ライフサイエンスコーパス: M. hyopneumoniae

1 M. hyopneumoniae serum antibody detection via commercial

2 A total of 48 M. hyopneumoniae PCR-positive field samples were pooled

3 ng infection, our analysis indicated that 79 M. hyopneumoniae genes were differentially expressed (P

4 an Escherichia coli opal suppressor host and M. hyopneumoniae bound specifically to swine cilia, and

5 evealed that pigs infected with both SIV and M. hyopneumoniae coughed significantly more than pigs in

6 In study 1, 6 commercial M. hyopneumoniae ELISAs were compared using serum sample

7 an assessment of the performance of current M. hyopneumoniae ELISAs and an understanding of their us

8 howing that P102 is expressed in vivo during M. hyopneumoniae infections.

9 were pooled 3-, 5-, and 10-times using field M. hyopneumoniae DNA-negative samples and tested in trip

10 r M. hyopneumoniae DNA and serum samples for M. hyopneumoniae antibody.

11 cheal and oral fluid samples were tested for M. hyopneumoniae DNA and serum samples for M. hyopneumon

12 How M. hyopneumoniae responds to changing environments in th

13 Thus, it is not clear how M. hyopneumoniae evades the immune response and establis

14 acheal pooling for Mycoplasma hyopneumoniae (M. hyopneumoniae) DNA detection allows for decreased dia

15 Most importantly, M. hyopneumoniae-infected pigs with minimal to nondetect

16 nd oral fluids, has the potential to improve M. hyopneumoniae surveillance.

17 ting that a processing event had occurred in M. hyopneumoniae.

18 c lesions were more severe and persistent in M. hyopneumoniae-infected pigs.

19 At 28 or 38 days after PRRSV inoculation, M. hyopneumoniae-infected pigs still exhibited lesions t

20 Unlike other mycoplasmas, M. hyopneumoniae contains few genes with tandem repeat s

21 In contrast, nonpathogenic M. hyopneumoniae and M. flocculare at concentrations of

22 enic Mycoplasma hyopneumoniae, nonpathogenic M. hyopneumoniae, and Mycoplasma flocculare on intracell

23 disease process is initiated by adherence of M. hyopneumoniae to the cilia of swine respiratory epith

24 s provided here will assist in the design of M. hyopneumoniae surveillance and monitoring programs fo

25 Numerous PCR assays for the detection of M. hyopneumoniae have been developed, targeting several

26 f pooled-sample testing for the detection of M. hyopneumoniae in tracheal samples and to develop prob

27 hospholipase C, also abolished the effect of M. hyopneumoniae.

28 ing of 632 of the 698 open reading frames of M. hyopneumoniae was constructed and used to study gene

29 In this study, a panel of isolates of M. hyopneumoniae, M. flocculare, M. hyorhinis, and M. hy

30 assays tested did not detect all isolates of M. hyopneumoniae.

31 urther delineate the molecular mechanisms of M. hyopneumoniae interactions with ciliated epithelium,

32 M. hyosynoviae were tested with a number of M. hyopneumoniae-specific PCR assays.

33 ay serve as a signal for the pathogenesis of M. hyopneumoniae.

34 ffective tool to maximize the performance of M. hyopneumoniae surveillance programs.

35 acheal samples and to develop probability of M. hyopneumoniae detection estimates for tracheal sample

36 A comparison of the R1 regions of M. hyopneumoniae strains displaying variation in cilium

37 infection did not influence the severity of M. hyopneumoniae infection, although microscopic lesions

38 Recently, genetic diversity among strains of M. hyopneumoniae was demonstrated.

39 e P97 cilium adhesin in different strains of M. hyopneumoniae, but the extent of genetic variation am

40 ion, although microscopic lesions typical of M. hyopneumoniae were more severe in PRRSV-infected pigs

41 in the probability of detecting at least one M. hyopneumoniae-infected pig given any pool size (3, 5,

42 In Ca2+-free medium, pathogenic M. hyopneumoniae still increased [Ca2+]i in tracheal cel

43 l within 100 s of the addition of pathogenic M. hyopneumoniae strain 91-3 (300 microg/ml), and this i

44 These results suggest that pathogenic M. hyopneumoniae activates receptors that are coupled to

45 tracheal samples (to establish the true pig M. hyopneumoniae status) were collected at 7- to 14-day

46 Since M. hyopneumoniae is a worldwide problem, it is reasonabl

47 Some M. hyopneumoniae PCR assays tested did not detect all is

48 is does not exist, although it is clear that M. hyopneumoniae adheres to porcine ciliated epithelium

49 Previous studies demonstrated that M. hyopneumoniae, which produces a chronic bronchopneumo

50 These results indicate that M. hyopneumoniae infection potentiates PRRSV-induced dis

51 ing gene was present as a single copy in the M. hyopneumoniae chromosome.

52 ermutation test identified a location in the M. hyopneumoniae genome where there is spatial clusterin

53 ups, with minimal levels of pneumonia in the M. hyopneumoniae-only-infected pigs.

54 specific and capable of detecting all of the M. hyopneumoniae isolates used in this study were develo

55 rsity on the accuracy and sensitivity of the M. hyopneumoniae PCR assays could result in false-negati

56 ual-infected pigs was similar to that of the M. hyopneumoniae-only-infected group, and the pneumonia

57 coplasma hyosynoviae (strain 34428), and (v) M. hyopneumoniae (strain 232).

58 potentiation found with dual infection with M. hyopneumoniae and PRRSV.

59 In this study, pigs were inoculated with M. hyopneumoniae 21 days prior to inoculation with SIV.

60 Pigs were inoculated with M. hyopneumoniae 21 days prior to, simultaneously with,

61 ld pigs were intratracheally inoculated with M. hyopneumoniae Matched serum and tracheal samples (to

62 46 pens were intratracheally inoculated with M. hyopneumoniae Thereafter, one tracheal sample, four s