ライフサイエンスコーパス: Langerhans' cell

1 of skin langerin-expressing cells (including Langerhans cells).

2 e, overproduction of IFNs, and an absence of Langerhans cells.

3 diated expression of interleukin-6 (IL-6) by Langerhans cells.

4 s (LCH) represents a clonal proliferation of Langerhans cells.

5 facilitate the interaction of M. leprae with Langerhans cells.

6 The latter also included CD1a(bright) Langerhans cells.

7 ts internalization in specific organelles of Langerhans cells.

8 of VIP on TLR regulation in macrophages and Langerhans cells.

9 kin dendritic cell subpopulations, including Langerhans cells.

10 elocalization toward the basal epidermis and Langerhans cells.

11 se T cells recognize CD1a-lipid complexes on Langerhans cells.

12 nterleukin 22 (IL-22) in response to CD1a on Langerhans cells.

13 1c(+)CD1a(+) dermal DCs but not to epidermal Langerhans cells.

14 mice induced in situ maturation of epidermal Langerhans cells.

15 conventional DC subsets and not by epidermal Langerhans cells.

16 s the only other cytokine expressed by human Langerhans cells.

17 al melanocyte hyperplasia and intraepidermal Langerhans cells.

18 DCs, including a population of inflammatory Langerhans cells.

19 ing molecule that is abundantly expressed on Langerhans cells.

20 lation by UVB radiation, because ablation of Langerhans cells abolished the UVB-induced phenotype.

21 particular conventional dendritic cells and Langerhans cells, accounting for the chronic inflammatio

22 ells and specifically increased the level of Langerhans cells activation.

23 Mice that are deficient in epidermal Langerhans cells allow the functions of these cells in v

24 ell generation in the presence or absence of Langerhans cells; analysis of BMP versus canonical TGF-b

25 selective expression of RUNX paralogs during Langerhans cell and inducible regulatory T cell differen

26 dent manner, suggesting an essential role of Langerhans cells and dendritic cells in CHQ-provoked pso

27 dermally delivered TLR agonists to stimulate Langerhans cells and dermal DCs in their natural complex

28 he MCs was efficiently taken up by epidermal Langerhans cells and dermal dendritic cells in the vicin

29 Langerhans cells and inflammatory dendritic epidermal ce

30 This infection of Langerhans cells and interstitial dermal dendritic cells

31 oinflammatory cytokines, and accumulation of Langerhans cells and macrophages within 3 days of tamoxi

32 In contrast, epidermal Langerhans cells and tissue macrophages were largely pre

33 s highly expressed by DC purified from skin (Langerhans cells) and bone marrow, and has been shown to

34 upffer cells), brain (microglia), epidermis (Langerhans cells) and lung (alveolar macrophages) origin

35 stinct types in the skin: branched, putative Langerhans cells, and amoeboid cells.

36 cluding ex vivo-generated DCs, skin-isolated Langerhans cells, and blood myeloid DCs and plasmacytoid

37 utrophils, macrophages, dendritic cells, and Langerhans cells, and colocalization of vaccine Ag withi

38 ll populations including endogenous T cells, Langerhans cells, and gammadelta T cells were not requir

39 dressed this in human ex vivo skin, in vitro Langerhans cells, and IDEC models generated from primary

40 tic cells: monocyte-derived dendritic cells, Langerhans cells, and interstitial dermal dendritic cell

41 in regulates the maturation and migration of Langerhans cells, and its ligation prevents the inductio

42 cells, dendritic cells, monocytes, NK cells, Langerhans cells, and leukocytes) had increasing mRNA ex

43 cytes, fibroblasts, melanocytes, mast cells, Langerhans cells, and Meissner's corpuscles, as well as

44 sues, such as liver Kupffer cells, epidermal Langerhans cells, and microglia--cell populations that a

45 mposed of classical and monocyte-derived DC, Langerhans cells, and plasmacytoid DC.

46 r with myeloid cell or with dedifferentiated Langerhans cell antigens, carry the BRAFV600E mutation.

47 Activation via RANKL prevents Langerhans cell apoptosis, thus leading to enhanced anti

48 Langerhans cells appeared in the dermis of skin borderin

49 Dermal dendritic cells and epidermal Langerhans cells are APCs that migrate from skin to drai

50 However, Flt3L-dependent DCs and resident Langerhans cells are dispensable for the inflammation.

51 Kupffer cells, microglia and Langerhans cells are only marginally replaced in one-yea

52 pids, whereas DHA abrogated the migration of Langerhans cells, as assessed by immunohistochemistry.

53 migratory dendritic cells and in particular Langerhans cells at governing T follicular helper and ge

54 Langerin, an endocytic receptor of Langerhans cells, binds pathogens such as human immunode

55 in the oesophagus, and the relative lack of langerhans cells (CD1a) may indicate this cell type is u

56 cific pattern recognition receptor TLR7, the Langerhans cell chemoattractant CCL20, and proinflammato

57 characterize the dermal T cell and epidermal Langerhans cell components of the skin immune system in

58 R signaling licenses inflammation-associated Langerhans cell/DC to gain an enhanced capacity to promo

59 logical heterogeneity and differentiation of Langerhans cells, delineate the signaling pathways respo

60 No differences were identified in corneal Langerhans cell density (19.84 cells/mm2 for the control

61 corneal nerve fiber tortuosity, and corneal Langerhans cell density between healthy controls and pat

62 ree main cutaneous DC populations: epidermal Langerhans cells, dermal myeloid DCs, and dermal plasmac

63 g cutaneous inflammation when it facilitates Langerhans cell egress from skin and enables the accumul

64 ) mice contained normal numbers of epidermal Langerhans cells (eLC) and increased numbers of CD11b(+)

65 Epidermal Langerhans cells (eLCs) uniquely express the C-type lect

66 inophils, macrophages, osteoclasts, DCs, and Langerhans cells from human embryonic stem cells (hESCs)

67 ify possible features that can differentiate Langerhans cells from malignant melanocytes to prevent t

68 eric C-type lectin specifically expressed in Langerhans cells, has been reported to be a pathogen rec

69 Precursors of pathological Langerhans cells have yet to be defined.

70 ations have been observed in 57% of cases of Langerhans cell histiocytosis (LCH) and 54% of cases of

71 novel somatic ARAF mutation in a child with Langerhans cell histiocytosis (LCH) and demonstrate that

72 enesis of 2 of the most common histiocytoses-Langerhans cell histiocytosis (LCH) and Erdheim-Chester

73 Langerhans cell histiocytosis (LCH) and Erdheim-Chester

74 have been observed in half of patients with Langerhans cell histiocytosis (LCH) and in 50% to 100% o

75 heterogeneous diseases that mostly comprise Langerhans cell histiocytosis (LCH) and non-LCH.

76 Langerhans cell histiocytosis (LCH) and the non-LCH neop

77 Langerhans cell histiocytosis (LCH) combines in one noso

78 Langerhans cell histiocytosis (LCH) has a broad spectrum

79 nase (ERK) signaling pathway is activated in Langerhans cell histiocytosis (LCH) histiocytes, but onl

80 atients with refractory, risk-organ-positive Langerhans cell histiocytosis (LCH) in 2005.

81 There is little data on treatment of Langerhans cell histiocytosis (LCH) in adults.

82 Langerhans cell histiocytosis (LCH) is a clinically and

83 Langerhans cell histiocytosis (LCH) is a clonal disorder

84 Langerhans cell histiocytosis (LCH) is a myeloid neoplas

85 Langerhans cell histiocytosis (LCH) is a myeloproliferat

86 Langerhans cell histiocytosis (LCH) is a rare disease af

87 Langerhans cell histiocytosis (LCH) is a rare disease ch

88 Langerhans cell histiocytosis (LCH) is a rare disease wi

89 Langerhans cell histiocytosis (LCH) is a rare histiocyti

90 Langerhans cell histiocytosis (LCH) is an enigmatic dise

91 Langerhans cell histiocytosis (LCH) is an inflammatory m

92 Langerhans cell histiocytosis (LCH) is an inflammatory m

93 Langerhans cell histiocytosis (LCH) is caused by clonal

94 Langerhans cell histiocytosis (LCH) is characterized by

95 Langerhans cell histiocytosis (LCH) represents a clonal

96 0E) mutation-positive, refractory, childhood Langerhans cell histiocytosis (LCH) was evaluated.

97 of two humans from skeletal collections with Langerhans Cell Histiocytosis (LCH), a benign osteolytic

98 ing data to date on a group of patients with Langerhans cell histiocytosis (LCH), which historically

99 Langerhans cell histiocytosis (LCH)-III tested risk-adju

100 BRAF mutations have been observed in Langerhans cell histiocytosis (LCH).

101 9 patients with ECD and ECD overlapping with Langerhans cell histiocytosis (so-called mixed histiocyt

102 hough lymphangioleiomyomatosis and pulmonary Langerhans cell histiocytosis are perhaps more frequentl

103 Erdheim-Chester disease (ECD) is a rare non-Langerhans cell histiocytosis that most commonly affects

104 d eruptive histiocytosis (GEH) is a rare non-Langerhans cell histiocytosis with a benign, self-healin

105 "Langerhans cell histiocytosis" (LCH) describes a spectru

106 most common histiocytic disorders, including Langerhans cell histiocytosis, Erdheim-Chester disease,

107 Erdheim-Chester disease (ECD) is a rare non-Langerhans cell histiocytosis, to whose pathogenesis neo

108 those of histiocytic human diseases, such as Langerhans cell histiocytosis.

109 ytic conditions, Erdheim-Chester disease and Langerhans cell histiocytosis.

110 recurrent and systemic viral infections and Langerhans cell histiocytosis.

111 lymphomas, neuroblastoma, some sarcomas, and Langerhans cell histiocytosis.

112 Rosai-Dorfman disease is a rare non-Langerhans cell histiocytosis.

113 us on lymphangioleiomyomatosis and pulmonary Langerhans cell histiocytosis.

114 n the cohort with Erdheim-Chester disease or Langerhans'-cell histiocytosis, the response rate was 43

115 ng cancer and in Erdheim-Chester disease and Langerhans'-cell histiocytosis.

116 scopy of the t.c. immunization site revealed Langerhans cells in areas of the skin containing the Abe

117 led an increased number of epidermal CD83(+) Langerhans cells in FLG-null subjects.

118 Although Langerhans cells in LCH are clonal, their benign morphol

119 NTB failed to induce emigration of epidermal Langerhans cells in naive individuals.

120 cts with FLG-null mutations have more mature Langerhans cells in nonlesional skin irrespective of whe

121 y an abnormality in corneal nerve fibers and Langerhans cells in patients with and without HIV-SN.

122 IL-10 production by (regulatory) T cells and Langerhans cells in regulating CHS has been established

123 Flt3L augmented the appearance of Langerhans cells in response to both injury and infectio

124 rai and coworkers explore the involvement of Langerhans cells in skin graft rejection and describe fa

125 es, highly significant reductions in CD1a(+) Langerhans cells in the dermis and CD11c(+) dermal dendr

126 ection against HIV infection to skin-derived Langerhans cells in the ex vivo system, suggesting Marav

127 arbohydrate-dependent uptake of pathogens by Langerhans cells in the first step of antigen presentati

128 oid and myeloid DCs, but also from epidermal Langerhans cells, indicating a distinct DC entity.

129 f Th1 and Th2 polarization in the setting of Langerhans cell (LC) Ag presentation to responsive T cel

130 identify corneal nerve damage and increased Langerhans cell (LC) density in adults with Type 1 diabe

131 RC1 but not mTORC2 is required for epidermal Langerhans cell (LC) homeostasis.

132 Cs in SDLNs, and augmented TRITC/DBP-induced Langerhans cell (LC) migration 72 hours post TRITC treat

133 e required for CCL19 production and adequate Langerhans cell (LC) migration both ex vivo and in vivo.

134 talin1 regulates not only integrin-dependent Langerhans cell (LC) migration, but also MyD88-dependent

135 Langerhans cell (LC) networks play key roles in immunity

136 Human Langerhans cell (LC) precursors populate the epidermis e

137 In this study, the role of the epidermal Langerhans cell (LC) subset of skin dendritic cells in t

138 ells from BM of MS-RO+ LCH patients produced Langerhans cell (LC)-like cells in vitro.

139 dritic cells (DC) in the skin and mucosa are Langerhans cells (LC) and interstitial dermal DC (iDDC).

140 1-negative adults and neonates (moDC) and by Langerhans cells (LC) and interstitial, dermal-intestina

141 ransit through the epidermis, which contains Langerhans cells (LC) and keratinocytes (KC), among othe

142 Langerhans cells (LC) are a subset of skin-resident dend

143 Langerhans cells (LC) are APC that reside at the barrier

144 Langerhans cells (LC) are epidermal dendritic cells capa

145 etween these cells and langerin(+) epidermal Langerhans cells (LC) are incompletely characterized.

146 Langerhans cells (LC) are the dendritic APC population o

147 Langerhans cells (LC) are the resident APCs at the site

148 Langerhans cells (LC) are thought to be the only mononuc

149 Langerhans cells (LC) can prime tolerogenic as well as i

150 Mice lacking epidermal Langerhans cells (LC) develop exaggerated contact-hypers

151 dly, huLangerin-DTA mice (DeltaLC) that lack Langerhans cells (LC) developed increased skin inflammat

152 Epidermal Langerhans cells (LC) expressing the high-affinity recep

153 e literature regarding the role of epidermal Langerhans cells (LC) in promoting skin immune responses

154 Presentation of foreign antigen by Langerhans cells (LC) in the absence of exogenous adjuva

155 isrupted, allowing easier access of HIV-1 to Langerhans cells (LC) in the epidermis and perhaps even

156 Langerhans cells (LC) in the epidermis of patients with

157 plasmacytoid DC (pDC) and in vitro-generated Langerhans cells (LC) obtained from AD patients with HSV

158 After activation, Langerhans cells (LC), a distinct subpopulation of epide

159 ce had multiorgan inflammation, lack of skin Langerhans cells (LC), and a shortened lifespan, consist

160 ll types, including keratinocytes, epidermal Langerhans cells (LC), and dermal dendritic cells (DC).

161 MHD1 is also expressed in epidermis-isolated Langerhans cells (LC), but degradation of SAMHD1 does no

162 There are at least three subsets of skin DC- Langerhans cells (LC), Langerin(+) dermal DCs (dDCs), an

163 Tissue-resident dendritic cells, such as Langerhans cells (LC), normally carry Ags from tissues t

164 HPV does not activate Langerhans cells (LC), the APC at the site of infection,

165 During infection, HPV16 also interacts with Langerhans cells (LC), the APC of the epithelium, induci

166 Langerhans cells (LC), the dendritic cells of the epider

167 or component of adherens junctions and marks Langerhans cells (LC), the only dendritic cell (DC) popu

168 leting or altering the function of epidermal Langerhans cells (LC).

169 CD207+ Langerhans cells (LCs) and CD11c+ myeloid dendritic cell

170 Langerhans cells (LCs) and CD8(+) tissue-resident memory

171 dritic cell (DC) subsets, epidermal CD207(+) Langerhans cells (LCs) and dermal CD14(+) DCs.

172 We tested the capacity of human Langerhans cells (LCs) and dermal dendritic cells (DDCs)

173 of TGF-beta1 on the differentiation of human Langerhans cells (LCs) and identified Axl as a key TGF-b

174 he two best-characterized skin-resident DCs, langerhans cells (LCs) and Langerin(+) dermal DCs (dDCs)

175 by heterogeneous lesions containing CD207(+) Langerhans cells (LCs) and lymphocytes that can arise in

176 Langerhans cells (LCs) and microglia develop from embryo

177 The development of both Langerhans cells (LCs) and microglia is highly dependent

178 for all mouse DC subsets revealed that human Langerhans cells (LCs) and the mouse XCR1(+)CD8alpha(+)C

179 Langerhans cells (LCs) are a distinct population of dend

180 Langerhans cells (LCs) are able to orchestrate adaptive

181 Langerhans cells (LCs) are dendritic cells (DCs) localiz

182 Langerhans cells (LCs) are dendritic cells (DCs) residin

183 eport that dermal dendritic cells (DDCs) and Langerhans cells (LCs) are differentially mobilized duri

184 Langerhans cells (LCs) are distinct dendritic cells (DCs

185 Langerhans cells (LCs) are epidermis-resident antigen-pr

186 Langerhans cells (LCs) are epithelial APCs that sense da

187 esident member of the dendritic cell family, Langerhans cells (LCs) are generally regarded to functio

188 Human Langerhans cells (LCs) are highly efficient at priming c

189 Langerhans cells (LCs) are immediate targets of exogenou

190 Langerhans cells (LCs) are known as "sentinels" of the i

191 We recently reported that human epidermal Langerhans cells (LCs) are more efficient than dermal CD

192 Langerhans cells (LCs) are myeloid cells of the epidermi

193 Langerhans cells (LCs) are professional antigen-presenti

194 Langerhans cells (LCs) are self-renewing epidermal myelo

195 Langerhans cells (LCs) are self-renewing in the steady s

196 Langerhans cells (LCs) are skin-resident dendritic cells

197 Langerhans cells (LCs) are the dendritic cells (DCs) of

198 Langerhans cells (LCs) are the only DC subset in the hea

199 Langerhans cells (LCs) are the sole dendritic cell type

200 Langerhans cells (LCs) are the unique dendritic cells fo

201 could be detected in the epidermal-resident Langerhans cells (LCs) as mRNA and protein.

202 Langerhans cells (LCs) comprise a dendritic network with

203 Langerhans cells (LCs) comprise a network of dendritic c

204 Langerhans cells (LCs) constitute a network of immune se

205 Following MN immunization, Langerhans cells (LCs) constituted the major skin DC sub

206 Human epidermal and mucosal Langerhans cells (LCs) express the C-type lectin recepto

207 dent DCs remain controversial, and epidermal Langerhans cells (LCs) have been referred to recently as

208 Although Langerhans cells (LCs) have been reported to express IL-

209 Langerhans cells (LCs) have been shown to be sufficient

210 However, the roles of skin-resident Langerhans cells (LCs) in eliciting immune responses hav

211 The precise role of human epidermal Langerhans cells (LCs) in immune response is highly cont

212 Since their discovery in 1868, the role of Langerhans cells (LCs) in skin immunity has been researc

213 ency virus type 1 (HIV-1) is internalized by Langerhans cells (LCs) in stratified epithelia and trans

214 Although implied by other models, proof that Langerhans cells (LCs) in the human vagina participate i

215 Langerhans cells (LCs) in the psoriatic epidermis engage

216 Langerhans cells (LCs) in the skin are a first line of d

217 Understanding the function of Langerhans cells (LCs) in vivo has been complicated by c

218 Langerhans cells (LCs) induce type 2 antibodies reactive

219 Langerhans cells (LCs) may function as inducers of immun

220 his issue, Su and Igyarto (2019) showed that Langerhans cells (LCs) obtain mRNA from keratinocytes (K

221 assumptions on the identity and functions of Langerhans cells (LCs) of the epidermis have undergone c

222 Langerhans cells (LCs) of the epidermis, although of mye

223 Epidermal Langerhans cells (LCs) of the skin represent the prototy

224 The ontogeny of human Langerhans cells (LCs) remains poorly characterized, in

225 nfection, we showed that in vivo ablation of Langerhans cells (LCs) resulted in enhanced bone loss.

226 Langerhans cells (LCs) serve as epidermal sentinels of t

227 We tested the contribution of mucosal Langerhans cells (LCs) to alveolar bone homeostasis in m

228 with monocyte-derived DCs (MDDCs) and MUTZ3 Langerhans cells (LCs) to investigate their relevance as

229 In vivo studies questioned the ability of Langerhans cells (LCs) to mediate CD8(+) T cell priming.

230 regulates the outcome of Ag presentation by Langerhans cells (LCs) to T cells through actions on mic

231 To determine the relative contribution of Langerhans cells (LCs) to the ensuing GVHD-like reaction

232 tion of the epidermal mononuclear phagocytes Langerhans cells (LCs) to this phenomenon because of the

233 At steady state, Langerhans cells (LCs) were lineage traced but also expr

234 ited in recent years, and the involvement of Langerhans cells (LCs), a population of epidermal dendri

235 Mice lacking Langerhans cells (LCs), a signatory epidermal dendritic

236 interplay of the C-type lectins, Langerin on Langerhans cells (LCs), and dendritic cell-specific inte

237 Based on our ability to discriminate Langerhans cells (LCs), conventional DCs, monocytes, mon

238 ered intradermally are taken up by epidermal Langerhans cells (LCs), dermal Langerin(neg) DCs, and de

239 ts of skin-resident DC have been identified: Langerhans cells (LCs), dermal Langerin+ DC (Lang+ dDC),

240 Skin-resident DCs, namely, Langerhans cells (LCs), have been implicated in regulati

241 n two distinct microanatomical compartments: Langerhans cells (LCs), mainly in the epidermis, and der

242 In addition to Langerhans cells (LCs), other dendritic cells (CD11c(+))

243 ance in mice critically depends on epidermal Langerhans cells (LCs), which capture DNTB and migrate t

244 ers the morphology and function of epidermal Langerhans cells (LCs), which play a role in UV-induced

245 tigated the age-related changes in epidermal Langerhans cells (LCs), which play a sentinel role in th

246 le of keratinocyte-derived cytokine TSLP and Langerhans cells (LCs).

247 henotypic characteristics of human epidermal Langerhans cells (LCs).

248 for maintaining the homeostasis of epidermal Langerhans cells (LCs).

249 ltrastructural features similar to cutaneous Langerhans cells (LCs).

250 intraepidermal dendritic cells (DCs) called Langerhans cells (LCs).

251 ation of graft antigen by resident epidermal Langerhans cells (LCs).

252 s; CD1c), plasmacytoid DCs (pDCs; CD303) and Langerhans cells (LCs; CD1a, CD207) in uncinate tissue (

253 Using FACS, we isolated Langerhans cells (LCs; HLA-DR(+)CD207(+) cells) and derm

254 Langerhans' cells (LCs) are a subset of periphery reside

255 matic disease defined by the accumulation of Langerhans cell-like dendritic cells (DCs).

256 nd T-helper type 1 skewing function in human Langerhans cell-like dendritic cells (LC-DCs).

257 o neutrophils, eosinophils, dendritic cells, Langerhans cells, macrophages and osteoclasts.

258 -derived IL-1beta alone reduced infection of Langerhans cells, macrophages, and dermal dendritic cell

259 culation led to recruitment and infection of Langerhans cells, macrophages, and dermal dendritic cell

260 Langerhans cells, macrophages, and T lymphocytes were st

261 NV infected a wide range of cells, including Langerhans cells, macrophages, dermal dendritic cells, m

262 A-DR, CD83, costimulatory molecules, and the Langerhans cell marker CD1a, whereas pDC expressed low l

263 Unconventional immune regulatory roles for Langerhans cells, mast cells, and natural killer T (NKT)

264 EP also induced Langerhans cell maturation, illustrated by CD86, CD83, a

265 Langerin, a C-type lectin on Langerhans cells, mediates carbohydrate-dependent uptake

266 response to oxazolone and oxazolone-induced Langerhans cell migration from epidermis were both preve

267 The presence of hyperreflective Langerhans cells mimicking malignant melanocytes was the

268 Strikingly, using Langerhans cells model systems (mutz-3-derived LC, monoc

269 Together, these data suggest that Langerhans cell/Mphi recognition of microbial LPS regula

270 of chronic inflammation, namely macrophages, Langerhans cells, myeloid-derived suppressor cells, and

271 langerin (Lang; CD207)(neg) DCs, but neither Langerhans cells nor Lang(+) DCs were required for CD8(+

272 is functional, resulting in slightly reduced Langerhans cell numbers.

273 uman immunodeficiency virus-1) transmission, Langerhans cells of genital mucosa play a protective rol

274 terstitial dendritic cells of the dermis and Langerhans cells of the epidermis, in a dose- and time-d

275 Langerhans cells participate in the immune response in l

276 nces in the numbers of T or B lymphocytes or Langerhans cells present in StrataGraft skin substitute

277 utrophils, macrophages, dendritic cells, and Langerhans cells remained in the tissue at least 1 wk.

278 y factors (IRFs) as controllers of the human Langerhans cell response to epidermal cytokines was reve

279 Antigen-presenting Langerhans cells show a differential migration phenotype

280 okine for mature DCs) in vitro, and in vivo, Langerhans cells show reduced emigration into draining l

281 Skin-migratory Langerhans cells (smiLCs) were the main cutaneous DC sub

282 Langerhans cells stained for phospho-mitogen-activated p

283 1 TCRgammadelta+ intraepithelial T cells and Langerhans cells, swiftly followed by epithelial infiltr

284 Langerin is a C-type lectin present on Langerhans cells that mediates capture of pathogens in a

285 ology of the skin, including the function of Langerhans cells, the migration of immune cells in skin,

286 The working theory is that niacin provokes Langerhans cells to produce prostaglandin D2 (PGD2), sti

287 (FLG and other skin barrier gene mutations, Langerhans cells, type 2 innate lymphoid cells, IL-33, T

288 )5V(delta)1+ T cells limited carcinogenesis, Langerhans cells unexpectedly promoted it.

289 s, mast cells, eosinophils, macrophages, and Langerhans cells; upregulation of chemokine and cytokine

290 epithelial, monocyte-derived dendritic, and Langerhans cells via direct cell-cell transmission.

291 of Flt3L at the burn wound, localization of Langerhans cells was examined.

292 Increased numbers of CD1a-expressing Langerhans cells were detected both in the epithelium an

293 Langerhans cells were required for eliciting immune resp

294 DCs were able to cross-prime CD8(+) T cells, Langerhans cells were unexpectedly found to potently cro

295 ted production of prostaglandin D2 and E2 in Langerhans' cells which act on DP1 and EP2/4 receptors i

296 These two markers are mainly expressed by Langerhans cells, which are one of several functionally

297 angerin and the ephrin A2 receptor to infect Langerhans cells, which support full HHV-8 lytic replica

298 S-100-positive cells included CD1a-positive Langerhans cells, while CK20 did not identify any Merkel

299 althy skin biopsies, human keratinocytes and Langerhans cells with IL-4.

300 angerin, the distinguishing C-type lectin of Langerhans cells, would recognize the highly mannosylate