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1 wal [4, 5] and feeding [6] in the pond snail Lymnaea.
2 o 6.2 mM for Aplysia and 0.12 to 0.22 mM for Lymnaea.
3 tructure of the multimillimeter-scale CNS of Lymnaea.
4                       In the isolated CNS of Lymnaea, a peptidergic neuron termed VD4 makes monosynap
5 mputer docking of the methylnicotines to the Lymnaea acetylcholine binding protein crystal structure
6                                    Using the Lymnaea acetylcholine-binding protein as a surrogate of
7  that has the highest known affinity for the Lymnaea AChBP and also potently blocks the alpha7 nAChR
8 on are possible for approximately 56% of the Lymnaea AChBP sequence, covering primarily the outer sur
9 r of 4,6-disubstituted 2-aminopyrimidines to Lymnaea AChBP, with different molecular variants exhibit
10 ide that was purified and characterized from Lymnaea albumen gland extracts.
11 f nitric oxide (NO) production in the CNS of Lymnaea, an established model for molecular analysis of
12          In this work, homomeric AChBPs from Lymnaea and Aplysia snails were used as in situ template
13 ing substituted anabaseines with AChBPs from Lymnaea, Aplysia, and Bulinus species and correlated the
14  Secretions of the exocrine albumen gland of Lymnaea are packaged in the eggs of an egg mass before t
15  we also establish a functional CNS atlas in Lymnaea as a shared and scalable resource for the resear
16 s contingent upon trophic factors present in Lymnaea brain-conditioned medium.
17                    Here, we characterize the Lymnaea CBP (LymCBP) gene and identify a conserved domai
18 lar structure and HAT activity of CBP in the Lymnaea central nervous system (CNS), hindering an inves
19 CBP mRNA in CNS are very similar to those of Lymnaea CREB1.
20  the albumen gland produces large amounts of Lymnaea epidermal growth factor.
21  the development of the embryo, for example, Lymnaea epidermal growth factor.
22   This is the maximum feeding rate of intact Lymnaea in sucrose.
23  provide evidence that: (1) LTM formation in Lymnaea is associated with the timed and targeted change
24 S1AS are co-expressed in a key neuron of the Lymnaea memory network.
25                                In the snail, Lymnaea, motoneurons such as the B4, B8, and B4CL cells
26 atively damped swelling-tension responses of Lymnaea neurons (no BAPTA) were consistent with feedback
27 hods to measure Ca2+ channel inactivation in Lymnaea neurons-one method, based upon the conventional
28 t membrane tension of swelling and shrinking Lymnaea neurons.
29         In the feeding system of the mollusc Lymnaea, one of the best-studied rhythmical networks, in
30      Specifically, the electrically synapsed Lymnaea pedal dorsal A cluster neurons were used to stud
31 al approach, we demonstrate that the mollusc Lymnaea performs a sophisticated form of decision-making
32 g masses of the freshwater gastropod mollusc Lymnaea provide a microenvironment for developing embryo
33 e of a single-trial conditioning paradigm in Lymnaea solves this problem.
34 a soluble acetylcholine-binding protein from Lymnaea stagnali.
35 availability in the model freshwater species Lymnaea stagnalis (Gastropoda).
36 ith distinct pharmacological profiles [i.e., Lymnaea stagnalis (Ls) AChBP of low neonicotinoid and hi
37 this intricate scenario, invertebrates, like Lymnaea stagnalis (LS), provide a flexible tool to unrav
38 2 and the acetylcholine-binding protein from Lymnaea stagnalis (Ls-AChBP) confirms Ls-AChBP as struct
39 , we chronically exposed freshwater snails ( Lymnaea stagnalis ) to synthetic water spiked with Cu th
40 alifornica, Pleurobranchaea californica, and Lymnaea stagnalis -three well-recognized models in cellu
41 erred to as model I, was built from both the Lymnaea stagnalis acetylcholine binding protein (AChBP)
42 constructed from the human alpha(7) AChR and Lymnaea stagnalis acetylcholine binding protein (AChBP),
43 tinic acetylcholine receptor (nAChR) and the Lymnaea stagnalis acetylcholine binding protein (Ls-AChB
44 L112Q, and M114T, into the binding pocket of Lymnaea stagnalis acetylcholine-binding protein (Ls-AChB
45                                              Lymnaea stagnalis efficiently accumulated (65)Cu after a
46 r and reward, and research on the pond snail Lymnaea stagnalis elucidated the role of a behavior-init
47 e of Cu were evaluated in the benthic grazer Lymnaea stagnalis following 4-5 h exposures to Cu adsorb
48 entity over a stretch of 294 residues with a Lymnaea stagnalis G-protein-linked receptor (LSGLR).
49                         The great pond snail Lymnaea stagnalis has served as a model organism for ove
50 -type calcium channels from great pond snail Lymnaea stagnalis have a selectivity-filter ring of five
51 rates Gammarus pulex, Gammarus fossarum, and Lymnaea stagnalis illustrates the approach.
52 ly, we trained intact specimens of the snail Lymnaea stagnalis in a single conditioning trial using a
53  of dietary U uptake in the freshwater snail Lymnaea stagnalis in laboratory experiments.
54 lower than reported for the freshwater snail Lymnaea stagnalis in similar experiments.
55                                              Lymnaea stagnalis is a model species that has relatively
56 ic behaviour we show that the feeding CPG of Lymnaea stagnalis is itself associated with another, and
57 ted during the maturation of the pond snail, Lymnaea stagnalis L., applying light and electron micros
58                                    The snail Lymnaea stagnalis produces a neuropeptide precursor prot
59 tral nervous system of the gastropod mollusk Lymnaea stagnalis produces a soluble protein that specif
60 dividually identified synaptic partners from Lymnaea stagnalis to interrogate the role of neuronal ac
61 re of the acetylcholine binding protein from Lymnaea stagnalis to model the chicken alpha7 agonist-bi
62 ntified neurons in the CNS of the pond snail Lymnaea stagnalis to study the role of endogenous NO sig
63 nervous system (CNS) of the freshwater snail Lymnaea stagnalis was conducted.
64         First, heterozygous (Dd) pond snails Lymnaea stagnalis were self-fertilised or backcrossed, a
65 re of the acetylcholine-binding protein from Lymnaea stagnalis with NS9283, a stoichiometry selective
66         We used the model learning system of Lymnaea stagnalis(1-3) to address the question of how an
67 udying ancestral channels in the pond snail (Lymnaea stagnalis), Guan et al.
68  these transposon families in the pond snail Lymnaea stagnalis, a cosmopolitan vector of trematodes i
69 re of the acetylcholine-binding protein from Lymnaea stagnalis, a nicotinic receptor surrogate.
70  protein (AChBP) from the fresh water snail, Lymnaea stagnalis, shows it to be a structural homolog o
71      We found that in neurons of the mollusk Lymnaea stagnalis, two different RXR antagonists (PA452
72  using central neurons from the invertebrate Lymnaea stagnalis, we demonstrate that menin coordinates
73 te circuit generating feeding in the mollusk Lymnaea stagnalis, we identified three independent pathw
74 lines: one of these is the great pond snail, Lymnaea stagnalis.
75 ntral nervous system of the freshwater snail Lymnaea stagnalis.
76 ontaining the learning circuits of the snail Lymnaea stagnalis.
77  bound to acetylcholine binding protein from Lymnaea stagnalis.
78 ferent identified neurons in the pond snail, Lymnaea stagnalis.
79 ng buccal neurons from the freshwater snail, Lymnaea stagnalis.
80  the binding protein previously derived from Lymnaea stagnalis.
81 e binding protein (AChBP) found in the snail Lymnaea stagnalis.
82 the isolated central nervous system (CNS) of Lymnaea stagnalis.
83 ed buccal feeding neurons in the pond snail, Lymnaea stagnalis.
84 experiments using the model freshwater snail Lymnaea stagnalis.
85 ole in neuronal plasticity in the pond snail Lymnaea stagnalis.
86 etopleura, the limpet Tectura, and the snail Lymnaea, the MAPK pathway is activated in the 3D cell bu
87                               In the mollusc Lymnaea, this pathway involves the functional interactio
88 rs to produce significant amounts of a novel Lymnaea trypsin inhibitor (LTI), a second peptide that w
89             Using the molluscan model system Lymnaea, we investigate here whether LTM formation is as
90                Using associative learning in Lymnaea, we show that reconsolidation after the retrieva
91    Here, we elucidate a circuit mechanism in Lymnaea, which enables past memory to shape new memory f