ライフサイエンスコーパス: L-ascorbic acid

1 in dipalmitate and 2-O-beta-d-glucopyranosyl-l-ascorbic acid.

2 ulated 2.2-fold by the addition of 50 microm l-ascorbic acid.

3 the de novo synthesis of GDP-L-galactose and L-ascorbic acid.

4 such as rutin and 2-O-beta-d-glucopyranosyl-l-ascorbic acid.

5 support the interactions between solutes and L-ascorbic acid.

6 rectly oxidized the major plant antioxidant, l-ascorbic acid.

7 of any trend supporting a protective role of L-ascorbic acid.

8 correlates of circulating concentrations of L-ascorbic acid.

9 gene locus and circulating concentrations of L-ascorbic acid.

10 arable for ascorbic acid and 6-bromo-6-deoxy-L-ascorbic acid.

11 served for ascorbic acid and 6-bromo-6-deoxy-L-ascorbic acid.

12 l beverages (citric acid, pH 3.0) containing l-ascorbic acid (0.050%) degraded with a first-order rea

13 sive and readily available starting material L-ascorbic acid (15) is described.

14 hree components: the basal medium RPMI 1640, L-ascorbic acid 2-phosphate and rice-derived recombinant

15 toxicant injury and stimulation of repair by L-ascorbic acid-2-phosphate (AscP), exogenous collagen I

16 a reduction in circulating concentrations of L-ascorbic acid (-4.15 micromol/L; 95% CI: -0.49, -7.81

17 drogenase was activated by either 150 microm L-ascorbic acid (56%) or 300 microm iron (Fe(2+) or Fe(3

18 The 2-O-beta-(d)-glucopyranosyl-(l)-ascorbic acid (AA-2betaG) was identified by UHPLC Q-T

19 hlorogenic acid (CG), (-) epicatechin (EPI), L-ascorbic acid (AA) and polyphenoloxidase (PPO) activit

20 optimised and evaluated for determination of l-ascorbic acid (AA) content in beverages.

21 ioxidant properties, total phenolic (TP) and l-ascorbic acid (AA) content.

22 Treatment with 2-phospho-L-ascorbic acid (AA2P) restored cell viability in both m

23 y DHA and GLUT transporters, 6-bromo-6-deoxy-L-ascorbic acid accumulation was <1% of accumulation whe

24 relation between rs33972313 and circulating L-ascorbic acid across all studies confirmed this and sh

25 nts and recovery percentages of vanillin and l-ascorbic acid among the coexisting phases.

26 incorporation into a topical solution of 15%l-ascorbic acid and 1%alpha-tocopherol improved chemical

27 We assessed the relation between L-ascorbic acid and 10 cardiometabolic traits by using a

28 , we have identified two inhibitors of SapM, L-ascorbic acid and 2-phospho-L-ascorbic, which define t

29 The antioxidants l-ascorbic acid and alpha-tocopherol, with better brain

30 gestion that observational relations between L-ascorbic acid and cardiometabolic health may be attrib

31 Two different reducing agents, namely, L-ascorbic acid and citric acid, were utilized for the r

32 ons of haemoglobin and natural antioxidants (l-ascorbic acid and Fuscus vesiculosus extract), were hy

33 vestigation of relations between circulating L-ascorbic acid and health outcomes.

34 The addition of L-ascorbic acid and iron resulted in a significant incre

35 (62%)) and 2.6-fold by the addition of both L-ascorbic acid and iron.

36 ared with that expected given the rs33972313-L-ascorbic acid and L-ascorbic acid-outcome associations

37 tes, inverse associations were shown between L-ascorbic acid and systolic blood pressure, triglycerid

38 utritional compounds (phenolic compounds and L-ascorbic acid), and antioxidant capacity of 4 Spanish

39 ased content of phenolics, anthocyanins, and l-ascorbic acid, and an increased antioxidant activity.

40 phenolic compounds, carotenoid, anthocyanin, L-ascorbic acid, and fibre content), antioxidant potenti

41 noreactor (NR) that comprises chlorophyll a, l-ascorbic acid, and gold nanoparticles that are encapsu

42 creased stem removal force, total flavonoid, l-ascorbic acid, and oxalic acid.

43 s of human AspH with respect to 2OG, Fe(II), l-ascorbic acid, and substrate and found that these para

44 07-SD change; P = 0.0001) per SD increase in L-ascorbic acid], and a positive association was shown w

45 volumes of transfer of solutes from water to L-ascorbic acid(aq) have been calculated.

46 The antioxidant and redox properties of l-ascorbic acid are closely associated with the electron

47 ue, we identified and tested 6-bromo-6-deoxy-L-ascorbic acid as a specific candidate for SVCTs.

48 L-Ascorbic acid (AsA) and its metabolic precursors give

49 l-Ascorbic acid (AsA) degradation at concentrations 0.13

50 is mutant vitamin c-1 (vtc1) is deficient in l-ascorbic acid (AsA) due to a mutation in GDP-Man pyrop

51 s developed and applied to the extraction of l-ascorbic acid (AsA) in apple.

52 L-Ascorbic acid (AsA) was found to be loaded into phloem

53 Vitamin C, or L-ascorbic acid (AsA), is the most abundant antioxidant

54 nderlying the regulation of fruit vitamin C (L-ascorbic acid [AsA]) concentrations, quantitative trai

55 Two biosynthetic pathways for ascorbate (l-ascorbic acid [AsA]; vitamin C) in plants are presentl

56 Vitamin C (L-ascorbic acid; AsA) acts as a potent antioxidant and c

57 nzyme activity is specifically stimulated by l-ascorbic acid (ascorbate, vitamin C), an effect not we

58 idue (Cys32) located close to the substrate (L-ascorbic acid) binding site has been modified to precl

59 alized using the Hill equation; oxidation of L-ascorbic acid by Cys32-modified rsAPX showed no eviden

60 ort of the reduced compound, 6-bromo-6-deoxy-L-ascorbic acid, by GLUT1 or GLUT3.

61 his study, nine different food constituents (l-ascorbic acid, caffeic acid, caffeine, curcumin, (-)-e

62 hocyanins (0.025%) in model beverages (0.05% l-ascorbic acid, citric acid, pH 3.0) stored at elevated

63 ene (rs33972313) associated with circulating L-ascorbic acid concentrations.

64 de participating in oxidation of an analyte, l-ascorbic acid, connected with electrode coated by poly

65 tenoid content (2.80-4.08 mg 100 g(-1)), and L-ascorbic acid content (34.63-63.84 mg 100g(-1)).

66 The highest l-ascorbic acid content (4.41mg/100g) was found in the p

67 ultivar on the antioxidant capacity, TP, and l-ascorbic acid content, whilst the breeding strategy an

68 lowest yielding genotype showed the highest l-ascorbic acid content.

69 c transients for reduction of Compound II by L-ascorbic acid for Cys32-modified rsAPX are monophasic

70 Clinical manifestations owing to l-ascorbic acid for scurvy as comparison to d-ascorbic a

71 ated on carbon electrodes working area using L(+) ascorbic acid, gold chroloauric acid and poly-l-lys

72 Vitamin C (L-ascorbic acid) has important antioxidant and metabolic

73 Vitamin C (L-ascorbic acid) has important antioxidant and metabolic

74 25 and 0.35) mol kg(-1) aqueous solutions of l-ascorbic acid have been determined from density data m

75 Simple derivatives of l-ascorbic acid have been shown to possess antioxidant,

76 Although the biosynthetic pathway of L-ascorbic acid in animals is well understood, the plant

77 valuating chemical behavior and stability of L-ascorbic acid in aqueous and buffer solutions.

78 r is capable of measuring d-ascorbic acid or l-ascorbic acid in aqueous as well as in real and commer

79 ssociated with circulating concentrations of L-ascorbic acid in the general population.

80 anches on a Pd core by reducing K2PtCl4 with L-ascorbic acid in the presence of uniform Pd nanocrysta

81 he combined data support the conclusion that l-ascorbic acid is a physiological activator of mitochon

82 l-Ascorbic acid is a versatile radical scavenger widely

83 L-ascorbic acid is an essential part of the human diet a

84 activity of PANI/SBA-15 toward oxidation of L-ascorbic acid is comparable to that obtained from a co

85 This shows that the addition sequence of l-ascorbic acid is crucial in determining the onset of d

86 We conclude that 6-bromo-6-deoxy-L-ascorbic acid is the first transport substrate identif

87 Vitamin C (l-ascorbic acid) is a potent antioxidant and cellular re

88 Vitamin C (L-ascorbic acid) is essential for many enzymatic reactio

89 Erythorbic acid, an epimer of L-ascorbic acid, is used in the United States as a food

90 which the vast majority of world vitamin C (L-ascorbic acid, L-AA) is produced.

91 In the presence of 2-phospho-L-ascorbic acid, miRNAs were quantitatively determined o

92 a reduction in circulating concentrations of L-ascorbic acid of -5.98 micromol/L (95% CI: -8.23, -3.7

93 The performance of the obtained l-ascorbic acid or d-ascorbic acid chiral selective sens

94 glycerol-3-phosphate dehydrogenase by either L-ascorbic acid or iron or its combination could be tota

95 ted given the rs33972313-L-ascorbic acid and L-ascorbic acid-outcome associations.

96 l-ascorbic acid, oxalic acid, citric acid and malic acid

97 -, 0.25-SD; P = 3.34 x 10(-)(6)) increase in L-ascorbic acid per effect allele.

98 culture in the presence of dexamethasone and L-ascorbic acid phosphate magnesium salt n-hydrate.

99 n model beverage systems (pH 3.0) containing L-ascorbic acid proceeded with a first-order reaction ra

100 a combination of a catalytic Fe(II) salt and l-ascorbic acid, proceeds under mild conditions with goo

101 ta-induced paralysis because the antioxidant L-ascorbic acid reduced intracellular levels of hydrogen

102 -O-allyl derivatives of 5,6-O-isopropylidene-l-ascorbic acid, respectively, followed by stereospecifi

103 L-(-)-Malic and L-(-)-ascorbic acids served well as starting materials f

104 the growth of the mutant mice, which require L-ascorbic acid supplemented in their drinking water (33

105 en made with the studies reported earlier in l-ascorbic acid, thiamine HCl and pyridoxine HCl.

106 irst step in investigating the importance of L-ascorbic acid transport in regulating the supply and m

107 T2 each mediate concentrative, high-affinity L-ascorbic acid transport that is stereospecific and is

108 erequisite for investigating 6-bromo-6-deoxy-L-ascorbic acid transported by SVCTs, SVCT2 transport ac

109 Here we describe the isolation of two L-ascorbic acid transporters, SVCT1 and SVCT2, from rat

110 Enzyme activity measurement showed that L-ascorbic acid (vitamin C (Vc)) competitively inhibits

111 oxoglutarate as a co-substrate(4), CMD1 uses L-ascorbic acid (vitamin C) as an essential co-substrate

112 eeler) pathway represents the major route to L-ascorbic acid (vitamin C) biosynthesis in higher plant

113 L-ascorbic acid (vitamin C) is a powerful reducing agent

114 bservational studies showed that circulating L-ascorbic acid (vitamin C) is inversely associated with

115 The biosynthesis of L-ascorbic acid (vitamin C) is not well understood in pl

116 cultured endothelial cells, the antioxidant, L-ascorbic acid (vitamin C), increases nitric oxide synt

117 r previously reported studies carried out in l-ascorbic acid (vitamin C).

118 llular uptake of the essential micronutrient l-ascorbic acid (vitamin C).

119 The effect of organic farming on l-ascorbic acid was dependent on cultivar and environmen

120 The accumulation of polyphenols and l-ascorbic acid was evaluated under conventional (integr

121 The reduction of 2,6-dichloroindophenol by l-ascorbic acid was performed using the theta-glass emit

122 2-O-beta-d-glucopyranosyl-l-ascorbic acid was stable at the conditions applied.

123 When L-ascorbic acid was used as the reducing agent, conforma

124 The content of L-ascorbic acid was very high and took values in the ran

125 yme activity, dependent upon the presence of l-ascorbic acid, was inhibited by lauryl gallate, propyl

126 processed food waste material, vanillin and l-ascorbic acid, was successfully accomplished.

127 rmine trace amounts of total arsenic, KI and l(+) ascorbic acid were used to reduce As(V) to the triv

128 ore and after the de-doping of chiral d- and l-ascorbic acid were characterized by scanning electron

129 ess this question, d-glucose, d-fructose and l-ascorbic acid were incubated with human serum albumin

130 compounds ascorbic acid and 6-bromo-6-deoxy-L-ascorbic acid were similar.

131 ch migrates for the alcohol-rich phase, from l-ascorbic acid, which preferentially partitions for the

132 The enzyme catalyzes the formation of L-ascorbic acid, which reduces Ag(+) in solution to yiel

133 aldono-1,4-lactone derivatives starting from l-ascorbic acid, which would be valuable in the synthesi

134 h electrocatalytic activity for oxidation of L-ascorbic acid, with very low overpotential and high cu