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1                                              L. pneumophila adhesion on these biofilm under low flow
2                                              L. pneumophila delivers almost 300 effector proteins int
3                                              L. pneumophila encodes more than 300 putative effectors,
4                                              L. pneumophila is able to infect alveolar macrophages in
5                                              L. pneumophila is ubiquitously found in freshwater envir
6                                              L. pneumophila is well known as the cause of Legionnaire
7                                              L. pneumophila lacking lppA replicated less efficiently
8                                              L. pneumophila mutant strains lacking EnhC (DeltaenhC) i
9                                              L. pneumophila pathogenicity relies on secretion of more
10                                              L. pneumophila replicates in protozoa and mammalian phag
11                                              L. pneumophila replicates within macrophages by using a
12                                              L. pneumophila serogroup 1 isolates (n = 106) from the s
13                                              L. pneumophila strain Lp02, which is attenuated in the a
14                                              L. pneumophila TLR ligands induced the splicing of mRNA
15                                              L. pneumophila translocates more than 300 effectors into
16                                              L. pneumophila triggers the reduction of several sphingo
17                                              L. pneumophila was able to inhibit both chemical and bac
18                                              L. pneumophila were not detected by either method; Legio
19                            The 2011 and 2013 L. pneumophila patient isolates were serogroup 1 and clo
20                            WGS data from 229 L. pneumophila ST1 isolates were analyzed, including 99
21 , we report the sequence and analyses of 364 L. pneumophila genomes, including 337 from the five dise
22 er treatment to characterize and eliminate a L. pneumophila population responsible for nosocomial inf
23 t Ab-mediated protection is effective across L. pneumophila serogroups, suggesting that Abs specific
24 ay an important role in host defense against L. pneumophila infection.
25  relevant for Ab-mediated protection against L. pneumophila.
26 ntribute to the host immune response against L. pneumophila during pulmonary infection.
27 ay contribute to the immune response against L. pneumophila.
28 prophylactic potential in restricting airway L. pneumophila replication.
29 y in response to iron limitation that allows L. pneumophila to abandon the host cell when nutrients a
30                                     Although L. pneumophila utilizes host amino acids as the main sou
31                              Inside amoebae, L. pneumophila was detected in 13.9% (6/43) of the isola
32 genetic diversity and clonal expansion among L. pneumophila bacteria.
33 ocated inefficiently into cultured cells, an L. pneumophila DeltalpdA mutant displayed reduced replic
34 n among disinfectant residual, biofilms, and L. pneumophila, which provides guidelines to assess and
35  Bordetella pertussis, Escherichia coli, and L. pneumophila.
36 racellular colocalization of B. neotomae and L. pneumophila was required for rescue and that colocali
37 ring the wet season than the dry season, and L. pneumophila was only observed during the wet season.
38 TBK1/IRF3 pathway during M. tuberculosis and L. pneumophila infection of macrophages, whereas L. mono
39  to recognize intracellular bacteria such as L. pneumophila, leading to potent inflammatory responses
40 sinfectant to release the biofilm-associated L. pneumophila from these two types of biofilms, the L.
41 his study, the release of biofilm-associated L. pneumophila under simulated drinking water flow conta
42         We determined the connection between L. pneumophila adhesion and subsequent detachment with b
43       Given the intimate interaction between L. pneumophila and the endoplasmic reticulum, we investi
44 hat LtpD is a novel phosphoinositide-binding L. pneumophila effector that has a role in intracellular
45 al de novo synthesis of these amino acids by L. pneumophila.
46 ously derived rhizoferrin are assimilated by L. pneumophila in an LbtU- and LbtC-dependent manner.
47 ytokines in experimental pneumonia caused by L. pneumophila Intratracheal (i.t.) administration of L.
48 mg/L, 10/11 sites (91%) were contaminated by L. pneumophila serogroups 3 and 10.
49                               Since death by L. pneumophila infection depends on the early anti-micro
50 creens reveal known host factors hijacked by L. pneumophila, as well as genes spanning diverse traffi
51 solized bacteria, and infection of humans by L. pneumophila can result in a severe pneumonia called L
52 -mediated inflammasome activation induced by L. pneumophila.
53 entify host factors that regulate killing by L. pneumophila.
54     However, the infection event mediated by L. pneumophila Cas2 appeared to be distinct from this fu
55 ortant for phagosomal membrane remodeling by L. pneumophila.
56  We show here an iron starvation response by L. pneumophila after infection of macrophages that was p
57 We now report that two molecules secreted by L. pneumophila, homogentisic acid (HGA) and its polymeri
58 ious proteomic analysis revealed that T2S by L. pneumophila 130b mediates the export of >25 proteins,
59 d molecular mimicry strategy that is used by L. pneumophila to take control of the host cell Hippo pa
60 troscopy to directly sort pellets containing L. pneumophila cells, expelled by T. thermophila, and to
61 ned interactions between vacuoles containing L. pneumophila and the host ER.
62 ost xenophagy system but vacuoles containing L. pneumophila avoided targeting.
63        Ubiquitin-labeled vacuoles containing L. pneumophila failed to recruit autophagy adaptors by a
64 al structure and local hydrodynamics control L. pneumophila adhesion to and detachment from simulated
65      As infection model results for critical L. pneumophila concentrations were often below a feasibl
66 spase-11-dependent pyroptosis by cytoplasmic L. pneumophila-derived LPS required Gbp(chr3) proteins.
67 ng bacterial products into the host cytosol, L. pneumophila also activates cytosolic immunosurveillan
68  to T4SS-sufficient, but not T4SS-deficient, L. pneumophila.
69                          Analysis of defined L. pneumophila knock-out mutants indicated Lsp-dependent
70     PlcA and PlcB are two previously defined L. pneumophila proteins with homology to the phosphatidy
71 fections with the cytosol-invading DeltasdhA L. pneumophila mutant was similarly dependent on Gbp(chr
72 lic sensors called inflammasomes that detect L. pneumophila in vitro and in vivo.
73 y, two primer/probe sets (one able to detect L. pneumophila and the other L. pneumophila Sg1) were de
74  TRIF, and TBK1 in cytokine secretion during L. pneumophila infection of macrophages.
75 l regulation of the activity of UBE2N during L. pneumophila infection.
76  host unfolded protein response (UPR) during L. pneumophila infection.
77 the gene encoding LegC4 resulted in enhanced L. pneumophila in the lungs of infected mice but not wit
78  enhanced the capacity of viable filamentous L. pneumophila to escape phagosomal killing in a length-
79 cance of PlaB-derived lipolytic activity for L. pneumophila intracellular replication.
80 s when molecular diagnostics are applied for L. pneumophila detection.
81 aired translocation of proteins critical for L. pneumophila intracellular growth.
82    The metaeffector Lpg2505 is essential for L. pneumophila intracellular replication only when its c
83                 Compared to the findings for L. pneumophila-infected wild-type (WT) mice, the i.t.
84               Although the natural hosts for L. pneumophila are free-living protozoa that reside in f
85 ion in vitro Moreover, LbtP is important for L. pneumophila growth within macrophages while LbtU is d
86 come the new gold standard typing method for L. pneumophila.
87  moth Galleria mellonella as a new model for L. pneumophila infection.
88 n Rab6A' function and the role of Rab6A' for L. pneumophila growth within host cells has been unclear
89  colony-forming units (CFU) per reaction for L. pneumophila and three CFU per reaction for S. typhimu
90    Thus, the T2SS is absolutely required for L. pneumophila to grow to larger numbers in its intravac
91 hils are both an intracellular reservoir for L. pneumophila and a source of proinflammatory cytokines
92 9, was previously identified in a screen for L. pneumophila IDTS that manipulate secretory traffic wh
93 ar isotopologue patterns in amino acids from L. pneumophila wild type and the mutants under study ref
94 of the immunoglobulin-cleaving protease from L. pneumophila revealed that the protease is conserved a
95 ify the LegC3 secreted effector protein from L. pneumophila as able to inhibit a SNARE- and Rab GTPas
96 oiled-coiled domain containing proteins from L. pneumophila, LegC7/YlfA and LegC2/YlfB, did not inhib
97                     The use of 16s rRNA from L. pneumophila allowed for the detection of metabolicall
98  we show that the effector protein VipD from L. pneumophila exhibits phospholipase A1 activity that i
99  environmental survival and virulence; e.g., L. pneumophila employs T2S for infection of amoebae, gro
100 erved in the same amino acids from LCV-grown L. pneumophila.
101                     These studies reveal how L. pneumophila creates a vacuole that supports intracell
102                                     However, L. pneumophila is able to suppress the UPR and block the
103 erestingly, we show that the host identifies L. pneumophila infection as a form of endoplasmic reticu
104 , loss of both LbtP and LbtU does not impair L. pneumophila growth in the amoebal host Acanthamoeba c
105  Although the ability of IFN-gamma to impede L. pneumophila growth is fully dependent on Stat1, IFN-a
106                                           In L. pneumophila, DsbA2 is maintained as a mixture of disu
107                                           In L. pneumophila, DsbA2 was maintained as a mixture of thi
108  exhibits the most prominent PLA activity in L. pneumophila.
109 approach to interfere with DsbA2 function in L. pneumophila determined that DSB oxidase activity was
110                        Expression of IcaA in L. pneumophila inhibited the caspase-11 activation in ma
111              Related DGRs were identified in L. pneumophila clinical isolates that encode unique targ
112 lly abolishes the synthesis of pyomelanin in L. pneumophila cultures at 10 muM.
113 -E3 biochemistry and play important roles in L. pneumophila virulence.
114 phtD loci contribute to thymidine salvage in L. pneumophila.
115 ch is encoded in a recombination hot spot in L. pneumophila Paris.
116 -based typing (SBT) analysis of all incoming L. pneumophila serogroup 1 (Lp1) isolates to identify po
117 delay in lung bacterial clearance, increased L. pneumophila dissemination to extrapulmonary organs, a
118                                     Instead, L. pneumophila cells secrete HGA only when they are cond
119 ba amino acids into the LCV and further into L. pneumophila where they served as precursors for bacte
120 alth of previously undescribed insights into L. pneumophila pathogenesis and mammalian cell function.
121 ng some that are important for intracellular L. pneumophila replication.
122 vo synthesis of amino acids by intravacuolar L. pneumophila contributes to its nutrition.
123          Instead, the absence of LbtP limits L. pneumophila replication and causes bacteria to premat
124 pellets, detection methods for packaged live L. pneumophila forms remaining in water should be cultiv
125       Finally, when cultured in macrophages, L. pneumophila required the phtC-phtD locus to replicate
126    Furthermore, approximately <10 CFU per mL L. pneumophila may be appropriate for healthcare or susc
127 er identify lpg1681 as a gene that modulates L. pneumophila susceptibility to HGA.
128 s for these cytokines in experimental murine L. pneumophila pneumonia.
129 infection with wild-type but not T4SS mutant L. pneumophila Using confocal microscopy, it was determi
130                          A total of 30 novel L. pneumophila B cell Ags were identified, the majority
131 cterial supernatants enhanced the ability of L. pneumophila and other species of Legionella to take u
132 '(Q72L) significantly reduced the ability of L. pneumophila to initiate intracellular replication in
133 enic evolution and nutritional adaptation of L. pneumophila and other intracellular bacteria to life
134  and macrophages, we show that adaptation of L. pneumophila to each amoeba causes the accumulation of
135 phila Intratracheal (i.t.) administration of L. pneumophila induced the upregulation of both IL-36alp
136                            administration of L. pneumophila to IL-36 receptor-deficient (IL-36R(-/-))
137        The results of phenotypic analyses of L. pneumophila strains lacking phtC or phtD strongly ind
138  together, genome-wide chromatin analysis of L. pneumophila-infected macrophages demonstrated inducti
139        Here, we show that upon attachment of L. pneumophila to human monocyte-derived macrophages (hM
140        However, after ingesting the cells of L. pneumophila, some protozoa expel them as compressed l
141 tematically comparing pulmonary clearance of L. pneumophila in C57BL/6 MyD88(-/-), TLR2(-/-), TLR3(-/
142  document the occurrence and colonization of L. pneumophila Sg1 in cold water delivered from point of
143 ection, T2SS is clearly a major component of L. pneumophila intracellular infection.
144 olling adhesion and subsequent detachment of L. pneumophila associated with biofilms remain unclear.
145 e direct, timely, and effective detection of L. pneumophila within man-made water systems.
146 viable Legionella, shown with the example of L. pneumophila, ranging in a total concentration between
147 ost cell infection, VipD reduces exposure of L. pneumophila to the endosomal compartment and protects
148 e role of two important virulence factors of L. pneumophila, the potent danger signal flagellin and t
149 ector SdeA is a member of the SidE family of L. pneumophila effector proteins.
150  that members of the SidE effector family of L. pneumophila ubiquitinate multiple Rab small GTPases a
151                             A key feature of L. pneumophila pathogenesis is the rapid influx of neutr
152                                   Fusions of L. pneumophila Icm/Dot-translocated substrates (IDTS) to
153 with iron to enhance intracellular growth of L. pneumophila DeltamavN strains, indicating a clear rol
154       Phytate reversibly abolished growth of L. pneumophila in broth, and growth inhibition was relie
155 etectable effects on intracellular growth of L. pneumophila within macrophages or amebae, the lack of
156 to support efficient intracellular growth of L. pneumophila.
157 monocytes via LILRA2 inhibited the growth of L. pneumophila.
158 two qPCR assays to evaluate the incidence of L. pneumophila Sg1.
159 yses indicate that environmental isolates of L. pneumophila have a potential positive selection for t
160                           Therefore, LamA of L. pneumophila is an amoebae host-adapted effector that
161 ) hospital plumbing contained high levels of L. pneumophila; c) Legionella control measures in hospit
162                                 A library of L. pneumophila effectors was screened using an expressio
163               The intracellular lifestyle of L. pneumophila within protozoa is considered to be a fun
164 or of neutrophil recruitment to the lungs of L. pneumophila-infected mice.
165 ectious mature intracellular forms (MIFs) of L. pneumophila are considered as infectious particles mo
166  can be easily adapted for the monitoring of L. pneumophila serogroups in clinical and environmental
167 lla pneumophila or a nonpathogenic mutant of L. pneumophila.
168 Genetic screening using flagellin mutants of L. pneumophila as a surrogate host, reveals a novel C. b
169    Here we show the aroB and aroE mutants of L. pneumophila to be defective in growth in human monocy
170           Here, we used flagellin mutants of L. pneumophila, which bypass the NAIP5/NLRC4-mediated re
171                          The large number of L. pneumophila effectors has been a limiting factor in a
172   This review aims to provide an overview of L. pneumophila pathogenesis in the context of the host i
173 th a role in the ecology and pathogenesis of L. pneumophila, HGA and HGA-melanin were effective at re
174 s not been characterized in the pathology of L. pneumophila TNFAIP2 messenger RNA and protein were up
175          Therefore, the shikimate pathway of L. pneumophila is differentially required for optimal gr
176 arly half of the taps showed the presence of L. pneumophila Sg1 in one sampling event, and 16% of tap
177               Intracellular proliferation of L. pneumophila in two evolutionarily distant hosts is fa
178 ient to power intracellular proliferation of L. pneumophila.
179       We now report that the Cas2 protein of L. pneumophila has both RNase and DNase activities, with
180 ntly, we determined that the Cas2 protein of L. pneumophila promotes intracellular infection of Acant
181 The bona fide F-box AnkB effector protein of L. pneumophila strain AA100/130b is anchored to the cyto
182 in different ways to the broad host range of L. pneumophila.
183  led to reduced intracellular replication of L. pneumophila Corby in A549 cells.
184 nsight into how intracellular replication of L. pneumophila is regulated by a metaeffector.
185 ot required for intracellular replication of L. pneumophila, RavD is a part of the molecular mechanis
186 s essential for intracellular replication of L. pneumophila.
187 t for efficient intracellular replication of L. pneumophila.
188        The higher disinfectant resistance of L. pneumophila released from untreated biofilms was pres
189  into a phase associated with restriction of L. pneumophila Furthermore, control of cyclin D1 may be
190 s the first step toward reducing the risk of L. pneumophila exposure and subsequent infections.
191          In a next step, clinical samples of L. pneumophila were analyzed using the FO-SPR sensor.
192  remediation of other significant sources of L. pneumophila.
193                           A single strain of L. pneumophila encodes a repertoire of over 300 differen
194 he vacuole was observed by using a strain of L. pneumophila in which all of the effector proteins in
195               Here, we developed a strain of L. pneumophila producing a fusion protein consisting of
196  its catalytic mutant form, into a strain of L. pneumophila that naturally lacks a CRISPR-Cas locus c
197 re conducted with a RavZ-deficient strain of L. pneumophila, L. monocytogenes was targeted by the hos
198 pneumophila subsp. pneumophila, 3 strains of L. pneumophila subsp. fraseri or L. pneumophila subsp. p
199 ionella species or subspecies: 15 strains of L. pneumophila subsp. pneumophila, 3 strains of L. pneum
200 Here, we determined the crystal structure of L. pneumophila RidL in complex with the human VPS29-VPS3
201  or phtD(+) alleles enhanced the survival of L. pneumophila thymidylate synthase (thyA)-deficient str
202                            DotL, the T4CP of L. pneumophila, contains an ATPase domain and a C-termin
203                      Upon further testing of L. pneumophila culture supernatants, we found that signi
204 genes of E. coli were replaced with those of L. pneumophila, motility was restored and DsbA2 was pres
205   This observation highlights the utility of L. pneumophila as a powerful tool for studying a critica
206                             The virulence of L. pneumophila depends on its Dot/Icm type IV secretion
207 its chaperone RABIF are required for optimal L. pneumophila replication and ER recruitment to the LCV
208  strains of L. pneumophila subsp. fraseri or L. pneumophila subsp. pascullei, 4 strains of "L. donald
209 cisella novicida, but unlike R. pickettii or L. pneumophila, Francisella species lack putrescine bios
210  domains are encoded in the genomes of other L. pneumophila isolates and species, suggesting that Ltp
211  able to detect L. pneumophila and the other L. pneumophila Sg1) were determined to be highly sensiti
212  spp. densities (likely including pathogenic L. pneumophila) were significantly higher in one type of
213               Only infection with pathogenic L. pneumophila resulted in ubiquitination of positive re
214 f genes characteristic of transmissive-phase L. pneumophila.
215  risks of pathogenic Legionella pneumophila (L. pneumophila), thus raising human health concerns.
216                               The preadhered L. pneumophila on selected rough and smooth biofilms wer
217 iminated culturable Legionella and prevented L. pneumophila from recolonizing biofilms, but M. avium
218 ose that PhtC and PhtD contribute to protect L. pneumophila from dTMP starvation during its intracell
219                     Indeed, lpg0273 protects L. pneumophila from toxic concentrations of nicotinic ac
220 and infectivity (to amoebae) of the released L. pneumophila were studied.
221 d by 16S rRNA qPCR) surrounding the released L. pneumophila.
222              In particular, when replicative L. pneumophila are treated with 5 mM nicotinic acid, the
223 ound a superior role of IgG2c in restricting L. pneumophila replication in a prophylactic setting.
224                                Here, we show L. pneumophila injects the effector LamA, an amylase, in
225                                        Since L. pneumophila cells are hardly culturable from these pe
226 enetic element profiles, suggesting a single L. pneumophila population as the source of nosocomial in
227 mimicked thymidine limitation or starvation, L. pneumophila exhibited a marked requirement for PhtC f
228 olving a major nosocomial-associated strain, L. pneumophila sequence type (ST) 1.
229 tion of the ER-derived vacuole that supports L. pneumophila replication.
230 Stat1, IFN-alphabeta unexpectedly suppresses L. pneumophila growth in both Stat1- and Stat2-deficient
231                    Thus, we demonstrate that L. pneumophila is able to inhibit the UPR by multiple me
232  tests with E. coli mutants established that L. pneumophila dsbA1, but not the dsbA2 strain, restored
233                           Here, we find that L. pneumophila can antagonize the growth of other Legion
234                                 We find that L. pneumophila sensitivity to HGA is density-dependent a
235                     Here, we have found that L. pneumophila induces monoubiquitination of the E2 enzy
236                    Previously, we found that L. pneumophila uses both a ferrisiderophore pathway and
237  lacking phtC or phtD strongly indicate that L. pneumophila requires PhtC and PhtD function under con
238                         Here, we report that L. pneumophila differentiation is also triggered by nico
239                          Here we report that L. pneumophila encodes an effector protein, named SidP,
240                          Here we report that L. pneumophila translocates the effector protein sphingo
241 Furthermore, biochemical studies reveal that L. pneumophila uses two effectors (Lgt1 and Lgt2) to inh
242           Our previous studies revealed that L. pneumophila encodes the effector kinase LegK7 which p
243                  In this study, we show that L. pneumophila avoids host S phase by blocking host DNA
244                               We showed that L. pneumophila-induced TNFAIP2 expression is dependent o
245                     Our studies suggest that L. pneumophila proteins interact with ER tubules at an e
246          Thus, the pulmonary cell types that L. pneumophila infects not only may act as an intracellu
247                                          The L. pneumophila DGR is found within a horizontally acquir
248                                          The L. pneumophila population has evolved to comprise 3 clon
249                                          The L. pneumophila-containing vacuole evades fusion with lys
250 ophila from these two types of biofilms, the L. pneumophila release kinetics values from predisinfect
251                                       In the L. pneumophila strain 130b, one mechanism used to acquir
252  paralog of LbtU, in iron acquisition in the L. pneumophila strain Philadelphia-1.
253                            Expression of the L. pneumophila DGR resulted in transfer of DNA sequence
254                 However, inactivation of the L. pneumophila released from predisinfected biofilms was
255 rs to the ubiquitin-decorated surface of the L. pneumophila-containing vacuole.
256 gether, our study provides evidence that the L. pneumophila effector GobX exploits two post-translati
257                       Here, we show that the L. pneumophila effector GobX possesses E3 ubiquitin liga
258                        Here we show that the L. pneumophila effector Lpg1137 is a serine protease tha
259                        Here we show that the L. pneumophila effector protein RavD targets host membra
260                 These data indicate that the L. pneumophila T2S system dampens the signaling of the T
261 is suggests a complex mechanism by which the L. pneumophila effector SidJ modulates the function of t
262 de novo assembly allowed comparison with the L. pneumophila Paris reference strain to infer phylogene
263  It remained unclear, however, whether these L. pneumophila proteins exhibit PLC activity.
264 nding interface on Rab5, explaining why this L. pneumophila effector can compete with cellular ligand
265                                        Thus, L. pneumophila can shut down ER-mitochondria communicati
266 htC and PhtD conferred a growth advantage to L. pneumophila thyA(+) strains.
267 large cohort of effectors that contribute to L. pneumophila virulence positively or negatively and ha
268 hat it is a virulence factor contributing to L. pneumophila infection in vivo.
269 ue and that colocalization came at a cost to L. pneumophila fitness.
270  estimated risks of illness from exposure to L. pneumophila and MAC via showering were generally low
271                            Human exposure to L. pneumophila Sg1 may occur from aerosolization and sub
272  signaling pathways previously not linked to L. pneumophila pathogenesis.
273 al initiator of the inflammatory response to L. pneumophila in vivo and point to an important role fo
274  and protein were upregulated in response to L. pneumophila infection of human-BDMs and human alveola
275 tral role in the transcriptional response to L. pneumophila METHODS: We infected human-blood-derived
276 o the production of IL-1alpha in response to L. pneumophila remain poorly defined.
277 phil recruitment to the lungs in response to L. pneumophila.
278 contrast, the Sec-7 domain was restricted to L. pneumophila and seven other species, indicating effec
279  ability to direct an innate response toward L. pneumophila.
280 host immunity and reveal a T4SS-translocated L. pneumophila phytase that counteracts intracellular ba
281  an essential carbon source for transmissive L. pneumophila.
282     Interference with AnkB function triggers L. pneumophila to exhibit a starvation response and diff
283 e able to promote Fe(3+) uptake by wild-type L. pneumophila as well as enhance growth of iron-starved
284        Furthermore, in contrast to wild-type L. pneumophila, a DeltasdhA mutant caused a transient de
285                                Unexpectedly, L. pneumophila can itself be inhibited by HGA secreted f
286 4SS-translocated effectors and harbor viable L. pneumophila during pulmonary infection of mice.
287 utrophil recruitment in response to virulent L. pneumophila requires the production of IL-1alpha spec
288 IL-1alpha production in response to virulent L. pneumophila.
289 ria to be inhaled into the human lung, where L. pneumophila can be phagocytosed by alveolar macrophag
290 us, there are at least two pathways by which L. pneumophila can disrupt xenophagic targeting of the v
291                Here, the mechanisms by which L. pneumophila can prevent the host xenophagy pathway fr
292 ranes for biogenesis of the vacuole in which L. pneumophila replicates, these studies have revealed t
293                                        While L. pneumophila-induced mortality in IL-36alpha- or IL-36
294 flexneri intercepts all host pyruvate, while L. pneumophila induces host protein degradation and bloc
295  human-blood-derived macrophages (BDMs) with L. pneumophila and used chromatin immunoprecipitation fo
296 se 13C-prelabeled amoebae were infected with L. pneumophila wild type or some mutants defective in pu
297                 When mice were infected with L. pneumophila, immunoglobulins were cleaved and recogni
298         We show that, in cells infected with L. pneumophila, SidJ mediates the glutamylation of SidE
299          Infection of mouse macrophages with L. pneumophila wild type, plaB knock-out mutant, and pla
300 evealed that hospitals have been seeded with L. pneumophila via both local and international spread o

 
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