ライフサイエンスコーパス: LVI

1 high grade without lymphovascular invasion (LVI).

2 cular spaces termed lymphovascular invasion (LVI).

3 ing microvial insert large volume injection (LVI).

4 re widely negative at > 5 mm and there is no LVI.

5 sis, all factors remained significant except LVI.

6 breast cancer who have grade 3 tumors and/or LVI.

7 et)) levels of spontaneous metastasis but no LVI.

8 53 expression while 26.6% showed evidence of LVI.

9 rade Group, Nuclear Grade, and more frequent LVI.

10 in the presence of lymphovascular invasion (LVI; 0.25 +/- 0.02 v 0.17 +/- 0.02; P < .0001) or lymph

11 gic features (nodal or margin involvement or LVI), adjuvant chemotherapy followed by radiation provid

12 75 mm, MR >/= 1, presence of ulceration, and LVI (all P = .001) were significantly associated with se

13 n T1 CRC, including validation, showing that LVI and perineural invasion, mucinous subtype, and low a

14 Our findings suggest including LVI and regression as new prognostic factors in the mela

15 carcinoma (IC), or lymphovascular invasion (LVI), and 8% lobular neoplasia (lobular carcinoma in sit

16 Lung weight index (LWI), lung volume index (LVI), and alveolar cell proliferation index (CPI) were m

17 ration, blood and lymphatic vessel invasion (LVI), and lymph node and lung metastasis.

18 grade, presence of lymphovascular invasion (LVI), and receipt of chemotherapy and/or radiation.

19 e (MR), ulceration, lymphovascular invasion (LVI), and regression; incidence was lower and subgroup d

20 ral invasion (PNI), lymphovascular invasion (LVI), and residual positive margins (R1) were found in 2

21 BE]), nodal status, lymphovascular invasion (LVI), and the presence of multifocal neoplasia (MFN) (hi

22 s regarding the role of submucosal invasion, LVI, and age.

23 in further significant enhancements of LWI, LVI, and CPI in group PK.

24 As expected, LWI, LVI, and CPI were significantly increased after pneumone

25 in adult rats as indicated by increased LWI, LVI, and CPI.

26 The combined high incidence of MFN, LVI, and occult nodal metastases does not support the us

27 On adjusted analysis, PNI, LVI, and poorly differentiated tumors were independently

28 R >/= 1, presence of ulceration, presence of LVI, and regression >/= 50%.

29 breast tumors is essential for BVI, but not LVI, and that MT1-MMP should be further explored as a pr

30 th postcolumn infusions) and compared across LVI- and SPE-based methods at constant (high and low) an

31 nts with associated lymphovascular invasion (LVI) are at increased risk of occult metastases.

32 esults from this study demonstrated that the LVI-based method produced analytical signals of quality

33 ogic feature (nodal or margin involvement or LVI) chemotherapy with subsequent radiation provided the

34 ted responses of interneurons in LII-III and LVI completely desensitized, while cholinergic responses

35 All 9 patients with LVI had T1b tumors.

36 atrixes using direct large-volume injection (LVI) high-performance liquid chromatography (HPLC) tande

37 rs are younger age, lymphovascular invasion (LVI), high Ki-67, and larger tumors within the T1a,b sub

38 versely, there was a significant decrease in LVI in re-stented segments B and C (P<0.05).

39 Prevalence of LVI increased with higher pathologic stage (9.0%, 23%, 6

40 p53 expression of > 5% and LVI, individually and jointly, are associated with poore

41 ma with 80% MMI and lymphovascular invasion (LVI) involving the outer half of the cervical stroma ( F

42 LVI is an independent predictor of recurrence and decrea

43 crete sequence of three amino acid residues (LVI), located at the carboxyl-terminal end of the impair

44 LVI maintained its independent predictor status in compe

45 Patients with high-grade tumors and/or LVI may have 10-year RFS rates of less than 75% in the a

46 status, presence of lymphovascular invasion (LVI), number of SLN(s) identified, number of positive SL

47 s similar in magnitude to that observed with LVI, or by ER/PR negativity.

48 to sham-operated animals did not alter LWI, LVI, or CPI.

49 P expression in lymphatic endothelial cells, LVI, or lymph node metastasis.

50 ith T1a and 25 of 45 (58%) with T1b had MFN, LVI, or nodal metastases.

51 LVI (P < 0.001), perineural invasion (P < 0.001), mucino

52 de of both DCIS (P = .002) and IC (P = .03), LVI (P = .03), and lymph node involvement (P = .02) by u

53 tension (P < 0.001), tumor size (P = 0.001), LVI (P = 0.019), and histology (P = 0.034).

54 Furthermore, LVI presented a clear advantage over SPE because it was

55 cholinergic responses in LV interneurons and LVI pyramidal cells showed less desensitization.

56 ized by exaggerated lymphovascular invasion (LVI), recapitulated in our human xenograft, MARY-X.

57 Age, MR, ulceration, LVI, regression, and sentinel node status were independe

58 nodal involvement, lymphovascular invasion (LVI), stage, lymph node ratio, not receiving chemotherap

59 with transitional-cell carcinoma (n = 776), LVI status was available for 750.

60 thogenic TP53 alterations and, together with LVI status, could enhance early prognostication of prost

61 sis (DM) as a function of p53 expression and LVI status.

62 combining the 4,871 and 8,596 Da peaks with LVI, the area under the receiver operating characteristi

63 e, and number of pelvic lymph nodes removed, LVI was an independent predictor of local (HR = 2.03, P

64 LVI was an independent predictor of overall (HR = 1.84,

65 Without lipid removal, LVI was counterproductive because method sensitivity suf

66 LVI was defined as the presence of tumor cells within an

67 In patients treated with (192)Ir radiation, LVI was maintained from baseline to follow-up only in no

68 LVI was not a predictor of recurrence or survival in nod

69 LVI was performed by the direct injection of 900 muL of

70 LVI was present in 36.4% (273 of 750) overall, involving

71 ed segment A (P<0.05).At follow-up, however, LVI was similar in all 4 segments secondary to the incre

72 onmental analysis by large-volume injection (LVI) was compared to solid-phase extraction (SPE) based

73 tients, postintervention lumen volume index (LVI) was significantly greater in re-stented segments B

74 Lymphovascular invasion (LVI) was the only clinicopathologic factor predictive of

75 Serum DNA integrity and LVI were significant for predicting LN metastasis in a m

76 tein expression and lymphovascular invasion (LVI) were evaluated for predicting metastatic progressio