ライフサイエンスコーパス: MRP

1 MRP obtained by ultrasound exhibited 1,1-diphenyl-2-picr

2 MRP resulted in less variability among estimates across

3 MRP RNA is an abundant, essential noncoding RNA whose fu

4 MRP was found to achieve generally superior performance

5 MRP-1 binds to both HSP90 and HSP70, although only inhib

6 MRP-1 inhibition as well as knockdown trapped nuclear GS

7 MRP-1 is also expressed in mitochondria, and we have exa

8 MRP-14(-/-) recipients also had significantly more lymph

9 MRP-8/14 in saliva might be a potential diagnostic param

10 MRP-8/14 levels were documented with receiver operating

11 MRPs exhibited low PV, p-anisidine and inhibited the for

12 MRPs possess excellent antioxidant ability in many food

13 MRPs showed higher PRS and RP than BHA.

14 MRPs were detected only in oil of flaxseeds roasted at 5

15 e multidrug resistance-associated protein 1 (MRP-1) in Ewing's sarcoma (ES) predicts poor outcome.

16 otein (P-gp), multidrug resistant protein 1 (MRP-1), B-cell lymphoma (BCL-2) and other targets that a

17 Multidrug resistance-associated protein-1 (MRP-1), an active GSSG efflux mechanism, showed 2-fold i

18 somewhat surprising given there are over 70 MRPs and suggest little functional redundancy.

19 We observed that MRP-14 and MRP-8/MRP-14 heterodimers (S100A8/A9) are expressed in and sec

20 latelets, purified MRP-14, or purified MRP-8/MRP-14 heterodimers into Mrp14(-)/(-) mice decreased the

21 calgranulin A/calgranulin B oligomer, MRP-8/MRP-14 oligomer) chelates a number of first-row transiti

22 protectin (CP, S100A8/S100A9 oligomer, MRP-8/MRP-14 oligomer) is an abundant host-defense protein tha

23 Here, we show that a mesodermal ABCC (MRP) transporter is necessary for endodermal gut morphog

24 The active MRP fraction (16.1g/L) completely inhibited the growth o

25 ion of a proteinaceous multi-repeat adhesin (MRP) required for binding to the host plant.

26 The lowest fluorescence of advanced MRP (FAST) index was observed for buckwheat roasted at 1

27 ded with the increase (p<0.05) in BI for all MRPs.

28 De novo generation of H2O2 by an MRP fraction, extracted from a ribose/lysine Maillard re

29 Whey or e-fructoselysine, an MRP in whey and many processed foods, selectively increa

30 We observed that MRP-14 and MRP-8/MRP-14 heterodimers (S100A8/A9) are expressed in a

31 ived MRP-14(-/-) DCs infected with MRP-8 and MRP-14 retroviral vectors showed significantly decreased

32 iated drug efflux and that CFTR channels and MRP pumps utilize similar allosteric mechanisms for coup

33 Alternatively, combined PRCP(gt/gt) and MRP-14(-/-) mice were protected from wire injury with le

34 methylation levels correlated with hsCRP and MRP 8/14 levels and with apnea-hypopnea index (AHI), BMI

35 Residual differences between observed and MRP expected values were smallest for LAs with large pro

36 rgy barrier to both CFTR channel opening and MRP-mediated drug efflux and that CFTR channels and MRP

37 RNase P and MRP are highly conserved, multi-protein/RNA complexes wi

38 s of the human endoribonucleases RNase P and MRP.

39 the function and organization of RNase P and MRP.

40 Augmented import of RNase P, 5S rRNA, and MRP RNAs depended on PNPASE expression and PNPASE-import

41 myeloid-related protein (MRP)-8 (S100A8) and MRP-14 (S100A9) form MRP-8/14 heterodimers (S100A8/A9, c

42 art and B6 recipients) in wild-type (WT) and MRP-14(-/-) recipients.

43 dology to predict the release of antioxidant MRP in vivo.

44 Consumption of the assayed MRPs did not affect the hepatic antioxidant defence whil

45 The average MRP concentration in summer exceeds that winter by facto

46 ely to the prevention of cAMP efflux because MRP inactivation is also associated with reduced cAMP de

47 The negative correlations between MRP and DO is marked (r = -0.89).

48 significant associations were found between MRP inhibitor exposure and abnormal protein, glucose, or

49 Associations were observed between MRP inhibitor exposure and eGFR declines of >10 mL/min (

50 et (containing negligible amounts of biscuit MRP).

51 Therefore, the biscuit MRP functional claims could be demonstrated by an in viv

52 purified MRP-14 into mice deficient for both MRP-14 and CD36 failed to reduce carotid occlusion times

53 nitoring the concentration of free and bound MRPs in low lactose milk during shelf life.

54 nzyme reduced the formation of protein-bound MRPs down to 79% after six days at 37 degrees C.

55 identified a regulatory module activated by MRP-1, a regulator of BETL differentiation and function.

56 d, and then analyzed for acrylamide content, MRPs absorbance, pH, color, antioxidant capacity, antiba

57 Control ATPase-defective MRP mutants could not be rescued by this mechanism.

58 Proximity ligand assay demonstrated MRP-1 and YB-1 binding.

59 Bone marrow-derived MRP-14(-/-) DCs infected with MRP-8 and MRP-14 retrovira

60 ter injury, indicating that platelet-derived MRP-14 directly regulates thrombosis.

61 Results showed that dietary MRP reduced the food efficiency, increased the antioxida

62 onserved with the thermodynamically distinct MRPs.

63 xpressed from four Drosophila genes, Dscam1, MRP, Mhc, and Rdl.

64 signaling cascades that are activated during MRP-induced tolerance of phagocytes.

65 l22, Mrpl44, Mrps18c and Mrps22 Loss of each MRP results in successful implantation and egg-cylinder

66 ed MRP loci loses this activity, and ectopic MRP RNA expression restores cleavage activity.

67 e RNP isolated from cells with CRISPR-edited MRP loci loses this activity, and ectopic MRP RNA expres

68 f import and assembly of the nucleus-encoded MRPs into ribosomes.

69 e-mediated knockdown of the endoribonuclease MRP did not result in a clear defect in ITS1 processing.

70 s that were markedly different from expected MRP values (>=90% posterior exceedance probability) had

71 (cells per lymph node: 23.7+/-0.7x10(5) for MRP-14(-/-) versus 6.0+/-0.2x10(5) for WT; P<0.0001).

72 Mutations in the genomic locus for MRP RNA cause pleiotropic human diseases, including cart

73 times, indicating that CD36 is required for MRP-14-dependent thrombosis.

74 y artery disease; however, a causal role for MRP-14 in acute coronary syndromes has not been establis

75 ndicating a previously unrecognized role for MRP-14 in immune cell biology.

76 These findings offer further support for MRP as a promising analytical approach for addressing pa

77 in (MRP)-8 (S100A8) and MRP-14 (S100A9) form MRP-8/14 heterodimers (S100A8/A9, calprotectin) that reg

78 Three hundred twenty-one patients had MRP inhibitor exposure, ranging from 0.02 to 120 mean DD

79 YB-1 and Ape/Ref-1 were responsible for high MRP-1 expression in EOMA.

80 ments that are defined by small volume, high MRP activity, limited PDE activity, and limited exchange

81 t the mito-specific extensions in homologous MRPs generally are involved in inter-MRP contacts and in

82 o-specific MRPs and extensions of homologous MRPs are situated on the peripheral regions, they also c

83 nome editing to disrupt the endogenous human MRP RNA locus, thereby attaining what has eluded RNAi an

84 A hyperactive MRP-1 system for GSSG efflux acts as a critical survival

85 nts with acute coronary syndromes identified MRP-14 as a novel predictor of myocardial infarction.

86 to reduced degradation by PDEs and identify MRP-dependent transport mechanisms as novel regulators o

87 orthern blots, and RNA sequencing-implicates MRP RNA in pre-rRNA processing.

88 survival averaged 5.9+/-2.9 weeks (n=10) in MRP-14(-/-) recipients compared with >12 weeks (n=15; P<

89 o weeks after transplantation, allografts in MRP-14(-/-) recipients had significantly higher parenchy

90 Compared with WT recipients, allografts in MRP-14(-/-) recipients had significantly increased T-cel

91 scover significant conformational changes in MRP mL45 at the nascent polypeptide-exit site within the

92 erved in animals fed with a diet enriched in MRP (p < 0.05), which indicated a possible prebiotic act

93 d dry-heating time, TOTOX values of SDASO in MRP-derived microcapsules were 29-87% lower than that of

94 ific amino acid extensions and insertions in MRPs that are homologous to bacterial ribosomal proteins

95 Although mutations in MRPs have been associated with human diseases, little is

96 Compared with healthy individuals, MRP-8/14 levels were significantly higher in both the sa

97 ity at concentrations widely used to inhibit MRPs.

98 After femoral artery wire injury, MRP-14(-/-) mice had significant reductions in leukocyte

99 Instead, MRP concentrations significantly increase with increasin

100 ologous MRPs generally are involved in inter-MRP contacts and in contacts with mito-specific MRPs, su

101 R indicated the predominance of intermediate MRPs, such as the Schiff base compounds.

102 sediments at a different fraction from known MRP RNA-containing complexes, the MRP ribonucleoprotein

103 t rejection using MRP-14(-/-) mice that lack MRP-8/14 complexes.

104 s (FIC) and FAST index, indicating the least MRP formation.

105 ing gain of function (GOF) mutants of a long MRP in yeast (Ycf1p cadmium transporter), a short yeast

106 Home cooking conditions leading to low MRP generation and pleasant colours were obtained and co

107 Boiling in water caused very low MRP formation.

108 S100 calcium-modulated protein family member MRP-14 (also known as S100A9) is elevated in platelets f

109 Mitochondrial MRP-1 is glycosylated and localized to the outer mitocho

110 The effect of disrupting mitochondrial MRP-1-dependent efflux activity on the cytotoxic effect

111 Disruption of mitochondrial MRP-1-dependent efflux significantly increases the cytot

112 ime, we have demonstrated that mitochondrial MRP-1 is expressed in the outer mitochondrial membrane a

113 ime, we have demonstrated that mitochondrial MRP-1 is expressed in the outer mitochondrial membrane a

114 The presence of model MRPs in the diet, especially melanoidins, led to a signi

115 se, asparagine and chitosan mixture had more MRPs compared to other mixtures.

116 signaling predict that selective ablation of MRP-dependent cAMP efflux per se does not affect bulk cy

117 The absence of MRP-14 markedly increased T-cell activation and exacerba

118 water); HT-B diet (containing high amount of MRP derived from biscuits) and LT-B diet (containing neg

119 reatment on biscuits increases the amount of MRP with antioxidant capacity (p < 0.05).

120 ean cumulative defined daily doses (DDDs) of MRP inhibitors (NSAIDs, PDE5-i, salicylates, dipyridamol

121 nd serum were collected for the detection of MRP-8/14 and calcium levels.

122 These findings suggest that the effects of MRP inactivation on intracellular cAMP levels reported p

123 The effects of MRP inhibitors on the estimated glomerular filtration ra

124 RNAi and RNase H experiments: elimination of MRP RNA in the majority of cells.

125 ibition of HSP90beta decreases expression of MRP-1 in the mitochondria.

126 rgery, we confirmed the initial induction of MRP expression and the tolerance induction of monocytes

127 ecreted MRP, with an altered localization of MRP observed in an ECA3265 mutant background.

128 xamined the submitochondrial localization of MRP-1 and investigated the mechanism of MRP-1 transport

129 Localization of MRP-1 to the outer mitochondrial membrane by the chapero

130 The mitochondrial localization of MRP-1 was examined in ES cell lines by differential cent

131 Moreover, genetic manipulation of MRP expression results in concomitant changes in PDE act

132 n of MRP-1 and investigated the mechanism of MRP-1 transport and role of this organelle in the respon

133 urther examine the accuracy and precision of MRP in a context where census data provided reasonable p

134 tors STAT3 and BCL-3 as master regulators of MRP-induced tolerance.

135 rRNA imbalance is achieved through rescue of MRP RNA levels by ectopic expression.

136 ory mediator, the pathophysiological role of MRP-8/14 complexes in cardiovascular disease is incomple

137 This study investigated the role of MRP-8/14 in cardiac allograft rejection using MRP-14(-/-

138 uch binding enabled the nuclear targeting of MRP-1 in EOMA in a leptomycin-B-sensitive manner.

139 etal chelation-based antibrowning ability of MRPs to inhibit the enzymatic browning reaction in fruit

140 ays to evaluate the antioxidative ability of MRPs, and parameters influencing their functional proper

141 s an overview of the antioxidant activity of MRPs in model and real food systems.

142 n must be paid to the overall consumption of MRPs from different sources in a conventional diet, due

143 ltrasound treatment resulted in formation of MRPs with all tested carbohydrates.

144 armacological and/or genetic inactivation of MRPs has been shown to augment intracellular cAMP signal

145 V and inhibition of TBARS similar to that of MRPs.

146 gomer, calgranulin A/calgranulin B oligomer, MRP-8/MRP-14 oligomer) chelates a number of first-row tr

147 an calprotectin (CP, S100A8/S100A9 oligomer, MRP-8/MRP-14 oligomer) is an abundant host-defense prote

148 With DPPH, only MRPs at 121 degrees C exhibited higher AA than BHA.

149 factors affecting the molybdate reactive P (MRP) in these waters were analyzed using the data of the

150 versally found in all enzymes of the RNase P/MRP family, as well as with a phylogenetically conserved

151 ion of the eukaryotic enzymes of the RNase P/MRP family.

152 affect TLC1 and the RNA subunits of RNase P/MRP in very different ways.

153 revisiae RNase MRP in a complex with RNase P/MRP proteins Pop6 and Pop7 solved to 2.7 A.

154 rotein-binding domain in the RNAs of RNase P/MRP.

155 ganization of the P3 RNA domains in RNases P/MRP and possible functions of the P3 domains and protein

156 ally distinct RNPs, telomerase, and RNases P/MRP from unrelated progenitor RNAs.

157 The eukaryotic RNases P/MRP have acquired an essential helix-loop-helix protein-

158 pe, in other words mutation rate plasticity (MRP), can be caused by stress or ageing.

159 pathway of thrombosis that involves platelet MRP-14 and CD36 and suggest that targeting MRP-14 has po

160 ultilevel regression and poststratification (MRP) is a model-based approach for estimating a populati

161 observation.The movement-related potential (MRP), investigated as intracortical low-frequency LFP ac

162 reactions with allo-endothelial cell-primed MRP-14(-/-) DCs resulted in significantly higher antigen

163 Ovalbumin-primed MRP-14(-/-) DCs augmented proliferation of OT-II (ovalbu

164 avage by RNase mitochondrial RNA processing (MRP) and mutations in the RNase MRP small nucleolar RNA

165 RNase mitochondrial RNA processing (MRP) is an essential, evolutionarily conserved endoribon

166 east RNase for mitochondrial RNA processing (MRP), a catalytic ribonucleoprotein (RNP), recently repo

167 PNC components mitochondrial RNA-processing (MRP) RNA, pyrimidine tract-binding protein (PTB), and CU

168 d content (TFC), Maillard reaction products (MRP) and phenolic profile in eight Tartary buckwheat (Fa

169 he generation of Maillard reaction products (MRP) in beef was investigated.

170 e anti-oxidative Maillard reaction products (MRP) which was used to encapsulate stearidonic acid soyb

171 lease of biscuit Maillard reaction products (MRP) with antioxidant capacity was compared by in vitro

172 n acrylamide and Maillard reaction products (MRPs) (>52.6%), respectively.

173 The Maillard reaction products (MRPs) are widely produced in foods containing reducing s

174 otic activity of Maillard reaction products (MRPs) derived from biscuits were investigated in Wistar

175 ommonly consumed Maillard reaction products (MRPs) from the glucose-lysine system.

176 of protein bound Maillard reaction products (MRPs) is still a challenge in food chemistry.

177 Maillard reaction products (MRPs) show antimicrobial activity that is at least parti

178 ls and poorer in Maillard reaction products (MRPs) than were GPE200 extracts.

179 Formation of Maillard reaction products (MRPs) was monitored by mass spectrometry, spectrophotome

180 ing capacity and Maillard reaction products (MRPs) were determined in ginger cakes after storage and

181 al properties of Maillard reaction products (MRPs) were investigated in a food model system.

182 Maillard reaction products (MRPs) were prepared from aqueous ribose-lysine (RL) and

183 ity index (OSI), Maillard reaction products (MRPs), chlorophyll and carotenoid contents were increase

184 The effect of Maillard reaction products (MRPs), formed during the production of dark malts, on th

185 the formation of Maillard reaction products (MRPs), protein hydrolysates and glycated compounds.

186 in colour of the Maillard reaction products (MRPs), were recorded.

187 Maillard reaction products (MRPs), which are common in processed foods, form upon he

188 formation of the Maillard reaction products (MRPs).

189 composition and Maillard reaction products (MRPs).

190 e UK Myeloma Research Alliance Risk Profile (MRP) for overall survival.

191 and multidrug-resistance-associated protein (MRP) 2 and 3 eliminate toxic compounds from tissues and

192 man multidrug-resistance associated protein (MRP) 2, and Mrp2 knockout mice displayed increased vinor

193 findings; hsCRP and myeloid-related protein (MRP) 8/14 levels were also assayed.

194 role of S100A8/A9 [myeloid-related protein (MRP) 8/14, calprotectin] in promoting glomerulonephritis

195 um-binding proteins myeloid-related protein (MRP)-8 (S100A8) and MRP-14 (S100A9) form MRP-8/14 hetero

196 ate calcium-binding myeloid-related protein (MRP)-8/14 in the saliva and serum of individuals with pe

197 The alarmins myeloid-related protein (MRP)8 and MRP14 are the most prevalent cytoplasmic prote

198 identified mitochondrial ribosomal protein (MRP)L14 as the specific interacting protein partner of C

199 ific (mito-specific) mitoribosomal proteins (MRPs) and a mito-specific segment of EF-G1(mt) in mitoch

200 Myeloid-related proteins (MRPs) 8 and 14 are cytosolic proteins secreted from myel

201 metabolites, multidrug-resistance proteins (MRPs) extrude the second-messenger cAMP from various cel

202 xporters (the multidrug resistance proteins (MRPs)) and a unique ATP-gated ion channel (cystic fibros

203 ong and short multidrug resistance proteins (MRPs), and an ATP-gated anion channel, the cystic fibros

204 ar-encoded mitochondrial ribosomal proteins (MRPs) are required for proper function of the organelle.

205 genome and mitochondrial ribosomal proteins (MRPs) that are encoded by nuclear genes and imported int

206 ge mass of mitochondrial ribosomal proteins (MRPs), including large mito-specific amino acid extensio

207 Several prototypical MRP inhibitors block PDE activity at concentrations wide

208 Furthermore, affinity-purified MRP ribonucleoprotein (RNP) from HeLa cells cleaves the

209 In contrast, infusion of purified MRP-14 into mice deficient for both MRP-14 and CD36 fail

210 f WT platelets, purified MRP-14, or purified MRP-8/MRP-14 heterodimers into Mrp14(-)/(-) mice decreas

211 Infusion of WT platelets, purified MRP-14, or purified MRP-8/MRP-14 heterodimers into Mrp14

212 s II(-/-) hosts and of B6 WT hosts receiving MRP-14(-/-) DCs had significantly augmented inflammatory

213 A3 and to the RNA component of ribonuclease MRP, which cleaves site A3.

214 RNase MRP has established roles in multiple pathways including

215 RNase MRP is a ribonucleoprotein with a large catalytic RNA mo

216 RNase MRP is an essential eukaryotic ribonucleoprotein complex

217 RNase MRP is related to the ribozyme-based RNase P, but it has

218 Ribonuclease P (RNase P) and RNase MRP are closely related ribonucleoprotein enzymes, which

219 ng we show that eukaryotic RNase P and RNase MRP have a modular architecture, where proteins stabiliz

220 The structures of RNase P and RNase MRP have not yet been solved, so it is unclear how the p

221 teins, known components of RNase P and RNase MRP, bind to yeast telomerase RNA and are essential cons

222 reatest difference between RNase P and RNase MRP, highlighting the importance of the adaptation of th

223 cryo-EM structure of the S. cerevisiae RNase MRP holoenzyme solved to 3.0 angstrom.

224 trate and the Saccharomyces cerevisiae RNase MRP holoenzyme.

225 A domain from Saccharomyces cerevisiae RNase MRP in a complex with RNase P/MRP proteins Pop6 and Pop7

226 s a role for the host endoribonuclease RNase MRP in viral RNA degradation and recombination.

227 ving rise to a closely related enzyme, RNase MRP, which has similar components but has evolved to hav

228 Thus, a role for RNase MRP in human pre-rRNA processing is established.

229 d several new potential substrates for RNase MRP including a cell cycle-regulated transcript, CTS1; t

230 precursors, as well as some mRNAs, for RNase MRP.

231 In addition, we found RNase MRP to be involved in the regulation of the Ty1 transpos

232 o RNase MRP, and demonstrate that four RNase MRP protein components, all shared with RNase P, interac

233 Application of PAIR-MaP to human RNase MRP and 2 bacterial messenger RNA 5' untranslated region

234 nknown how CHH-pathogenic mutations in RNase MRP snoRNA interfere with skeletal development, and aber

235 To identify novel RNase MRP substrates, we utilized RNA immunoprecipitation and

236 s in the functional diversification of RNase MRP and its progenitor, RNase P, and demonstrate structu

237 ons for the structural organization of RNase MRP and the roles of its components are discussed.

238 ults reinforce and broaden the role of RNase MRP in cell cycle regulation and help to identify new ro

239 evelopment, and aberrant processing of RNase MRP substrate RNAs is thought to be involved.

240 gene, coding for the RNA component of RNase MRP, lead to reduced production of the endoribonucleolyt

241 in eukaryotes: snRNA, snoRNA, RNase P, RNase MRP, Y RNA or telomerase RNA.

242 f interactions between a photoreactive RNase MRP substrate and the Saccharomyces cerevisiae RNase MRP

243 d TBSV RNA, suggesting that in plants, RNase MRP is involved in TBSV RNA degradation.

244 nucleolar RNA (snoRNA) subunit of the RNase MRP complex cause cartilage-hair hypoplasia (CHH), a hum

245 We show that some of the RNase MRP proteins shared with RNase P undergo an unexpected R

246 vidual subunits with the P3 arm of the RNase MRP RNA is revealed to be negligible whereas the 1:1 Rpp

247 processing (MRP) and mutations in the RNase MRP small nucleolar RNA (snoRNA) subunit of the RNase MR

248 n of SNM1, encoding a component of the RNase MRP, and a temperature-sensitive mutation in the NME1 ge

249 conserved RNA region that is unique to RNase MRP, and demonstrate that four RNase MRP protein compone

250 fy RNA that physically associates with RNase MRP.

251 so show that the highly purified yeast RNase MRP cleaves the TBSV RNA in vitro, resulting in TBSV RNA

252 Ribonuclease (RNase) MRP is a ubiquitous and essential site-specific eukaryot

253 Salivary MRP-8/14 levels correlated significantly with the presen

254 A high diagnostic potential of salivary MRP-8/14 was detected for periodontitis (ROC = 0.86).

255 d that ECA3270 and ECA3271 regulate secreted MRP levels by increasing and decreasing, respectively, t

256 We show that c-di-GMP regulates secreted MRP levels in Pba1043 through the action of two genes en

257 ays an important role in regulating secreted MRP, with an altered localization of MRP observed in an

258 Correlations between salivary and serum MRP-8/14 levels and clinical parameters, bacteria, and c

259 ition to their role in the ribosome, several MRPs have auxiliary functions or have been implicated in

260 Although most of the mito-specific MRPs and extensions of homologous MRPs are situated on t

261 55S mitoribosome reveals that mito-specific MRPs of both the SSU and LSU are involved directly in th

262 contacts and in contacts with mito-specific MRPs, suggesting a stepwise evolution of the current arc

263 proteins and an additional 35 mito-specific MRPs.

264 the interesting findings for these specific MRPs, attention must be paid to the overall consumption

265 tilevel Regression with Post-stratification (MRP) to synthesise data from multiple sources, using chl

266 t MRP-14 and CD36 and suggest that targeting MRP-14 has potential for treating atherothrombotic disor

267 t with their potency in inhibiting PDEs than MRPs.

268 ution localization analyses demonstrate that MRP RNA, CUGBP, and PTB colocalize at the PNC as a retic

269 Our results indicate that MRP-14 regulates B7 molecule expression and reduces anti

270 sion compared with controls, indicating that MRP-8/14 regulates B7-costimulatory molecule expression.

271 We observed that MRP-14 and MRP-8/MRP-14 heterodimers (S100A8/A9) are exp

272 The MRP concentrations are geographically unrelated to erosi

273 The MRP groups defining low-risk, medium-risk, and high-risk

274 The MRP groups remained prognostic in patients exposed to di

275 The MRP included WHO performance status, International Stagi

276 The MRP was internally validated in NCRI-XI and externally v

277 The MRP was prognostic of overall survival and was successfu

278 Although the MRP was detected only during dynamic periods of the task

279 from known MRP RNA-containing complexes, the MRP ribonucleoprotein ribozyme and human telomerase reve

280 The genes encoding the MRP and the T1SS are tightly linked to genes encoding se

281 f the legacy P in the sediments explains the MRP concentrations which are most pronounced at low DO c

282 d substantial information about grasp in the MRP corroborating its suitability for brain-machine inte

283 The dendritic cells (DCs) of the MRP-14(-/-) recipients of bm12 hearts expressed signific

284 p35 and Cfd1 are prototypical members of the MRP/Nbp35 class of iron-sulfur (FeS) cluster scaffolds t

285 indings support a working model in which the MRP RNA-protein complex becomes nucleated at the PNC in

286 It was shown that the MRPs derived from electro-activated whey at a concentrat

287 This cytotoxicity could be related to their MRPs content.

288 This MRP is genetically switchable, dependent on the quorum-s

289 Thus, MRPs can be considered as active ingredients for antimic

290 Concomitant incidental exposure to MRP inhibitors and TDF did not result in major additiona

291 Exposure to MRP inhibitors was associated with an additional mean eG

292 ance-associated protein efflux-transporters (MRPs) in the renal proximal tubule could enhance this un

293 RP-8/14 in cardiac allograft rejection using MRP-14(-/-) mice that lack MRP-8/14 complexes.

294 dence of small gains in precision when using MRP, particularly for smaller population subsets.

295 Whether MRP-1 is chaperoned by heat shock proteins (HSPs) was in

296 Beyond its serving as a risk marker, whether MRP-8/14 participates directly in vascular inflammation

297 marrow-derived MRP-14(-/-) DCs infected with MRP-8 and MRP-14 retroviral vectors showed significantly

298 lso rescued defective drug export by a yeast MRP (Yor1p) with ATP binding defects in its NBDs.

299 t (Ycf1p cadmium transporter), a short yeast MRP (Yor1p oligomycin exporter), and human CFTR channels

300 ons rescued ATP binding mutants of the yeast MRPs and CFTR by increasing ATP sensitivity.