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1 ptor binding and toxicity toward gypsy moth (Lymantria dispar).
2  by 4-fold, while retaining activity against Lymantria dispar.
3 binding proteins (PBPs) from the gypsy moth, Lymantria dispar.
4 ing preference of the generalist caterpillar Lymantria dispar A potential role of the sulfated salici
5 n-specific toxin, bound purified gypsy moth (Lymantria dispar) aminopeptidase N (APN) biphasically.
6 ight trajectories of a moth and a butterfly (Lymantria dispar and Pieris rapae), hindwing removal cau
7    Two subspecies of Asian gypsy moth (AGM), Lymantria dispar asiatica and L. dispar japonica, pose a
8 ngy moth (ASM), which consists of subspecies Lymantria dispar asiatica and Lymantria dispar japonica
9 trast, females of the Asian gypsy moth (AGM; Lymantria dispar asiatica) subspecies have fully develop
10 ased survival and weight gain of gypsy moth (Lymantria dispar) caterpillars, suggesting that aldoxime
11 of the destructive lepidopteran forest pest, Lymantria dispar dispar (European spongy moth), were sep
12 he late 1800s, the European gypsy moth (EGM; Lymantria dispar dispar) has become a major defoliator i
13 nt biotypes: the European spongy moth (ESM), Lymantria dispar dispar, which is distributed in Europe
14                              The spongy moth Lymantria dispar, formerly known as the gypsy moth, is a
15 giensis toxin against the forest insect pest Lymantria dispar (gypsy moth), unfortunately it is also
16 9, populations of North American gypsy moth, Lymantria dispar, in seven contiguous northeastern state
17                              The gypsy moth (Lymantria dispar) is nonpermissive for Autographa califo
18                  The invasion of gypsy moth (Lymantria dispar) is well-documented from its introducti
19  of subspecies Lymantria dispar asiatica and Lymantria dispar japonica and is distributed in China, R
20                                  Gypsy moth (Lymantria dispar L.) is one of the world's worst hardwoo
21                              The gypsy moth, Lymantria dispar L., is one of the most destructive fore
22 he population growth rate of the gypsy moth, Lymantria dispar (L.), along its invasion front in Virgi
23 es of the mutant toxins on Manduca sexta and Lymantria dispar larvae were examined.
24  with a baculovirus that infects gypsy moth (Lymantria dispar) larvae.
25 AMVsod is expressed late during infection of Lymantria dispar (Ld652) cells and produces a discrete n
26  nonpermissive for AcMNPV, was identified in Lymantria dispar M nuclear polyhedrosis virus (LdMNPV).
27 ingle gene, hrf-1, from another baculovirus, Lymantria dispar M nucleopolyhedrovirus, is able to prec
28                              F proteins from Lymantria dispar MNPV (LdMNPV) and Spodoptera exigua MNP
29 MNPV (OpMNPV) GP64 and F, thogotovirus GP75, Lymantria dispar MNPV (LdMNPV) F, human immunodeficiency
30 lyses of the genome of one of these viruses, Lymantria dispar MNPV (LdMNPV), revealed a single open r
31                     Sequence analysis of the Lymantria dispar multicapsid nucleopolyhedrovirus (LdMNP
32 tified two enhancin genes (E1 and E2) in the Lymantria dispar multinucleocapsid NPV (LdMNPV) and show
33 usly identified an enhancin gene (E1) in the Lymantria dispar multinucleocapsid nucleopolyhedrovirus
34                                            A Lymantria dispar nuclear polyhedrosis virus (LdMNPV) gen
35  high frequency during serial passage of the Lymantria dispar nucleopolyhedrovirus (LdMNPV) in the L.
36 nces of the establishment of the gypsy moth (Lymantria dispar) surfaced as a general threat.
37 c in outbreak populations of the gypsy moth, Lymantria dispar, throughout many forested and residenti