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1                                              L. interrogans LpxA furthermore displays absolute select
2 were challenged intraperitoneally with 10(8) L. interrogans serovar Pomona bacteria (NVSL 1427-35-093
3                         Mice infected with a L. interrogans lic11254 transposon mutant shed tenfold f
4  alleys where the patients may have acquired L. interrogans; polymerase chain reaction (PCR) analysis
5 n was discovered to be at risk for acquiring L. interrogans: urban residents who are sporadically exp
6 igA is a potential vaccine candidate against L. interrogans serovar Pomona infection.
7 mer was unable to discriminate strains among L. interrogans serovar copenhageni isolates, it was able
8 o Leptospira species, L. borgpetersenii, and L. interrogans.
9 ward rLipL32 protein in phosphate buffer and L. interrogans-spiked healthy human serum samples within
10             Infection led to weight loss and L. interrogans dissemination from blood to urine, and sp
11 e inoculum of high-passage-number attenuated L. interrogans strains resulted in dissemination to all
12 No significant association was found between L. interrogans or helminth infections and health profile
13 l (i.p.) and intradermal (i.d.) challenge by L. interrogans serovar Copenhageni strain Fiocruz L1-130
14 orses demonstrated that LfhA is expressed by L. interrogans during mammalian infection.
15  as a vaccine candidate against infection by L. interrogans serovar Pomona in a hamster model.
16                  Inner-city rats often carry L. interrogans.
17  rats by microagglutination assay to confirm L. interrogans infection.
18                         A PCR assay detected L. interrogans DNA in samples of body fluid obtained fro
19       Our R-LFI sensor successfully detected L. interrogans-positive clinical samples as confirmed by
20                                  We examined L. interrogans LigA, domains 7 to 13 (LigA7-13), as an o
21 tively acylated by LpxAs of A. ferrooxidans, L. interrogans, and M. loti.
22 nt, suggesting that additional receptors for L. interrogans await identification.
23                                     LPS from L. interrogans cells is unusual in that it activates TLR
24 gomyelinases Sph2 and Sph1 (N terminus) from L. interrogans serovar Lai.
25                     To better understand how L. interrogans serovar Copenhageni adapts to osmolar con
26 notyping directly from the kidney identified L. interrogans.
27 lso required for acyltransferase activity in L. interrogans.
28  revealed a candidate gene (renamed lmtA) in L. interrogans showing distant homology to the yeast iso
29 fects of temperature on protein synthesis in L. interrogans.
30                 We also show that for intact L. interrogans, it is LPS, not lipoprotein, that constit
31 antly altered the transcript levels of 6% of L. interrogans genes.
32              Intact hexa-acylated lipid A of L. interrogans Pomona was also analyzed by NMR, confirmi
33 ty is relevant in studying the adaptation of L. interrogans to host conditions.
34   An in-depth structural characterization of L. interrogans is needed to understand its biology and p
35 f glycolipoprotein, a cell wall component of L. interrogans that is known to inhibit the expression a
36 ral difference between the cell envelopes of L. interrogans and Leptospira biflexa involving variatio
37 f both the virulent and nonvirulent forms of L. interrogans serovar Icterohaemorrhagiae (strain Verdu
38 s intrinsically labeled during incubation of L. interrogans in medium containing [14C]palmitic acid,
39 ese assays to several new animal isolates of L. interrogans from Nicaragua, which recently had an out
40 gned to identify clones in a gene library of L. interrogans serovar Pomona expressing host-inducible
41                               Indeed, LPS of L. interrogans efficiently prevents caspase 11 dimerizat
42             Lipid A was released from LPS of L. interrogans serovar Pomona by 100 degrees C hydrolysi
43                             The mechanism of L. interrogans LpxA appears to be similar to that of E.
44  Differences may be related to the number of L. interrogans spirochetes that succeed in overcoming th
45   Chronic shedding can enable persistence of L. interrogans within the sea lion population.
46 ues were positive by PCR for the presence of L. interrogans.
47 s supported the isolation and propagation of L. interrogans serovar Copenhageni strain IC:20:001 in s
48                                 Screening of L. interrogans and L. kirschneri expression libraries wi
49                     Given the selectivity of L. interrogans LpxA for 3-hydroxylaurate, we propose tha
50                 The substrate selectivity of L. interrogans LpxA is consistent with the structure of
51 und as a single gene copy in all serovars of L. interrogans but not in other Leptospira spp. except L
52 of qlp42 and hsp15 in pathogenic serovars of L. interrogans but not in the nonpathogenic Leptospira b
53                             The structure of L. interrogans lipid A has now been determined by a comb
54 ans LpxA is consistent with the structure of L. interrogans lipid A.
55  B PGs appeared to be the primary targets of L. interrogans attachment, while heparan sulfate PGs wer
56 e (strain Verdun) were identical to those of L. interrogans Pomona by the above criteria.
57 ctor in the invasiveness and transmission of L. interrogans.
58 tified an abundant protein from the pathogen L. interrogans, exposed on the PF surface, and named it
59 d a potent antagonistic effect of pathogenic L. interrogans and their atypical LPS on spontaneous and
60  major outer membrane proteins of pathogenic L. interrogans.
61 inity blot analyses revealed that pathogenic L. interrogans produces at least two factor H-binding pr
62                                     Purified L. interrogans lipid A is inactive against human THP-1 c
63 and urine were used to identify and quantify L. interrogans, while fecal samples were analyzed for he
64 ogen, Salmonella: In contrast to Salmonella, L. interrogans did not alter transepithelial electrical
65 core group of pathogenic Leptospira species: L. interrogans, L. kirschneri, L. noguchii, L. borgpeter
66            In this work, we demonstrate that L. interrogans induces NLRP3 inflammasome-dependent secr
67 oratories and another have demonstrated that L. interrogans can acquire host plasminogen on its surfa
68 s LpxA for 3-hydroxylaurate, we propose that L. interrogans lipid A is acylated with R-3-hydroxylaura
69                Interestingly, we showed that L. interrogans and Leptospira biflexa do not trigger cel
70 D protein orthologs were not detected in the L. interrogans genome.
71              The recent determination of the L. interrogans lipid A structure revealed an unprecedent
72 atients had high levels of antibodies to the L. interrogans serogroup icterohaemorrhagiae.
73 PS, is not involved in cellular responses to L. interrogans.
74 ar to those of hosts infected with wild-type L. interrogans.
75                  Characterization of various L. interrogans exoprotein mutants in the animal infectio
76 thin 1 h after inoculation of 10(8) virulent L. interrogans bacteria.
77  LigB proteins was substantially higher when L. interrogans proliferated at 37 degrees C instead of t
78 cent sera from mares naturally infected with L. interrogans suggest that Qlp42 is expressed during le
79 nse to an IgG3 response after infection with L. interrogans Histological periodic acid-Schiff D stain
80  result of naturally acquired infection with L. interrogans serovar pomona type kennewicki recognize
81 TLR2/4-deficient mice 3 d postinfection with L. interrogans.