ライフサイエンスコーパス: NST

1 NST patients were more likely to have unrelated donors (

2 NST projection targets identified by these two approache

3 NSTs are linked to several developmental and immune diso

4 es a novel N-linked glycosylation site ((131)NST(133)).

5 ical stimulation of the LH on 36 DMNV and 14 NST neurons.

6 Each of 50 NST cells was classified as S-, N-, H-, or Q-best on the

7 ence suggests that, in patients over age 50, NST with fludarabine and low-dose busulfan leads to an o

8 the firing rates of 107 DMNV neurons and 68 NST neurons.

9 Of the 68 NST neurons characterized, 25 neurons were inhibited and

10 endogenously produced TNF(alpha) to activate NST neurons.

11 major sensory input into the NST, activated NST astrocytes, as indicated by increases in astrocytic

12 sensory inputs to NST neurons also activates NST astrocytes.

13 After NST (n = 473), the clipped TLN was intraoperatively rese

14 After NST, the MARI node was selectively removed using a gamma

15 Breast pCR achieved after NST is strongly correlated with ypN0 in cN0 patients, es

16 Before and after NST, treating surgeons evaluated BCT candidacy by clinic

17 es to ALND to identify axillary burden after NST in patients with pathologically confirmed node-posit

18 ty-seven (90%) remained BCT candidates after NST, of whom 138 (70%) chose BCT, which was successful i

19 g for the identification of breast pCR after NST raise the question whether breast surgery is a redun

20 of late enhancement and high ADC ratio after NST are associated with pCR.

21 be most accurate for axillary staging after NST.

22 se-negative rate (FNR) of TLNB and TAD after NST.

23 d to tailor further axillary treatment after NST.

24 Although NST activities are widespread, mammalian cells lack a GD

25 itivity demonstrating the role of Y499 as an NST facilitator.

26 Here we present the crystal structure of an NST, the GDP-mannose transporter Vrg4, in both the subst

27 ecimens compared with non-HIV-related KS and NST.

28 S cases compared with non-HIV-related KS and NST.

29 an association between CDKN2A mutations and NST (P = 0.52) or UM (P = 0.25).

30 exhibit the most differences between ST and NST sites, whereas subjects using ST for <10 years show

31 ween solution containing only AfB1, AfB1 and NSTs and no AfB1.

32 Ts causes several debilitating diseases, and NSTs are virulence factors for many pathogens.

33 were used to analyze a clade of Arabidopsis NSTs, resulting in the identification and characterizati

34 n this setting it can be difficult to assess NST properties because of the presence of glycosyltransf

35 leptin pre-treatment significantly augments NST neurons' responsiveness to TRH.

36 nce that the increased reliance on BAT-based NST depends on increased autonomic input, as indicated b

37 s demonstrate that both BAT and muscle-based NST are equally recruited during mild and severe cold ad

38 However, muscle-based NST in mammals remains poorly characterized.

39 genesis in rodents, the role of muscle-based NST is less obvious.

40 ated the importance of skeletal muscle-based NST.

41 185 (46%) who were not BCT candidates before NST, 78 (42%) converted to candidates with NST.

42 ed nineteen (54%) were BCT candidates before NST.

43 a marginally significant association between NST and ARF (P = 0.05).

44 neration of mixed hematopoietic chimerism by NST prior to tumor cell vaccination.

45 immunodeficient hosts could not be cured by NST, DLI, and vaccine administration.

46 characterize substrate-specific transport by NSTs; however, the availability of certain substrates an

47 Data base mining revealed 12 candidate NST genes in the L. major genome, including LPG2 as well

48 l (Ce) subnucleus in the intermediate/caudal NST.

49 as microinjected bilaterally into the caudal NST in adult rats.

50 pamine beta hydroxylase (DbH)] in the caudal NST were lesioned to determine their role in mediating a

51 o generate human norovirus-specific T cells (NSTs) that can recognize different viral sequences.

52 a-1 agonist, phenylephrine (PHE), to control NST neurones responsible for vago-vagal reflex regulatio

53 Telephone-delivered NST consisted of 10 sessions focused on providing a supp

54 ered CBT was superior to telephone-delivered NST in reducing worry, GAD symptoms, and depressive symp

55 includes a phasic response sent to different NST cells than a later tonic response, and only the latt

56 uses gastric stasis by activating downstream NST neurons that, in turn, suppress gastric motility via

57 DLH), while the spontaneous activity of each NST cell was recorded.

58 major ABO incompatibility undergoing either NST (fludarabine/cyclophosphamide conditioning) or myelo

59 TbNST4 is the first NST shown to transport both pyrimidine and purine nucleo

60 LPG2 is the first NST to be associated with multiple substrate specificiti

61 on to full donor myeloid chimerism following NST occurred significantly sooner in cases with, compare

62 pheral blood) was markedly delayed following NST versus myeloablative SCT (median, 114 versus 40 days

63 occurred in 4 of 14 (29%) patients following NST, while neither event occurred in 12 patients followi

64 inin levels and resolution of PRCA following NST.

65 y insignificant levels more slowly following NST than myeloablative SCT (median, 83 versus 44 days; P

66 The nonrelapse mortality rate was lower for NST patients (32% versus 50%; P = .01), but the relapse

67 a single polypeptide is solely required for NST activity and is able to mediate the uptake of multip

68 In addition, four NST neurons were antidromically invaded from the ipsilat

69 cterized the substrate specificities of four NSTs, TbNST1-4, which are expressed in both the insect p

70 recordings of miniature EPSCs (mEPSCs) from NST neurons show that astrocytes control presynaptic vag

71 ractices affected the effectiveness of FST + NST to increase yield but benefits were minimal ranging

72 vidence of a break-even cost of FST or FST + NST.

73 e seed treatment) + neonicotinoid use (FST + NST) reached 0.13 Mg/ha.

74 Furthermore, NSTs showed a high degree of cross-reactivity to multipl

75 cells drives the activity of adjacent (e.g., NST) neurons.

76 two genes (LPG5A and LPG5B) encoded UDP-Gal NSTs.

77 administered alone has no effect on gastric-NST or -DMN neuron responsiveness, or on gastric motilit

78 silateral RF using the location of gustatory NST as a point of reference.

79 oss the rostrocaudal extent of the gustatory NST (gNST), especially within its dorsomedial portion (s

80 mmunoreactive (FLI) neurons in the gustatory NST (gNST), particularly in the medial portion (subfield

81 ajority of descending input to the gustatory NST and PBN originates from distinct neuronal population

82 e TH-immunoreactive neurons of the gustatory NST receive direct input from the CT and taste receptors

83 vary with its target neuron in the gustatory NST.

84 H-NST proteins are found in large genomic islands in patho

85 at the protein level, the UPEC homologue (H-NST(UPEC)) has only a weak anti-H-NS activity, correlati

86 This correlates with the ability of H-NST(EPEC) to co-purify with H-NS in vitro, and can be ab

87 . coli (UPEC) shows that the EPEC protein (H-NST(EPEC)) has a potent anti-H-NS function at the classi

88 the DNA-binding domain, which we term the H-NST family.

89 Detailed analysis of the H-NST proteins from enteropathogenic E. coli (EPEC) and ur

90 n contrast, despite being 90% identical to H-NST(EPEC) at the protein level, the UPEC homologue (H-NS

91 cally deployed as seed treatments (hereafter NST) in many grain and oilseed crops, including soybeans

92 The hierarchical NST-Ni electrode was used to develop a sensitive and sel

93 How NSTs recognize and transport activated monosaccharides,

94 Dysregulation of human NSTs causes several debilitating diseases, and NSTs are

95 obular and tubular carcinomas); group B (IBC-NST, metaplastic, invasive apocrine and micropapillary c

96 ive breast carcinoma of no special type (IBC-NST) were submitted to evaluation of collagen parameters

97 produced rapid increases in [Ca(2)(+)](i) in NST astrocytes as well as neurons.

98 llowed us to highlight the role of muscle in NST.

99 Mutations in NST genes cause human and cattle diseases and impaired c

100 d dramatic cytosolic calcium oscillations in NST neurones.

101 ds later by an increase in calcium signal in NST neurons.

102 (1) common following major ABO-incompatible NST and (2) associated with prolonged persistence of hos

103 le mitochondrial uncoupling to the increased NST.

104 pplication of 2-deoxyglucose (2-DG) inhibits NST neurons and activates dorsal motor nucleus (DMN) neu

105 ar formation (RF), and those interconnecting NST subnuclei.

106 edominantly in V at rostral and intermediate NST levels.

107 Identification of these key NSTs in Leishmania will facilitate the dissection of gly

108 lls were also located in the rostral lateral NST subdivision (RL), a site of trigeminal and sparse ge

109 the first crystal structures of a mammalian NST, the mouse CMP-sialic acid transporter (mCST), in co

110 e widespread, mammalian cells lack a GDP-Man NST, thereby providing an ideal heterologous system for

111 e fibers in sampled subregions of the medial NST and DMV increased approximately 23-fold and 94-fold,

112 c fibers in gastric subregions of the medial NST-DMV.

113 alcium imaging study shows that while medial NST neurons are rarely activated by leptin alone, leptin

114 ed obesity, which suggests that Sln-mediated NST is recruited during metabolic overload.

115 n complex mixture with as high as 1000 ng/ml NSTs.

116 egulation by SLN can be the basis for muscle NST.

117 AT, strengthening the hypothesis that muscle NST has likely played an important role in the evolution

118 Our results suggest that muscle NST may be the primary mechanism of heat production duri

119 To evaluate whether muscle NST could indeed play an important role in thermoregulat

120 based on radially oriented NiO nanostrands (NSTs) onto 3D porous Ni foam substrate for monitoring, a

121 was developed to classify a nonsubtypeable (NST) strain of N. meningitidis, 7967.

122 so, meningococcal strains of phenotype NG:NT:NST were isolated from cerebrospinal fluid samples from

123 as well as neurons of the solitary nucleus (NST) and dorsal motor nucleus (DMN) of the vagus.

124 ubset of astrocytes in the solitary nucleus (NST) is activated by low glucose.

125 re highly expressed in the solitary nucleus [NST], dorsal motor nucleus of the vagus [DMN] and catech

126 curs within neurons of the solitary nucleus [NST], though this interaction had not been verified.

127 pipettes were used to record the activity of NST neurons extracellularly and to apply the GABA(A) ant

128 is often cited to support administration of NST.

129 e shape-dependent structural architecture of NST-Ni electrode.

130 ed, indicating an increasing contribution of NST.

131 The radial orientation of NST-Ni electrode onto the interior of the 3D porous subs

132 This leptin-mediated priming of NST neurons was uncoupled by pre-treatment with the phos

133 ion, provide evidence for the recruitment of NST in skeletal muscle.

134 he parasite highlights the essential role of NST(s) in glycosylation of T. brucei.

135 at skeletal muscle becomes the major site of NST when BAT activity is minimized.

136 ther muscle will become an important site of NST when BAT function is conditionally minimized in mice

137 Our immunohistochemical staining of NST cells further verified the presence of the AMPAR sub

138 These studies are relevant to the study of NST structure and function in both protozoan parasites a

139 Neurons had properties similar to those of NST cells in other species, including mean breadth-of-tu

140 lateral RF, subjacent to the rostral tip of NST.

141 omes with respect to BRCA status and type of NST received.

142 t the current widespread prophylactic use of NST in the key soybean-producing areas of the US should

143 hemical evidence for a multimeric complex of NSTs, a finding with important implications to the struc

144 concentration and not with concentration of NSTs and is found to be capable of detecting sub-femtomo

145 ucted specifically to evaluate the effect of NSTs on soybean seed yield at sites within 14 states fro

146 vel approach that combines reconstitution of NSTs into liposomes and the subsequent assessment of nuc

147 ications to the structure and specificity of NSTs in both Leishmania and other organisms.

148 life cycle and further our understanding of NSTs generally.

149 appreciated, but the nature of NE action on NST neurones themselves, especially on the alpha-1 recep

150 In vivo microinjection of PHE onto NST neurones caused a significant reduction in gastric t

151 control of the pattern of radially oriented NSTs onto 3D porous Ni foam substrate.

152 for conducting functional analyses of other NSTs identified in T. brucei.

153 neurons appear to project to targets outside NST, this suggests that most of these cells have local,

154 Toxin-induced loss of DbH-positive NST neurons was positively correlated with loss of CCK-i

155 olling the excitability of both postsynaptic NST neurons and presynaptic vagal afferent terminals.

156 trate that it is feasible to generate potent NSTs from third-party donors for use in antiviral immuno

157 re consisting of SLNB with excision of a pre-NST marked positive lymph node appears to be most accura

158 tumors, extramedullary tumors (predominantly NSTs) were present in higher numbers and were associated

159 randomized to receive CBT and 71 to receive NST.

160 a surprising dearth of information regarding NST effectiveness in increasing soybean seed yield, and

161 we found that isolates from closely related NSTs were often similar by PFGE profile as well, further

162 main 1 (AD1), the flanking Asn/Ser/Thr-rich (NST) domain and AD2] are transiently translocated into t

163 connections with the anterior, rostrolateral NST.

164 wing reduced-intensity nonmyeloablative SCT (NST), consecutive series of patients with major ABO inco

165 lly identified, second-order gastric sensory NST neurones.

166 ably, the fact that inactivation of a single NST gene results in measurable defects in surface glycop

167 n images from the 1.6 m New Solar Telescope (NST) equipped with high order adaptive optics at Big Bea

168 ose transporters in the Arabidopsis thaliana NST family and designated them UDP-XYLOSE TRANSPORTER1 (

169 ecession on ST-site (approximately 20%) than NST-site (approximately 10%; P = 0.0001).

170 It seems likely that NST neurons are involved in the leptin-mediated increase

171 These observations suggest that NST astrocytes may be active participants in the regulat

172 It has been well-established that NSTs are antiporters that exchange nucleotide sugars wit

173 The NST is essential to the maintenance of behavioural and a

174 The NST-based transport of UDP-xylose into the Golgi lumen w

175 The NST-Ni electrode shows significant glucose sensing perfo

176 enhancement probably eliminating W506 as the NST.

177 umbers of double-labeled neurons in both the NST and RF, suggesting that some medullary gustatory neu

178 This MAb bound to both the NST strain and the prototype subtype P1.14 strain, S3446

179 The significance of NE projections from the NST to other CNS regions has long been appreciated, but

180 s when taste cell recording changes from the NST to the PBN.

181 port of cholera toxin neural tracer from the NST-DMV in newborn rats confirmed that PVN neurons were

182 with GSP-innervated receptive fields in the NST and PBN.

183 stributions of double-labeled neurons in the NST and RF suggest a role for NO in stimulus-specific gu

184 Fibers in the NST and RP that possess TRHR1 receptors were phenotypica

185 Fibers in the NST and the raphe pallidus [RP] and obscurrus [RO] that

186 Many taste-responsive cells in the NST are inhibited by gamma-aminobutyric acid (GABA).

187 es suggest that noradrenergic neurons in the NST are particularly important to the generation of refl

188 istics, overall survival was improved in the NST group at 1 year (51% versus 39%) and 2 years (39% ve

189 uggest that a subset of taste neurons in the NST is under the influence from the bilateral VPMpc and

190 led us to hypothesize that TNF action in the NST may preferentially affect putative noradrenergic neu

191 entity of these TNF-activated neurons in the NST was approximately 29% tyrosine hydroxylase [TH]-posi

192 other 54% of the cFOS-activated cells in the NST were phenotypically identified to be astrocytes.

193 e terminal fields of all three nerves in the NST were up to 2.7 times greater in alphaENaC knock-out

194 cytes act within a tripartite synapse in the NST, controlling the excitability of both postsynaptic N

195 In the NST, double-labeled neurons were most numerous in the ro

196 ng these 165 taste-responsive neurons in the NST, the activity of 39 (23.6%) was suppressed by Met-EN

197 After identifying a taste neuron in the NST, the VPMpc was stimulated bilaterally.

198 lls [as indicated by cFOS production] in the NST.

199 the VPMpc on taste-responsive neurons in the NST.

200 a tympani terminal fibers and boutons in the NST.

201 on the activity of gustatory neurons in the NST.

202 BICM microinjected into the NST blocked the cortical-induced inhibition but had no e

203 afferents, the major sensory input into the NST, activated NST astrocytes, as indicated by increases

204 si) into the taste-responsive regions of the NST and the ipsilateral PBN in six rats.

205 hitectonic parcellation (Nissl stain) of the NST into rostral, intermediate, and caudal divisions.

206 e central third of the rostral 1.0 mm of the NST ipsilateral to the stimulated nerve.

207 Cyclical activation of the NST may function to increase the responsiveness of these

208 urons were inhibited and the majority of the NST neurons were excited by gastrointestinal distention.

209 to the caudal visceral sensory region of the NST, and also by immunocytochemical localization of gluc

210 king advantage of the resolving power of the NST, we measure the cross-sectional widths of flare ribb

211 caudal reaches of the gustatory zone of the NST, where taste neurons receive inputs from the IXth ne

212 increasing oxytocinergic innervation of the NST-DMV.

213 om the visceral and gustatory regions of the NST.

214 eceptors within the gustatory portion of the NST; previous studies had shown numerous fiber terminals

215 uclei confined to the caudal division of the NST; they also connect with the area postrema.

216 T-site teeth did not differ from that on the NST-site teeth (P = 0.0875).

217 In this study, the NST-Ni electrode is fabricated to develop a simple, sele

218 dulation of gustatory processing through the NST.

219 the CeA is the major source of input to the NST (82.3+/-7.6 cells/section) and the PBN (76.7+/-11.5)

220 tryptophan at position 506 homologous to the NST in other myosins.

221 data suggest that corticofugal input to the NST may differentially inhibit gustatory afferent activi

222 re sources of catecholaminergic input to the NST.

223 ranslocated into the ER lumen, whereupon the NST domain is glycosylated to yield an inactive 120-kDa

224 ar recording of neuronal activity within the NST in response to taste input was combined with local m

225 Small CTb injections within the NST label extensive projections from the rostral divisio

226 itive fibers already were present within the NST-DMV in rats on the day of birth.

227 ergic projection pathways arising within the NST.

228 s treated with neoadjuvant systemic therapy (NST) for different breast cancer subtypes.

229 staging after neoadjuvant systemic therapy (NST) in node-positive breast cancer is an area of contro

230 or response to neoadjuvant systemic therapy (NST) in patients with breast cancer and to outline a mod

231 nts undergoing neoadjuvant systemic therapy (NST); however, clinical evidence is limited.

232 ted as a site of nonshivering thermogenesis (NST) besides brown adipose tissue (BAT).

233 higher levels of nonshivering thermogenesis (NST) in brown adipose tissue (BAT) of animals that huddl

234 eletal muscle in nonshivering thermogenesis (NST) is not well understood.

235 Muscle nonshivering thermogenesis (NST) was recently suggested to play an important role in

236 h an increase in nonshivering thermogenesis (NST).

237 layer in muscle non-shivering thermogenesis (NST) and can compensate for loss of BAT activity.

238 tant lines affected in the function of these NSTs confirmed their role as UDP-Araf transporters in vi

239 simple, scalable one-pot fabrication of this NST-Ni sensor design enabled control of the pattern of r

240 oxin subunit B (CTb) labeling revealed those NST subnuclei receiving chorda tympani nerve (CT) affere

241 osi sarcoma (KS), and 7 normal skin tissues (NSTs) of Dutch origin were analyzed.

242 eater in ST-site quadrants (36%) compared to NST-site quadrants (18%; P <0.001).

243 determine whether afferent sensory inputs to NST neurons also activates NST astrocytes.

244 subject's unilateral ST keratosis lesion) to NST-site teeth (contralateral corresponding teeth).

245 Prior to NST, proven tumor-positive axillary lymph nodes were mar

246 ng-confirmed partial or complete response to NST and underwent study-specific image-guided VAB before

247 shift from myeloablative transplantation to NST on relapse, transplant complications, and outcome ha

248 rving surgery in patients responding well to NST.

249 gement-specific yield benefits attributed to NSTs.

250 ture of AfB1 with other non-specific toxins (NSTs), thus leading to erroneous quantification of AfB1

251 eurons in the nucleus of the solitary tract (NST) activated by the intraoral infusion of quinine usin

252 ctions to the nucleus of the solitary tract (NST) and dorsal motor nucleus of the vagus (DMV) in adul

253 eurons in the nucleus of the solitary tract (NST) and subjacent reticular formation (RF).

254 s to both the nucleus of the solitary tract (NST) and the dorsal motor nucleus of the vagus (DMNV).

255 rojections to the nucleus of solitary tract (NST) and the parabrachial nucleus (PBN) that modulate ta

256 asured in the nucleus of the solitary tract (NST) in anesthetized B6 and 129 mice to address this con

257 e buds to the nucleus of the solitary tract (NST) in the medulla.

258 eurons in the nucleus of the solitary tract (NST) not only send axons to the parabrachial nuclei (PbN

259 The nucleus of the solitary tract (NST) processes gustatory and related somatosensory infor

260 The nucleus of the solitary tract (NST) processes substantial visceral afferent input and s

261 The nucleus of the solitary tract (NST) receives descending connections from several forebr

262 The nucleus of the solitary tract (NST), located in the dorsomedial medulla, is the site of

263 n (RC) of the nucleus of the solitary tract (NST), the principal site where geniculate axons synapse,

264 in the caudal nucleus of the solitary tract (NST), with signals subsequently relayed to higher brain

265 the hindbrain nucleus of the solitary tract (NST).

266 ortion of the nucleus of the solitary tract (NST).

267 nerves in the nucleus of the solitary tract (NST).

268 y zone of the nucleus of the solitary tract (NST).

269 ns in the rat nucleus of the solitary tract (NST).

270 eurons in the nucleus of the solitary tract (NST; principal locus integrating visceral afferent input

271 ts within the nucleus of the solitary tract [NST].

272 Nonmyeloablative stem cell transplantation (NST) is increasingly used in older patients.

273 lative allogeneic stem cell transplantation (NST) protocol that achieves stable mixed bone marrow chi

274 rter (TPT) and nucleotide-sugar transporter (NST) families.

275 members of the nucleotide sugar transporter (NST) family can efficiently transport UDP-Araf in vitro.

276 Nucleotide sugar transporters (NSTs) are an essential component of the glycosylation pa

277 Nucleotide-sugar transporters (NSTs) are critical components of the cellular glycosylat

278 Nucleotide sugar transporters (NSTs) are indispensible for the biosynthesis of glycopro

279 Nucleotide sugar transporters (NSTs) regulate the flux of activated sugars from the cyt

280 y a family of nucleotide sugar transporters (NSTs).

281 olgi lumen by nucleotide sugar transporters (NSTs).

282 the action of nucleotide sugar transporters (NSTs).

283 of eukaryotic nucleotide-sugar transporters (NSTs).

284 olgi lumen by nucleotide sugar transporters (NSTs).

285 Neoadjuvant systemic treatment (NST) elicits a pathologic complete response in up to 80%

286 stases after neoadjuvant systemic treatment (NST) in patients with breast cancer.

287 ease in the nucleotide sensitive tryptophan (NST) accompanies nucleotide binding and hydrolysis in se

288 ancreatic cancer (PC), neural system tumors (NST), and uveal melanoma (UM).

289 y tumors (P <.001), and nerve sheath tumors (NSTs) (P <.001).

290 es, we defined 38 S. Newport sequence types (NSTs), all of which were novel compared to our previous

291 Typically, NSTs are studied in microsomal preparations from wild-ty

292 d invasive breast cancer who were undergoing NST were included (mean age, 54 years; range, 27-84 year

293 152 patients older than 50 years undergoing NST or myeloablative transplantation.

294 and sparse geniculate input, and the ventral NST (V) and medullary reticular formation (RF), a caudal

295 axonal projections from the caudal visceral NST to the hypothalamus and limbic forebrain, discussion

296 ity rate rose from 54% to 68% (275/404) with NST.

297 Treatment of animals with NST, posttransplantation donor lymphocyte infusions (DLI

298 e NST, 78 (42%) converted to candidates with NST.

299 tivated astrocytes communicate directly with NST neurons by releasing glutamate.

300 ith cT1-3N0-1 breast cancer and treated with NST, followed by surgery between 2010 and 2016, were sel