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1 ic infection, and birth on site as risks for MAC infection.
2 aB to promote monocyte migration to sites of MAC infection.
3 ted diagnostic, and treatment strategies for MAC infections.
4 or disseminated Mycobacterium avium complex (MAC) infection.
5 d prevalence of Mycobacterium avium complex (MAC) infections.
6 ly resistant to Mycobacterium avium complex (MAC) infections.
7  investigating the influence of the level of MAC infection and the effect of other drugs on the frequ
8 xic, IL-12 is an effective immunotherapy for MAC infection, and combination with clarithromycin reduc
9                                  Thus, while MAC infections are typically opportunistic in humans and
10 RIM5alpha(AGM) is essential for blocking SIV(mac) infection at the stage of capsid uncoating.
11 nt of disseminated Myocardium avium complex (MAC) infection, beige mice were infected with MAC strain
12                 It appears that disseminated MAC infection can be cured by prolonged antimycobacteria
13 disorder causing increased susceptibility to MAC infections in a canine breed.
14 epidemiology of Mycobacterium avium complex (MAC) infections in children with and without human immun
15 nd disseminated Mycobacterium avium-complex [MAC] infection) in persons whose CD4(+) T lymphocyte cou
16 week study to determine whether treatment of MAC infection is associated with decreases in plasma tum
17 stages of AIDS, Mycobacterium avium complex (MAC) infection is the most common disseminated bacterial
18                 Mycobacterium avium complex (MAC) infection is the most common disseminated bacterial
19 nts of less than 50 cells/mL and subclinical MAC infection may be associated with a severe illness, c
20                       Thus, similar to SIVsm/mac infection of macaques and human AIDS, viral load is
21                        However, unlike SIVsm/mac infection of macaques, SIVlhoest and SIVsun infectio
22 ents to further define the mechanism whereby MAC infection of macrophages initiates monocyte migratio
23 ionship between Mycobacterium avium complex (MAC) infection of blood and bone marrow was studied in h
24     Prophylaxis or screening for subclinical MAC infection, or both, should therefore be done before
25  to the time of last sample, irrespective of MAC infection (P = .015).
26 suggest that with an initially high level of MAC infection, the addition of ethambutol may only delay
27 prophylaxis for Mycobacterium avium complex (MAC) infection to describe the specific signs, symptoms,
28 g therapy was 77 weeks, and the incidence of MAC infection was 1.44/100 person-years (95% confidence
29 lammatory response to previously subclinical MAC infection was associated with leucocytosis in all pa
30 e Schnauzers developing progressive systemic MAC infections were related to a common founder, and ped
31 rophages were treated either before or after MAC infection with different concentrations of IL-7.
32  of experimental murine Mycobacterium avium (MAC) infection with interleukin-12 (IL-12) significantly