ライフサイエンスコーパス: Neisseria meningitidis

1 ecifically inhibit the CRISPR-Cas9 system of Neisseria meningitidis.

2 s with N. lactamica prevents colonization by Neisseria meningitidis.

3 , influenza, Mycobacterium tuberculosis, and Neisseria meningitidis.

4 sing a dynamic transmission model of group A Neisseria meningitidis.

5 cillus anthracis, Neisseria gonorrhoeae, and Neisseria meningitidis.

6 l investigations frequently fail to identify Neisseria meningitidis.

7 ningococcal disease caused by infection with Neisseria meningitidis.

8 orms of ClpP from the Gram-negative pathogen Neisseria meningitidis.

9 an unexpectedly versatile Cas9 protein from Neisseria meningitidis.

10 nse of primary human meningothelial cells to Neisseria meningitidis.

11 key virulence factor and vaccine antigen of Neisseria meningitidis.

12 rm kinetic studies on PglL, the O-OTase from Neisseria meningitidis.

13 ream infection caused by the human pathogen, Neisseria meningitidis.

14 s catalysed by three DsbA oxidoreductases in Neisseria meningitidis.

15 ond most prevalent outer membrane protein in Neisseria meningitidis.

16 genetic island (GGI), as do a few strains of Neisseria meningitidis.

17 y to polymyxin B, as reported previously for Neisseria meningitidis.

18 nt mechanism of such phase variation (PV) in Neisseria meningitidis.

19 infection with the important human pathogen Neisseria meningitidis.

20 noclonal antibodies (MAbs) with encapsulated Neisseria meningitidis.

21 ival of infections with certain serotypes of Neisseria meningitidis.

22 ndonuclease paralogues in the human pathogen Neisseria meningitidis.

23 the porin PorB from the pathogenic bacterium Neisseria meningitidis.

24 rial pathogens including Vibrio cholerae and Neisseria meningitidis.

25 ation rates with Streptococcus pneumoniae or Neisseria meningitidis.

26 ; for Listeria monocytogenes, 0.12/0.12; for Neisseria meningitidis, 0.12/0.25; for Haemophilus spp.,

27 Overall, S. pneumoniae (53.4%), Neisseria meningitidis (13.7%), and Haemophilus influenz

28 % (3790/6286) of bacterial meningitis cases: Neisseria meningitidis (1350 cases, 22%), Streptococcus

29 ]), Haemophilus influenzae (18 [14.3%]), and Neisseria meningitidis (15 [11.9%]).

30 le dynamic regulation mechanism observed for Neisseria meningitidis 3-deoxy-d-arabino-heptulosonate 7

31 Neisseria meningitidis A epidemics have been eliminated

32 Neisseria meningitidis, a causative agent of bacterial m

33 allelic profiles to characterize strains of Neisseria meningitidis, a major cause of bacterial menin

34 of human umbilical vein endothelial cells or Neisseria meningitidis after incubation with human serum

35 e Control and Prevention for the analysis of Neisseria meningitidis and Bordetella bronchiseptica gen

36 clinically relevant bacterial CNS pathogens, Neisseria meningitidis and Borrelia burgdorferi.

37 cillin-tazobactam, cefepime, and gentamicin, Neisseria meningitidis and ceftriaxone, and Haemophilus

38 Many gram-negative pathogens such as Neisseria meningitidis and Escherichia coli express acid

39 ownregulates complement activation, binds to Neisseria meningitidis and increases resistance to serum

40 eficiency and compares it to studies done on Neisseria meningitidis and Moraxella catarrhalis; the tw

41 hown that the lipooligosaccharide (LOS) from Neisseria meningitidis and N. gonorrhoeae engages the TL

42 erdin in AP activation on diverse strains of Neisseria meningitidis and N. gonorrhoeae specifically u

43 These would include the beta-proteobacteria Neisseria meningitidis and Neisseria gonnorhoeae, in whi

44 t activity against the Gram-negative species Neisseria meningitidis and Neisseria gonorrheae and impr

45 Both Neisseria meningitidis and Neisseria gonorrhoeae recruit

46 NrrF, a trans-acting sRNA in Neisseria meningitidis and Neisseria gonorrhoeae, has be

47 pathology, while the two pathogenic species, Neisseria meningitidis and Neisseria gonorrhoeae, stradd

48 Neisseria meningitidis and S. pneumoniae remain importan

49 coccus pneumoniae compared with responses to Neisseria meningitidis and that in each case, the bacter

50 such as the nitric oxide reductase (NorB) of Neisseria meningitidis and the flavohemoglobins (Hmp) of

51 e use the method to analyze the porB gene of Neisseria meningitidis and verify the inferences using p

52 des two pathogens, Neisseria gonorrhoeae and Neisseria meningitidis, and at least 13 species of comme

53 e in vitro against Streptococcus pneumoniae, Neisseria meningitidis, and H. influenzae.

54 Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus accounted for 66

55 ry-confirmed Streptococcus pneumoniae (Spn), Neisseria meningitidis, and Haemophilus influenzae menin

56 ry tract pathogens Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae, but

57 igen) and qPCR for Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae.

58 rocesses of pathogenic Neisseria gonorrheae, Neisseria meningitidis, and Haemophilus influenzae.

59 negative pathogens, including P. aeruginosa, Neisseria meningitidis, and Helicobacter pylori.

60 Vaccines against Streptococcus pneumoniae, Neisseria meningitidis, and Hemophilus influenzae type b

61 ophilus influenzae type b (Hib), serogroup C Neisseria meningitidis, and multiple capsular serotypes

62 ten human-restricted Neisseria species two, Neisseria meningitidis, and Neisseria gonorrhoeae, cause

63 ampylobacter jejuni, Haemophilus influenzae, Neisseria meningitidis, and Pasteurella multocida.

64 -MS assay identified Pseudomonas aeruginosa, Neisseria meningitidis, and Staphylococcus aureus; these

65 eported to occur in Lactobacillus plantarum, Neisseria meningitidis, and Streptococcus agalactiae, an

66 Infection with Haemophilus influenzae, Neisseria meningitidis, and Streptococcus pneumoniae cau

67 Neisseria gonorrhoeae and Neisseria meningitidis are closely related organisms tha

68 Neisseria gonorrhoeae and Neisseria meningitidis are closely-related bacteria that

69 methods for detecting pharyngeal carriage of Neisseria meningitidis are complex.

70 The HrpAB proteins of Neisseria meningitidis are members of a two-partner secr

71 Several outer membrane proteins of Neisseria meningitidis are subject to phase variation du

72 the responses of human tonsil-derived DC to Neisseria meningitidis as a model organism.

73 esent in numerous bacterial pathogens, using Neisseria meningitidis as a model organism.

74 previously identified lipooligosaccharide on Neisseria meningitidis as an acceptor for complement C4b

75 cterizing bacteria, using the human pathogen Neisseria meningitidis as an example.

76 that initiate complement-mediated killing of Neisseria meningitidis as they enter the bloodstream fro

77 dentified as a ligand for these molecules on Neisseria meningitidis As with N. meningitidis NspA (Nm-

78 ucture of a bacterial homologue of ASBT from Neisseria meningitidis (ASBT(NM)) at 2.2 A.

79 crystal structure of an ASBT homologue from Neisseria meningitidis (ASBT(NM)) in detergent was repor

80 es of the substrate-bound ClpXP complex from Neisseria meningitidis at 2.3 to 3.3 angstrom resolution

81 d O-antigen capsular polysaccharide (CPS) of Neisseria meningitidis B (NmB) have been investigated by

82 Administration to respond to an outbreak of Neisseria meningitidis B at a U.S. university.

83 domonas aeruginosa, Acinetobacter baumannii, Neisseria meningitidis, Bacteroides fragilis, Bacillus a

84 Neisseria meningitidis binds factor H (fH), a key regula

85 Neisseria meningitidis binds the complement downregulati

86 Neisseria gonorrhoeae and Neisseria meningitidis both express the lacto-N-neotetra

87 diated engulfment of Neisseria gonorrheae or Neisseria meningitidis by human cells and can offer deep

88 Invasive bacterial meningitis caused by Neisseria meningitidis can be prevented by active immuni

89 Neisseria meningitidis can be transmitted via asymptomat

90 h intact, heat-killed cells of Gram-negative Neisseria meningitidis, capsular serogroup C (MenC) or G

91 , and AcrIIC3 proteins were found to inhibit Neisseria meningitidis Cas9 (NmeCas9) activity in bacter

92 Cas9 orthologs (including Neisseria meningitidis Cas9 [NmeCas9]) have also been ad

93 Neisseria meningitidis causes 500 000 cases of septicemi

94 Neisseria meningitidis causes bacterial meningitis and s

95 The pathogen Neisseria meningitidis causes disease amongst infants an

96 The bacterium Neisseria meningitidis causes life-threatening meningiti

97 Neisseria meningitidis changes its capsular phenotype th

98 Among 25 serogroup B Neisseria meningitidis clinical isolates, we identified

99 lP, an autotransporter of spherically shaped Neisseria meningitidis contains the molecular informatio

100 lipid A phosphoethanolamine transferase from Neisseria meningitidis, determined to 2.75-A resolution.

101 ainment strategies for outbreaks of invasive Neisseria meningitidis disease are informed by serogroup

102 e (SPR) based biosensor for the detection of Neisseria meningitidis DNA employing Kretschmann configu

103 nd impairs the fitness of the human pathogen Neisseria meningitidis during infection.

104 ningococcal disease (IMD) due to serogroup Y Neisseria meningitidis emerged in Europe during the 2000

105 Neisseria meningitidis employs redundant heme acquisitio

106 Neisseria meningitidis encodes up to five TpsA proteins

107 Lipooligosaccharide (LOS) from Neisseria meningitidis enhances the respiratory burst in

108 ts sporadic nature and the high diversity of Neisseria meningitidis, epidemiological surveillance inc

109 ding patients with Streptococcus pneumoniae, Neisseria meningitidis, Escherichia coli, and Pseudomona

110 m, and understanding the mechanisms by which Neisseria meningitidis evades host innate and acquired i

111 antibodies raised against sheaths presenting Neisseria meningitidis factor H binding protein (fHbp) a

112 s 3 main recombinant proteins, including the Neisseria meningitidis factor H binding protein (fHbp),

113 with a radiolabeled lipooligosaccharide from Neisseria meningitidis for binding to LBP or to the clos

114 In the human pathogen Neisseria meningitidis for example, 23 proteins are dedi

115 admission to hospital and identification of Neisseria meningitidis from a sterile site.

116 ce diversity in the Campylobacter jejuni and Neisseria meningitidis genomes encoded hypothetical prot

117 ta that the class III Fic protein NmFic from Neisseria meningitidis gets autoadenylylated in cis, the

118 ced meningitis incidence and carriage due to Neisseria meningitidis group A (MenA).

119 ampaign was launched using a newly developed Neisseria meningitidis group A (NmA) polysaccharide-teta

120 ine containing the N-propionyl derivative of Neisseria meningitidis group B (MenB) capsular polysacch

121 S) or with LPS as a noncovalent complex with Neisseria meningitidis group B outer membrane protein (L

122 l meningitis (Haemophilus influenzae type b, Neisseria meningitidis group C and seven serotypes of St

123 Vaccines against Neisseria meningitidis group C are based on its alpha-2,

124 as oropharyngeal carriage of disease-causing Neisseria meningitidis (group A, B, C, W, X, or Y) in st

125 ncluding pathogens such as Escherichia coli, Neisseria meningitidis, Haemophilus influenzae, and Past

126 Neisseria meningitidis, Haemophilus influenzae, and Stre

127 eillance targeted meningitis cases caused by Neisseria meningitidis, Haemophilus influenzae, and Stre

128 terial meningitis, which is caused mainly by Neisseria meningitidis, Haemophilus influenzae, and Stre

129 terial infections (Streptococcus pneumoniae, Neisseria meningitidis, Haemophilus influenzae, S suis)

130 Group A Neisseria meningitidis has been a major cause of bacteri

131 Neisseria meningitidis has been recognized as a cause of

132 e substrate complexes of heme oxygenase from Neisseria meningitidis has been systematically perturbed

133 Neisseria meningitidis has several strategies to evade c

134 ative bacteria, including the human pathogen Neisseria meningitidis, have evolved means to preferenti

135 identified small-molecule inhibitors of the Neisseria meningitidis HO (nm-HO).

136 Recombinant forms of Neisseria meningitidis human factor H binding protein (f

137 H binding protein (fHbp) is a lipoprotein of Neisseria meningitidis important for the survival of the

138 pneumoniae, GBS, Listeria monocytogenes, or Neisseria meningitidis in cerebrospinal fluid or other n

139 dent responses except that made with group C Neisseria meningitidis; in the latter case, only peptide

140 ge infectivity potentiator (rMIP) protein of Neisseria meningitidis induces significant serum bacteri

141 The incidence of Neisseria meningitidis infection decreased from 0.721 pe

142 sential to understanding the epidemiology of Neisseria meningitidis infection.

143 specific protection against capsular group B Neisseria meningitidis infections, but the full breadth

144 ts used for either therapy or prophylaxis of Neisseria meningitidis infections.

145 Neisseria meningitidis inhibits the alternative pathway

146 We previously reported that Neisseria meningitidis internalization into human brain

147 Neisseria meningitidis is a cause of fatal sepsis and ep

148 Neisseria meningitidis is a commensal microbe that colon

149 Neisseria meningitidis is a commensal of humans that can

150 Neisseria meningitidis is a frequent colonizer of the hu

151 Neisseria meningitidis is a human commensal that can als

152 Neisseria meningitidis is a human pathogen causing bacte

153 Neisseria meningitidis is a human-specific bacterium tha

154 Neisseria meningitidis is a human-specific pathogen and

155 Neisseria meningitidis is a leading cause of bacterial m

156 GNA2091 of Neisseria meningitidis is a lipoprotein of unknown funct

157 Neisseria meningitidis is a major cause of bacterial men

158 Neisseria meningitidis is a major cause of bacterial men

159 Neisseria meningitidis is a major global pathogen causin

160 Neisseria meningitidis is a strict human pathogen that c

161 We show that the closely related bacterium Neisseria meningitidis is also polyploid, while the comm

162 Neisseria meningitidis is an encapsulated pathogen, and

163 Neisseria meningitidis is an important cause of invasive

164 Neisseria meningitidis is an important cause of septicem

165 Neisseria meningitidis is an important human pathogen th

166 Neisseria meningitidis is an obligate human commensal ba

167 Neisseria meningitidis is an obligate human nasopharynge

168 nt inhibitor factor H (fH) to the surface of Neisseria meningitidis is critical for evasion of innate

169 Although the opportunistic pathogen Neisseria meningitidis is enriched for colonization in t

170 Although Neisseria meningitidis is naturally competent and porB g

171 Neisseria meningitidis is one of the few commensal bacte

172 Neisseria meningitidis is one of the main agents of bact

173 ron-regulated FetA outer membrane protein of Neisseria meningitidis is one of various outer membrane

174 riation systems of Neisseria gonorrhoeae and Neisseria meningitidis is presented.

175 The LTTR, CrgA, from the human pathogen Neisseria meningitidis, is upregulated during bacterial-

176 An apparently rare Neisseria meningitidis isolate containing one copy of a

177 Almost all invasive Neisseria meningitidis isolates express capsular polysac

178 Our whole-genome microarray studies of Neisseria meningitidis MC58 previously identified a set

179 a genomic DNA sequence of lpt3, derived from Neisseria meningitidis MC58, to search the genomic seque

180 nation with glyco-conjugate capsular group C Neisseria meningitidis (Men C) vaccines in infancy.

181 ulation-level protection against serogroup A Neisseria meningitidis (MenA) are unknown.

182 outbreaks, largely attributed to serogroup A Neisseria meningitidis (MenA).

183 Serogroup B Neisseria meningitidis (MenB) is a major cause of severe

184 absence of an effective vaccine, serogroup B Neisseria meningitidis (MenB) remains a major cause of i

185 Neisseria meningitidis (meningococcus) is a symbiont of

186 (pneumococcus), Haemophilus influenzae, and Neisseria meningitidis (meningococcus) was performed by

187 Streptococcus pneumoniae (pneumococcus), Neisseria meningitidis (meningococcus), and Haemophilus

188 ing Streptococcus pneumoniae (pneumococcus), Neisseria meningitidis (meningococcus), and Haemophilus

189 of Streptococcus pneumoniae (pneumococcus), Neisseria meningitidis (meningococcus), and Haemophilus

190 (pneumococcus), Haemophilus influenzae, and Neisseria meningitidis (meningococcus).

191 ce identity, PilE is structurally similar to Neisseria meningitidis minor pilins PilXNm and PilVNm, r

192 lation of lipopolysaccharide-null mutants in Neisseria meningitidis, Moraxella catarrhalis, and most

193 tococcus pneumoniae, Haemophilus influenzae, Neisseria meningitidis, Mycoplasma pneumoniae, Mycobacte

194 Neisseria meningitidis (N. meningitidis), Streptococcus

195 ion of the main meningitis-causing bacteria, Neisseria meningitidis (N. meningitidis).

196 s present within the obligate human pathogen Neisseria meningitidis, NApe and NExo, are important for

197 The three species Neisseria meningitidis, Neisseria gonorrheae, and Neisse

198 nships among three closely related bacteria, Neisseria meningitidis, Neisseria gonorrhoeae and Neisse

199 ajor human pathogens Haemophilus influenzae, Neisseria meningitidis, Neisseria gonorrhoeae, Helicobac

200 Neisseria meningitidis (Nm) clonal complex 11 (cc11) lin

201 evasion is an important survival strategy of Neisseria meningitidis (Nm) during colonization and infe

202 Neisseria meningitidis (Nm) is a Gram-negative diplococc

203 The human pathogen Neisseria meningitidis (Nm) is a leading cause of bacter

204 Neisseria meningitidis (Nm) is a leading cause of bacter

205 Neisseria meningitidis (Nm) is a nasopharyngeal commensa

206 The human pathogen Neisseria meningitidis (Nm) is known to possess several

207 Neisseria meningitidis (Nm) strains infecting these pati

208 Clusters of Neisseria meningitidis (Nm) urethritis among primarily h

209 meningitis pathogens, 16 (73%) of which were Neisseria meningitidis (Nm).

210 ed cases of meningitis, 5590 were confirmed: Neisseria meningitidis ([Nm] 85%), Streptococcus pneumon

211 k in 2009 due to a large epidemic of group A Neisseria meningitidis (NmA) meningitis.

212 The conjugate vaccine against serogroup A Neisseria meningitidis (NmA), MenAfriVac, was first intr

213 ed Cas9s to identify a compact ortholog from Neisseria meningitidis-Nme2Cas9-that recognizes a simple

214 The HO from the pathogenic bacterium Neisseria meningitidis (NmHO) possesses a crystallograph

215 The HO from the pathogenic bacterium Neisseria meningitidis, NmHO, possesses C-terminal His20

216 neither inactivated, unencapsulated, intact Neisseria meningitidis nor Streptococcus agalactiae inhi

217 observed in this loop when compared with the Neisseria meningitidis NT domain structure.

218 lonization of the upper respiratory tract by Neisseria meningitidis occurs despite elicitation of ada

219 duals cocolonized by Neisseria lactamica and Neisseria meningitidis One HGT event resulted in the acq

220 al disease caused by ciprofloxacin-resistant Neisseria meningitidis, one in North Dakota and two in M

221 Carriage for Neisseria meningitidis (P < 0.05) and Neisseria lactamic

222 Asymptomatic oropharyngeal carriage of Neisseria meningitidis peaks in adolescence and young ad

223 Neisseria meningitidis phasevarions regulate genes inclu

224 rs of bacterial pathogenic strains including Neisseria meningitidis, Pseudomonas aeruginosa and Esche

225 ion of a purified polysialyltransferase from Neisseria meningitidis (PST(Nm)) to the extracellular en

226 t infectious diseases over the past century, Neisseria meningitidis remains a major causative agent o

227 he availability of antibiotics and vaccines, Neisseria meningitidis remains a major cause of meningit

228 Inactivation of the misR/misS system in Neisseria meningitidis results in the loss of phosphoryl

229 uman pathogens, Streptococcus pneumoniae and Neisseria meningitidis, revealed both previously identif

230 asmodium falciparum infections, and virulent Neisseria meningitidis samples.

231 Respiratory pathogens, such as Neisseria meningitidis, secrete site-specific proteases

232 ing in 2010, the burden of meningitis due to Neisseria meningitidis serogroup A (NmA) has substantial

233 serological correlates of protection against Neisseria meningitidis serogroup A (NmA) in Burkina Faso

234 To combat Neisseria meningitidis serogroup A epidemics in the meni

235 ecially in the African meningitis belt where Neisseria meningitidis serogroup A historically caused l

236 the countries of the meningitis belt, where Neisseria meningitidis serogroup A historically caused l

237 An affordable, highly immunogenic Neisseria meningitidis serogroup A meningococcal conjuga

238 Vaccination campaign against Neisseria meningitidis serogroup A was carried out in 20

239 rrelate of protection against infection with Neisseria meningitidis serogroup A, we use an assumed SB

240 belt undergoes recurrent epidemics caused by Neisseria meningitidis serogroup A.

241 The prediction of efficacy of Neisseria meningitidis serogroup B (MenB) vaccines is cu

242 Neisseria meningitidis serogroup B (MnB) is a leading ca

243 Use of recently licensed vaccines against Neisseria meningitidis serogroup B (NmB) will depend par

244 The capsular polysaccharide of the pathogens Neisseria meningitidis serogroup B and of Escherichia co

245 Candidate Neisseria meningitidis serogroup B vaccines that are bas

246 abeled ([(14)C]-acetate) blebs purified from Neisseria meningitidis serogroup B with either human mon

247 tococcus pneumoniae, Listeria monocytogenes, Neisseria meningitidis serogroup B, Candida albicans, an

248 Neisseria meningitidis serogroup C (NmC) was responsible

249 We characterized five Neisseria meningitidis serogroup C isolates from a Chica

250 (at 2, 3, and 4 months or 2 and 4 months) or Neisseria meningitidis serogroup C monovalent meningococ

251 Acetylated sialic acid has been found in the Neisseria meningitidis serogroup W (NmW) capsular polysa

252 July 2016, Togo experienced its first major Neisseria meningitidis serogroup W (NmW) outbreak.

253 he United Kingdom due to a sublineage of the Neisseria meningitidis serogroup W ST-11 clonal complex

254 d States, South Africa, and Israel caused by Neisseria meningitidis serogroup Y (NmY) was greater tha

255 vasive meningococcal disease (IMD) caused by Neisseria meningitidis serogroup Y has increased in Euro

256 esponse to i.p.-injected intact, heat-killed Neisseria meningitidis, serogroup C (MenC), a gram-negat

257 des of recombinant capsular polymerases from Neisseria meningitidis serogroups A (CsaB) and X (CsxA)

258 Neisseria meningitidis serogroups A and X are among the

259 ve meningococcal disease is mainly caused by Neisseria meningitidis serogroups A, B, C, X, W, and Y.

260 al virulence determinants of disease causing Neisseria meningitidis species are their extracellular p

261 o human pathogens, Neisseria gonorrhoeae and Neisseria meningitidis, stimulate PS/gammaS processing o

262 The native lipooligosaccharide (LOS) from Neisseria meningitidis strain 89I was analyzed by matrix

263 Neisseria meningitidis strain H44/76 was modified by exp

264 age prevention against antigenically diverse Neisseria meningitidis strains and to compare this prote

265 Neisseria meningitidis strains are characterized with ML

266 membrane vesicle (OMV) vaccines from mutant Neisseria meningitidis strains engineered to overexpress

267 Laboratory data on 4,735 Neisseria meningitidis strains was collected and reporte

268 Neisseria meningitidis, Streptococcus pneumoniae, or Hae

269 mophilus influenzae, Listeria monocytogenes, Neisseria meningitidis, Streptococcus pneumoniae, Strept

270 erized a TE6 thioesterase from the bacterium Neisseria meningitidis Structural analysis with X-ray cr

271 his reveals how the important human pathogen Neisseria meningitidis subverts immune responses by mimi

272 ned H influenzae type b and capsular group C Neisseria meningitidis tetanus toxoid conjugate vaccine

273 iochemical study of the AP endonuclease from Neisseria meningitidis that has allowed us to capture st

274 In Neisseria meningitidis the majority of iron-responsive g

275 ccus pneumoniae, Haemophilus influenzae, and Neisseria meningitidis, the 3 most common bacteria causi

276 OpcA from Neisseria meningitidis, the causative agent of meningoco

277 sm of action has been studied extensively in Neisseria meningitidis, the specific subset of genes tha

278 o the closely related opportunistic pathogen Neisseria meningitidis, there is an absence of adaptive

279 d from the surface of the bacterial pathogen Neisseria meningitidis; they play a key role in adhesion

280 The ability of the human bacterial pathogen Neisseria meningitidis to cause invasive disease depends

281 Neisseria meningitidis, typically a resident of the oro-

282 Neisseria meningitidis use Type IV pili (T4P) to adhere

283 Neisseria meningitidis utilizes capsular polysaccharide,

284 method for the direct quantification of two Neisseria meningitidis vaccine antigens, in mono- and mu

285 Gram-negative bacterial pathogens including Neisseria meningitidis, Vibrio cholerae, and Salmonella

286 tor H binding protein (FHbp) is an important Neisseria meningitidis virulence factor that binds a neg

287 A 2.9-kilobase pair locus in Neisseria meningitidis was identified as containing tran

288 A model pathogen, Neisseria meningitidis, was used to validate the technol

289 NmLgtB-B beta1-4 galactosyltransferase from Neisseria meningitidis we demonstrate fast and robust ac

290 egative pathogens Haemophilus influenzae and Neisseria meningitidis We hypothesized that activation o

291 ere, using a distinct CRISPR-Cas system from Neisseria meningitidis, we demonstrate efficient targeti

292 ausative agent of meningitis and septicemia, Neisseria meningitidis, we showed that the Pfs reaction

293 n antigenic variation (Av) of two strains of Neisseria meningitidis were determined using an unbiased

294 known TNF (-308) genotype after exposure to Neisseria meningitidis were measured.

295 cord by a number of microorganisms including Neisseria meningitidis, which can lead to permanent neur

296 a family of outer membrane lipoproteins from Neisseria meningitidis, which elicits bactericidal antib

297 Hyperinvasive lineages of Neisseria meningitidis, which persist despite extensive

298 ere we expressed the gene encoding LpxA from Neisseria meningitidis, which specifically attaches 3OH-

299 abeling of hexaacylated endotoxin (LOS) from Neisseria meningitidis with [(13)C]acetate allowed the u

300 ve isolates and 25 isolates from carriers of Neisseria meningitidis without disease.