 of their target g
234 of a negative regulator of dendritic spines, Kruppel-like factor 9 (Klf9), in mature DGCs enhanced in
235 GR1), forkhead box protein A3 (FOXA3), JUNB, Kruppel-like factor 9 (KLF9), KLF10, and REV-ERBalpha-we
236  Nrf2 stimulates expression of transcription Kruppel-like factor 9 (Klf9), resulting in further Klf9-
237 o3), thyroid receptors (tralpha, trbeta) and Kruppel-like factor 9 (klf9), suggesting regulation by t
238 identified a circadian transcription factor, Kruppel-like factor 9 (Klf9), that is substantially up-r
239                     Using keratinocytes with Kruppel-like factor 9 knockdown, we revealed a feedforwa
240 -of-function approaches, we also showed that Kruppel-like factor 9 was a key mediator of this effect
241                                    Erythroid Kruppel-like factor, a pivotal regulator of erythropoies
242                                          The Kruppel-like factor, BTEB1/KLF9, was expressed in both p
243                                    Erythroid Kruppel-like factor (EKLF or KLF1) is a transcription fa
244                                    Erythroid Kruppel-like factor (EKLF or KLF1) is a transcriptional
245             Mutations in the human erythroid Kruppel-like factor (EKLF) can lead to either anemia or
246 man erythroid cells, expression of erythroid Kruppel-like factor (EKLF) precedes PlGF, and its enforc
247 thin the second zinc finger of the erythroid Kruppel-like factor (EKLF), a critical erythroid regulat
248 here transcription factors such as erythroid Kruppel-like factor (EKLF), which is also known as Krupp
249                            Because Erythroid Kruppel-like Factor (EKLF)-knockout mice showed similar
250 , including the erythroid-specific erythroid Kruppel-like factor (EKLF).
251 ing the erythroid master regulator erythroid Kruppel-like factor (EKLF, also known as KLF1).
252                                    Erythroid Kruppel-like Factor (EKLF/KLF1) is a master transcriptio
253                                    Erythroid Kruppel-like factor (EKLF/KLF1) is one of a very small n
254 tic leukemia 1 protein (Tal1), and Erythroid Kruppel-like factor (EKLF; henceforth referred to as Klf
255                                    Erythroid Kruppel-like factor (EKLF; KLF1) is an erythroid-specifi
256                                    Erythroid Kruppel-like factor (EKLF; KLF1), a crucial zinc finger
257     Our group and others have implicated the Kruppel-like factor family of transcription factors as c
258                                          The Kruppel-like factor family of transcription factors play
259                      KLF4 is a member of the Kruppel-like factor family of transcriptional regulators
260 codes a novel transcription regulator in the Kruppel-like factor family.
261  signals implicate lower-frequency variants, Kruppel-like factors, Hedgehog signaling, Hippo-YAP sign
262 review focuses on the relevant literature of Kruppel-like factors in leukocyte biology and their impl
263 OXO may represent a broad mechanism by which Kruppel-like factors integrate with insulin signaling to
264 ctor 4 (Klf; previously known a gut-enriched Kruppel-like factor) is a DNA-binding transcriptional re
265 c mice deficient in the transcription factor Kruppel-like factor (KLF) 13 have comparable numbers of
266                                              Kruppel-like factor (KLF) 13 is a transcription factor t
267  and decreased induction of antiinflammatory kruppel-like factor (KLF) 2 and KLF4.
268 entified a role for the transcription factor Kruppel-like factor (KLF) 4 in the development of IL-17-
269 nomics, we have identified conserved ETS and Kruppel-like factor (KLF) binding sites within the Flk1
270 he Sp1 transcription factor, a member of the Kruppel-like factor (KLF) family of transcriptional regu
271                                              Kruppel-like factor (KLF) motifs were enriched in cornea
272                                              Kruppel-like factor (KLF) proteins are emerging as key r
273                                              Kruppel-like factor (KLF) proteins have elicited signifi
274 scription factors involved in axonal growth, Kruppel-like factor (KLF) transcription factors-7 and -9
275 chanistically, ORAI1 induced upregulation of Kruppel-like factor (KLF)-2 and KLF4 in the flow-activat
276 Regulating these states is the presence of a Kruppel-like factor (KLF)-containing Polycomb response e
277                            The importance of Kruppel-like factor (KLF)-mediated transcriptional pathw
278         Several recent studies reported that Kruppel-like factor (KLF)2 controls trafficking, develop
279 leted mice have injured vessels with reduced Kruppel-like factor (KLF)2, KLF4, endothelial nitric oxi
280 duced activation of the transcription factor Kruppel-like factor (KLF)4 may have an important role in
281 dent pathway by serving as a coactivator for Kruppel-like factor (KLF)4-a driver of tissue-resident m
282 croarray and experimental data revealed that Kruppel-like factor (Klf)5 was the most up-regulated tra
283                       Here, we identify that Kruppel-like factor (KLF)6 promotes inflammation by rest
284              Here we identify the C. elegans Kruppel-like factor, KLF-1, as an essential and specific
285 erein, we report that a TGF-beta1-responsive Kruppel- like factor, KLF10, is strongly expressed in PA
286 expression of FABP5 is down-regulated by the Kruppel-like factor KLF2, suggesting a tumor suppressor
287                                          The Kruppel-like factor Klf4 is implicated in tumorigenesis
288 titution of the sole sumoylation site of the Kruppel-like factor (KLF4), a well known reprogramming f
289                   TGFbeta/Smad3 up-regulated Kruppel-like factor (KLF5) protein, and SMC de-different
290                                              Kruppel-like factors (KLFs) are a family of 17 transcrip
291                                              Kruppel-like factors (KLFs) are a family of zinc-finger
292                                              Kruppel-like factors (KLFs) are transcription factors th
293                                              Kruppel-like factors (KLFs) are zinc finger transcriptio
294              While recent insights implicate Kruppel-like factors (KLFs) as important regulators of v
295               Specificity proteins (SPs) and Kruppel-like factors (KLFs) belong to the family of tran
296                                              Kruppel-like factors (KLFs) control cell differentiation
297                                              Kruppel-like factors (KLFs) play a critical role in regu
298  of enhancer RNAs pointed a central role for Kruppel-like factor, MEF2C, ETS, NFY, ATF, E2F2, and NRF
299                              KLF1 (erythroid Kruppel-like factor) plays essential roles in embryonic
300 e isoform 2 (PKM2) and T-helper-inducing POZ-Kruppel-like factor (ThPOK).

 
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