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1                                              L. lactis HR279 and JHK24 demonstrates two datasets with
2                                              L. lactis I-1631 may represent a promising vehicle to de
3                                              L. lactis I-1631 possesses genes encoding enzymes that d
4                                              L. lactis K1 was rapidly destroyed by the macrophages, a
5                                              L. lactis KF147, a strain originally isolated from plant
6                                              L. lactis NCDO2118 significantly reduced visceral hypers
7                                              L. lactis NCDO2727 with similar genes for GABA metabolis
8                                              L. lactis(pLM1) invaded epithelial cells efficiently in
9                              We identified a L. lactis strain, able to attenuate esophageal eosinophi
10      Inactivation of the sodA gene abolished L. lactis I-1631's beneficial effect in the T-bet(-/-) R
11 a at baseline predicts a poor response after L. lactis-based immunotherapy in nonobese mice with new-
12 redation and that LTA galactosylation alters L. lactis resistance to bacteriocin.
13 deficient in binding fluid-phase gp-340, and L. lactis cells expressing AspA were not agglutinated by
14 hydroorotate dehydrogenases from E. coli and L. lactis also have a lysine near N5 of the flavin.
15 oming mechanism in both Escherichia coli and L. lactis.
16 ased internalization of both E. faecalis and L. lactis (with and without AS expression).
17 nted on the cell surfaces of E. faecalis and L. lactis but not on that of S. gordonii.
18 sc10 on the cell surfaces of E. faecalis and L. lactis revealed a significant increase in cell surfac
19 osed by Lactococcus lactis subsp. lactis and L. lactis subsp. cremoris (R704); QLA - with Lactobacill
20 or of S. pyogenes LDH, E. faecalis LDH1, and L. lactis LDH1 and LDH2 at pH 6.
21 e growth of VRE is inhibited by BP(SCSK) and L. lactis in vitro, only BP(SCSK) colonizes the colon an
22 ese experiments, the E. faecalis strains and L. lactis K1 were grown in brain heart infusion (BHI) br
23 oup II intron in the Gram-positive bacterium L. lactis and demonstrate for the first time in bacteria
24 ant genes present in the probiotic bacterium L. lactis.
25 in can also be used to differentiate between L. lactis and L. garvieae.
26 ermined the ability to uptake (3)H-biotin by L. lactis.
27              Unlike the scenario in E. coli, L. lactis appears to be auxotrophic for biotin in that i
28                     Therefore, like E. coli, L. lactis has a single beta-ketoacyl-ACP reductase activ
29 owed transients that occurred when colliding L. lactis reduced transport of FcM to the electrode.
30 ti-CD3 with a clinical-grade self-containing L. lactis, appropriate for human application, secreting
31 d significantly better (P < 0.0001) than did L. lactis K1 at each time point.
32 rable to therapy results with plasmid-driven L. lactis Initial blood glucose concentrations (<350 mg/
33                               The engineered L. lactis is able to develop biofilms on different surfa
34 ontrast, a strong benefit for the engineered L. lactis strain was observed in acid stress survival.
35                                          For L. lactis, the particle velocity due to convection drive
36 rtance of specific plant-inducible genes for L. lactis growth in ATL, xylose metabolism was targeted
37 a number of different P335 phages, lytic for L. lactis NCK203, have a common operator region which ca
38 , possibly, cephalothin were higher than for L. lactis, and unlike L. lactis, L. garvieae was resista
39  occur when introduced into the plasmid-free L. lactis LM0230 during growth in galactose or lactose,
40  inhibitor binding to the Class 1A DHOD from L. lactis has now been studied in detail and is reported
41    It has recently been shown that HisZ from L. lactis binds to the ATP-PRPP transferase (HisG) and t
42 ssess HisZ) requires both HisG and HisZ from L. lactis.
43 idrug transcriptional repressor protein from L. lactis.
44 bound riboflavin transport protein RibU from L. lactis.
45 a homodimeric multidrug ABC transporter from L. lactis.
46                                           In L. lactis, LlPC is required for efficient milk acidifica
47  but different median values (3-4 x 10(4) in L. lactis and yeast versus 4 x 10(5) in Arabidopsis).
48 ly, overexpression of the pilB gene alone in L. lactis enhanced resistance to phagocyte killing, incr
49 endonuclease-independent pathway, and, as in L. lactis, such events have a more random integration pa
50 NA, LtrA, intron splicing and conjugation in L. lactis.
51 to enable primary-active multidrug efflux in L. lactis.
52  and expressed this fusion protein (MBP*) in L. lactis.
53                        The aspartate pool in L. lactis is negatively regulated by c-di-AMP, and high
54     Expression of the recombinant protein in L. lactis also markedly increased biofilm formation.
55 high levels in ATL, indicating redundancy in L. lactis carbohydrate metabolism on plant tissues.
56 x LlaI on enhancement of LlaI restriction in L. lactis revealed that growth at elevated temperatures
57 romoter region abolished LlaI restriction in L. lactis.
58            However, in retrotransposition in L. lactis, the intron inserts predominantly into single-
59 monstrated that the AbiR operon was toxic in L. lactis without the presence of the LlaKR2I methylase,
60                                    Unlike in L. lactis, in E. coli, Ll.LtrB retrotransposed frequentl
61                     This effect is weaker in L. lactis and Hfx.
62                      Inactivation of ylgG in L. lactis resulted in DHNTP accumulation and folate depl
63  Ll.LtrB intron from Lactococcus lactis into L. lactis 23S rRNA.
64 gens via the gut through Lactococcus lactis (L. lactis) has been demonstrated to be a promising appro
65 urification method using Lactococcus lactis (L. lactis), a generally recognized as safe (GRAS) bacter
66 Lactococcus lactis LDH2 < E. faecalis LDH1 &lt; L. lactis LDH1 </= Streptococcus pyogenes LDH.
67 rucial role of the microbial GAD activity of L. lactis NCDO2118 to deliver GABA into the gastro-intes
68 ray crystallographic and kinetic analyses of L. lactis galactose mutarotase complexed with D-glucose,
69 n of E. faecalis by HT-29 enterocytes and of L. lactis by HT-29 and Caco-2 enterocytes.
70                       In phage challenges of L. lactis(pTRK414H) with phi31, the efficiency of plaqui
71 modify the L. lactis AcpA and the chimera of L. lactis AcpA helix II in AcpP).
72 proved MIC values against liquid cultures of L. lactis HP.
73 lated microscopy of collision experiments of L. lactis using a 5 mum radius Pt disk UME in 2 mM ferro
74  AcpP helix II was due to incompatibility of L. lactis AcpA helix I with the downstream elements of A
75 lypeptides mediated higher binding levels of L. lactis cells to surface immobilized gp340 than did S.
76 tion of protective blends for manufacture of L. lactis probiotic powders was optimized using a statis
77                        By testing a panel of L. lactis cell wall mutants, we observed that Lc-Lys and
78  These findings show that certain strains of L. lactis are well adapted for growth on plants and poss
79 expression of AspA protein on the surface of L. lactis conferred biofilm-forming ability.
80 n the tip of pili external to the surface of L. lactis might constitute a successful vaccine strategy
81 yCEP expression was required for survival of L. lactis but not S. pyogenes.
82                                Velocities of L. lactis extracted from correlated microscopy movies co
83 t form of this protein either in vesicles of L. lactis or B. subtilis or in intact cells of B. subtil
84 % trehalose ensures the highest viability of L. lactis bacteria upon both drying techniques (viabilit
85          Heterologous expression of Asc10 on L. lactis also allowed the recognition of its binding li
86  P335 group phages failed to form plaques on L. lactis harboring pTRKH2::CI-per2, while 4 phages form
87 tion activity was not apparent in E. coli or L. lactis prior to phage infection.
88 on indicator gene previously employed in our L. lactis studies.
89                    In contrast to the parent L. lactis strain (lacks SpyCEP), which was avirulent whe
90 h DNA interacts with crystalline Dps phases, L. lactis DNA:Dps complexes appeared as non-crystalline
91                   Furthermore, such piliated L. lactis cells evoked a higher TNF-alpha response durin
92 KR2I methylase, which is required to protect L. lactis from AbiR toxicity.
93               Expression of the AcpP protein L. lactis AcpA helix II allowed weak growth, whereas the
94 in accordance with the growth rate, provides L. lactis cells the means to ensure optimal CW plasticit
95 owth in ATL than the dairy-associated strain L. lactis IL1403.
96 th either 0.035 or 0.1 mM KCl confirmed that L. lactis experienced transport by convection due to ele
97                                          The L. lactis expression system was also used to study the r
98                                          The L. lactis strain recapitulated in the compartment system
99 crobiota composition were observed after the L. lactis NCDO2118 treatment.
100 essment of the immune changes induced by the L. lactis-based therapy revealed elevated frequencies of
101  presented as a predictive signature for the L. lactis-based immunotherapy outcome in new-onset type
102                                 However, the L. lactis fabH deletion mutant requires only long-chain
103 entified a potential flippase encoded in the L. lactis genome (llnz_02975, cflA) and confirmed that i
104 er)-L-Lys(3); moreover, they do not lyse the L. lactis mutant containing only the nonamidated D-Asp c
105 cuous Sfp transferase required to modify the L. lactis AcpA and the chimera of L. lactis AcpA helix I
106                           Replacement of the L. lactis AcpA helix II residues in this protein showed
107 ucts showed that the lack of function of the L. lactis AcpA-derived protein containing E. coli AcpP h
108                                  Both of the L. lactis genes are adjacent to (and predicted to be cot
109 olecular mass and subunit composition of the L. lactis HisZ-HisG heteromeric ATP-PRTase is investigat
110 low-resolution cryo-EM reconstruction of the L. lactis ribosome fused to the intron-LtrA RNP of a spl
111 entially be used in the future to tailor the L. lactis-based combination therapy for individual patie
112 8 warrant further consideration of using the L. lactis system for the production of circumsporozoite
113 pA helix II allowed weak growth, whereas the L. lactis AcpA-derived protein that contained E. coli Ac
114  type 1 diabetes in clinical trials with the L. lactis-based immunotherapy.
115       Mucosal immunization of mice with this L. lactis strain expressing pilus-linked MBP* results in
116 of the phenotypic variation in resistance to L. lactis and E. faecalis, respectively, most of the mol
117 omic analyses, we identified genes unique to L. lactis I-1631 involved in oxygen respiration.
118              The in vivo function of the two L. lactis birA genes was judged by their abilities to co
119 eport that expression of only one of the two L. lactis proteins (that annotated as FabG1) allows grow
120                                    Wild-type L. lactis strain KF147 but not an xylA deletion mutant w
121 n were higher than for L. lactis, and unlike L. lactis, L. garvieae was resistant to clindamycin, ind
122                                      We used L. lactis strain to colonize material surfaces and produ
123                                 When we used L. lactis-derived PfCSP4/38 to immunize mice, it elicite
124 crophages, and by 24 h postinfection, viable L. lactis could not be recovered.
125 d with pretreated HT-29 enterocytes and when L. lactis was incubated with pretreated Caco-2 and HT-29
126  The current transients had step shapes when L. lactis collided and adsorbed and spike shapes when th
127 ise, chimeric ACPs were constructed in which L. lactis helix II replaced helix II of E. coli AcpP and
128 reducing flavin and Lys 168 (by analogy with L. lactis DHODase A).
129 e induction of those genes corresponded with L. lactis KF147 nutrient consumption and production of m
130 ults demonstrate that oral immunization with L. lactis expressing an Ag on the tip of the group A Str
131      In this EoE model, supplementation with L. lactis NCC 2287 significantly decreased esophageal an

 
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