ライフサイエンスコーパス: NADH dehydrogenase

1 ster, which encodes the proton-translocating NADH dehydrogenase.

2 itch from transcription of type I to type II NADH dehydrogenase.

3 thesis of two isoforms of the ND3 subunit of NADH dehydrogenase.

4 -cytochrome P450 reductase and mitochondrial NADH dehydrogenase.

5 ND1 and ND3) encoding additional subunits of NADH dehydrogenase.

6 ts defective in the energy-conserving type I NADH dehydrogenase.

7 rovides further evidence for the presence of NADH dehydrogenase.

8 nia as a bona fide, matrix-oriented, type II NADH dehydrogenase.

9 -bond flipping upon NAD(+) binding in proper NADH dehydrogenases.

10 tion that is dependent on acetate kinase and NADH dehydrogenases.

11 and major Amb a 1 allergens, and as unique, NADH dehydrogenases.

12 are closer to the FMN than they are in other NADH dehydrogenases.

13 itochondrial DNA: the matR gene found within NADH dehydrogenase 1 (nad1) intron 4.

14 ated from the heavy strand promoter 2 [i.e., NADH dehydrogenase 1 (ND1) by 11-fold, P < 0.005; cytoch

15 assessed by measuring the copy number of the NADH dehydrogenase 1 gene using quantitative real-time P

16 t on mtRNA expression and that expression of NADH dehydrogenase 1, 3, and 6 (ND-1, ND-3, ND-6) and AT

17 Cancer cells expressing yeast NADH dehydrogenase-1, which recycles NADH to NAD(+) inde

18 ins including the phosphate carrier protein, NADH dehydrogenase 1alpha subcomplexes 2 and 3, transloc

19 Silencing of the phosphate carrier or NADH dehydrogenase 1alpha subcomplexes 2 or 3 in 3T3-L1

20 change from threonine to alanine within the NADH dehydrogenase 3 (ND3) of complex I.

21 ochrome c oxidase-IV, ATP synthase-beta, and NADH dehydrogenase-3 decreased markedly in FPG, and thes

22 ariant mapped to the mitochondrially-encoded NADH dehydrogenase 4 (MT-ND4) gene, was confirmed in an

23 A novel mitochondrial NADH dehydrogenase 5 (ND5) m.12955A > G mutation was ide

24 the hepatoma cytochrome c oxidase I, II, and NADH dehydrogenase 5, 6, the downstream targets of Tfam,

25 of somatic truncating mutations occurred in NADH dehydrogenase 5.

26 ng mitochondrial genes encoded in the mtDNA [NADH dehydrogenase 6 (ND6) and cytochrome c oxidase subu

27 region of the mtDNA that contained the gene NADH dehydrogenase 6 (ND6), which encodes an essential c

28 it 6 (ATP6) by 6.5-fold, P < 0.005); but not NADH dehydrogenase 6 (ND6)], which is initiated from the

29 Their NADH dehydrogenase activities were almost normal.

30 Mutations that reduce NADH dehydrogenase activity (Ndh; type II) cause multipl

31 Therefore, maintenance of mitochondrial NADH dehydrogenase activity and the electron transport c

32 Thus, aberration in mitochondrial complex I NADH dehydrogenase activity can profoundly enhance the a

33 a threefold reduction in total mitochondrial NADH dehydrogenase activity in cells cultivated with glu

34 cerevisiae (NDI1) can completely restore the NADH dehydrogenase activity in mutant human cells that l

35 detection of a 700 kDa subcomplex retaining NADH dehydrogenase activity indicates an arrest in the a

36 phenotype analyses suggest that the external NADH dehydrogenase activity of Ndh1p is important for op

37 ed with protein folding, cell-cell adhesion, NADH dehydrogenase activity, ATP-binding, proteasome com

38 contrary, no decrease in rotenone-sensitive NADH dehydrogenase activity, using a water-soluble ubiqu

39 In terms of immunochemical and NADH dehydrogenase activity-staining analyses, all site-

40 n of a mouse A9 cell derivative defective in NADH dehydrogenase activity.

41 metformin inhibits mitochondrial complex I (NADH dehydrogenase) activity and cellular respiration.

42 marked decrease in mitochondrial complex I (NADH dehydrogenase) activity, coupled to decreased ATP s

43 are consistent with a protective role of the NADH dehydrogenases against oxidative stress, thus, when

44 atory electron transport chain that included NADH dehydrogenase, alternative complex III and cytochro

45 s of rotenone (an inhibitor of mitochondrial NADH dehydrogenase and a naturally occurring toxicant) o

46 Thus, multiple hits in NADH dehydrogenase and COX activity-impairing genes repr

47 e pathogenic mtDNA point mutations affecting NADH dehydrogenase and COX genes as well as regulatory e

48 erved among Black women and genes coding for NADH dehydrogenase and cytochrome c oxidase subunits.

49 erved among Black women and genes coding for NADH dehydrogenase and cytochrome c oxidase subunits.

50 are decreased by about 90%, whereas that of NADH dehydrogenase and cytochrome c reductase are unchan

51 rroborated that WSUCF1 biofilms uses type-II NADH dehydrogenase and demethylmenaquinone methyltransfe

52 and acetic acid tolerance; overexpression of NADH dehydrogenase and methylmalonyl-CoA epimerase impro

53 strains expressed binary combinations of one NADH dehydrogenase and one quinol oxidase.

54 The proton-pumping type-I NADH dehydrogenase and the aa3-type cytochrome c oxidase

55 , suggesting that the external mitochondrial NADH dehydrogenase and the malate-aspartate shuttle may

56 rrected by expression of one of two enzymes: NADH dehydrogenase and the NADH-dependent malate dehydro

57 not caspase 3, and significantly suppressed NADH dehydrogenases and cytochrome c oxidases, consisten

58 t low RSV doses (1-5 muM) directly stimulate NADH dehydrogenases and, more specifically, mitochondria

59 (ND2 and ND5) encode interacting subunits of NADH dehydrogenase, and amino residues that were inferre

60 uo genes not present in C. jejuni encode the NADH dehydrogenase, and in their place in the operon are

61 of flavoenzymes, including xanthine oxidase, NADH dehydrogenase, and NADPH oxidase.

62 native oxidase activity and that alternative NADH dehydrogenases are also up-regulated in these plant

63 Four NADH dehydrogenases are encoded in the genome, suggestin

64 e or when devoid of quinones, implicating an NADH dehydrogenase as their source.

65 ochondrial genes, those encoding subunits of NADH-dehydrogenase as well as cytochrome c oxidase subun

66 ays (additional photosystem genes, duplicate NADH dehydrogenase, ATP synthases), whose functionality

67 ransport proteins from Arabidopsis thaliana, NADH dehydrogenase B14.7 like (B14.7 [encoded by At2g422

68 tered respiratory function, as inhibition of NADH dehydrogenase brought ROS levels back to wild-type

69 er, in permeabilized cells NDI1 (alternative NADH dehydrogenase) bypassed complex I inhibition, where

70 Related to the respiratory NADH dehydrogenase complex (complex I), NDH transfers el

71 t represents a minor form of the respiratory NADH dehydrogenase complex (complex I).

72 Defects of the NADH dehydrogenase complex are predominantly manifested

73 proteins and to a subunit of the eukaryotic NADH dehydrogenase complex I.

74 ND6 is a subunit of the NADH dehydrogenase complex, also known as Complex I, the

75 mutant lacking nuoG, a subunit of the type I NADH dehydrogenase complex, exhibits attenuated growth i

76 The ND4 gene encodes one subunit of the NADH dehydrogenase complex, part of the oxidative phosph

77 systems but up-regulation of the chloroplast NADH dehydrogenase complex, plastocyanin, and Ca(2+) sen

78 nd to those of the isolated P. denitrificans NADH-dehydrogenase complex.

79 ely 40 subunits of the mammalian respiratory NADH dehydrogenase (Complex I) are encoded in mitochondr

80 iratory activity concurs with that of type I NADH dehydrogenase (complex I) in some Leishmania specie

81 nate, which suggests that rotenone-sensitive NADH dehydrogenase (complex I) is present in these mitoc

82 en observed in the level of the mRNA for the NADH dehydrogenase (complex I) ND6 subunit gene, which i

83 Enzymologic analysis of mitochondrial NADH dehydrogenase (complex I) with submitochondrial par

84 ha blocked electron transfer at three sites, NADH dehydrogenase (complex I), succinate dehydrogenase

85 ting evolutionary links to the mitochondrial NADH dehydrogenase (Complex I).

86 ng type I nicotinamide adenine dinucleotide (NADH) dehydrogenase (Complex I) is found in low abundanc

87 inhibition of the respiratory chain enzymes NADH-dehydrogenase (complex I) and succinate dehydrogena

88 ulation of the chloroplast photosystem I and NADH dehydrogenase complexes and had been proposed to fa

89 Our study reveals that the same NADH dehydrogenase complexes are utilized under oxic con

90 hat functional variation in cytochrome b and NADH dehydrogenase could mechanistically contribute to l

91 n transport chain, a large membrane-embedded NADH dehydrogenase, couples electron transfer to the rel

92 c for each type of mitochondrial lesion: the NADH dehydrogenase-defective NCS2 mutant has high expres

93 e was just sufficient to support the maximum NADH dehydrogenase-dependent respiration rate, with no u

94 However, NADH dehydrogenase-dependent respiration, as measured in

95 Saccharomyces cerevisiae is catalyzed by an NADH dehydrogenase designated Ndi1p.

96 rity to the 18-kD Fe-S subunit of complex I (NADH dehydrogenase, EC 1.6.5.3) in the mitochondrial ele

97 NDI1 is a yeast NADH dehydrogenase enzyme that complements the loss of m

98 le-genome scanning technique to identify the NADH dehydrogenase gamma subunit (nuoG) primer set that

99 NADH dehydrogenase gene mutations preferentially accumul

100 We report the first molecular defect in an NADH-dehydrogenase gene presenting as isolated myopathy.

101 iochlorophyll biosynthesis, cbb3 oxidase and NADH dehydrogenase genes, as well as genes for autotroph

102 l DNA-encoded ND5 subunit of the respiratory NADH dehydrogenase has been isolated and characterized.

103 A rotenone-insensitive NADH dehydrogenase has been isolated from the mitochondr

104 ng a bacterial microcompartment (BMC), and a NADH dehydrogenase (hdrABC-flxABCD) complex.

105 tides (CsmR, CsmS, CsmT, CsmU, and a type II NADH dehydrogenase homolog).

106 lcarbodiimide, an inhibitor of mitochondrial NADH dehydrogenase I (also called complex I), inhibits t

107 ase A, and FNR) and membrane-bound proteins (NADH dehydrogenase I and succinate dehydrogenase).

108 nic message with the putative nuoF (encoding NADH dehydrogenase I chain F), secF (encoding protein ex

109 ansport chain, especially those encoding the NADH dehydrogenase I complex.

110 -containing aconitase, serine deaminase, and NADH dehydrogenase I enzymes of S. Typhimurium under bas

111 energetically efficient proton-translocating NADH dehydrogenase I is used in preference to the non-pr

112 DH-quinone oxidoreductase (energy-conserving NADH dehydrogenase I) from various eukaryotic and prokar

113 Aer-mediated responses were linked to NADH dehydrogenase I, although there was no absolute req

114 port chain, including cytochrome bo oxidase, NADH dehydrogenase I, NADH dehydrogenase II, and succina

115 Thus NADH dehydrogenase II but not NADH dehydrogenase I, respiratory quinones, or cytochrom

116 oxygen are associated with redox changes in NADH dehydrogenase I.

117 ssed the proton pumps cytochrome o (cyo) and NADH dehydrogenases I and II.

118 However, mutants that lacked NADH dehydrogenase II and fumarate reductase, the most o

119 Null mutations in the gene encoding NADH dehydrogenase II averted autoxidation of vesicles,

120 Thus NADH dehydrogenase II but not NADH dehydrogenase I, resp

121 NADH dehydrogenase II did generate substantial H2O2 when

122 n preference to the non-proton translocating NADH dehydrogenase II during periods of rapid growth, by

123 ne that encodes the non-proton-translocating NADH dehydrogenase II of Escherichia coli is anaerobical

124 NADH dehydrogenase II that was purified from both wild-t

125 cytochrome bo oxidase, NADH dehydrogenase I, NADH dehydrogenase II, and succinate dehydrogenase.

126 uired for the activity of the copper-related NADH dehydrogenase II.

127 ided additional evidence for the presence of NADH dehydrogenase in bloodstream forms of T. brucei.

128 lude that AtNDB2 is the predominant external NADH dehydrogenase in mitochondria and together with AtA

129 een well characterized, however, the role of NADH dehydrogenases in feeding electrons to Mtr has been

130 ine, coding for ND5, a subunit of complex I (NADH dehydrogenase) in the electron transport chain.

131 on in the subunit for respiratory complex I, NADH dehydrogenase, in the ND6 gene.

132 terized the iron-sulfur protein required for NADH dehydrogenase (INDH) in the model plant Arabidopsis

133 where ndhF is the ND5 protein of chloroplast NADH dehydrogenase) indicate that Hesperomannia belongs

134 ion to MPP(+) by a monoamine oxidase and (b) NADH dehydrogenase inhibition by MPP(+).

135 itochondrial ATP production, and rotenone, a NADH dehydrogenase inhibitor, were also tested.

136 ent of intact mitochondria revealed that the NADH dehydrogenase is located in the inner membrane/matr

137 ncentrations of digitonin suggested that the NADH dehydrogenase is loosely bound to the inner mitocho

138 spectively, by rotenone, which suggests that NADH dehydrogenase is present in these cells.

139 onella strain bearing mutations in complex I NADH dehydrogenases is refractory to the early NADPH oxi

140 Mitochondrial complex I (NADH dehydrogenase) is a major contributor to neuronal e

141 it synthesis, but that assembly of RCI (i.e. NADH dehydrogenase) is far less efficient, with dramatic

142 e BAT-specific first mammalian NDE (external NADH dehydrogenase)-like enzyme, Aifm2 oxidizes NADH to

143 NADH dehydrogenase-like (NDH) complex is enriched in the

144 Chloroplast NADH dehydrogenase-like (NDH) complex mediates cyclic el

145 y plays a role in the down-regulation of the NADH dehydrogenase-like complex-dependent plastoquinone

146 tor ACHT2 results in increased levels of the NADH dehydrogenase-like complex.

147 mplex (PGR) and one that is dependent of the NADH dehydrogenase-like complex.

148 ROS are produced from complex I by the NADH dehydrogenase located in the matrix side of the inn

149 xidized PQ pool upon inactivation of type II NADH dehydrogenase may be related to the facts that the

150 lasmic mtDNA mutations affecting subunits of NADH dehydrogenase may play a synergistic role in the es

151 alternative NADH:ubiquinone oxidoreductases (NADH dehydrogenases) may protect against oxidative stres

152 Nicotinamide Adenine Dinucleotide Hydrogen (NADH) dehydrogenase (MNdh).

153 Here we describe a novel NADH dehydrogenase module of respiratory complex I that

154 n regulatory mtDNA elements, but only rarely NADH dehydrogenase mutations.

155 The sodium-dependent NADH dehydrogenase (Na(+)-NQR) is a key component of the

156 The sodium-dependent NADH dehydrogenase (Na(+)-NQR) is the main ion transport

157 ith no change in CcO activity, and inhibited NADH dehydrogenase (NADH-DH) activity (P<0.01) without a

158 the ATPase 6 subunit gene (ATP), ATC for the NADH dehydrogenase (ND) 2 subunit gene, and ATT for the

159 perfectly conserved regions upstream of two NADH dehydrogenase (ND) genes are transcribed and likely

160 ncoding subunit ND4 of the respiratory chain NADH dehydrogenase (ND), did not affect the synthesis, s

161 We determined the activities of NADH dehydrogenase (ND), succinate dehydrogenase, and cy

162 gene for subunit 1 of the respiratory chain NADH dehydrogenase (ND1), complete genes for cytochrome

163 n of NDE1, Saccharomyces cerevisiae external NADH dehydrogenase (NDE), ameliorates impairment in gluc

164 In contrast, deletion of the external NADH dehydrogenases NDE1 and NDE2, which feed electrons

165 Deletion of cytochrome aa(3)-600 or NADH dehydrogenase (NDH) genes also increased B. subtili

166 both major clades of Erodium contain intact NADH dehydrogenase (ndh) genes, but the 11 ndh genes are

167 While NADH dehydrogenase (NDH) is a critical site of this O(2)

168 Exposure of NADH dehydrogenase (NDH), the flavin subcomplex of compl

169 Cytochrome bc1-aa3 oxidase and type-2 NADH dehydrogenase (NDH-2) are respiratory chain compone

170 Mycobacterial type II NADH dehydrogenase (NDH-2) is a promising drug target be

171 of the electron transport chain such as the NADH dehydrogenases (NDH-2 and NdhA) and the terminal re

172 s to act as allosteric inhibitors of type II NADH dehydrogenases (Ndh-2 of the electron transport cha

173 type 2 NADH:quinone oxidoreductase complex (NADH dehydrogenase [NDH]) from M. capsulatus Bath, along

174 hybrid metabolism is dependent on a type-II NADH dehydrogenase (Ndh2) and conditionally requires a f

175 t on the reduction of the quinone by type II NADH dehydrogenase (NDH2) for the generation of bacteric

176 mediators by L. plantarum requires a type-II NADH dehydrogenase (Ndh2); however, structural variation

177 Type II NADH dehydrogenases (NDH2) are monotopic enzymes present

178 Expression of NADH dehydrogenase NDI1 restores mitochondrial functions

179 metformin-resistant Saccharomyces cerevisiae NADH dehydrogenase NDI1 was overexpressed.

180 s rescued by bypassing complex I using yeast NADH dehydrogenase Ndi1.

181 restored by ectopic expression of the yeast NADH dehydrogenase Ndi1.

182 topic expression of Saccharomyces cerevisiae NADH dehydrogenase (NDI1) or Ciona intestinalis alternat

183 Conditional expression of yeast NADH dehydrogenase (NDI1) protein that regenerates NAD(+

184 accharomyces cerevisiae internal alternative NADH dehydrogenase (NDI1) protein to determine whether t

185 on anesthetic sensitivity in mice expressing NADH dehydrogenase (NDi1).

186 Complex I (NADH dehydrogenase, NDU) and complex IV (cytochrome-c-ox

187 nteny group of THRSP and its flanking genes [NADH dehydrogenase (NDUFC2) and glucosyltransferase (ALG

188 with the rotenone-insensitive single-subunit NADH dehydrogenase of Saccharomyces cerevisiae (NDI1), w

189 s, which encode the major, energy-generating NADH dehydrogenase of the cell.

190 veloped to specifically target the alternate NADH dehydrogenase of the malaria parasite respiratory c

191 NADH dehydrogenase or complex I (CI) is affected in most

192 Mutations of respiratory NADH dehydrogenases prevent nitrotyrosine formation and

193 enol and the presence of subunits 7 and 8 of NADH dehydrogenase provided additional evidence for the

194 g human cell lines carrying a frame-shift at NADH dehydrogenase (respiratory complex I) subunit 5 gen

195 Mutants that lacked both NADH dehydrogenases respired very slowly, as expected; h

196 lipolytica carrying an internal alternative NADH dehydrogenase resulted in slower growth and strongl

197 itochondrial genes (cytochrome b (Cytb), the NADH dehydrogenase subunit 1 (ND1) and cytochrome oxidas

198 nding peptides allowed us to characterize an NADH dehydrogenase subunit 1 (ND1)-derived peptide as th

199 Here, we demonstrate that the NADH dehydrogenase subunit 1 self-peptide is seen by mat

200 arms and different conformations of the ND1 (NADH dehydrogenase subunit 1) loop near the quinol bindi

201 various DE genes, including cytochrome P450, NADH dehydrogenase subunit 1, cuticle protein and heat s

202 s allowed us to characterize a mitochondrial NADH dehydrogenase subunit 1-derived 9-mer peptide as th

203 transversion in the mitochondrially encoded NADH dehydrogenase subunit 2 (mt-ND2, human; mt-Nd2, mou

204 n unusual ATC (non-AUG) codon initiating the NADH dehydrogenase subunit 2 (ND2) gene.

205 l function of the mtDNA mutations, we cloned NADH dehydrogenase subunit 2 (ND2) mutants based on prim

206 By using mitochondrial NADH dehydrogenase subunit 2 (ND2) sequences and 467 amp

207 rial genes, namely, cytochrome b (CYT B) and NADH dehydrogenase subunit 2 (ND2), from 383 archived sp

208 europilin (ESDN), prostatic binding protein, NADH dehydrogenase subunit 2, and an unknown protein.

209 NADH dehydrogenase subunit 2, encoded by the mtDNA, has

210 s (cardiac alpha-actin, cyclin G1, stathmin, NADH dehydrogenase subunit 2, titin and prostatic bindin

211 3'-terminal sequence of mitochondria-encoded NADH dehydrogenase subunit 3 (ND-3).

212 We sequenced the NADH dehydrogenase subunit 3 (ND3) gene from a sample of

213 nce, containing part of the tRNA glycine and NADH dehydrogenase subunit 3 genes, is the target of our

214 scripts in a 24-hour time course showed that NADH dehydrogenase subunit 4 mRNA decreased by 2-fold as

215 -annotated 28 C-terminal residues within the NADH dehydrogenase subunit 4.

216 id evolutionary rates, 16S rRNA (379 bp) and NADH dehydrogenase subunit 5 (NADH-5, 318 bp), from mult

217 the cytochrome c oxidase subunit I (COI) and NADH dehydrogenase subunit 5 (ND5) genes each include a

218 chain (ETC), is a key source of ROS and the NADH dehydrogenase subunit 5 (ND5) is a highly conserved

219 for cytochrome c oxidase subunit I (COI) and NADH dehydrogenase subunit 5 (ND5) of the sea anemone Me

220 acids in cytochrome c oxidase subunit 1 and NADH dehydrogenase subunit 5.

221 within the mitochondrial DNA (mtDNA)-encoded NADH dehydrogenase subunit 6 (ND6) gene has been identif

222 led a novel variant of the gene encoding the NADH dehydrogenase subunit 6 (ND6) protein in the tumor

223 rotein S12 (RPS12), the 5' editing domain of NADH dehydrogenase subunit 7 (ND7 5'), and C-rich region

224 g CO2/HCO3 (-)-uptake mutant DeltandhD3 (for NADH dehydrogenase subunit D3)/ndhD4 (for NADH dehydroge

225 or NADH dehydrogenase subunit D3)/ndhD4 (for NADH dehydrogenase subunit D4)/cmpA (for bicarbonate tra

226 creased mRNA levels from both mitochondrial (NADH dehydrogenase subunit IV) and nuclear [cytochrome c

227 anscripts nicotinamide adenine dinucleotide (NADH) dehydrogenase subunit 4 and cytochrome b were down

228 rring peptide derived from the mitochondrial NADH-dehydrogenase subunit 1 gene (9-mer peptide).

229 t low peptide concentrations, shortening the NADH-dehydrogenase subunit 1 gene 9-mer peptide or mutat

230 emoglobin [HBG] gene or the amount of mtDNA (NADH dehydrogenase, subunit 1) relative to HBG.

231 ng events in cytochrome oxidase subunit2 and NADH dehydrogenase subunit4 transcripts, encoding subuni

232 the slo3 mutant was defective in splicing of NADH dehydrogenase subunit7 (nad7) intron 2.

233 ial for the translation of the mitochondrial NADH dehydrogenase subunit7 (nad7) mRNA.

234 3.8-kb mtDNA deletion in the region encoding NADH dehydrogenase subunits 1 and 2 and cytochrome c oxi

235 ndrial DNA (mtDNA) mutations associated with NADH dehydrogenase subunits and nuclear gene mutations t

236 We observed that transcripts of most NADH dehydrogenase subunits are edited inefficiently in

237 nes, including 11 ndh genes which encode the NADH dehydrogenase subunits, and a significant size redu

238 including those for ATP6, ATP8 synthase, and NADH dehydrogenase subunits, supporting electron microsc

239 ion, especially for ATP6, ATP8 synthase, and NADH dehydrogenase subunits.

240 f the mitochondrial genes encoding the seven NADH-dehydrogenase subunits showed a G-to-A transition a

241 nt formate dehydrogenase and a member of the NADH dehydrogenase superfamily.

242 sidered to be the minimal form of the type I NADH dehydrogenase, the first enzyme complex in the resp

243 A new family of NADH dehydrogenases, the flavin oxidoreductase (FlxABCD,

244 fully reduced in the mutant without type II NADH dehydrogenase, thus causing regulatory inhibition.

245 in vitro, in the gene for the ND4 subunit of NADH dehydrogenase) to undergo transcomplementation of t

246 Multiple NADH dehydrogenases, transcription factors of unknown fu

247 mical reduction of the plastoquinone pool by NADH dehydrogenases type-1 and type-2 (NDH-1 and NDH-2).

248 ncoding a nuclear DNA-encoded subunit of CI (NADH dehydrogenase ubiquinone Fe-S protein 4), typically

249 ndrial membrane, where it interacts with the NADH dehydrogenase (ubiquinone) 1 alpha subcomplex subun

250 (ubiquinone) 1beta subcomplex subunit 8, and NADH dehydrogenase (ubiquinone) 1alpha subcomplex subuni

251 rogenase (ubiquinone) iron-sulfur protein 3, NADH dehydrogenase (ubiquinone) 1beta subcomplex subunit

252 e show that human fibroblasts mutant for the NADH dehydrogenase (ubiquinone) Fe-S protein 1 (NDUFS1)

253 t the 49-kDa mitochondrial complex I subunit NADH dehydrogenase (ubiquinone) Fe-S protein 2 (NDUFS2)

254 ochondrial respiratory chain subunit Ndufs4 [NADH dehydrogenase (ubiquinone) Fe-S protein 4], delays

255 chondrial membranes, decreased levels of the NADH dehydrogenase (ubiquinone) iron-sulfur protein 3, N

256 Loss of murine Ndufs4, which encodes NADH dehydrogenase (ubiquinone) iron-sulfur protein 4, r

257 and backbone carbonyl group of M1 of NDUFA1 (NADH dehydrogenase [ubiquinone] 1 alpha subcomplex subun

258 Furthermore, we also identified NADH dehydrogenase [ubiquinone] 1 alpha subcomplex subun

259 Mice missing the Complex I subunit NADH dehydrogenase [ubiquinone] iron-sulfur protein 4 (N

260 he unexpected loss of respiratory complex I (NADH dehydrogenase), universally present in all 300+ oth

261 As anticipated, however, NADH dehydrogenase was inhibited by these rotenoids.

262 The presence of three subunits of NADH dehydrogenase was observed in immunoblots of mitoch

263 l and oxidative phosphorylation pathways and NADH dehydrogenase were downregulated, and there was upr

264 ochondrial proteins, including aconitase and NADH dehydrogenases, were oxidized and their activities