ライフサイエンスコーパス: LDH

1 LDH and MTT assays demonstrate that peptide 1a is toxic

2 LDH in the mitochondrial matrix is not compatible with t

3 LDH inhibition combined with IL-21 increased the formati

4 LDH inhibition did not significantly affect IL-21-induce

5 LDH inhibition rewired IL-2-induced effects, promoting p

6 LDH inhibitors affected Ewing sarcoma cell viability bot

7 LDH patients have increased BL firing rate and insuffici

8 LDH was the marker with the strongest prognostic value,

9 LDH, which is a non-limiting enzyme of glycolysis in dif

10 LDH/PK and colorimetric enzymatic assays revealed two pr

11 LDHs have found widespread application as catalysts, ani

12 This study enrolled 17 LDH patients and 17 sex- and age-matched healthy individ

13 acrophage-expressed lactate dehydrogenase-A (LDH-A) to tumor formation in a K-Ras murine model of lun

14 ctate by the enzyme lactate dehydrogenase-A (LDH-A), suggesting a possible vulnerability at this targ

15 HPS and TMAO did not affect LDH protein structure.

16 ared by in-situ growth of ZIF-8 on the Zn-Al LDH surface.

17 In this research, nanoporous Zn-Al LDH/ZIF-8 composite was prepared by in-situ growth of ZI

18 ls of hepatobiliary injury markers AST, ALT, LDH, and gamma-GT after 6 hours of NMP.

19 PP) was significantly enhanced by adding AMO-LDH-OCNT hybrids.

20 egree of dispersion (mixability) between AMO-LDH and OCNT has a significant effect on the flame retar

21 3)0.5.yH2O LDH-oxidized carbon nanotube (AMO-LDH-OCNT) hybrids are shown to perform better than the e

22 Further, a system with 10 wt% of AMO-LDH and 1 wt% OCNT showed a peak heat release rate (PHRR

23 In addition, the incorporation of AMO-LDH-OCNT hybrids led to better mechanical properties, su

24 ), than those composites containing only AMO-LDH (25.6 MPa and 7.5%, respectively).

25 perform better than the equivalent pure AMO-LDH.

26 A synergistic effect between the AMO-LDH and OCNT was observed; this endows the hybrid materi

27 For PP mixed with AMO-LDH-OCNT hybrids to produce a composite with 10 wt% LDH

28 r than the PHRR reduction with PP/20 wt% AMO-LDH (31%).

29 cible organic layered double hydroxides (AMO-LDHs) can act as organophilic inorganic flame retardant

30 controlled their fate through the AMPKalpha1/LDH pathway, emphasizing the importance of metabolism in

31 ncentrations of total protein, AST, ALT, and LDH than the CN group.

32 ncentrations of total protein, AST, ALT, and LDH, decreased salivary flow rate and a significant adve

33 ts with CTCs >/= 5 cells/7.5 mL of blood and LDH > 250 U/L (high risk) at 12 weeks was 46% and 2%, re

34 l-2-yl)-2,5-diphenyltetrazolium bromide) and LDH (lactate dehydrogenase) assays performed with the de

35 at proliferating cells rely on both MDH1 and LDH to replenish cytosolic NAD, and that therapies desig

36 0.25 mg/ml extract as measured with MTT and LDH-leakage assays.

37 Both cellular viability and LDH level remained unchanged with up to 100 microg/mL of

38 The composite AO/LDH reaches the highest fluorescence intensity when the

39 nce intensity was achieved with a pulp to AO/LDH ratio of 1:5 which can be used to detect Hg(2+) in w

40 Lactate dehydrogenase assay (LDH), electronic microscopy analysis, collagen I product

41 Targeting HER2 or Src attenuated LDH activity as well as invasive potential in head and n

42 is review we summarize the current available LDH exfoliation methods.

43 eved partial remission had a normal baseline LDH.

44 rrelation test showed no correlation between LDH and D-dimer levels.

45 ntly, Oshima et al. developed a bioavailable LDH inhibitor that decreases tumor growth in mice and fu

46 hnology was validated using five biomarkers; LDH, CRP, PSA, MMP-7, and C3a.

47 t with this possibility, larvae lacking both LDH and G3P dehydrogenase (GPDH1) exhibit growth defects

48 multifidus muscles with or without LDH, but LDH accelerates this process rather than being a result

49 Cell death was assayed by LDH and MTT methods.

50 plex interplay of anion uptake mechanisms by LDH phases, by using changes in Mo geometry as powerful

51 ucose, pyruvate was metabolized primarily by LDH to generate lactate and NAD(+) and by SpxB and PDHc

52 Cell death was proven by LDH assay and cell viability by IL-8 ELISA.

53 over, an anion exchange process on both CaAl LDHs was followed by in situ time-resolved synchrotron-b

54 bdate (MoO4(2-)) sorption mechanisms on CaAl LDHs with increasing loadings of molybdate.

55 the circulatory system and protects cardiac LDH exposed to HPS produced by the contracting heart.

56 aluminum layered double hydroxide chloride (LDH), is synthesized and characterized with X-ray powder

57 Compared to pristine CoFe LDHs, the as-exfoliated ultrathin CoFe LDHs nanosheets e

58 CoFe LDHs, the as-exfoliated ultrathin CoFe LDHs nanosheets exhibit excellent catalytic activity for

59 rst time, this study prepares ultrathin CoFe LDHs nanosheets with multivacancies as OER electrocataly

60 flow cytometry method (FCM), a colorimetric LDH-based ELISA : DELI), and standard microscopic slide

61 as achieved in 4T1 breast cancer with (64)Cu-LDH-BSA via passive targeting alone (7.7 +/- 0.1%ID/g at

62 ide (LDH) photocatalysts, in particular CuCr-LDH nanosheets, possess remarkable photocatalytic activi

63 daily monitoring of changes in cytotoxicity (LDH), energy metabolism (glucose), and liver function (t

64 d-Lactate dehydrogenase (d-LDH) is encoded by a single gene in Arabidopsis (Arabido

65 In addition, overexpression of d-LDH and CYTc increased tolerance to d-lactate and MGO To

66 icture of the organization and function of d-LDH in the plant cell and exemplify how the plant mitoch

67 and MGO Together with fine-localization of d-LDH, the functional interaction with CYTc in vivo strong

68 c acts as the in vivo electron acceptor of d-LDH.

69 rometry allowed us to definitely show that d-LDH acts specifically on d-lactate, is active as a dimer

70 c loss-of-function mutants, as well as the d-LDH mutants, were more sensitive to d-lactate and MGO, i

71 cent mice in hyperoxia by 24 h and decreased LDH release and lung cell apoptosis after 72 h of exposu

72 lar protein (48%) and lactate dehydrogenase (LDH) (68%) following 72 hours of hyperoxia.

73 (P = .0040); elevated lactate dehydrogenase (LDH) (P < .0001); and increased procalcitonin (P < .0001

74 Lactate dehydrogenase (LDH) accounts for the fermentative component of aerobic

75 ability, cell number, lactate dehydrogenase (LDH) activity, cell necrosis, transepithelial electrical

76 d by protein leak and lactate dehydrogenase (LDH) activity.

77 ydrate metabolism and lactate dehydrogenase (LDH) activity.

78 inotransferase (AST), lactate dehydrogenase (LDH) and creatine kinase (CK), which cardiac troponins b

79 Increased levels of lactate dehydrogenase (LDH) and decreased levels of succinate dehydrogenase (SD

80 is, robustly inducing lactate dehydrogenase (LDH) and lactate production, whereas IL-21 maintained a

81 ude to increase serum lactate dehydrogenase (LDH) and was oxidative in nature as shown by increased e

82 (HRP2), aldolase, and lactate dehydrogenase (LDH) antigens were analyzed by Bayesian latent class mod

83 alpha (HIF1alpha) and Lactate dehydrogenase (LDH) are required for macrophage activation, their bacte

84 We identified lactate dehydrogenase (LDH) as a new functional target of AMPKalpha1.

85 scence microscopy and Lactate dehydrogenase (LDH) assay on the supernatant.

86 viability, autophagy, lactate dehydrogenase (LDH) assay, and mammalian target of rapamycin (mTOR) pat

87 oti lacks a canonical lactate dehydrogenase (LDH) but instead expresses a unique enzyme, B. microti L

88 drives expression of lactate dehydrogenase (LDH) but not hexokinase 2 (HK-II).

89 Lactate dehydrogenase (LDH) catalyzes the conversion of pyruvate to lactate, wi

90 Patients with normal lactate dehydrogenase (LDH) concentration and fewer than three organ sites cont

91 The release of lactate dehydrogenase (LDH) during brain death was reduced in the NOD group.

92 osed that human heart lactate dehydrogenase (LDH) employs protein promoting vibrations (PPVs) on the

93 ria-conserved antigen lactate dehydrogenase (LDH) for P. vivax and other malaria species.

94 ON) were modified for lactate dehydrogenase (LDH) immobilization using (3-aminopropyl)triethoxysilane

95 y: cell viability and lactate dehydrogenase (LDH) leakage.

96 lymphodepletion serum lactate dehydrogenase (LDH) level and a favorable cytokine profile, defined as

97 l using CTC count and lactate dehydrogenase (LDH) level was shown to satisfy the four Prentice criter

98 active protein level, lactate dehydrogenase (LDH) level, ferritin level, d-dimer level, neutrophil co

99 h the products of the lactate dehydrogenase (LDH) reaction and the intermediates of the malate-aspart

100 bromide (MTT) assay, lactate dehydrogenase (LDH) release assay, Hoechst 33342 staining, annexin V/PI

101 ide (MTT) assay and a lactate dehydrogenase (LDH) release assay, respectively.

102 r, and 8i in lowering lactate dehydrogenase (LDH) release induced by ischemia-like conditions in rat

103 ion of caspase-3, and lactate dehydrogenase (LDH) release.

104 urification and serum lactate dehydrogenase (LDH) to evaluate their prognostic value in newly diagnos

105 t the inactivation of lactate dehydrogenase (LDH) under heat, dehydration-rehydration and freeze-thaw

106 rogenase (GAPDH) to L-lactate Dehydrogenase (LDH) using enzymes from different cells.

107 ide (RGO-AuNPs) and l-lactate dehydrogenase (LDH) was developed for the sensing of l-lactate.

108 Serum containing lactate dehydrogenase (LDH) was directly spotted on to the pullulan-coated bioa

109 ect proteins, such as lactate dehydrogenase (LDH), against hydrostatic pressure stress (HPS).

110 ecific antigen (PSA), lactate dehydrogenase (LDH), and CgA at baseline and in follow-up of PSMA RLT.

111 inotransferase (AST), lactate dehydrogenase (LDH), and thiobarbituric acid reactive substance (TBARS)

112 rum albumin, elevated lactate dehydrogenase (LDH), high Creactive protein (CRP) and chest radiographi

113 ver, while release of lactate dehydrogenase (LDH), HMGB1, and IL-1beta occurred without rupture, rupt

114 o-unstable (EF-Tu), l-lactate dehydrogenase (LDH), protein D (PD), and peptidoglycan-associated lipop

115 the glycolytic enzyme lactate dehydrogenase (LDH), which is overexpressed and plays a critical role i

116 total bile acids, and lactate dehydrogenase (LDH).

117 al glycolytic enzyme, lactate dehydrogenase (LDH).

118 d inhibitors of human lactate dehydrogenase (LDH).

119 Lactate dehydrogenases (LDHs) are tetrameric enzymes of major significance in ca

120 y first-in-class inhibitors that demonstrate LDH inhibition in vivo.

121 Herein, we demonstrate LDH detection using porous silicon (pSi) microcavities a

122 on domain, thus disrupting and destabilizing LDH tetramers.

123 so, we designed a protein model of a dimeric LDH-H.

124 rall, our study demonstrates that disrupting LDH oligomerization state by targeting their tetrameriza

125 NiFe-LDH due to (i) amorphous and distorted LDH structure, (ii) enhanced active surface area, and (i

126 To explore the potential role of Drosophila LDH in promoting biosynthesis, we examined how Ldh mutat

127 Elevated LDH implied a reduced chance for partial remission, with

128 sed leukocyte count (P = .0005) and elevated LDH (P < .0001).

129 the strongest prognostic value, and elevated LDH increased the risk for progression of disease under

130 ARDS was predicted by elevated LDH (P < .0001), while mortality was predicted by increa

131 work provides a novel strategy to exfoliate LDHs and to produce multivacancies simultaneously as hig

132 The ability to exfoliate LDHs into ultrathin nanosheets enables a range of new op

133 In functional experiments, LDH overexpression phenocopied AMPKalpha1(-/-) phenotype

134 rathin Ni-Fe layered-double-hydroxide (Ni-Fe LDH) nanosheets or porous Ni-Fe oxides (NiFeO(x) ) assem

135 cious pCNT@Fe@GL/CNF ORR electrode and Ni-Fe LDH/NiF oxygen evolution reaction (OER) electrode exhibi

136 The remarkable activity of the Ni-Fe LDH@NiCu and NiFeO(x) @NiCu is further demonstrated by a

137 In alkaline solution, the as-prepared Ni-Fe LDH@NiCu possesses outstanding OER activity, achieving a

138 On the other hand, the spent Cu/Fe-LDH could be employed to produce porous carbon materials

139 spent Cu/Fe layered double hydroxide (Cu/Fe-LDH) which is generated from the adsorption of dyes by c

140 pacity (+25% for cell viability and +30% for LDH leakage) were observed in grape juices following PEF

141 e metals solution herein could be reused for LDH synthesis.

142 These findings indicate a pivotal role for LDH in modulating cytokine-mediated T cell differentiati

143 The biosensor was selective for LDH and did not produce a signal upon incubation with an

144 of two enzyme pathways (G6pDH-MDH and G6pDH-LDH) through the control of NAD(+) substrate channeling

145 Heavy LDH has slowed chemistry in single turnover experiments,

146 prepared the native (light) LDH and a heavy LDH labeled with (13)C, (15)N, and nonexchangeable (2)H

147 ier-crossing probability is reduced in heavy LDH, the concerted mechanism of the hydride-proton trans

148 and proton transfer for both light and heavy LDHs.

149 pattern maintained with both light and heavy LDHs.

150 hogenesis of pain in lumbar disc herniation (LDH) remains poorly understood.

151 cle in patients with lumbar disc herniation (LDH).

152 ant differences between B. microti and human LDH enzymes and suggest that BmLDH could be a suitable t

153 ng wild-type and mutant B. microti and human LDHs identified Arg99 as a critical residue for the cata

154 Hydrotalcite (LDH) is one of the most excellent carrier materials.

155 toxic, degradable, layered double hydroxide (LDH) clay nanosheets.

156 n rust, that is, a layered double hydroxide (LDH) containing iron (Fe).

157 Layered double hydroxide (LDH) nanomaterial has emerged as a novel delivery agent

158 ratio 2D non-toxic layered double hydroxide (LDH) nanosheet dispersions using a non-toxic exfoliation

159 ary Fe(II)-Al(III)-layered double hydroxide (LDH) phases during reaction with the Al-oxide sorbent, w

160 Layered double hydroxide (LDH)-based nanomaterials are considered as promising ele

161 tes that ultrathin layered-double-hydroxide (LDH) photocatalysts, in particular CuCr-LDH nanosheets,

162 ized in Mg2Al-NO3 Layered Double Hydroxides (LDH) and the electrochemical detection was achieved with

163 Fe and CoFe (MFe) layered double hydroxides (LDHs) are among the most active electrocatalysts for the

164 Iron-doped nickel layered double hydroxides (LDHs) are among the most active heterogeneous water oxid

165 on the surface of layered double hydroxides (LDHs) for preparation of porous nanocomposites is a favo

166 Layered double hydroxides (LDHs) have been considered as effective phases for the r

167 wastewater using layered double hydroxides (LDHs) through their formation is presented.

168 Layered double hydroxides (LDHs) with similar compositions to the minerals shigaite

169 Layered double hydroxides (LDHs) with their highly flexible and tunable chemical co

170 including lactate sensors using immobilized LDH on the ISFET surface.

171 Dysfunction in LDH patients was associated with immoderate intermuscula

172 ficantly attenuated pain hypersensitivity in LDH rats.

173 ed (~1.3 fold) and a significant increase in LDH release at 24 hrs.

174 co-contraction of the TA-LG was increased in LDH patients than in the control group (p < 0.05).

175 and the Oswestry disability index scores in LDH patients.

176 shrinkage, F-actin alterations or increased LDH activity); we hypothesize that oxidative modificatio

177 hemolysis, thrombophilia, and inflammation (LDH, bilirubin, D-dimer, C-reactive protein [CRP]) impro

178 rapeutic potential of transiently inhibiting LDH during adoptive T cell-based immunotherapy, with an

179 sed cell viability, aggravated intracellular LDH release, intracellular Ca(2+), ROS levels, apoptosis

180 nts for the direct synthesis of single-layer LDH nanosheets, as well as the emerging applications of

181 We prepared the native (light) LDH and a heavy LDH labeled with (13)C, (15)N, and nonex

182 and inhibited cell injury indicated by lower LDH activity in the media.

183 decrease in lactate dehydrogenase isoform M (LDH-M) activity and an increase in cellular protection a

184 ation of HRP2, P. vivax LDH, and all-malaria LDH (pan LDH) was developed to accurately measure circul

185 d circulating levels of muscle damage marker LDH.

186 ression of ALDH1 in lung metastasis and MDR1/LDH-A expression in liver metastasis compared to human p

187 nting for the high catalytic activity of MFe LDHs.

188 e that, under applied anodic potentials, MFe LDHs oxidize from as-prepared alpha-phases to activated

189 nstead expresses a unique enzyme, B. microti LDH (BmLDH), acquired through evolution by horizontal tr

190 localization of ssDNA-FITC suggest that nano-LDHs have potential application as a novel gene carrier

191 layered double hydroxide lactate nanosheets (LDH-lactate-NS) with a 0.52 nm thickness and 3060 nm dia

192 r I(FITC) and DNA molecules, forming neutral LDH-nanosheet conjugates.

193 ckel-aluminum double layered hydroxide (NiAl-LDH) nanoplates on carbon nanotubes (CNTs) network.

194 t results in fully integrated amorphous NiFe-LDH/C nanohybrid, allowing the harness of the high intri

195 The confined growth of both NiFe-LDH and carbon in one single sheet results in fully inte

196 morphous NiFe-layered double hydroxide (NiFe-LDH) (<5 nm) and nanocarbon using the molecular precurso

197 rness of the high intrinsic activity of NiFe-LDH due to (i) amorphous and distorted LDH structure, (i

198 During the solvothermal synthesis of NiFe-LDH, the organic ligand decomposes and transforms to amo

199 As such, the resultant NiFe-LDH/C exhibits superior activity and stability.

200 et count < 50 x 109/L, albumin below normal, LDH above normal at time of admission), and physician-re

201 Intravenous administration of LDH inhibitors resulted in the greatest intratumoral dru

202 ets, as well as the emerging applications of LDH nanosheets in catalyzing oxygen evolution reactions

203 Here, we used a novel class of LDH inhibitors to demonstrate, for the first time, that

204 We provide evidence for the degradation of LDH, dsRNA uptake in plant cells and silencing of homolo

205 Myeloid-specific deletion of LDH-A promoted accumulation of macrophages with a CD86(h

206 d expression of NaV1.7 and NaV1.8 in DRGs of LDH rats.

207 nanticipated cooperative antitumor effect of LDH inhibition and IL-21.

208 In vitro, exposure of LDH to HPS with or without TMAO did not affect protein s

209 hat exhibit oncogene-dependent expression of LDH and increased glycolysis.

210 Our results suggest that expressions of LDH-A and lactate by macrophage in the tumor microenviro

211 c self-assembly process for the formation of LDH/C composite, this method offers one new opportunity

212 Herein we identified unspliced forms of LDH and ENO transcripts produced during transition betwe

213 for the de novo design and identification of LDH tetramerization disruptors.

214 Inhibition of LDH activity by small hairpin ribonucleic acid or expres

215 ir return to quiescence, while inhibition of LDH activity rescued AMPKalpha1(-/-) MuSC self-renewal.

216 otent enzymatic and cell-based inhibition of LDH enzymatic activity.

217 t targeting glycolysis through inhibition of LDH should be further investigated as a potential therap

218 is, phenocopying pharmacologic inhibition of LDH.

219 s are exquisitely sensitive to inhibition of LDH.

220 Thus, potent, selective inhibitors of LDH represent an attractive therapeutic approach.

221 nce of anion adsorption in the interlayer of LDH has often been highlighted, but we are still missing

222 tudy is to explore chelator-free labeling of LDH nanoparticles with radioisotopes for in vivo PET ima

223 -droplet assays revealed increased levels of LDH as well as reduction in bile acid synthesis-results

224 icity as demonstrated by increased levels of LDH, in parallel to the presence of numerous vacuoles in

225 effects to resolve the chemical mechanism of LDH and establish the coupling of fs-ps protein dynamics

226 macrocycles, along with the dimeric model of LDH-H, constitute promising pharmacological tools for th

227 VGSCs in a previously validated rat model of LDH.

228 G) neurons were sensitized in a rat model of LDH.

229 anges can therefore be detected using MRI of LDH-catalyzed hyperpolarized (13)C label exchange betwee

230 Complete recovery of LDH, CRP, and PSA levels was achieved post-rehydration w

231 ions were utilized to construct the base of LDHs in an alkaline solution.

232 nly exhibits the alternative exploitation of LDHs, but also provides new insights into the removal of

233 ion of AY25 molecules into the interlayer of LDHs during their structural arrangement, where Mg(2+) a

234 However, the stacking structure of LDHs limits the exposure of the active sites.

235 ificantly, a single spray of dsRNA loaded on LDH (BioClay) afforded virus protection for at least 20

236 Once loaded on LDH, the dsRNA does not wash off, shows sustained releas

237 ic field between the NAD(H) binding sites on LDH and GAPDH tetramers can merge in the LDH-GAPDH compl

238 Among patients with aldolase or LDH levels detectable with a bead-based assay, the conce

239 viability was determined by MTT reduction or LDH release assays.

240 areas under the curve achieved with S100 or LDH markers.

241 ly 186 mg g(-1), comparable to that of other LDH-based methods.

242 Our LDH/RGO-AuNPs/SPCE based biosensor thus performs as elec

243 At thresholds for HRP2, pan LDH, and P. vivax LDH of 2.3 pg/ml, 47.8 pg/ml, and 75.1

244 ssay lower limits of detection for HRP2, pan LDH, P. vivax LDH, and CRP were 0.2 pg/ml, 9.3 pg/ml, 1.

245 HRP2, P. vivax LDH, and all-malaria LDH (pan LDH) was developed to accurately measure circulating ant

246 (-/-) BMDMs showed reduced MSU phagocytosis, LDH release, IL-1beta expression and production compared

247 the heart allograft recipients, lower plasma LDH levels were observed.

248 Plasmodium LDH (pLDH) concentration, in contrast to that of HRP2, c

249 d low temperature incubation did not prevent LDH-lactate-NS internalization, suggesting that LDH-lact

250 iO(2) ratio, neutrophil-to-lymphocyte ratio, LDH level, IL-6 level, and age was developed and showed

251 hours OGD (0.3% O(2)) significantly reduced LDH release and increased MTT activity compared to vehic

252 oncogenic driver of Ewing sarcoma, regulated LDH A (LDHA) expression.

253 sts in the spleen and bone marrow, and serum LDH level, consistent with ineffective erythropoiesis.

254 risk patients with pre-lymphodepletion serum LDH levels above normal, a favorable cytokine profile af

255 ted to the high Grade, pathological T stage, LDH, and NLR.

256 ysis time threefold compared to the standard LDH assay in solution.

257 supporting the concept of targeting stromal LDH-A as an effective strategy to blunt tumoral immune e

258 In this work, we conceived to target LDH tetramerization sites with the ambition of disruptin

259 The present work demonstrates that LDH is an effective sorbent for selective extraction of

260 tants grow at a normal rate, indicating that LDH is dispensable for larval biomass production.

261 Enzyme kinetics studies showed that LDH activity with free NADH and GAPDH-NADH complex alway

262 tein-protein interaction studies showed that LDH and GAPDH can form a leaky channeling complex only a

263 However, we suggest that LDH isoenzymes should be studied in further research.

264 -lactate-NS internalization, suggesting that LDH-lactate-NS penetrated the plasma membrane via non-en

265 The LDH patients showed enhanced BL RMS during the single su

266 nd the erector spina is more affected by the LDH process.

267 on the overlap between the off-rates for the LDH-(GAPDH-NADH) complex and the GAPDH-NADH complex.

268 rted structure and compressive strain in the LDH nanosheets, which significantly enhances N2 chemisor

269 on LDH and GAPDH tetramers can merge in the LDH-GAPDH complex.

270 nd APTES-GA treatments successfully link the LDH molecule to those surfaces while retaining its activ

271 The turnover frequency (TOF) of the LDH-Au/CNTs COE catalyst was much higher than the previo

272 e orange (AO) was successfully loaded on the LDH layers, which significantly inhibited fluorescence q

273 th changes in adsorbed amount of dyes on the LDH.

274 d derivatives that specifically targeted the LDH tetramerization sites.

275 a macrocyclic peptide that competes with the LDH tetramerization domain, thus disrupting and destabil

276 NAD(H)-channeling within the LDH-GAPDH complex can be an extension of NAD(H)-channeli

277 hemical detection was achieved with the TKec/LDH modified glassy carbon electrode (GCE).

278 The TKec/LDH/GCE biosensor was optimized using the best TK donor

279 dehydrogenase 1 (MDH1) is an alternative to LDH as a supplier of NAD.

280 y in glycolysis reduces the carbon supply to LDH.

281 sites is achievable and paves the way toward LDH inhibition through this novel molecular mechanism.

282 In the case of a transient LDH-(GAPDH-NADH) complex, the relative contribution from

283 with antitumor effector function, transient LDH inhibition enhanced the generation of memory cells c

284 esion and penetration protein [Hap]), EF-Tu, LDH, PD, and P6 exhibited interactions with laminin, and

285 ultivacancies in the as-exfoliated ultrathin LDHs nanosheets.

286 In vitro, LDH with or without TMAO was exposed to HPS and was eval

287 t thresholds for HRP2, pan LDH, and P. vivax LDH of 2.3 pg/ml, 47.8 pg/ml, and 75.1 pg/ml, respective

288 say for the quantification of HRP2, P. vivax LDH, and all-malaria LDH (pan LDH) was developed to accu

289 its of detection for HRP2, pan LDH, P. vivax LDH, and CRP were 0.2 pg/ml, 9.3 pg/ml, 1.5 pg/ml, and 2

290 s for further studying the effect of in vivo LDH inhibition.

291 urvival (75% [70-81]), whereas patients with LDH concentration at least two times the upper limit of

292 In patients with LDH, fatty degeneration in the erector spina is more pro

293 egy for chronic pain relief in patients with LDH.

294 ES modified surfaces can directly react with LDH via physical attachment.

295 spina and multifidus muscles with or without LDH, but LDH accelerates this process rather than being

296 T hybrids to produce a composite with 10 wt% LDH and 2 wt% OCNT, the 50% weight loss temperature was

297 contribution, AMO [Mg3Al(OH)8](CO3)0.5.yH2O LDH-oxidized carbon nanotube (AMO-LDH-OCNT) hybrids are

298 istinct Fe(III) sites (S = 5/2) in [Fe:ZnAl]-LDH.

299 We observed a similar effect in [Ni:ZnAl]-LDH materials; notably, Ni(II) (S = 1) atoms displayed a

300 kel atoms were doped into nonmagnetic [ZnAl]-LDH materials and the coordination environments of the i