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1 donor for patient 1, which subsequently grew M. hominis.
2 d to 24 (29.6%) of the 81 women negative for M. hominis.
3 a suggest donor-to-recipient transmission of M. hominis.
4 mutator element in the vaa gene that governs M. hominis adherence and highlight the importance of mut
5 T. vaginalis in symbiosis with M. hominis or M. hominis alone triggers a noncanonical type I interfer
7 ergence of Vaa could affect the adherence of M. hominis and evasion of antibody-mediated immunity, th
8 he results of these studies demonstrate that M. hominis and M. hominis antigen are potent stimulators
9 acteria will not be reliable for recovery of M. hominis and that specialized media and incubation con
10 ase demonstrates the pathogenic potential of M. hominis and the need for rapid recognition of the org
11 hree log(10) bacterial counts (G. vaginalis, M. hominis, and lactobacilli) in our model improved the
12 hese studies demonstrate that M. hominis and M. hominis antigen are potent stimulators of type II epi
13 erial vaginosis was strongly associated with M. hominis (aOR = 8.01, 95%CI:5.99-10.71), but was not a
14 tative bacterial counts for lactobacilli and M. hominis are better correlates of CVL HIV RNA than are
15 m the joint synovial fluid of a patient with M. hominis-associated arthritis, which indicated that Va
16 terial counts (P=.006; inverse association), M. hominis bacterial counts (P=.0001; positive associati
17 ate analyses, we found that G. vaginalis and M. hominis bacterial counts, Candida vaginitis, and herp
19 had log(10) G. vaginalis counts and log(10) M. hominis counts greater than 6.81 and 4.82, respective
24 e data strongly suggest that the presence of M. hominis in the lungs of premature infants may initiat
26 ng chronic, active arthritis associated with M. hominis infection and is highly immunogenic in the hu
28 ransplant recipients presented with invasive M. hominis infections at multiple sites characterized by
31 ICU were found to have genetically unrelated M. hominis isolates, excluding patient-to-patient transm
35 nfection with T. vaginalis in symbiosis with M. hominis or M. hominis alone triggers a noncanonical t
39 findings do not support routine testing for M. hominis, U. urealyticum, and U. parvum in nonpregnant
41 lvovaginal candidiasis (VVC), and tested for M. hominis, U. urealyticum, and U. parvum, and 4 nonvira
43 n requirements and relatively slow growth of M. hominis warrant that dependence on automated systems
47 s, Ureaplasma spp. was detected in 23 (82%), M. hominis was detected in 3 (11%), and both were detect