ライフサイエンスコーパス: MCA

1 MCA fitness (1+s) was determined by comparing the change

2 MCA pseudofeeders and hydrocephalus were risk factors fo

3 MCA recanalization after endovascular treatment was achi

4 MCA V(mean), P(aCO2) and cerebral vascular conductance i

5 MCA-related symptoms may be mild, moderate, severe, or e

6 MCAs were mounted in a wire myograph.

7 3 arterial segments were treated (10 ICA, 15 MCA, and 8 ACA).

8 sic electronic structure of the [B12X12](2-) MCAs provides the basis for a molecular level understand

9 , and t-butoxycarbonyl-Gln-Ala-Arg-7-amido-4-MCA fluorescent derivatives were used to assess chymotry

10 CA), benzyloxycarbonyl-Leu-Leu-Glu-7-amido-4-MCA, and t-butoxycarbonyl-Gln-Ala-Arg-7-amido-4-MCA fluo

11 Forty patients with 41 MCA aneurysms treated in a single centre were included i

12 with 5-methylphenazine-1-carboxylic acid (5-MCA) and pyocyanin (PYO) localized to the colony edge.

13 PYO), 5-methylphenazine-1-carboxylic acid (5-MCA), and 1-hydroxyphenazine (OHPHZ), which have toxic e

14 indicates an effect of O2 availability on 5-MCA synthesis.

15 concentrations of PYO and highly reactive 5-MCA were determined in different polymicrobial culture s

16 A MCA prepared for chemical sensing was exposed to the sam

17 gas chromatograph and then introduced into a MCA flow cell.

18 ntional 7-methoxy-coumarin-4-yl acetic acid (MCA)/Lys(DNP) substrates; thus, substantially lower amou

19 he implementation of melamine-cyanuric acid (MCA) supramolecular aggregates and microwave-assisted th

20 of blood-brain barrier monocarboxylic acid (MCA) transport may contribute to the maintenance of brai

21 nonglucose substrates (monocarboxylic acids [MCAs]) by upregulation of the MCA transporters may contr

22 Mast cell activation (MCA) occurs in a number of different clinical conditions

23 rder to maximize its milk-clotting activity (MCA).

24 well as double mid1-complementing activity (MCA) channel mutants did not affect the response.

25 tive treatment option in patients with acute MCA occlusion with contraindication to IVT or after IVT

26 Neurologic deficit at admission, MCA recanalization, symptomatic intracerebral hemorrhage

27 ed threshold of 120 cm/sec for less advanced MCA spasm was significantly better than that of TCD at s

28 vanced, and 200 cm/sec for the more advanced MCA narrowing.

29 le, Medieval Climate Anomaly-Little Ice Age, MCA-LIA).

30 tivity and safety, microbial control agents (MCAs) appear to be ready-made components of integrated p

31 s were cultured as multicellular aggregates (MCA), cyclin D1 was induced through a serum-dependent EG

32 ure the growth of multi-cellular aggregates (MCAs), of increasing metastatic potential in different e

33 on the surface of multicellular aggregates (MCAs) are removed by treatment with LPA.

34 ation of cells and multicellular aggregates (MCAs) from the tumor into the peritoneal cavity, adhesio

35 ng tumor cells and multicellular aggregates (MCAs) present in ascites fluid adhere to the peritoneum

36 dividual cells and multicellular aggregates (MCAs).

37 monstrated Km and kcat/Km values for Lys-Ala-MCA of 688 muM and 11.02 muM(-1) s(-1), respectively, an

38 rithm and a medoid classification algorithm (MCA), are examined.

39 well as tauro-alpha-muricholic acid (T-alpha-MCA) are potential markers for Geniposide-induced hepati

40 , making lactate the most likely alternative MCA as brain fuel.

41 ation enzyme mycothiol- S-conjugate amidase (MCA) from Mycobacterium tuberculosis are reported.

42 Michaelis complex analogue AK:Arg:Mg.AMPPNP (MCA), a product complex analogue AK:pAIE:Mg.ADP (PCA), a

43 We developed an MCA and an SCA that utilized either intact or env-delete

44 tion into tumor-associated macrophages in an MCA-203 fibrosarcoma mice model.

45 ory processing with maximal causes analysis (MCA), a sparse coding model which captures both the non-

46 oposes a microfluidic colorimetric analysis (MCA) system composing of a wax-printed paper-microchip a

47 t the methods of metabolic control analysis (MCA) to determine which modifications are important for

48 d analysis using metabolic control analysis (MCA), metabolomics data, and statistical simulations.

49 is by multidimensional correlation analysis (MCA) is revisited.

50 The DT and MCA classifiers are sufficiently flexible and can produc

51 /- 11 vs. 336 +/- 14 W), and lowered ICA and MCA Vmean by 12-23% without compromising CCA blood flow.

52 In all conditions, reductions in ICA and MCA Vmean were associated with declining cerebral vascul

53 38.5%), and 26 (54.2%) with ICA, MCA-M1, and MCA-M2 occlusions, respectively, achieved a modified Ran

54 of 22) among patients with ICA, MCA-M1, and MCA-M2 occlusions, respectively.

55 me 16q24.1q24.2 in patients with ACD/MPV and MCA.

56 unrelated patients with sporadic ACD/MPV and MCA.

57 ciated behaviors of ovarian cancer cells and MCAs are influenced by cellular cadherin composition.

58 ymal N-cadherin-expressing (Ncad+) cells and MCAs invade much more efficiently than E-cadherin-expres

59 he adhesive capacity of EOC single cells and MCAs to murine peritoneal explants and impaired MCA surv

60 oulomb repulsion in multiply charged anions (MCAs) creates a potential barrier that provides dynamic

61 process of numerous multiply charged anions (MCAs) have been traditionally explained in terms of the

62 sic MA, i.e., magnetocrystalline anisotropy (MCA), typically depends upon crystal symmetry.

63 ers (ASD), or multiple congenital anomalies (MCA).

64 sociated with multiple congenital anomalies (MCA).

65 te that during the Medieval Climate Anomaly (MCA) (900-1300 AD) the Pacific Northwest experienced exc

66 nt) and during the Medieval Climate Anomaly (MCA) (~1 ka to 0.7 ka).

67 ynamics during the Medieval Climate Anomaly (MCA) and the Little Ice Age (LIA) are poorly characteriz

68 ability during the Medieval Climate Anomaly (MCA) between 1,200 and 850 y B.P., when temperatures wer

69 peaked during the Medieval Climate Anomaly (MCA) from A.D. 900 to 1100, providing evidence that the

70 the effect of the Medieval Climate Anomaly (MCA) on the precipitation over the tropics is still poor

71 routes during the Medieval Climate Anomaly (MCA), A.D. 800-1300, when the initial colonization of CE

72 states, during the Medieval Climate Anomaly (MCA).

73 During the Medieval Climate Anomaly (MCA; approximately 1,000-500 cal B.P.), the period most

74 B12X12](2-), X = F, Cl, Br, I, At, which are MCAs with not well-separated excess charges and cannot b

75 age site peptide (Ac-Asp-Arg-Glu-Ile-Nle-Arg-MCA) is used as the substrate of separase.

76 The AONP-modified MC array (MCA) were subsequently functionalized by being immersed

77 chromatograph with a microcantilever array (MCA) is demonstrated for enhanced selectivity and potent

78 nvestigated in rat middle cerebral arteries (MCA).

79 ed and pressurized middle cerebral arteries (MCAs) and PAs taken from the same animals.

80 halus, and so-called middle cerebral artery (MCA) "pseudofeeders" were correlated with cardiac failur

81 bserved in the right middle cerebral artery (MCA) (166 +/- 27 cm/s to 135 +/- 27 cm/s, P < .001) and

82 gations of the right middle cerebral artery (MCA) and both common carotid arteries (CCAs).

83 ary occlusion of the middle cerebral artery (MCA) causing damage to brain tissue occurs in the majori

84 he M1 segment of the middle cerebral artery (MCA) for 1 h.

85 ures after malignant middle cerebral artery (MCA) infarction with decompressive hemicraniectomy (DHC)

86 The middle cerebral artery (MCA) is the second most common location of intracerebral

87 nal occlusion of the middle cerebral artery (MCA) is used extensively in cerebral ischemia research.

88 n, 10 min, or 2 h of middle cerebral artery (MCA) ischemia using the suture (thread) model.

89 ly attenuated in the middle cerebral artery (MCA) isolated from female compared to male rats.

90 ing 1-hour bilateral middle cerebral artery (MCA) microembolic signals (MES).

91 tients with isolated middle cerebral artery (MCA) occlusion and early recanalization at MR angiograph

92 jury associated with middle cerebral artery (MCA) occlusion and reperfusion.

93 stroke (AIS) due to middle cerebral artery (MCA) occlusion were enrolled; 75 underwent IVT.

94 cose deprivation and middle cerebral artery (MCA) occlusion.

95 -rich thrombi in the middle cerebral artery (MCA) of mice.

96 tery (ACA), and / or middle cerebral artery (MCA) secondary to SAH due to an aneurysm treated with en

97 al carotid artery or middle cerebral artery (MCA) stroke and to evaluate the relationship with treatm

98 ischemic changes in middle cerebral artery (MCA) stroke and to retrospectively compare admission non

99 acrificed for either middle cerebral artery (MCA) structure and function assessments via ex vivo pres

100 nist tirofiban, in a middle cerebral artery (MCA) thrombosis model in guinea pigs.

101 opic emboli into the middle cerebral artery (MCA) via an indwelling carotid artery catheter.

102 Occlusion of the middle cerebral artery (MCA) with an endovascular filament is a widely used mode

103 clusion of the right middle cerebral artery (MCA), and anesthesia was either switched to alpha-chlora

104 (at proximal/distal middle cerebral artery (MCA), within/beyond diffusion-weighted imaging (DWI) les

105 emical injury to the middle cerebral artery (MCA).

106 ent occlusion of the middle cerebral artery (MCA).

107 cauterization of the middle cerebral artery (MCA).

108 CBF velocity at the middle cerebral artery (MCA).

109 oppler (aTCD) on the middle cerebral artery (MCA): MCA pulsatility index (PIa) and an estimator based

110 basilar artery (BA), middle cerebral artery (MCA)], the submandibular gland (SMG), and pineal gland w

111 Eln(+/+) mice in the middle cerebral artery (MCA, P < 0.001), but was similar between groups in the g

112 he M1 segment of the middle cerebral artery (MCA; 52 patients: median NIHSS score, 13 [interquartile

113 an those with tandem internal carotid artery-MCA occlusion and early recanalization (P = .05).

114 ither multiple-cycle or single-cycle assays (MCAs or SCAs); these assays have not been systematically

115 n the flanks of normal mice and mice bearing MCA 205 lung metastases.

116 T-beta-MCA is an farnesoid X receptor (FXR) nuclear receptor an

117 ntestinal tauro-beta-muricholic acid (T-beta-MCA).

118 alignant potential of lesions established by MCA inoculation, whereas inhibition of IL-12/23p40 enhan

119 by 25% following an acute stroke induced by MCA occlusion for 90 min.

120 rize the generative fields (GFs) inferred by MCA with respect to in vivo neural responses in A1 by ap

121 velocity (MCA V(mean)), and calculated CBVC (MCA V(mean)/MAP) were assessed in seven healthy individu

122 he Medieval Climate Anomaly (950 to 1250 CE, MCA), a period which coincided with glacier retreat.

123 tro however, the fate of free-floating cells/MCAs and the response of host peritoneal tissues to LPA

124 rowth and morphology of these cell clusters; MCAs grow larger and faster in the more rigid environmen

125 , OVA-expressing EG7 lymphoma cells and CMS5 MCA-induced fibrosarcoma cells naturally expressing muta

126 dditional infarction [Subtotal MCAAI] and co-MCA with additional infarction [Co-MCAAI].

127 difference in MIV of subtotal infarction, co-MCA, Subtotal MCAAI but the outcome was significantly di

128 subgroups by CT, subtotal, complete MCA [co-MCA], Subtotal MCA with additional infarction [Subtotal

129 le outcome were co-MCAAI, subtotal MCAAI, co-MCA and subtotal MCA infarction.

130 ial sheets and the formation of more compact MCAs relative to parental cells.

131 analysis of baseline variables, and complete MCA recanalization for the prediction of favorable clini

132 nto four subgroups by CT, subtotal, complete MCA [co-MCA], Subtotal MCA with additional infarction [S

133 small clusters compared to untreated control MCAs.

134 ducts, the actual feasibility of the current MCA system can be demonstrated.

135 72 h later in neurons throughout the entire MCA territory in many but not all animals.

136 EOC MCAs displayed significant LPA-induced changes in surfac

137 effectively suppressed growth of established MCA-induced tumors or TRAMP-C1 prostate tumors and inhib

138 Despite restoration of P(ETCO2), MCA V(mean) (P = 0.005) and CBVC (P = 0.03) remained red

139 C, which resulted in decreases in P(ETCO2), MCA V(mean) and calculated CBVC (P < 0.001 for all varia

140 MP levels were comparable in male and female MCAs (1.7 pmol/mg protein).

141 After 1 h of focal MCA ischemia, the coil was retrieved to cause reperfusio

142 Tumors from MCA/BHT-treated Rosa26-Foxm1 mice displayed a significan

143 he CARS signals from cellular sheddings from MCAs with LPA treatment are consistent with cleavage of

144 Furthermore, MCAs formed from MT1-MMP-T567E-expressing cells adhere a

145 Gly-MCA is a selective high-affinity FXR inhibitor that can

146 mice with glycine-beta-muricholic acid (Gly-MCA) inhibits FXR signalling exclusively in intestine, a

147 high-affinity FXR agonist GW4064 blocks Gly-MCA action in the gut, and intestine-specific Fxr-null m

148 eramide treatment reverses the action of Gly-MCA in high-fat diet-induced obese mice.

149 nresponsive to the beneficial effects of Gly-MCA.

150 These data suggest that Gly-MCA may be a candidate for the treatment of metabolic di

151 tically, the metabolic improvements with Gly-MCA depend on reduced biosynthesis of intestinal-derived

152 Thirty-one fetuses (53%) had MCA pseudofeeders.

153 tom onset in 51 patients suspected of having MCA stroke were reviewed.

154 To our knowledge, togetherness of hyperdense MCA sign and pulmonary thromboembolism is extremely rare

155 We conclude that during hypoglycemia MCA transport in the brain was increased by approximatel

156 ate day, however, exercise capacity and ICA, MCA Vmean and CCA dynamics were preserved.

157 38.5%), 20 (38.5%), and 26 (54.2%) with ICA, MCA-M1, and MCA-M2 occlusions, respectively, achieved a

158 and 40.9% (9 of 22) among patients with ICA, MCA-M1, and MCA-M2 occlusions, respectively.

159 s to murine peritoneal explants and impaired MCA survival and mesothelial clearance competence.

160 Research toward improving MCA efficacy and economic competitiveness is required to

161 d by P(ETCO2), contribute to the decrease in MCA V(mean) and CBVC; however, other factors (e.g. perha

162 are known to play important roles either in MCA tumorigenesis or in the elimination phase of cancer

163 We show that the endothelium in MCA/129 fibrosarcomas and B16 melanomas exhibits a wild-

164 ve stress marker nitrotyrosine was higher in MCAs from Eln(+/-) compared with Eln(+/+) mice (P < 0.05

165 6 mum in vehicle; P<0.05) in PAs, but not in MCAs.

166 An understanding of these processes in MCAs with not well-separated excess charges is still lac

167 normothermic level (P = 0.99) and increased MCA V(mean) (P = 0.002) and CBVC (P = 0.008) relative to

168 e data suggest that in addition to increased MCA transport at the blood-brain barrier, there may be a

169 O2) and cerebral vascular conductance index (MCA V(mean)/MAP) were lower in the older group at each e

170 ts previously shown to competitively inhibit MCA.

171 INTERPRETATION: MCA pseudofeeders are the result of impaired cerebral bl

172 ons during the Early (925-1150 C.E.) to Late-MCA (1150-1350 C.E.).

173 ements from Paredao during the Early to Late-MCA are coherent with a reduction in water supply.

174 cm/s to 135 +/- 27 cm/s, P < .001) and left (MCA) (168 +/- 26 cm/s to 142 +/- 27 cm/s, P < .001) velo

175 risk of developing seizures after malignant MCA stroke with DHC, especially after cranioplasty.

176 s increases growth rates, and change maximum MCA sizes.

177 raction conditions, corresponding to maximum MCA (9.550CAU/mL), were selected as follows: Grinding ti

178 (aTCD) on the middle cerebral artery (MCA): MCA pulsatility index (PIa) and an estimator based on di

179 recently observed as a long-lived metastable MCA with a large negative electron binding energy of -0.

180 rbamate (urethane), or 3-methylcholanthrene (MCA) and butylated hydroxytoluene (BHT) was used to indu

181 sed the development of 3-methylcholanthrene (MCA)-induced fibrosarcomas through a mechanism relying u

182 6-Foxm1 mice using the 3-methylcholanthrene (MCA)/butylated hydroxytoluene (BHT) lung tumor initiatio

183 tal tumor metastases and methylcholanthrene (MCA)-induced sarcomas in mice deficient for NLRP3 was NK

184 nged with the carcinogen methylcholanthrene (MCA).

185 cal skin carcinogenesis [methylcholanthrene (MCA) or 7,12-dimethylbenz(a)anthracene (DMBA)/12-O-tetra

186 We inoculated methylcholanthrene (MCA) 205 in the flanks of normal mice and mice bearing M

187 during immune control of methylcholanthrene (MCA)-induced or p53 mutant cancers and showed the critic

188 uccinyl-Leu-Leu-Val-Tyr-methylcoumarinamide (MCA), benzyloxycarbonyl-Leu-Leu-Glu-7-amido-4-MCA, and t

189 Following 5- and 10-minute MCA occlusions, 9 of 32 animals (28%) had microinfarcts

190 g a separate tumor initiator/promoter model (MCA+BHT) indicated that NF-kappaB functions as an indepe

191 The prion state, which we name [MCA], was transmitted to the chromosomally encoded Mca1p

192 Ncad+ MCAs exhibit rapid lateral dispersal prior to penetratio

193 Using intact tissue explants, Ncad+ MCAs were also shown to efficiently rupture peritoneal m

194 ry 2002 and 31 June 2011 for space-occupying MCA ischaemic stroke and who underwent DHC.

195 more sensitive than TCD in the detection of MCA spasm.

196 detected TP in the endothelial cell layer of MCA.

197 rarchical framework and the methodologies of MCA may prove useful both for thinking about aging and f

198 Neuroimaging results showed that 0 min of MCA occlusion did not produce a lesion, whereas occlusio

199 we previously developed an embolic model of MCA occlusion in the rat, which recapitulates the key co

200 ism from acetate (P = 0.015) and the rate of MCA transport (P = 0.01) were also approximately twofold

201 ant thrombi resulting in fast restoration of MCA patency and consequently reduced cerebral infarct si

202 The severity of MCA depends on a number of different factors, including

203 Endovascular treatment of MCA aneurysms is feasible, and our results are convergen

204 our department in endovascular treatment of MCA aneurysms.

205 ent with the hypothesis that upregulation of MCA transport may contribute to the maintenance of brain

206 itiveness is required to enhance the role of MCAs in IPM.

207 In this series of MCAs, comprising a "boron core" surrounded by a "halogen

208 respectively; P = .004) and was dependent on MCA recanalization (odds ratio, 5.55; P = .006).

209 individuals with unexplained DD/ID, ASD, or MCA than a G-banded karyotype ( approximately 3%, exclud

210 nostic test for patients with DD/ID, ASD, or MCA.

211 r twofold increase in the transport of other MCAs, including lactate, during insulin-induced hypoglyc

212 onses in a low-dimensional space (e.g., PCA, MCA, or t-SNE).

213 Vascular diameter was measured in perfused MCA segments using videomicroscopy in the presence and a

214 and directly after (0-5 h) distal, permanent MCA occlusion (pMCAo) in a rat model.

215 After induction of permanent MCA occlusion (pMCAO), the rats received an initial intr

216 PFR-MCA hydrolysis was greater for patients' IgG than for a

217 nylalanine-arginine-methylcoumarinamide (PFR-MCA) substrate.

218 tor VIII, did not correlate with that of PFR-MCA prior transplantation, whereas it did 12 mo posttran

219 Prenatal MCA pseudofeeders were a risk factor for encephalomalaci

220 ALX-0081 prevented MCA thrombosis and induced reperfusion when given immedi

221 pH 3 had the best milk-clotting properties (MCA/PA ratio).

222 g protein) and female (11.2 pmol/mg protein) MCAs.

223 Contrary to FLAIR-HAs at proximal MCA or within DWI lesions, FLAIR-HAs beyond DWI lesions

224 There were 160 patients with proximal MCA occlusion at six centers in this institutional revie

225 te that reduced EDHF dilations in female rat MCA cannot be solely attributed to impaired cAMP signali

226 nhance EDHF-mediated dilations in female rat MCA.

227 duced in the female compared to the male rat MCA.

228 In severe reactions, MCA can be documented by a substantial increase in the s

229 The right MCA was occluded in spontaneously hypertensive rats for

230 Ruptured MCA aneurysms may be treated endovascularly with similar

231 A second MCA functionalized with two different immunological rece

232 Furthermore, we show that in serum-starved MCA, expression of constitutively active AKT re-establis

233 In serum-starved MCAs, EGFR activation was associated with active ERK1/2,

234 o-MCAAI, subtotal MCAAI, co-MCA and subtotal MCA infarction.

235 T, subtotal, complete MCA [co-MCA], Subtotal MCA with additional infarction [Subtotal MCAAI] and co-M

236 s has been achieved, in most orchard systems MCAs account for a relatively small proportion of the pe

237 elial collagen matrix on which MT1-MMP-T567E MCAs rapidly disperse.

238 Following temporary MCA occlusion EEG was monitored from a screw electrode a

239 The MCA infarctions were classified into four subgroups by C

240 The MCA was graded as normal, narrowed <50%, and >50% using

241 femoral artery catheterization to access the MCA and therefore eliminated the occurrence of head trau

242 tatistically different fitness values by the MCA versus the SCA: the D30N, G48V, I50V, I54L, and I54M

243 est dataset (300 Cases and 300 Controls) the MCA procedure finds several unique classifiers that have

244 Diminished SASM precipitation during the MCA and CWP tracks reconstructed Northern Hemisphere and

245 Lake-based fire rotation during the MCA decreased to an estimated 120 y, representing a 260%

246 t change in the hydrologic regime during the MCA in the eastern Amazon and 'Cerrado', characterized b

247 imilar to feedbacks that occurred during the MCA may stabilize the fire regime, despite additional wa

248 patterns at bidecadal resolution during the MCA.

249 nt truncated EM algorithm is used to fit the MCA model to cochleagram data.

250 We propose that discordant results in the MCA and SCA are due to alterations in late events in the

251 tion did not affect tumor development in the MCA model; however, CXCR3 was important in the DMBA/TPA

252 In the MCA, after incubation with the superoxide scavenger TEMP

253 +/-11.65% in the ICA, 30.67%+/-18.45% in the MCA, and 28.38%+/-15.49% in the ACA.

254 protects the input of K(Ca)2.3 to EDH in the MCA, and represents a beneficial pleiotropic effect of s

255 In the MCA, EDD did not differ between groups after incubation

256 By occluding the MCA with a fibrin-rich allogeneic clot, we previously de

257 e range, 6-16]), or of the M2 segment of the MCA (48 patients: median NIHSS score, 7 [interquartile r

258 The peak of the MCA (900-1150 CE) was a warm period in the eastern Pacif

259 or flushed into the superior division of the MCA (M2).

260 ti-decadal cold conditions at the end of the MCA are neither resolved in temperature reconstructions

261 The average lifetime of the MCA is 36 ms and the free energy difference to the TSA-l

262 on of infarcts smaller than one-third of the MCA territory (32%, 48%, and 69% for ASITN/SIR grades of

263 boxylic acids [MCAs]) by upregulation of the MCA transporters may contribute metabolic substrates dur

264 lium-dependent dilatation (carbachol) of the MCA were not different between groups.

265 s suggestive of ischaemia in the area of the MCA within 12 hours following onset of the first symptom

266 millennium, the first one at the end of the MCA, in ~1200 CE, was of similar amplitude to two other

267 60 minutes after the total occlusion of the MCA.

268 e abrupt and sustained than the onset of the MCA.

269 re restricted to cortical territories of the MCA.

270 unts of mus-ms time scale dynamics, only the MCA samples a dominant excited state that resembles the

271 After T3-SCI, the MCA had more collagen I (42%), collagen III (24%), trans

272 ing, however, did not persist throughout the MCA.

273 ging wind field patterns associated with the MCA provided conditions in which voyaging to and from th

274 Infusion of t-PA did not dissolve these MCA occlusions.

275 e trend in the electronic stability of these MCAs is determined by the interplay between the Coulomb

276 gm for understanding the properties of these MCAs with not well-separated charges that departs from t

277 Interaction of these MCAs with peritoneal mesothelium disrupts mesothelial in

278 cetate is transported into the brain through MCA transporters that are also used for lactate and keto

279 re difficult to elucidate when introduced to MCA systems as mixtures, although pattern recognition te

280 The apparent limit to MCA burning has been surpassed by the regional fire regi

281 l barrier that provides dynamic stability to MCAs and allows electronically metastable species to be

282 exercise were attenuated following transient MCA occlusion (MCAO) and reperfusion, mediated via alter

283 n by single cells as well as epithelial-type MCAs.

284 ascularly with similar effects as unruptured MCA aneurysms.

285 B. thuringiensis is the most used MCA for control of lepidopteran orchard pests.

286 ontrol of several orchard pest insects using MCAs, including viruses, Bacillus thuringiensis, fungi,

287 V(MCA), V(tICA), and V(tICA)/V(ICA) ratio were not age dep

288 not age dependent, contrary to V(ICA) and V(MCA)/V(ICA) ratio, after controlling for hematocrit and

289 meters per second for mean angle-corrected V(MCA) on the left and right sides were 62-198 and 69-153;

290 Reference ranges for mean angle-corrected V(MCA)/V(ICA) ratio on the left and right sides were 1.2-4

291 nography (US), and carotid US to determine V(MCA)/V(ICA) and V(tICA)/V(ICA) ratios from angle-correct

292 The study provides reference limits for V(MCA), V(tICA), V(ICA), and velocity ratios obtained from

293 lood flow), middle cerebral artery velocity (MCA Vmean), arterial-venous differences and blood temper

294 CO2), middle cerebral artery blood velocity (MCA V(mean)), and calculated CBVC (MCA V(mean)/MAP) were

295 Mean middle cerebral artery blood velocity (MCA V(mean)), mean arterial pressure (MAP), cardiac outp

296 imal intracranial arterial occlusion (ICA vs MCA-M1 vs MCA-M2) was not.

297 cranial arterial occlusion (ICA vs MCA-M1 vs MCA-M2) was not.

298 y involved intracranial stenosis segment was MCA, present in 10 (41.6%) out of 24 intracranial segmen

299 formed in 26 (35%) IVT-treated patients with MCA recanalization (group 1).

300 l to use pattern recognition techniques with MCAs as a new type of detector for chromatography with a