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1 ats received pre-session injections of known MAO inhibitors.
2 thesized and evaluated as monoamine oxidase (MAO) inhibitors.
3 onooxygenase (DbetaM) and monoamine oxidase (MAO) inhibitors.
4 ) revealed a surprisingly high percentage of MAO inhibitors (16%) with a low false hit rate (0.9%).
5  the differentiation of the selectivity of a MAO inhibitor against rat brain MAO A and B in vivo.
6                                      The two MAO inhibitors also counteracted the increase in the DOP
7 viors that can be reversed by treatment with MAO inhibitors, as well as the tricyclic antidepressant
8  rats pretreated with the monoamine oxidase (MAO) inhibitor clorgyline.
9 rs were also treated with the antidepressant MAO inhibitor drug, tranylcypromine (10 mg/day for 3 day
10 line ligand agmatine, the monoamine oxidase (MAO) inhibitor harmane, the alpha(2)-adrenoceptor agonis
11 arge dietary intake of tyramine while taking MAO inhibitors has been reported; therefore, the measure
12 nal flexibility and structural properties of MAO inhibitors in tuning their isoform specificities.
13 show that (1) tranylcypromine (TCP), a known MAO inhibitor, increases sensitivity to the primary rein
14  unilaterally 2 h after pretreatment with an MAO inhibitor into left or right nucleus accumbens septi
15                   Addition of the monoamine (MAO) inhibitors, l-deprenyl, clorgyline, pargyline, or i
16 or its function in the brain; small molecule MAO inhibitors (MAOIs) are clinically used for treating
17 s the application and therapeutic benefit of MAO inhibitors (MAOIs) for cancer treatment, highlightin
18 ake; accordingly, it has been suggested that MAO inhibitors may represent a therapeutic alternative i
19            The effects of monoamine oxidase (MAO) inhibitors on the metabolism of dopamine synthesize
20 y the amine uptake inhibitor imipramine, the MAO inhibitor pargyline and the MEK inhibitor PD 98059.
21 as inhibited at the enzymatic level with the MAO inhibitor pargyline.
22 brain cortex in vitro in the presence of the MAO inhibitor pargyline.
23                                              MAO inhibitors prevent this action.
24     It is believed that a monoamine oxidase (MAO) inhibitor specific for MAO A, which is reversibly b
25 ministration of 15 mg/kg of the nonselective MAO inhibitor tranylcypromine.
26 vo were evaluated after application to three MAO inhibitors, tranylcypromine, clorgyline, and pargyli
27  effect" (hypertension during treatment with MAO inhibitors usually caused by ingestion of foods cont
28 ses (MAO) A and B following a single dose of MAO inhibitors was developed through the simultaneous de
29 cal features with a preferential response to MAO inhibitors, which is especially well-evidenced for r