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1 panosoma cruzi , Plasmodium falciparum , and Leishmania amazonensis .
2 howed that ATP-treated macrophages eliminate Leishmania amazonensis.
3 /6 mice following intradermal infection with Leishmania amazonensis.
4  2 hours of adding it to cells infected with Leishmania amazonensis.
5 reductase activity exposed on the surface of Leishmania amazonensis.
6 t parasite loads when they are infected with Leishmania amazonensis.
7 nes also recognize macrophages infected with Leishmania amazonensis.
8 infected CD40L knockout (CD40L-/-) mice with Leishmania amazonensis.
9 tion of the arginase enzyme from Leishmania (Leishmania) amazonensis.
10                  In cutaneous leishmaniasis, Leishmania amazonensis activates macrophage double-stran
11 d sera from infected BALB/c mice to screen a Leishmania amazonensis amastigote cDNA expression librar
12    We previously reported that the growth of Leishmania amazonensis amastigotes in murine macrophages
13 onstrated that infection of macrophages with Leishmania amazonensis amastigotes led to the activation
14 s a major trigger for the differentiation of Leishmania amazonensis amastigotes, independently of tem
15  with Leishmania mexicana complex parasites (Leishmania amazonensis and Leishmania pifanoi) is impair
16 ted resistance in Caenorhabditis elegans and Leishmania amazonensis and predicted sensitivity in Aede
17 mania braziliensis , Leishmania guyanensis , Leishmania amazonensis , and Trypanosoma cruzi .
18  mediate both phagocytosis and the uptake of Leishmania amazonensis by macrophages (Ms).
19 on against New World leishmaniasis caused by Leishmania amazonensis can be elicited by immunization w
20        Infection with the protozoan parasite Leishmania amazonensis can cause diverse clinical forms
21                                              Leishmania amazonensis can cause progressive disease in
22  Th1 response and subsequently heal, whereas Leishmania amazonensis challenge leads to chronic lesion
23 hown to protectively immunize mice against a Leishmania amazonensis challenge.
24            Infection of mammalian hosts with Leishmania amazonensis depends on the remarkable ability
25 h extracts of the related protozoan parasite Leishmania amazonensis did not induce myosin autoimmunit
26                       Antiserum specific for Leishmania amazonensis GP63 specifically reacted with a
27  modulation effect on their efficacy against Leishmania amazonensis in vitro infection.
28                                              Leishmania amazonensis induces a nonhealing infection in
29                            Upon contact with Leishmania amazonensis-infected macrophages, these FN-ac
30                                           In Leishmania amazonensis-infected patients, host SOD1 leve
31            The progressive disease following Leishmania amazonensis infection in mice requires functi
32      Cutaneous leishmaniasis associated with Leishmania amazonensis infection is characterized by unc
33                The susceptibility of mice to Leishmania amazonensis infection is thought to result fr
34 signed to ascertain the role of IFN-gamma in Leishmania amazonensis infection, we were surprised to f
35 Leishmania and this pathway is important for Leishmania amazonensis infection.
36 st inbred strains of mice are susceptible to Leishmania amazonensis infection.
37                   Lecoeur et al. show us how Leishmania amazonensis inhibits macrophage inflammasomes
38                       The protozoan parasite Leishmania amazonensis is a heme auxotroph and must acqu
39     The major surface glycoprotein (gp63) of Leishmania amazonensis is a metalloprotease implicated i
40                                              Leishmania amazonensis is an etiological agent of diffus
41  the role of LTB4 in neutrophil infection by Leishmania amazonensis is not clear.
42                                              Leishmania amazonensis is the etiological agent of cutan
43 the causative species in Brazil, Leishmania (Leishmania) amazonensis is an important etiological agen
44  Leishmania prevalent in Brazil [Leishmania (Leishmania) amazonensis, L (Viannia) braziliensis, and L
45 n of CD4(+) T cells mediates pathogenesis in Leishmania amazonensis (La)-infected mice, these suscept
46 eJ (C3H) mice with both Leishmania major and Leishmania amazonensis leads to a healed footpad lesion,
47 leucyl aminopeptidase (lap) were cloned from Leishmania amazonensis, Leishmania donovani, and Leishma
48                            Lesions caused by Leishmania amazonensis normally heal, but relapses occur
49                               Infection with Leishmania amazonensis, on the other hand, causes progre
50 le of SODA, the mitochondrial SOD isoform in Leishmania amazonensis Our inability to generate L. amaz
51 ules and plaques containing large numbers of Leishmania amazonensis parasites and few lymphoid elemen
52 ic immune responses following infection with Leishmania amazonensis parasites.
53 activation of DCs that were preinfected with Leishmania amazonensis promastigotes and that these acti
54 E, MPO and PAD4 in the netosis stimulated by Leishmania amazonensis promastigotes in human neutrophil
55 the in vivo growth of Leishmania chagasi and Leishmania amazonensis promastigotes, by impairing the f
56 ecifically inhibit Plasmodium falciparum and Leishmania amazonensis, respectively.
57 inally, FCaBP was expressed in the protozoan Leishmania amazonensis, which lacks FCaBP.
58 , we transfected a gp63-deficient variant of Leishmania amazonensis with constructs expressing gp63 a
59  efficacy of a vaccine combining heat-killed Leishmania amazonensis with human rIL-12 (rhIL-12) and a