ライフサイエンスコーパス: MAG

1 MAG (0-2.5wt%) had no remarkable impact on the chemical

2 MAG can display clarithromycin resistance through the in

3 MAG inhibition of axon outgrowth in some neurons is reve

4 MAG percentage was maximum (7mol%) at lower water activi

5 MAG significantly decreased the interfacial tension of S

6 "MAG" PTLs for ionotropic and metabotropic glutamate rece

7 MAG- and CSPG-dependent deacetylation of Miro1 requires

8 MAG-bound vesicles are deformed similarly, regardless of

9 MAG-PEI25 reached a maximum adsorption capacity of 6.11

10 iated glycoprotein to proteolipid protein 1 (MAG:PLP1) ratio, which declines in chronically hypoperfu

11 and transport of FAs, membrane lipids, and 2-MAG in rhizobia-soybean symbioses via the RAML-WRI-FatM-

12 These data show that active FA, 2-MAG and membrane lipid biosynthesis are essential for no

13 ed in glycolysis and the synthesis of FAs, 2-MAG, TAG, and membrane lipids compared to GmWRI1b-OE hai

14 ain that results in sn-2 monoacylglycerol (2-MAG) rather than LPA as the major product.

15 ysis, fatty acid (FA), 2-monoacylglycerol (2-MAG), and membrane lipid biosynthesis and transport duri

16 At low concentrations of 2-MAG (<50 microm), the major acylation product by DGAT1 w

17 TAG; however, increased concentrations of 2-MAG (50-200 microm) resulted in decreased TAG formation.

18 The possible roles of 2-MAGs as intermediates in cutin synthesis are discussed.

19 We recovered 36 MAGs, 29 of medium to high quality, and inferred their p

20 ) of an acylglycerol mixture (containing 67% MAGs) produced by enzymatic glycerolysis of sardine oil

21 d modulate alternative exon inclusion from a MAG minigene reporter.

22 rts binding of the myelin inhibitors Nogo-A, MAG (myelin-associated glycoprotein), and OMgp (oligoden

23 Three molecules, Nogo-A, MAG, and OMgp, are produced by oligodendrocytes and shar

24 ith anti-MAG demyelinating polyneuropathy (A-MAG-DP).

25 Thirteen A-MAG-DP patients were randomized to rituximab and 13 to p

26 irst drug that improves some patients with A-MAG-DP in a controlled study.

27 -Cys(336) is deleted and followed by a 13 aa MAG-binding motif of the NgR2 stalk, shows superior bind

28 MASS1 RNAi or a specific inhibitor abrogates MAG up-regulation.

29 osynthesis and export were identified across MAGs.

30 ns (based on Mash distances) between African MAGs and other publicly available genomes from the rumen

31 Although MAG:PLP1 tended to be lowest in cortex from patients wit

32 Significantly, an MAG basal to organisms from the phylum Thaumarchaeota th

33 The 12 analysed MAGs include representatives from four orders basal to t

34 shows superior binding of OMgp, Nogo-66, and MAG compared with wild-type NgR1 or NgR2.

35 Genetic deletion of either Cadm4 and MAG or Cadm4 and Caspr resulted in the formation of mult

36 The physical properties of DAG and MAG in the SSO may be related to the chemical stability

37 We also examined the influence of DAG and MAG on the physical properties of SSO.

38 lled or in assays where the gangliosides and MAG are not presented as part of fluid lipid bilayers.

39 We found that mating and MAG extract lower HC occurrence by 53% compared with all

40 To test the hypothesis that mating and MAG fluids inhibit a female's ability to induce HC in ma

41 asion showed the expression of both MUC1 and MAG.

42 me the inhibitory effects of both myelin and MAG for cortical, hippocampal, and DRG neurons.

43 ctively, act in concert to promote NF155 and MAG in maintaining the stable axo-glial interactions ess

44 of the axo-glial junction proteins NF155 and MAG, which interact to maintain the nodal complex.

45 ent in two major myelin inhibitors, Nogo and MAG, and their common receptor NgR1 (or NgR).

46 nal levels are commonly regulated by NT3 and MAG.

47 This NT3- and MAG-dependent axonal mRNA transport requires activation

48 NgR1 supports binding of Nogo-66, OMgp, and MAG.

49 SAG and MAG patients were propensity matched using 38 baseline c

50 zed with a supported lipid bilayer (SLB) and MAG, we detect vesicular GD1a and GT1b binding and deter

51 The release of FFAs and MAGs from TAGs proceeded faster than their incorporation

52 of triacylglycerides, formation of FFAs and MAGs, and micellar incorporation of carotenoids, FFAs an

53 ellar incorporation of carotenoids, FFAs and MAGs.

54 p 6.5 years) for patients receiving SAGs and MAGs.

55 Anti-MAG (myelin-associated glycoprotein) neuropathy is a dis

56 At month 8, IgM was reduced by 34% and anti-MAG titers by 50%.

57 immunological surrogate mouse model for anti-MAG neuropathy producing high levels of anti-MAG IgM was

58 toward an antigen-specific therapy for anti-MAG neuropathy.

59 th anti-myelin-associated glycoprotein (anti-MAG) neuropathy, an autoimmune disease of the peripheral

60 improved patients were those with high anti-MAG titers and most severe sensory deficits at baseline.

61 evidence of a treatment effect for IgM anti-MAG neuropathy and diabetic amyotrophy (radiculoplexus n

62 IgM levels, anti-MAG titers, B cells, antigen-presenting cells, and immun

63 enicity and demyelinating properties of anti-MAG autoantibodies are well established, current treatme

64 a correlation between the reduction of anti-MAG IgM levels and clinical improvement, an immunologica

65 MAG neuropathy producing high levels of anti-MAG IgM was developed.

66 selectively neutralizing the pathogenic anti-MAG antibodies with carbohydrate-based ligands mimicking

67 led study of rituximab in patients with anti-MAG demyelinating polyneuropathy (A-MAG-DP).

68 Patients with anti-MAG neuropathy showed substantial clonal expansions of b

69 Here, we study the mcr-containing archaeal MAGs from several hot springs, which reveal further expa

70 Analysis of all mcr-containing archaeal MAGs/genomes suggests a hydrothermal origin for these mi

71 hly expressed on adhesion molecules, such as MAG, present in myelinated nerve fibers.

72 d the composite outcome were similar between MAG and SAG patients at 1 year, but lower for MAG after

73 Finally, we show that hnRNPA1 binds MAG pre-mRNA and modulates alternative inclusion of MAG

74 itors and promotes neurite outgrowth on both MAG and CNS myelin substrates.

75 Conversely, the repulsion conferred by MAG or netrin-1 on adult growth cones is mediated by pro

76 ulture and in vivo to overcome inhibition by MAG and that spermidine can promote optic nerve regenera

77 he ability of cAMP to overcome inhibition by MAG in culture involves the upregulation of the enzyme a

78 AMP and putrescine to overcome inhibition by MAG is abolished in the presence of roscovitine, a Cdk i

79 ng lesion effect in overcoming inhibition by MAG is initially dependent on ongoing polyamine synthesi

80 trescine's ability to overcome inhibition by MAG.

81 verexpressing p35 can overcome inhibition by MAG.

82 Here we report that Cadm4-, MAG-, and Caspr-mediated adhesion cooperate during myeli

83 Here, we present an approach to characterize MAG-ganglioside interactions in real time, where MAG, GD

84 the concentration of triacylglycerols, DAG, MAG and free fatty acids (FFA) and the concentration of

85 aired trafficking of plasma membrane-derived MAG through the endolysosomal system in primary cells an

86 We report 1200 newly discovered MAGs from the rumen of Boran cattle.

87 AGs reveals two high-coverage, low-diversity MAGs from Piccard enriched in unique genes related to th

88 that hydrolyzes sialic acids and eliminates MAG-sialoglycan binding.

89 Before matching, 20% of procedures employed MAG.

90 or scalable production of n-3 PUFAs enriched MAGs as potential food emulsifier and ingredient.

91 in the acyltransferase function but exhibits MAG and LPC hydrolase activities.

92 The remaining five MAGs correspond to lineages that are only distantly rela

93 A focal MAG gradient induced polarized endocytosis and concomita

94 role for Nogo-A and synergistic actions for MAG and OMgp, presumably through shared receptors.

95 AG and SAG patients at 1 year, but lower for MAG after 7 years.

96 gated whether MUC1 is a counter-receptor for MAG and if their interaction contributed to pancreatic p

97 esults demonstrate an important new role for MAG molecules in mediating female post-mating behavior.

98 These results suggest a novel role for MAG/myelin in poor SC-myelin interaction and identify p7

99 on of NgR2, but not NgR1, are sufficient for MAG binding, and when expressed in neurons, exhibit cons

100 of the in vitro TAG synthesis initiated from MAG is mediated by DGAT1 in Caco-2 cell and rat intestin

101 nd that triacylglycerol (TAG) synthesis from MAG by DGAT1 does not behave according to classic Michae

102 Ganglioside-MAG interactions are often studied in cellular or animal

103 es, achieve complete (circularized, no gaps) MAGs (CMAGs).

104 ic lesions of multifocal atrophic gastritis (MAG) and intestinal metaplasia (IM) have occurred.

105 s of the ~2000 metagenome-assembled genomes (MAGs) available in the database revealed strong ecologic

106 Thermoplasmata metagenome-assembled genomes (MAGs) basal to the Methanomassiliicoccales, we show that

107 and obtaining metagenome-assembled genomes (MAGs) becomes more effective.

108 e obtained ten metagenome-assembled genomes (MAGs) belonging to potential methanogenic, anaerobic met

109 tase (Mcr), in metagenome-assembled genomes (MAGs) divergent to existing archaeal lineages.

110 ssembled draft metagenome-assembled genomes (MAGs) from environmental DNA extracted from two hot spri

111 reconstruct 73 metagenome-assembled genomes (MAGs) from two geochemically distinct vent fields in the

112 mes (SAGs) and metagenome-assembled genomes (MAGs) has led to a surge in genome-based discoveries of

113 entially novel metagenome-assembled genomes (MAGs) of core bacteria in Daphnia magna.

114 Metagenome-assembled genomes (MAGs) of these Petromonas sp. were obtained and used to

115 Our set of metagenome-assembled genomes (MAGs) represents >400 yet-unnamed genomospecies, substan

116 Metagenome-assembled genomes (MAGs) revealed that phenanthrene degradation is likely m

117 umen microbial metagenome-assembled genomes (MAGs) using approximately 6.5 terabases of short- and lo

118 near-complete metagenome-assembled genomes (MAGs), and there is a need for reproducible pipelines th

119 g obtained 527 metagenome-assembled genomes (MAGs), representing 150 bacterial species.

120 pathways in 64 metagenome-assembled genomes (MAGs), we show that MAG transcript abundance can be tied

121 vo assembly of metagenome-assembled genomes (MAGs).

122 mes (SAGs) and metagenome-assembled genomes (MAGs).

123 n by obtaining metagenome-assembled genomes (MAGs); but gaps, local assembly errors, chimeras, and co

124 Thermoplasmatota genomes/MAGs found no evidence of mcrA homologues outside of the

125 and maturation of the male accessory gland (MAG) in the red flour beetle, Tribolium castaneum.

126 nt with extracts from male accessory glands (MAG), which make seminal fluid molecules, female Ae. aeg

127 e substrates myelin-associated glycoprotein (MAG) and chondroitin sulfate proteoglycans (CSPGs).

128 (NF155) and myelin-associated glycoprotein (MAG) at axo-glial junctions.

129 contain the myelin-associated glycoprotein (MAG) but not P(0) or proteolipid protein (PLP), the stru

130 ant for PirB-myelin-associated glycoprotein (MAG) ectodomain interactions.

131 covered that myelin-associated glycoprotein (MAG) expression is dramatically decreased in Frings mice

132 umulation of myelin-associated glycoprotein (MAG) in LAMP1(+)perinuclear vesicles that fail to migrat

133 irectly with myelin associated glycoprotein (MAG) in myelin, resulting in reduced activation of growt

134 Myelin-associated glycoprotein (MAG) is a sialic acid-binding Ig-family lectin that func

135 in (MBP) and myelin-associated glycoprotein (MAG) myelin proteins were markedly increased in the cort

136 the axon and myelin-associated glycoprotein (MAG) on myelin.

137 (NF155) and myelin-associated glycoprotein (MAG) require membrane microdomains containing either sul

138 nstrate that myelin-associated glycoprotein (MAG), a well known inhibitor of neurite outgrowth, inhib

139 , as well as myelin-associated glycoprotein (MAG), are enriched at the internodes below the compact m

140 e inhibitor, myelin-associated glycoprotein (MAG), binds to sialoglycans and other receptors on axons

141 nous lectin, myelin-associated glycoprotein (MAG), is reported to bind to axonal gangliosides (GD1a a

142 olecule, the myelin-associated glycoprotein (MAG), located in the adaxonal plasmalemma of myelin-prod

143 tors such as myelin-associated glycoprotein (MAG), Nogo-A, and oligodendrocyte-myelin glycoprotein.

144 lin, such as myelin-associated glycoprotein (MAG), represent major obstacles to axonal regeneration f

145 nts encoding myelin-associated glycoprotein (MAG), which generates two protein isoforms that associat

146 (JAM-C) and myelin-associated glycoprotein (MAG).

147 lin, such as myelin-associated glycoprotein (MAG).

148 n induced by myelin-associated glycoprotein (MAG).

149 3 (NT3) and myelin-associated glycoprotein (MAG).

150 tion such as myelin-associated glycoprotein (MAG).

151 netrin-1 and myelin-associated glycoprotein (MAG).

152 -1beta) and myelin-associated glycoproteins (MAG, Nogo).

153 val advantage of multiple arterial grafting (MAG) vs the standard use of left internal thoracic arter

154 ry isolated coronary artery bypass grafting (MAG, n = 5580; LITA+SVG, n = 14496) in the province of B

155 tion in the use of multiple arterial grafts (MAGs) versus single arterial grafts (SAGs) for patients

156 embers of the Mycobacterium abscessus group (MAG) cause lung, soft tissue, and disseminated infection

157 tional resistance, as well as slowly growing MAG strains and also strains displaying an inducible res

158 We highlight MAGs that were taxonomically assigned to groups previous

159 We further demonstrate how MAG-ganglioside interactions can be disrupted by antigan

160 However, MAG:PLP1 showed a significant negative correlation with

161 The decline in MAG:PLP1 strongly suggests pathological hypoperfusion of

162 We show here that incisures are present in MAG-null and absent from P(0)-null PNS internodes.

163 that SPD is able to concentrate n-3 PUFAs in MAG form by distilling at proper TE e.g. 125 degrees C,

164 -T (-/-) mice exhibited a major reduction in MAG protein levels in CNS myelin compared with WT and si

165 Amino acid exporters were identified in MAGs identified as important for host fitness, and pathw

166 C marker S100 and MBP expressions increased; MAG, GFAP, and SCMP expressions were very low.

167 that from wild-type mice, but myelin lacking MAG and OMgp is indistinguishable from control.

168 higher volume MAG surgeons experienced lower MAG mortality.

169 was no mortality difference between matched MAG and SAG patients (2.4% vs. 2.2%, adjusted hazard rat

170 The MILLIPLEX-MAG Rat cytokine-chemokine magnetic bead array was used

171 resulted in rapid identification of modified MAGs in biofluids by displaying doublet peak characteris

172 f boron specialized the presence of modified MAGs in MS and led to distinctive identification.

173 e fatty acids, FFAs, and monoacylglycerides, MAGs) during in vitro digestion of oil-in-water emulsion

174 tic activity for acylating monoacylglycerol (MAG).

175 f diacylglycerol (DAG) and monoacylglycerol (MAG) on the oxidative stability of stripped soybean oil

176 yzes the acylation of both monoacylglycerol (MAG) and diacylglycerol (DAG) to generate DAG and TAG, r

177 s revealed the presence of monoacylglycerol (MAG) and lysophosphatidylcholine (LPC) hydrolytic activi

178 Pretreatment with the monoacylglycerol (MAG) lipase inhibitor JZL-184 also reduced affective dis

179 aroyl lactylate (SSL) and monoacylglycerols (MAG) and Bacillus stearothermophilus maltogenic alpha-am

180 diacylglycerols (DAG) and monoacylglycerols (MAG) with a high content of polyunsaturated fatty acids

181 Monoacylglycerols (MAGs) are active mediators involved in multiple biologic

182 Production of monoacylglycerols (MAGs) rich in omega-3 polyunsaturated fatty acids (n-3 P

183 Antibodies against the MPZ, MAG, S100, and SCMP proteins immunostained along pericor

184 nd flow cytometry, and this occurred in NAG, MAG and IM.

185 d substrata adsorbed with full-length native MAG extracted from purified myelin.

186 A soluble proteolytic fragment of native MAG, dMAG, also inhibited neurite outgrowth.

187 DGAT1 (IC(50) of human DGAT1: 16.6+/-4.0 nM (MAG as substrate) and 1499+/-318 nM (DAG as substrate);

188 Nogo, MAG and OMgp are three prototypical myelin inhibitors th

189 f myelin-associated inhibitors such as Nogo, MAG, and OMgp.

190 icity, is a high-affinity receptor for Nogo, MAG, and OMgp.

191 proteins present in myelin, including Nogo, MAG, and oligodendrocyte-myelin glycoprotein (OMgp), hav

192 of the three major myelin inhibitors, Nogo, MAG, and OMgp, in injury-induced axonal growth, includin

193 Three proteins in mature myelin (Nogo, MAG, and OMgp) have been purported to be critical in cau

194 Three proteins found in myelin--Nogo, MAG, and OMgp--inhibit axon regeneration in vitro and bi

195 Our data indicate that while Nogo, MAG, and OMgp may modulate axon sprouting, they do not p

196 ration beyond the injury site in either Nogo/MAG/NgR1 triple mutants or NgR1 single mutants.

197 tially rescues neurite inhibition by Nogo66, MAG, OMgp, and myelin in cultured neurons.

198 erebral amyloid angiopathy, neither VEGF nor MAG:PLP1 correlated significantly with the severity of s

199 We here showed that SSL but not MAG delays wheat starch hydrolysis by BStA.

200 VEGF but not MAG:PLP1 increased with Alzheimer's disease severity, as

201 r that selectively inhibits the acylation of MAG by DGAT1 (IC(50) of human DGAT1: 16.6+/-4.0 nM (MAG

202 The addition of MAG (0.5wt%) suppressed the effectiveness of alpha-tocop

203 s and subgroups support the consideration of MAG for a broader spectrum of patients who are undergoin

204 iency was thought to underlie the defects of MAG splicing in the qk(v) mutant.

205 In contrast, deletion of MAG and OMgp stimulates neither axonal growth nor enhanc

206 show that the first three Ig-like domains of MAG bind with high affinity and in a sialic acid-depende

207 OLs completely rescues the dysregulation of MAG splicing without increasing expression or nuclear ab

208 ing I (QKI) leads to severe dysregulation of MAG splicing.

209 The ectodomain of MAG is comprised of five Ig-like domains and uses neuron

210 dbcAMP), can block the inhibitory effects of MAG and myelin.

211 inhibitory and repulsive turning effects of MAG in vitro.

212 viously shown to bind an intronic element of MAG and modulate alternative exon inclusion from a MAG m

213 maturation of MAG by promoting the growth of MAG cells.

214 The growth of MAG was impaired after double-stranded RNA (dsRNA)-media

215 Domains Ig3-Ig5 of MAG are sufficient to inhibit neurite outgrowth but fail

216 -mRNA and modulates alternative inclusion of MAG exons.

217 ral HNK-1 epitope blocked the interaction of MAG with pathogenic IgM antibodies from patient sera but

218 g through IIS pathway regulate maturation of MAG by promoting the growth of MAG cells.

219 ilar effects on the growth and maturation of MAG.

220 pathways to regulate myelination by means of MAG protein stability in myelin-forming cells of the aud

221 nsistent with C1q-mediated neutralization of MAG.

222 compare the safety and long-term outcomes of MAG vs LITA+SVG among overall and selected subgroups of

223 the binding pocket in the 2 photoisomers of MAG and (ii) the degree of clamshell closure that is pos

224 arginine 118 in the extracellular portion of MAG, but it is independent of Nogo signaling in the axon

225 enhance neurite outgrowth in the presence of MAG.

226 romotes neurite outgrowth in the presence of MAG.

227 hibit HC indirectly via the broader range of MAG-induced female refractory mating behaviors.

228 The size of MAG increased from day 1 to day 5 post-adult emergence (

229 This increase in the size of MAG is contributed by an increase in cell size, but not

230 ed to be essential in the metabolic study of MAG-related diseases.

231 MASS1 inhibits the ubiquitylation of MAG, thus enhancing the stability of this protein, and t

232 A synthesized short version of MAG turns the channel on in either the cis or trans stat

233 on emulsion presented a higher conversion of MAGs to FFAs during digestion, which led to a higher con

234 traightforward approach by derivatization of MAGs with 3-nitrophenylboronic acid (3-NPB) for sensitiv

235 Sensitive and unambiguous detection of MAGs is thus essential; however, previous methods are bo

236 The majority of MAGs linked to oil-contaminated ecosystems were detectab

237 between boronic acid and cis-diol moiety of MAGs blocked the formation of multiple adduct ions and t

238 with 91% purity and 94% overall recovery of MAGs.

239 r evoluted to reveal the amount variation of MAGs by d(0)-NPB and d(4)-NPB separately derivatized in

240 ce extend significantly shorter processes on MAG compared with wild-type DRG neurons, and regeneratio

241 showed that the sialic acid binding site on MAG maps to arginine 118 in Ig domain 1.

242 ttraction to gradients of cAMP, netrin-1, or MAG is mediated by Epac.

243 en virgin males and either virgin, mated, or MAG extract-injected females and analyzed these recordin

244 evealed that increased expression of MUC1 or MAG enhanced adhesion.

245 Finally, when adding SSL or MAG on top of BStA to starch suspensions, the effect of

246 an assign genomes from isolate sequencing or MAGs to species-level genome bins built from >230,000 pu

247 Collectively, our MAGs represented about half of the total microbial commu

248 ssential source of cAMP for BDNF to overcome MAG-dependent inhibition of neurite outgrowth.

249 Mechanistically, MT-I/II ability to overcome MAG-mediated inhibition is transcription-dependent, and

250 quired for dbcAMP and putrescine to overcome MAG-mediated inhibition.

251 by antiganglioside antibodies that override MAG-based neuron growth inhibition.

252 uivalently premixed with d(0)-NPB to perform MAG derivatization, which resulted in rapid identificati

253 iGluR6 using a family of photoiosomerizable MAG (maleimide-azobenzene-glutamate) PTLs that covalentl

254 only at monoacylglycerol:native phytosterol (MAG:NPS) ratios of 10:0, 7:3 and 5:5.

255 se structural findings and the observed PirB-MAG interactions are compatible with a model for interce

256 r myelin genes, such as proteolipid protein, MAG, MBP, and myelin oligodendrocyte glycoprotein, were

257 nomassiliicoccales, including a high-quality MAG that likely represents a new order, Ca. Lunaplasma l

258 tially increasing the number of high-quality MAGs from freshwater lakes.

259 .1 (4.5-11.7) years for the groups receiving MAG and LITA+SVG, respectively.

260 of blood IgM memory B cells that recognized MAG antigen.

261 Over 95% of the recovered MAGs represented novel taxa underscoring the limited rep

262 echanism in which C1q binding to MAG reduces MAG signaling to neurons, complement C1q blocked both th

263 Our set of rumen MAGs increases the rate of mapping of rumen metagenomic

264 Metagenome-assembled genome sequences (MAGs) representing allochthonous populations were detect

265 vation of PKC and Rho in response to soluble MAG.

266 ome metabolic pathways were specific to some MAGs, including sulfur oxidation, nitrate reduction, and

267 ify 1200 high-quality African rumen-specific MAGs and provides further insight into the rumen functio

268 sted the presence of two separate substrate (MAG and LPC)-binding sites in a single polypeptide.

269 Comparison of nine Sulfurovum MAGs reveals two high-coverage, low-diversity MAGs from

270 Compared with LITA+SVG, MAG is associated with reduced mortality, repeated revas

271 Compared with LITA+SVG, MAG was associated with reduced mortality rates (hazard

272 is study sought to compare intermediate-term MAG and SAG outcomes with enhanced matching to reduce se

273 enome-assembled genomes (MAGs), we show that MAG transcript abundance can be tied to differences in m

274 s report establishes for the first time that MAG also promotes resistance to axonal injury and preven

275 In frontal cortex the MAG:PLP1 ratio was significantly reduced in Alzheimer's

276 hat is possible given the disposition of the MAG linker.

277 e, considering the multivalent nature of the MAG-IgM interaction, polylysine polymers of different si

278 mune system evasion were detected across the MAGs.

279 These MAGs are predicted to use diverse energy conservation pa

280 These MAGs were used as reference database to investigate the

281 We compared these MAGs to closely related genomes and show that these subs

282 total, our examination of functions in these MAGs shows a diversity of nutrient acquisition and metab

283 Five of these MAGs represent under-sampled (Verstraetearchaeota, Metha

284 Only approximately 8% of these MAGs were abundant in U.S. freshwater ecosystems, reveal

285 glycerol was proven to be an alternative to MAG as acyl-group acceptor.

286 is mediated by IgM autoantibodies binding to MAG antigen.

287 -enhancing mechanism in which C1q binding to MAG reduces MAG signaling to neurons, complement C1q blo

288 gamma-secretase activity before exposing to MAG or CNS myelin improves SC migration and survival in

289 Exposing neurons to MAG and CSPGs decreases acetylation of Miro1 on Lysine 1

290 formation of multiple adduct ions and tuned MAGs to negatively charged carrying species.

291 ficacy of two-photon (2P) excitation for two MAG molecules using nonlinear spectroscopy.

292 Of 5580 participants who underwent MAG, 586 (11%) were women and the mean (SD) age was 60 (

293 Patients of higher volume MAG surgeons experienced lower MAG mortality.

294 ganglioside interactions in real time, where MAG, GD1a, and GT1b contents are controlled and they are

295 which remained in an amorphous form, whereas MAG led to strong scattering, indicating the formation o

296 urther work is required to determine whether MAG dysregulation is a cause or consequence of audiogeni

297 At 7 years, the subgroups for which MAG did not have a lower mortality rate included patient

298 ction in EDN1, and positive correlation with MAG:PLP1, in the hypoperfused white matter in Alzheimer'

299 NPS co-crystallised with MAG in the oleogel mixtures and influenced the growth of

300 At 7 years, MAG patients had lower mortality (12.7% vs. 14.3%, AHR: