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1 ells infected with a heterologous RNA virus (Newcastle disease virus).
2 ing a control or fusion protein derived from Newcastle disease virus.
3 idase (HN) glycoprotein of the paramyxovirus Newcastle disease virus.
4 onse to HSV-1, but not to Sendai virus or to Newcastle disease virus.
5 d to rescue replication of the IFN-sensitive Newcastle disease virus.
6 nts similar to the one for cells infected by Newcastle disease virus.
7 omography to show that the matrix protein of Newcastle disease virus, a paramyxovirus and relative of
8 mutation is demonstrated in the F protein of Newcastle disease virus, a paramyxovirus of a different
9 nduced by all IL-33-inducing agonists except Newcastle disease virus, a RIG-I agonist that induced ex
10 We found that matrix proteins purified from Newcastle disease virus adsorb on a phospholipid bilayer
11 d RNA viruses that have dsRNA intermediates, Newcastle disease virus and Sendai virus, and a DNA viru
14 from non-target viruses such as H6N2, H9N2, Newcastle disease virus, and infectious bronchitis virus
16 ense RNA viruses (influenza viruses A and B, Newcastle disease virus, and vesicular stomatitis virus)
17 simplex virus type-1 (HSV-1), Sendai virus, Newcastle disease virus, and vesicular stomatitis virus.
18 W proteins) were expressed from recombinant Newcastle disease viruses, and the responses of infected
21 PV701, a replication-competent strain of Newcastle disease virus, causes regression of tumor xeno
22 packaging revealed a majority of infectious Newcastle disease viruses contain one functional genome.
24 ns for the ability to rescue the growth of a Newcastle disease virus expressing green fluorescent pro
25 mutational analysis of the HR1 domain of the Newcastle disease virus fusion protein, focusing on the
27 on of HN of parainfluenza virus 5 (PIV5) and Newcastle disease virus HN abolishes cell-cell fusion, w
29 embrane fusion, we characterized a series of Newcastle disease virus HN proteins whose surface residu
30 ith functionally active "headless" mumps and Newcastle disease virus HN proteins, provide insights in
34 paramyxoviruses measles virus, mumps virus, Newcastle disease virus, human parainfluenza virus 3, an
35 d receptor binding site of the paramyxovirus Newcastle disease virus in its function and its relation
36 the growth of a highly IFN-sensitive virus (Newcastle disease virus) in the presence of IFN, suggest
37 ways associated with immune response to NDV (Newcastle disease virus) in the trachea, an essential or
38 nd tumor necrosis factor alpha (TNFalpha) in Newcastle disease virus-infected Irf5(-)(/)(-) mice.
39 not type I interferon gene transcription in Newcastle disease virus-infected peritoneal macrophages.
41 le NF-kappaB activity, both Sendai virus and Newcastle disease virus infection led to robust IFN-beta
46 opathological characterization of a virulent Newcastle disease virus isolate (NDV-Peru/08) obtained f
48 ed to mimic sites that are found in virulent Newcastle disease virus isolates and to contain 4 or 5 b
51 AVX/COVID-12 vaccine using a cost-effective Newcastle disease virus LaSota platform to express a sta
52 DCs) by negative-strand RNA viruses, such as Newcastle disease virus, leads to the induction of the I
55 hemagglutinin-neuraminidase (HN) protein of Newcastle disease virus mediates attachment to sialic ac
57 the response of DCs to virus infection with Newcastle disease virus (NDV) after a 24-hour E2 treatme
58 VLP is composed of the NP and M proteins of Newcastle disease virus (NDV) and a chimeric protein con
61 he level of IFN-beta protein induced by both Newcastle disease virus (NDV) and Sendai virus infection
64 linical development of a mesogenic strain of Newcastle disease virus (NDV) as an oncolytic agent for
68 Virus-like particles (VLPs) built on the Newcastle disease virus (NDV) core proteins, NP and M, a
69 h virus-like particles (VLPs) containing the Newcastle disease virus (NDV) core proteins, NP and M, a
70 several paramyxoviruses.IMPORTANCE Oncolytic Newcastle disease virus (NDV) could establish persistent
74 respiratory tract based on recombinant avian Newcastle disease virus (NDV) expressing the hemagglutin
75 such a stimulus, we generated a recombinant Newcastle disease virus (NDV) expressing the MV hemagglu
76 iotin-labeled peptides with sequences of the Newcastle disease virus (NDV) F protein heptad repeat 2
77 and 289 in the structure and function of the Newcastle disease virus (NDV) F protein was explored by
78 the basis of the coordinates of the related Newcastle disease virus (NDV) F protein, Valine-94, a de
79 ith those of the parainfluenza virus SV5 and Newcastle disease virus (NDV) F proteins, the structures
80 has been demonstrated that the V protein of Newcastle disease virus (NDV) functions as an alpha/beta
84 It has been shown that the L289A-mutated Newcastle disease virus (NDV) fusion (F) protein gains t
86 equences in the transmembrane (TM) domain of Newcastle disease virus (NDV) fusion (F) protein in the
87 The role of N-linked glycosylation of the Newcastle disease virus (NDV) fusion (F) protein in vira
90 y viral fusion proteins, the sequence of the Newcastle disease virus (NDV) fusion protein has several
91 uman parainfluenza virus type 3 (HPIV-3) and Newcastle disease virus (NDV) have been previously evalu
94 onstrate that the F-interactive sites on the Newcastle disease virus (NDV) hemagglutinin-neuraminidas
98 alysis of the three-dimensional structure of Newcastle disease virus (NDV) HN protein revealed the pr
100 tion, mutations in a conserved domain in the Newcastle disease virus (NDV) HN stalk result in a sharp
103 morbidity-mortality event involving virulent Newcastle disease virus (NDV) in wild double-crested cor
106 hemagglutinin-neuraminidase (HN) protein of Newcastle disease virus (NDV) is a multifunctional prote
111 hemagglutinin-neuraminidase (HN) protein of Newcastle disease virus (NDV) is an important determinan
113 ug-resistant cancers.IMPORTANCEThe oncolytic Newcastle disease virus (NDV) is being developed for use
118 hemagglutinin-neuraminidase (HN) protein of Newcastle disease virus (NDV) plays a crucial role in th
122 tinin-neuraminidase (HN) cytoplasmic tail in Newcastle disease virus (NDV) replication and pathogenic
125 Ps) released from avian cells expressing the Newcastle disease virus (NDV) strain AV proteins NP, M,
126 The complete genome sequence of an African Newcastle disease virus (NDV) strain isolated from a chi
128 onomic losses, but little is known about the Newcastle disease virus (NDV) strains circulating in Afr
132 ystallographic structure of an HN dimer from Newcastle disease virus (NDV) suggests that a single sit
133 eloped a COVID-19 vaccine candidate based on Newcastle disease virus (NDV) that can be manufactured a
135 combination of gold nanoparticles (GNPs) and Newcastle disease virus (NDV) to enhance the antitumor e
138 he present study, we generated a recombinant Newcastle disease virus (NDV) vectoring the fusion (F) p
141 ression of the fusion glycoprotein of RSV by Newcastle disease virus (NDV) would stimulate a more rob
142 xpression, replication, and pathogenicity of Newcastle disease virus (NDV), a green fluorescent prote
144 Pteropus vampyrus bat kidney (PVK) cells to Newcastle disease virus (NDV), an avian paramyxovirus kn
153 the F proteins of SV5 (W3A and WR isolates), Newcastle disease virus (NDV), and human parainfluenza v
154 cular stomatitis virus (VSV), Sindbis virus, Newcastle disease virus (NDV), and Sendai virus (SeV), w
155 of the H5 and H7 hemagglutinin subtypes, and Newcastle disease virus (NDV), are important pathogens i
157 es such as avian metapneumovirus (aMPV), and Newcastle disease virus (NDV), human pathogens such as h
158 Here we find that intratumoral therapy with Newcastle disease virus (NDV), in addition to the activa
159 yxovirus 1 (APMV-1) RNA, also referred to as Newcastle disease virus (NDV), in clinical samples from
162 Virulent and moderately virulent strains of Newcastle disease virus (NDV), representing avian paramy
163 DCs showed a reduced type I IFN response to Newcastle disease virus (NDV), Sendai virus (SeV), and S
165 ns (UTRs) in replication and pathogenesis of Newcastle disease virus (NDV), we generated recombinant
166 is hypothesis, L929 cells were infected with Newcastle disease virus (NDV), which led to the inductio
170 e effects of lipopolysaccharide (LPS) on the Newcastle disease virus (NDV)-mediated induction of cyto
178 ession after infection with either Sendai or Newcastle disease viruses or after engagement of the Tol
180 ion with vectored vaccine, using recombinant Newcastle disease virus (rNDV) expressing glycoprotein D
184 A genetically modified, recombinant form of Newcastle disease virus (rNDV) undergoes ionic strength-
186 me this obstacle, we generated a recombinant Newcastle disease virus (rNDV)-vectored experimental nor
187 Using reverse genetics, three recombinant Newcastle disease viruses (rNDVs) were engineered, each
188 ed against CPE resulting from infection with Newcastle disease virus, Sendai virus, canine distemper
192 rs by about 2.5-fold, and neuraminidase from Newcastle disease virus typically increased infectivity
195 aramyxovirus 1 (APMV-1; a group encompassing Newcastle disease virus), which is a highly contagious p
196 y this network, we studied DCs infected with Newcastle disease virus, which is able to stimulate inna
197 in primary human dendritic cells infected by Newcastle disease virus, with copy numbers varying from