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3 l grass species Brachypodium distachyon with Magnaporthe grisea (rice blast), together with modificat
4 Podospora anserina and the pathogenic fungi Magnaporthe grisea and Cryphonectria parasitica, respect
5 t in glycans isolated from the rice pathogen Magnaporthe grisea and from the plant pathogen Botryotin
6 al for the biosynthesis of fungal melanin by Magnaporthe grisea and is a focus of inhibitor design st
8 were constructed for the ascomycete fungus, Magnaporthe grisea and the basidiomycete fungus, Ustilag
9 hydroxy-2-butanone-4-phosphate synthase from Magnaporthe grisea are reported for the E-SO(4)(2-), E-S
10 O39 was previously mapped to chromosome 1 of Magnaporthe grisea between cosegregating markers CH5-120
15 on alleles of the MPG1 hydrophobin gene from Magnaporthe grisea causes severe defects in development
22 ene resistance against strains of the fungus Magnaporthe grisea that express avirulent alleles of AVR
23 we show that in the plant pathogenic fungus Magnaporthe grisea trehalose-6-phosphate (T6P) synthase
24 ases of unknown function from the ascomycete Magnaporthe grisea were found to be the closest relative
25 significantly enhanced resistance to fungal (Magnaporthe grisea) and bacterial (Burkholderia glumae)
26 wing challenge with the rice blast pathogen (Magnaporthe grisea) and wheat/barley yellow stripe rusts
28 ense response against the rice blast fungus (Magnaporthe grisea), a large-scale expressed sequence ta
31 erization of ICL1 from the rice blast fungus Magnaporthe grisea, a gene that encodes isocitrate lyase
32 s were found to be present on the conidia of Magnaporthe grisea, and plant surface waxes were found t
33 ine synthases of Mycobacterium tuberculosis, Magnaporthe grisea, Candida albicans, and Schizosaccharo
35 sistant to strains of the rice blast fungus, Magnaporthe grisea, expressing AVR-Pita in a gene-for-ge
37 blast disease, caused by the fungal pathogen Magnaporthe grisea, is an excellent model system to stud
38 d a federated database for genome studies of Magnaporthe grisea, the causal agent of rice blast disea
40 sing metabolite fingerprinting, we show that Magnaporthe grisea, the causal agent of rice blast disea
42 of F. graminearum with the myosin I gene of Magnaporthe grisea, the causal agent of rice blast, also
43 ressorium formation and infectious growth in Magnaporthe grisea, the fungal pathogen responsible for
47 egion in O-137, a rice pathogenic isolate of Magnaporthe grisea, uncovered a novel gene, designated T
49 e economically important rice blast pathogen Magnaporthe grisea, which is required for exocytosis dur
50 ite changes occurring during interactions of Magnaporthe grisea--the cause of rice blast disease--wit
69 -related genes, are discussed in detail with Magnaporthe oryzae (M. grisea) and Fusarium graminearum
70 OsRING113 plants showed enhanced BSR against Magnaporthe oryzae (M. oryzae) and Xanthomonas oryzae pv
71 disease, caused by the Triticum pathotype of Magnaporthe oryzae (MoT), poses a significant threat to
72 d field populations of the rice blast fungus Magnaporthe oryzae (syn: Pyricularia oryzae) into three
73 rassa [Ncdcl-1 (50.5%); Ncdcl-2 (38.0%)] and Magnaporthe oryzae [MDL-1 (45.6%); MDL-2 (38.0%)], respe
74 ase in cereal plants is caused by the fungus Magnaporthe oryzae and accounts for a significant loss i
75 t greater susceptibility to the hemibiotroph Magnaporthe oryzae and biotroph Xanthomonas oryzae pv. o
76 stating disease of rice caused by the fungus Magnaporthe oryzae and can result in loss of a third of
77 plication impairs growth of the blast fungus Magnaporthe oryzae and has a pronounced effect on appres
78 t of OsDRB1.4 increased rice defense against Magnaporthe oryzae and increased the expression of 34 ge
79 to world agriculture, but also because both Magnaporthe oryzae and its host are amenable to advanced
80 gal species, including Zymoseptoria tritici, Magnaporthe oryzae and Neurospora crassa, exhibited PAMP
81 ances resistance against the fungal pathogen Magnaporthe oryzae and the bacterial pathogen Xanthomona
82 d-spectrum resistance to the fungal pathogen Magnaporthe oryzae and the RING-type E3 ligase AVRPIZ-T
83 ice disease resistance against the pathogens Magnaporthe oryzae and Xanthomonas oryzae pv. oryzae.
91 ause rice blast disease, the fungal pathogen Magnaporthe oryzae develops a specialized infection stru
93 rice (Oryza sativa) NLR RGA5 recognizes the Magnaporthe oryzae effector AVR-Pia through direct inter
99 tant strain of the devastating rice pathogen Magnaporthe oryzae impaired for de novo methionine biosy
103 prevents activation of Bsr-d1 expression by Magnaporthe oryzae infection and degradation of H(2) O(2
105 formation and reduce lesion sizes caused by Magnaporthe oryzae Inhibition of EXO70 by ES2-14 in Botr
106 thogenic life cycle of the rice blast fungus Magnaporthe oryzae involves a series of morphogenetic ch
109 at Sirt5-mediated protein desuccinylation in Magnaporthe oryzae is central to host ROS detoxification
113 poplastic effectors of the rice blast fungus Magnaporthe oryzae modulate the apoplast redox state of
114 well as biological analysis, we showed that Magnaporthe oryzae MTA1 gene is an orthologue of human M
115 he crystal structure of the DBD of PCG2, the Magnaporthe oryzae orthologue of MBP1, bound to MCB-DNA.
116 Here, we show that the rice blast fungus Magnaporthe oryzae overcomes this first line of plant de
117 e genetically tractable wheat blast pathogen Magnaporthe oryzae pathotype Triticum (MoT) as a suitabl
120 Here we report that the rice blast fungus Magnaporthe oryzae possesses two distinct secretion syst
121 effector AvrPiz-t from the rice blast fungus Magnaporthe oryzae preferentially accumulates in the spe
124 truction by the hemibiotrophic rice pathogen Magnaporthe oryzae requires plant defence suppression to
125 hic invasive hyphae (IH) of the blast fungus Magnaporthe oryzae secrete effectors to alter host defen
127 e rice blast disease, the filamentous fungus Magnaporthe oryzae secretes a battery of effector protei
132 iated with the presence of retrotransposons (Magnaporthe oryzae Telomeric Retrotransposons-MoTeRs) in
134 tating disease caused by the fungal pathogen Magnaporthe oryzae that threatens rice production around
135 Various surface signals are recognized by Magnaporthe oryzae to activate the Pmk1 MAP kinase that
139 gle metabolic gene, we engineered strains of Magnaporthe oryzae with different nutrient acquisition a
141 llb shows enhanced resistance to rice blast (Magnaporthe oryzae) and bacterial blight (Xanthomonas or
145 n developed for use in the rice blast fungus Magnaporthe oryzae, allowing rapid generation of transfo
146 h corresponds to MAP kinase kinase kinase in Magnaporthe oryzae, and urate oxidase (designated ClUras
147 ae and to the hemibiotrophic fungal pathogen Magnaporthe oryzae, but enhanced susceptibility to the n
148 e deletion experiments of Nudix effectors in Magnaporthe oryzae, Colletotrichum higginsianum, and Col
150 MoNLE1, an effector from the fungal pathogen Magnaporthe oryzae, is a core virulence factor that supp
151 (Oryza sativa, host) and rice blast fungus (Magnaporthe oryzae, pathogen) and uncover a new pathogen
152 against the devastating rice blast pathogen Magnaporthe oryzae, Pi9 functions as an intracellular re
153 -mediated infection in the rice blast fungus Magnaporthe oryzae, requires very-long-chain fatty acids
154 r challenge with three major rice pathogens (Magnaporthe oryzae, Rhizoctonia solani, and Xanthomonas
155 ied from different fungal species, including Magnaporthe oryzae, Sporisorium scitamineum, and Sporiso
159 otein genome editing in the fungal pathogen, Magnaporthe oryzae, we detail non-canonical DNA repair o
160 ing in pathogenesis of the rice blast fungus Magnaporthe oryzae, we identified MoGlo3 as an ArfGAP pr
162 sease is caused by the hemibiotrophic fungus Magnaporthe oryzae, which invades living plant cells usi
163 related development by the rice blast fungus Magnaporthe oryzae, which threatens global food security
197 trated the evolutionary relationship between Magnaporthe species and the influence of host differenti
198 we compared genome sequence of 6 isolates of Magnaporthe species obtained from three different host p
199 ergence are host adaptability mechanisms for Magnaporthe species, and this coevolution processes is g
201 o filamentous fungi, but homologous genes in Magnaporthe, Ustilago, Aspergillus, Fusarium, Epichloe,