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1 probes), and 3 (7%) were M. avium; none were M. intracellulare.
2 enicum and M. phocaicum, and M. chimaera and M. intracellulare.
3 5% confidence interval = 1.25 to 22.73) than M. intracellulare.
4                      Most patients (77%) had M. intracellulare.
5 nvestigate the public health significance of M. intracellulare.
6 larly, 130 divergent ORFs were identified in M. intracellulare.
7  M. avium could invade more efficiently than M. intracellulare.
8 the isolates from HIV-negative patients were M. intracellulare.
9 es within the 16S rRNA genes of M. avium and M. intracellulare.
10 l mechanism of host defense against M. avium-M. intracellulare.
11 trains genetically diverse from M. avium and M. intracellulare.
12 sis induce killing of intracellular M. avium-M. intracellulare.
13 ecies, including M. smegmatis, M. avium, and M. intracellulare.
14 clinical relapse/reinfection than those with M. intracellulare.
15  57.82 degrees C (57.05 to 58.60 degrees C); M. intracellulare, 54.46 degrees C (53.69 to 55.23 degre
16  species distribution, comprising 54 (81.8%) M. intracellulare, 6 (9.1%) M. avium, 5 (7.6%) M. colomb
17  However, concentrations of Legionella spp., M. intracellulare, Acanthamoeba spp., and M. avium peake
18      A marked age trend for the isolation of M. intracellulare among women was noted: 0.27% (1-fold)
19                          Mycobacterium avium-M. intracellulare, an intracellular parasite of mononucl
20 encing, 49 (90.7%) respiratory isolates were M. intracellulare and 4 (7.4%) were Mycobacterium chimae
21 nts has not been epidemiologically linked to M. intracellulare and appears to be unique to M. avium.
22 rdia sp., M. nonchromogenicum, M. monacense, M. intracellulare and M. avium subsp.
23 distinguish closely related species (such as M. intracellulare and M. chimaera).
24 h clarithromycin resistance were detected in M. intracellulare and M. colombiense.
25 er evaluate the interaction between M. avium-M. intracellulare and primary human monocytes.
26  Mycobacterium avium complex (MAC; M. avium, M. intracellulare, and "nonspecific or X" MAC) are emerg
27 cluded patients, 54% were M. avium, 18% were M. intracellulare, and 28% were M. chimaera.
28 The HD of M. nonchromogenicum, M. monacense, M. intracellulare, and M. avium subsp.
29 robe Culture Confirmation kits for M. avium, M. intracellulare, and MAC species; Gen-Probe).
30  95% confidence interval [CI], 1.33-3.44) or M. intracellulare (AOR, 3.12; 95% CI, 1.62-5.99) were mo
31                             Of 5126 genes of M. intracellulare ATCC13950, 506 genes were identified a
32 ellulare complex, and all were identified as M. intracellulare by the PCR-RFLP analysis.
33 ncy virus type 1-infected patients, M. avium-M. intracellulare can infect almost every tissue and org
34 subtractive hybridization using M. avium and M. intracellulare chromosomal DNAs.
35 were identified as belonging to the M. avium-M. intracellulare complex (but not M. paratuberculosis),
36 r the rapid diagnosis of Mycobacterium avium-M. intracellulare complex (MAC) bacteremia in patients w
37 icate that the currently identified M. avium-M. intracellulare complex includes strains genetically d
38 elerate the diagnosis of Mycobacterium avium-M. intracellulare complex infections, an immunomagnetic
39 r understand the role of Mycobacterium avium-M. intracellulare complex isolates in human disease.
40                  The 159 Mycobacterium avium-M. intracellulare complex isolates were further identifi
41 ional differentiation of Mycobacterium avium-M. intracellulare complex strains into M. avium and M. i
42 or 26 M. tuberculosis complex, 9 M. avium, 3 M. intracellulare complex, 3 M. kansasii, 4 M. gordonae,
43 ient samples were LiPA positive for M. avium-M. intracellulare complex, and all were identified as M.
44 erentiates M. tuberculosis complex, M. avium-M. intracellulare complex, and the following mycobacteri
45 terium tuberculosis complex and the M. avium-M. intracellulare complex, as well as rapid- and slow-gr
46 y coupled to magnetic beads with an M. avium-M. intracellulare complex-specific PCR protocol based on
47 ntified as mycobacteria outside the M. avium-M. intracellulare complex.
48  in specimens containing Mycobacterium avium-M. intracellulare complex.
49             The clinical isolate of M. avium-M. intracellulare did not replicate in freshly explanted
50 or fingerprinting of respiratory isolates of M. intracellulare from patients with underlying bronchie
51 contrast, 41 of the 65 (63.1%) patients with M. intracellulare had probable to definite infection, a
52 each of 10 clinical isolates of M. avium and M. intracellulare identified by conventional methods wer
53 isolated in 62 (0.83%) of 7,472 patients and M. intracellulare in 65 (0.87%).
54 tracellulare was observed only when M. avium-M. intracellulare-infected cells were treated with 10 mM
55 eath of intracellular mycobacteria, M. avium-M. intracellulare-infected human monocytes were treated
56 ed, H2O2-induced apoptotic death of M. avium-M. intracellulare-infected monocytes and its association
57 esent study, a long-term culture of M. avium-M. intracellulare-infected monocytes was used to further
58  intracellulare isolates, VNTR distinguished M. intracellulare into 42 clonal groups.
59 sults suggest that, among non-AIDS patients, M. intracellulare is more pathogenic and tends to infect
60 enetic analysis revealed a high diversity of M. intracellulare isolates and their evolutionary relati
61 solates did not contain IS1245 and 7% of the M. intracellulare isolates were found to carry IS1245.
62  used to characterize 32 Mycobacterium avium-M. intracellulare isolates, 4 Pseudomonas aeruginosa iso
63            Starting with a collection of 167 M. intracellulare isolates, VNTR distinguished M. intrac
64                  These results indicate that M. intracellulare lung disease in the United States is a
65 quiline shows potential for the treatment of M. intracellulare lung disease, but optimization of trea
66  NTM-PD patients due to Mycobacterium avium, M. intracellulare, M. abscessus, or M. massiliense and t
67 rdia sp., M. nonchromogenicum, M. monacense, M. intracellulare, M. avium subsp.
68 e (HD) of M. nonchromogenicum, M. monacense, M. intracellulare, M. avium subsp.
69 e following mycobacterial species: M. avium, M. intracellulare, M. kansasii, M. chelonae group, M. go
70  an inhibitory effect on Mycobacterium avium-M. intracellulare (MAI) when blood collected and process
71 acellulare complex strains into M. avium and M. intracellulare may provide a tool to better understan
72 lare (n = 57; 35.8%), and mixed M. avium and M. intracellulare (n = 2; 1.3%).
73 by MycoID as being M. avium (n = 98; 61.1%), M. intracellulare (n = 57; 35.8%), and mixed M. avium an
74 Rep-PCR also generated DNA fingerprints from M. intracellulare (n = 8) and MAC(x) (n = 2) strains.
75 ered more frequently from sterile sites than M. intracellulare (odds ratio, 4.6; P = 0.0092).
76                      In our system, M. avium-M. intracellulare parasitized the human monocytes and ap
77 cellulare were found to cross-react with the M. intracellulare probe in the assay.
78  negative with species-specific M. avium and M. intracellulare probes), and 3 (7%) were M. avium; non
79 e synthesized: MAV and MIN, for M. avium and M. intracellulare, respectively, and MYCOB, for the slow
80 stered in the distinct clades separated from M. intracellulare strains originating from other countri
81 um strains were mig positive, and all of the M. intracellulare strains were mig negative.
82                                 In addition, M. intracellulare subsp.
83 ps which could be divided into 2 subspecies: M. intracellulare subsp.
84 alysis presented clade-specific proteins for M. intracellulare, such as PE and PPE protein families.
85 otide probes that specifically detect either M. intracellulare, the two M. avium subspecies associate
86    We compared the abilities of M. avium and M. intracellulare to tolerate the acidic conditions of t
87 ison of pretreatment and relapse isolates of M. intracellulare uncovered mutations in a previously un
88 identified functions essential for growth of M. intracellulare under conditions relevant to the host
89 as-induced apoptosis did not affect M. avium-M. intracellulare viability.
90                                              M. intracellulare was identified by nonsequencing method
91                                              M. intracellulare was more prevalent in women (1.33% of
92  reduction in CFU) of intracellular M. avium-M. intracellulare was observed only when M. avium-M. int
93                               One isolate of M. intracellulare was subsequently found to have been mi
94                We observed that M. avium and M. intracellulare were both tolerant to the acidic condi
95                                 Genotypes of M. intracellulare were confirmed by internal transcribed
96        However, when strains of M. avium and M. intracellulare were examined for their ability to ent
97 nd a clinical isolate of Mycobacterium avium-M. intracellulare were examined.
98                 Three MAC strains other than M. intracellulare were found to cross-react with the M.
99  that were present in M. avium but absent in M. intracellulare were identified, including some that m