ライフサイエンスコーパス: NO

1 NO activates protein kinase G with the subsequent produc

2 NO and iron nitrosyl species (FeNOs), are relatively uns

3 NO decay in SMCs was measured following bolus addition o

4 NO permeates this tunnel and leverages upon the gating s

5 NO receptor machinery includes both canonical and novel

6 NO(2)-OA also significantly reduced RyR2-phosphorylation

7 For the whole tree, (15) NO(3) (-) contributed 39% to 90% to total (15) N tracer

8 labeling experiment ((15) NH(4) (+) vs (15) NO(3) (-) ) over 15 d to quantify ammonium and nitrate u

9 of nitromethane (i.e., CD(3)NO(2), CH(3)(15)NO(2), and (13)CH(3)NO(2)), revealing this easy-to-handl

10 th varying concentrations of liquid C(6)H(15)NO (assuming it presented as Diethylethanolamine).

11 precursors NO(x) (nitrogen oxides = NO(2) + NO) and VOC (volatile organic compounds) have decreased.

12 ce of common anions (Cl(-), Br(-), NO(2)(-), NO(3)(-), SO(4)(2-), and HCO(3)(-)).

13 l substances in the product water (NO(2)(-), NO(x)(-), NH(4)(+), SO(4)(2-), and heavy metals) or in u

14 Under hydrocarbon free air, CO, SO(2), NO, NO(2) and VOCs (mainly aldehydes, ketones and a carb

15 by the physiologically relevant MPO-H(2)O(2)-NO(2) (-) system.

16 le isotopologues of nitromethane (i.e., CD(3)NO(2), CH(3)(15)NO(2), and (13)CH(3)NO(2)), revealing th

17 e., CD(3)NO(2), CH(3)(15)NO(2), and (13)CH(3)NO(2)), revealing this easy-to-handle compound as a vers

18 o(2) (S-mandelate)(2) (4,4'-bipyridine)(3) ](NO(3) )(2) , CMOM-1S, is a modular MOF; five new variant

19 The contribution of NO(3)(-)/NO(2)(-) to the nitration and nitrosation processes was

20 )) LPt(IV)F(py)(Ar)X (X = CN, Cl, 4-OC(6)H(4)NO(2)) and LPt(IV)F(2)(Ar)(HX) (X = NHAlk; Alk = n-Bu, P

21 retain laboratory-generated NaNO(3) and NH(4)NO(3) particles, with maximum standard deviations for de

22 for complete dissociation of HNO(3) and NH(4)NO(3), suggesting that this channel may not constitute a

23 In this study, a NO nanoreporter (NO-NR) is reported that enables real-ti

24 a or hyperoxia could be explained by altered NO degradation in the parenchyma.

25 e accumulation, reduces vasoactivity, alters NO production, which leads to endothelial cell activatio

26 In addition, ambient NO(2) concentrations at the residence exceeded those mea

27 tation declines and the reduction of ambient NO(2) exposure.

28 ng a dielectric layer, is able to achieve an NO reduction in excess of 2X that of a platinum group me

29 ClopHensor is not only a Cl(-) but also, an NO(3) (-) sensor.

30 Actuating electron transfer with O(2) and NO movements averts irreversible NO poisoning and reduct

31 re poorly defined but could involve O(2) and NO transport and/or metabolism, respectively.

32 tly less sensitive to reaction with O(2) and NO when SvWhiD was bound to sigma(HrdB) (4), consistent

33 uggests air quality rules aimed at SO(2) and NO(x) emissions induce the cobenefit of reducing organic

34 ns and externality costs of GHGs, SO(2), and NO(x) from direct and upstream effects.

35 ization of ISEs for K(+), Na(+), Ca(2+), and NO(3)(-).

36 e, and found synergism between WO(4)(2-) and NO(2)(-), while additive effects were observed with ClO(

37 ive effects were observed with ClO(4)(-) and NO(3)(-).

38 ations of primary combustion tracers (BC and NO(x)) near 30% of metal recycling and concrete batch pl

39 Both CO and NO inhibited respiration, and treatment with Ngb-H64Q-CC

40 ion by increasing eNOS complex formation and NO production.

41 SA exhibited excellent HOO(*) and NO(2) scavenging activity in water (k(overall)(HOO(*)) =

42 e removal percentages for both NO(2)(-)N and NO(3)(-)N of approximately 99%.

43 d high removal efficiencies of NO(2)(-)N and NO(3)(-)N, with the removal percentages for both NO(2)(-

44 mean (GM) of outdoor PM(2.5), BaP, NAP, and NO(2) were 45.3 ug/m(3), 9.7 ng/m(3), 707.7 ng/m(3), and

45 atible, especially compared to other Nef and NO(x)-generating processes, and reveal selectivity over

46 eduction of both ammonia-derived nitrite and NO could lead to nitrous oxide (N(2)O) production.

47 e methane partial oxidation reaction, NO and NO(2) were not reduced to N(2) .

48 ineage-specific diversifications of NOSs and NO/nitrite/nitrate sensors from the common ancestor of M

49 l age (<~1 d) and high NO are needed, OH and NO generation by organic-nitrite photolysis in the UVA r

50 terferences of the molecular bands of PO and NO as well as the iron lines with Se line at 196.026 wer

51 od gases, Hct and [Hb], blood viscosity, and NO metabolites (ozone-based chemiluminescence) were meas

52 based increases of the emissions of VOCs and NO(x) stemming from U.S. oil and natural gas (O&NG) sour

53 proposed to create a screening layer around NO sensors, protecting against such chemical interferenc

54 The average NO(x) emission rate increases with engine load and decre

55 udents who exhibited no relationship between NO(2) exposure and symptom days (OR, 0.90; 95% CI, 0.57-

56 )(-)N, with the removal percentages for both NO(2)(-)N and NO(3)(-)N of approximately 99%.

57 the presence of common anions (Cl(-), Br(-), NO(2)(-), NO(3)(-), SO(4)(2-), and HCO(3)(-)).

58 cortex, these electrodes could detect brain NO released by local microinjection of the glutamatergic

59 dues containing fluorine, chlorine, bromine, NO(2), methyl, dimethyl, and methoxy, as well as 2-pyrid

60 ble fast aqueous-phase oxidation of SO(2) by NO(2), producing HONO which can in turn oxidize SO(2) to

61 amics may be emergent phenomena generated by NO degradation by the blood or parenchyma.

62 dine monoxide radical (IO) is intercepted by NO(2) to form the iodine nitrate (IONO(2)).

63 aging of BB tar proxy aerosols processed by NO(3)(*) under dark conditions followed by the photochem

64 Solutions of variable NaNO(3), Ca(NO(3))(2), and humic acid (HA) concentrations were used

65 sothiols (P = 0.03) and total red blood cell NO (P = 0.001) were collectively reduced by ~15-40%.

66 site reducing systems that regulate cellular NO decay, we assessed the intracellular concentrations o

67 c oxide (NO) produced through the citrulline-NO pathway promotes CR-triggered hypothermia and that le

68 g metal salts: NiX(2) with X = Br(-), Cl(-), NO(3)(-), and OAc(-).

69 Classroom NO(2) data, linked to enrolled students, were collected

70 to evaluate relationships between classroom NO(2) exposure and asthma symptoms and morbidity by body

71 etermined the relationship between classroom NO(2) levels and asthma outcomes by BMI stratification.

72 ears to increase susceptibility to classroom NO(2) exposure effects on asthma symptoms in inner-city

73 allows us to detect the intermediates of (CO+NO) reaction via IR measurements on Rh cations on zeolit

74 pollutant anomalies attributable to complex NO(x) chemistry and long-distance transport of fine part

75 demonstrated 80-120% benefit of controlling NO(x) emissions on NO(y) deposition.

76 viant subgroup is the cytochrome c dependent NO reductases (cNOR), which reduce nitric oxide to nitro

77 through constraint of endothelium-dependent NO-mediated vasodilatation in healthy humans.

78 mpatible with a role for the fast-diffusible NO gas in signaling and cell-cell communication via the

79 s a reducing agent in the SCR and diminishes NO conversion and N(2) selectivity.

80 ulfur dioxide (SO(2)), and nitrogen dioxide (NO(2)) over two consecutive 24-h sampling periods in 29

81 -cost passive samplers for nitrogen dioxide (NO(2)), which complement data from the sparse reference

82 evated levels of gas-phase nitrogen dioxide (NO(2)).

83 luded regional pollutants (nitrogen dioxide [NO(2)] or particulate matter with an aerodynamic diamete

84 branching ratio <0.2%, indicating efficient NO(x) recycling.

85 ion for hypothetical interventions on either NO(2) or PM(2.5).

86 multiphase chemical processing from elevated NO(x) and SO(2) within the oil field.

87 Specifically, nitric oxide emissions (NO) lead to increased smog, acid rain, climate change, a

88 enhancement of sGC sensitivity to endogenous NO.

89 ivated protein kinase (MAPK) and endothelial NO synthase (eNOS) in EA.hy926 cells treated with condit

90 285222 treatment enhanced dermis endothelial NO synthase expression and plasma NO levels of diabetic

91 mice resulting from a disturbed endothelial NO pathway and increased endothelial oxidative stress.

92 stically, higher Abeta42 reduced endothelial NO synthase (eNOS), cyclic GMP (cGMP), and protein kinas

93 , localizes in SECs with eNOS in a GIT1/eNOS/NO signaling module.

94 NMDA-evoked NO release peaked at 1.1 muM and lasted more than 20 min

95 For example, NO(2) groups positioned over a biaryl bond exhibited sim

96 The metal-free flavin/NO(x)/TEMPO catalytic cycles are uniquely compatible, es

97 ease in volatile reactive nitrogen gas flux (NO(y) = NO, NO(2) , HONO) as ECM tree abundance increase

98 entration increases ranging from 2 to 3x for NO(2), and >9x for NO.

99 ranging from 2 to 3x for NO(2), and >9x for NO.

100 ose with, but interaction was found only for NO(2.) CONCLUSIONS: People with rhinitis who live in are

101 s per trillion by volume (10(-12), pptv) for NO(2) and 148 pptv for ammonium nitrate.

102 Here, we identify a novel role for NO in strengthening inhibitory GABA(A) receptor-mediated

103 Arginine, the substrate for NO production, becomes acutely deficient in SCD patients

104 gesting that these tyrosines are targets for NO-dependent downregulation.

105 les, and biomarkers of endothelial function (NO(2)(-) and ADMA).

106 Furthermore, NO reduction by both pathways is restricted to chlorophy

107 reverse Hofmeister series (Cl(-) >= Br(-) > NO(3)(-) > ClO(4)(-) > SCN(-)).

108 t for the photolytic process HNO(3) + hnu -> NO(2) + OH, leading to 2 x 10(-6) s(-1), about twice the

109 lly, we describe the reaction HNO(3) + OH -> NO(3) + H(2)O using a cluster model containing 21 water

110 trieved ratio of formaldehyde to NO(2) (HCHO/NO(2)), developed from theory and modeling, has previous

111 uggest promise for applying space-based HCHO/NO(2) to interpret local O(3) chemistry, particularly wi

112 Two-decade (1996-2016) multisatellite HCHO/NO(2) captures the timing and location of the transition

113 quivalent photochemical age (<~1 d) and high NO are needed, OH and NO generation by organic-nitrite p

114 ally, low and no sulphide, coupled with high NO(3) (-) , favoured the activity of Campylobacterales,

115 ed flight circuits, we find 37 +/- 6% higher NO(2) for non-whites and Hispanics living in low-income

116 O(2)N)(ClO(4)) (1a), this report illustrates NO release from nitrite at copper(II) following a proton

117 n brain tissue, though how this might impact NO signaling dynamics is not completely understood.

118 lglyoxal and glycerol, which in turn impacts NO detoxification.

119 diet-fed mice to mimic obese levels impaired NO production, vascular relaxation, and raised blood pre

120 CI = 1.01-1.25] per 10-mug/m(3) increase in NO(2) exposure).

121 For each 10-parts per billion increase in NO(2), obese students had a significant increase in the

122 s in eNOS-PAI-1 binding promote increases in NO production and enhance vasodilation in vivo.

123 the reduction of nitrites and participate in NO homeostasis.

124 Also, a 19% reduction in NO(2) was observed during the 2020-lockdown as compared

125 ased, offsetting the benefit of reduction in NO(y) deposition.

126 root juice (BRJ), has been shown to increase NO bioavailability and improve cardiovascular function i

127 e knockdown or antagonism of PAI-1 increases NO bioavailability.

128 deficient granuloma formation and inducible NO synthase (iNOS) induction, increased dissemination of

129 properties of a heme chaperone for inducible NO synthase, here we investigated whether heme delivery

130 nd differential cross sections for inelastic NO-He collisions in the 0.2 to 8.5 centimeter(-1) range

131 r 1 receptor (CSF1R) using a CSF1R inhibitor-NO-NR system leads to enhanced efficacy and better imagi

132 erapy response within PDAC and blocking iNOS/NO signaling may improve radiotherapy outcomes.

133 These data show the important role that iNOS/NO signaling plays in the effectiveness of radiotherapy

134 in leafy green vegetables is converted into NO in vivo to improve cardiovascular function.

135 th O(2) and NO movements averts irreversible NO poisoning and reductive inactivation of the enzyme.

136 We also find that isoprene/NO(x) pathway SOA mass primarily comprises organosulfate

137 velope were treated with FNA at 6.09 mg N/L (NO(2)(-) = 250 mg N/L, pH 5.0) for 24 h (conditions typi

138 eekend variability, to attribute tract-level NO(2) disparities to industrial sources and heavy-duty d

139 with the Hofmeister series (Cl(-) < Br(-) < NO(3)(-) <= ClO(4)(-) < SCN(-)).

140 To better define the Cygb-mediated NO dioxygenation process in vascular smooth muscle cells

141 jor reducing system supporting Cygb-mediated NO metabolism in SMCs with changes in cellular B5/B5R le

142 ular reducing systems regulate Cygb-mediated NO metabolism.

143 e involved in hypoxia responses and modulate NO concentration, which may explain their roles in plant

144 Among these gaseous molecules, NO, H(2)S, and CO occupy a special place because of thei

145 Over eight months of monitoring, NO(2) removal efficiency was 96% initially and decreased

146 ionship between the generation of myocardial NO and the occurrence of myocardial fibrosis.

147 Here we probe them in the case of Myoglobin-NO (MbNO) using element- and spin-sensitive femtosecond

148 In this study, a NO nanoreporter (NO-NR) is reported that enables real-time monitoring of

149 We utilize the high-resolution FIVE and NEI NO(x) inventories, plus one year of TROPOMI weekday-week

150 take up ammonium (NH(4) (+) ) over nitrate (NO(3) (-) ).

151 g the few eukaryotes known to store nitrate (NO(3) (-) ) and to use it as an electron acceptor for re

152 cellular electron acceptors such as nitrite (NO(2)(-)) or nitric oxide (NO).

153 ransition-metal-mediated routes for nitrite (NO(2)(-)) to nitric oxide (NO) conversion and phenol oxi

154 ach via aqueous reactions involving nitrite (NO(2)(-)) and ammonia (NH(3)), respectively.

155 lusive action of the acid (H(+)) or nitrite (NO(2)(-)) counterparts.

156 a robust measurement of total odd nitrogen (NO(y)) in environments where NaNO(3) particles may be pr

157 eduction in deposition of oxidized nitrogen (NO(y), the sum of all oxidized nitrogen species, except

158 Control of nitrogen oxides (NO(x) = NO + NO(2)) emissions has led to reduction in deposition of o

159 Under hydrocarbon free air, CO, SO(2), NO, NO(2) and VOCs (mainly aldehydes, ketones and a carboxyl

160 tile reactive nitrogen gas flux (NO(y) = NO, NO(2) , HONO) as ECM tree abundance increases.

161 e central body (CBL), and the paired noduli (NO).

162 lateral accessory lobe (LAL) to the noduli (NO).

163 knockdown experiments indicated that ~78% of NO metabolism in SMCs is Cygb-dependent.

164 MCs was measured following bolus addition of NO to air-equilibrated cells.

165 plest free-living animals, the complexity of NO-cGMP-mediated signaling in Placozoa is greater to tho

166 guanylyl cyclase (sGC) is a key component of NO-cGMP signaling in mammals.

167 ffected by acute increased concentrations of NO precursors in hypoxia.

168 The contribution of NO(3)(-)/NO(2)(-) to the nitration and nitrosation proce

169 tions driven by the increased degradation of NO by the blood, and vasomotion-like 0.1-0.3 Hz oscillat

170 in F2 exhibited high removal efficiencies of NO(2)(-)N and NO(3)(-)N, with the removal percentages fo

171 fers from our results that the hydrolysis of NO(2) in clouds must be catalyzed by organic or inorgani

172 oping stable pro-drugs with slow kinetics of NO release.

173 on P. tricornutum prey with lower levels of NO, suggesting that this molecule and its effect on oxyl

174 vailable about the sources and metabolism of NO and its bioactive metabolites (NOx) in both normal an

175 Using a mixture of NO + O(2) as the oxidant enabled the direct selective ox

176 (3))(2)](+) at 200 degrees C in a mixture of NO and NH(3).

177 e effects of sulphide on the partitioning of NO(3) (-) between complete denitrification and DNRA and

178 s and examined their roles in the process of NO decay.

179 ular B5/B5R levels modulating the process of NO decay.

180 O oxidation) and L-citrulline (co-product of NO synthesis from L-arginine), which were affected by NO

181 The activity of iNOS and production of NO(-) by macrophages following stimulation is one of the

182 resulting in their activation, production of NO, and subsequent destruction of parasites.

183 Although products of NO metabolism (NOx) also have significant bioactivity, l

184 assays, we quantified nitrites (products of NO oxidation) and L-citrulline (co-product of NO synthes

185 The much higher rate of NO degradation and scavenging of NO in the blood relativ

186 However, the rate of NO degradation in hemoglobin is orders of magnitude high

187 Loss rates of NO(2) and O(3) through the air-cleaning system were ~1.5

188 The heterogeneous reaction of NO(2) with water on diverse surfaces is broadly consider

189 The reduction of NO to N(2)O by flavodiiron nitric oxide reductases (FNOR

190 oactivity, little is known about the role of NO and NOx under conditions of aberrant placental inflam

191 ings indicate that haemoglobin scavenging of NO appears to be an important factor in the regulation o

192 her rate of NO degradation and scavenging of NO in the blood relative to the tissue drove emergent va

193 2)O(5)-WO(3)/TiO(2) catalyst in NH(3)-SCR of NO(x) under dry conditions has been analyzed in detail b

194 nsor platform achieves continuous sensing of NO levels in living mammals for several days.

195 and robustly establish the full spectrum of NO bioactivity in plants, it will be essential to apply

196 ) Regulated 1 (SRG1), is a central target of NO bioactivity during plant immunity.

197 te of binding of O(2) is faster than that of NO and also leads to l-Trp nitration, while little evide

198 were then able to quantify the variations of NO(3) (-) and pH in the cytosol.

199 lassified as high and low responder based on NO(-) production.

200 0% benefit of controlling NO(x) emissions on NO(y) deposition.

201 l, our results highlight COVID-19 impacts on NO(2), and the results can inform pollution mitigation e

202 This is the first study on NO(*)-related molecular changes and SNO-signaling in the

203 )) = 1.53 x 10(8) M(-1) s(-1) and k(overall)(NO(2)) = 1.98 x 10(8) M(-1) s(-1)), whereas it did not s

204 Protein S-nitrosylation, the nitric oxide (NO(*))-mediated posttranslational modification (PTM) of

205 Nitric oxide (NO(-)) is a member of RNS produced from arginine by indu

206 , due in part to reductions in nitric oxide (NO) bioavailability.

207 utes for nitrite (NO(2)(-)) to nitric oxide (NO) conversion and phenol oxidation are of prime importa

208 Constant therapeutic gas phase nitric oxide (NO) delivery is achieved from S-nitrosothiol (RSNO) type

209 n (Cygb) functions as a potent nitric oxide (NO) dioxygenase and regulates NO metabolism and vascular

210 Real-time sensing of nitric oxide (NO) in physiological environments is critically importan

211 Nitric oxide (NO) is a gasotransmitter with important roles in pregnan

212 Nitric oxide (NO) is a key signaling molecule that regulates diverse b

213 Nitric oxide (NO) is a ubiquitous gaseous messenger, but we know littl

214 th and photosynthesis, altered nitric oxide (NO) level and leaf and root anatomy, inhibited enzyme ac

215 rning the effects of augmented nitric oxide (NO) on skeletal muscle force production and oxygen consu

216 mechanism, we designed a smart nitric oxide (NO) probe, PYSNO, with high sensitivity and selectivity.

217 Nitric oxide (NO) produced as a result of activation of macrophages to

218 harmacological approaches that nitric oxide (NO) produced through the citrulline-NO pathway promotes

219 lar complications and impaired nitric oxide (NO) production.

220 sociated with perturbations to nitric oxide (NO) signaling and impaired glucose metabolism.

221 human vasodilatory endothelial nitric oxide (NO) signaling.

222 ished by pretreatment with the nitric oxide (NO) synthase inhibitor l-N (G)-nitro-l-arginine methyl e

223 antly fills the phagosome with nitric oxide (NO) to clear the pathogen.

224 and immunosuppressive molecule nitric oxide (NO), whereas macrophages largely express antitumor prope

225 Among molecules modulating the nitric oxide (NO)-GMP-phosphodiesterase (PDE) pathway, the evaluation

226 n of endothelium-dependent and nitric oxide (NO)-mediated vasodilator activity, given its contributio

227 o O(2) and highly sensitive to nitric oxide (NO).

228 such as nitrite (NO(2)(-)) or nitric oxide (NO).

229 by converting hydroxylamine to nitric oxide (NO).

230 Control of nitrogen oxides (NO(x) = NO + NO(2)) emissions has led to reduction in de

231 he ozone precursors NO(x) (nitrogen oxides = NO(2) + NO) and VOC (volatile organic compounds) have de

232 lts indicate that inhibition of the PGE2/p50/NO axis prevents MDSC-suppressive functions and restores

233 ,N2,N2-tetramethylcyclohexane-1,2-diamine)Pd(NO(3))(2)] have been used to design enantiopure Pd(II) t

234 ripyridyl donor L.HNO(3) with cis-[(tmeda)Pd(NO(3))(2)] (M) [tmeda = N,N,N',N'-tetramethylethane-1,2-

235 were more active against lipid peroxidation, NO production, and tumour cells growth.

236 By mapping phagosomal NO produced in microglia of live zebrafish brains, we fo

237 s impressive progress has been made in plant NO research.

238 In plants, NO is generated in pollen tubes (PTs) and affects intrac

239 ndothelial NO synthase expression and plasma NO levels of diabetic mice.

240 responsible for variation in peak potential NO(y) flux.

241 bornly frequent even as the ozone precursors NO(x) (nitrogen oxides = NO(2) + NO) and VOC (volatile o

242 While copper enzymes promote NO release from RSNOs by serving as Lewis acids for intr

243 ding to the nitrate reductase (NR) promoter, NO production, and virulence in F. graminearum Our resul

244 ining soluble guanylate cyclases as putative NO/nitrite/nitrate sensors.

245 istribution of nitrogen oxide free radicals (NO(x)).

246 ring the methane partial oxidation reaction, NO and NO(2) were not reduced to N(2) .

247 g that the lifetime of the putative reactive NO(2) dimer on the surface of pure water droplets is too

248 cur as a result of interventions that reduce NO(2) or PM(2.5) concentrations.

249 cal catalytic membrane significantly reduces NO emissions.

250 -1 directly inhibits eNOS activity, reducing NO synthesis, and the knockdown or antagonism of PAI-1 i

251 nitric oxide (NO) dioxygenase and regulates NO metabolism and vascular tone.

252 sions of P. nitroreducens completely removed NO(2)(-) at various concentrations (1, 2, and 5 mM) from

253 iving in high-income tracts (HIW) and report NO(2) disparities separately by race ethnicity (11-32%)

254 hibition (with negative or neutral results), NO-independent soluble guanylate cyclase (sGC) activatio

255 On-road NO(x) emission rates have been much higher than type app

256 proach to reliably transform passive sampler NO(2) data from multiweek averages to annual-averaged va

257 he suboxic zone and coincided with secondary NO(2) (-) maxima and available NH(4) (+) .

258 10(8) M(-1) s(-1)), whereas it did not show NO scavenging activity in any of the studied environment

259 tch-clamp technique, we detected significant NO(3) (-) conductance of OsPIP1;3 in mammalian cells.

260 Since NO(-) production is strongly associated with host geneti

261 ion of highly specific microsensors to study NO physio-pathological actions in the brain.

262 tios to provide new perspectives on sulfate, NO(x,) and particle acidity influencing isoprene-derived

263 In addition, we also summarize NO detoxification and protective mechanisms against nitr

264 We additionally demonstrate that NO might have a crucial role in these responses.

265 Molecular dynamics simulations show that NO(3) (-) participates in the solvation sheath of lithiu

266 re we collate emerging evidence showing that NO bioactivity regulates a growing number of diverse pos

267 Several studies have suggested that NO plays a vast and diverse signaling role in molds.

268 The NO(2) temporal patterns matched the AOD signal; however,

269 The NO(3)(*)-aged wood tar aerosols are more susceptible to

270 distance from the city center increased, the NO(2) levels decreased exponentially.

271 ine methyl ester, while iontophoresis of the NO donor sodium nitroprusside eliminated the observed di

272 Additionally, the presence of the NO scavenging protein haemoglobin alpha (Hbalpha) within

273 monstrate the influence of isomerism of the -NO(2) substituents for the electrocatalytic multi electr

274 perated electrochemical sensor for real-time NO detection with a low detection limit (3.97 nmol), a w

275 In contrast to NO(3)(-) and SO(4)(2-) ions, the presence of PO(4)(3-) a

276 450 (CYP55), we show that FLVs contribute to NO reduction in the light, while CYP55 operates in the d

277 green leafy vegetables, can be converted to NO in vivo and demonstrates antidiabetic and antiobesity

278 n an increase in the contribution of DNRA to NO(3) (-) respiration.

279 Higher exposure to NO(2) was associated with an increased severity of rhini

280 satellite-retrieved ratio of formaldehyde to NO(2) (HCHO/NO(2)), developed from theory and modeling,

281 cation of the transition from VOC-limited to NO(x)-limited O(3) production regimes in major U.S. citi

282 oreductase-catalyzed reduction of nitrate to NO and independently of peroxisome proliferator-activate

283 VdAtf1 responds to NO stress by strengthening the fungal cell wall, and by

284 optimized for fast elimination of the toxic NO molecules.

285 In addition to traditional NO generators, a number of sGC activators and stimulator

286 is achieved from S-nitrosothiol (RSNO) type NO donor doped silicone rubber films using feedback-cont

287 Ultimately, NO accumulation leads to suppression and loss of mitocho

288 e impacts of COVID-19 on air pollution using NO(2) and Aerosol Optical Depth (AOD) from TROPOMI and M

289 NO from complex 1 with the pendant SH versus NO from 2 with the pendant SMe is achieved by the employ

290 lly harmful substances in the product water (NO(2)(-), NO(x)(-), NH(4)(+), SO(4)(2-), and heavy metal

291 Both pathways are active when NO is produced in vivo during the reduction of nitrites

292 The top cities where NO(2) reduction occurred were New Delhi (61.74%), Delhi

293 teristics of bovine MDMs are associated with NO-based classification.

294 ceeds via a reaction of these complexes with NO.

295 The reaction of BCP-peroxy with NO produces bicyclic hydroxy nitrate with a branching ra

296 The reaction with NO results in an almost complete reduction to Cu(I), und

297 imuli and diverse biochemical reactions with NO.

298 hydride equilibrium is explored for the {WTp(NO)(PBu(3))} (Bu = n-butyl; Tp = trispyrazoylborate) sys

299 Control of nitrogen oxides (NO(x) = NO + NO(2)) emissions has led to reduction in deposition

300 volatile reactive nitrogen gas flux (NO(y) = NO, NO(2) , HONO) as ECM tree abundance increases.