ライフサイエンスコーパス: O157

1 for epidemiological surveillance of E. coli O157.

2 ric culture (0.7%), with 65 cases of E. coli O157.

3 e region yielded the outbreak strain of STEC O157.

4 epared only from Adj-Vac group feces blocked O157 adherence to epithelial cells.

5 n mutant of Shiga toxin 2 negative (stx2(-)) O157 (Adj-Vac); non-adjuvanted (NoAdj-Vac); or non-vacci

6 lated, 28 (90%) were attributable to E. coli O157 and 3 (10%) were attributable to non-O157 STEC.

7 nce for the geographic divergence of E. coli O157 and for a prominent role of stx2a in total Stx prod

8 EIA and/or culture and distinguishes between O157 and non-O157 STEC in clinical samples and that E. c

9 E. coli O157 and non-O157 STEC were detected in 35 and 18 cases,

10 ce) and cultured in attempts to recover both O157 and non-O157 STEC.

11 Interestingly, the IEE sequences from O157 and the top 10 non-O157 STEC serotypes fell into cl

12 associated with highly pathogenic serotypes (O157 and top non-O157 Shiga toxin-producing Escherichia

13 and epidemiological surveillance of E. coli O157, and the data were used to identify discernible ass

14 Cattle are the main reservoir for E. coli O157, and vaccines for cattle now exist.

15 o identify sublineages and clades of E. coli O157, and when they were correlated with the clinical ou

16 uvanted, inactivated whole-cell vaccines for O157 can induce O157-specific cellular and mucosal immun

17 ga toxin-producing Escherichia coli O157:H7 (O157) can cause mild to severe gastrointestinal disease

18 prevent substantial numbers of human E. coli O157 cases.

19 bacter jejuni, Escherichia coli O157:H7, non-O157 E. coli, Listeria monocytogenes, and Salmonella spp

20 modified by enzymes in the E. coli serotype O157 (E. coli O157) O-PS biosynthetic pathway.

21 found espW in the sequenced O157:H7 and non-O157 EHEC strains as well as in Shigella boydii Furtherm

22 Most EPEC and non-O157 EHEC strains express lymphostatin (also known as Li

23 -based simulation model for Escherichia coli O157 environmental transmission in cattle to simulate fo

24 LD-PCR differentiated STEC O157 from non-O157 using rfbEO157, and LD-PCR results pr

25 differential association of Escherichia coli O157 genotypes with animal and human hosts has recently

26 Adherence of E. coli O157 : H-expressing flagella of serotype H7, H6 or H48 t

27 s of endotoxins and bacterial cells (E. coli O157:H19).

28 or both glucose (0.08+/-0.02muM) and E. coli O157:H7 ( approximately 4 CFUmL(-1)) were competitive wi

29 ion of the food contaminant Escherichia coli O157:H7 (E. coli O157:H7) in complex food products due t

30 etection of food pathogenic Escherichia coli O157:H7 (E.coli O157:H7), a dangerous strain among 225 E

31 PCR) was used to test for genes from E. coli O157:H7 (eaeO157), shiga-toxin producing E. coli (stx2),

32 Enterohemorrhagic Escherichia coli O157:H7 (EHEC) is a foodborne pathogen that causes blood

33 rate that enterohemorrhagic Escherichia coli O157:H7 (EHEC) regulates virulence via the oxygen-respon

34 licated type I locus within Escherichia coli O157:H7 (EHEC), which we have named the gene pairs zorO-

35 Shiga toxin-producing Escherichia coli O157:H7 (O157) can cause mild to severe gastrointestinal

36 olves PCR-amplified gene markers for E. coli O157:H7 (rfbO157, eae, vt1, and vt2) incorporating a det

37 ly, a homologue of lymphostatin from E. coli O157:H7 (ToxB; L7095) was also found to possess comparab

38 Extremely low concentration of E. coli O157:H7 (~10 CFU/mL) could be detected within 1h and 3h

39 Positive selection for E. coli O157:H7 across the farms identified only one positive is

40 inase (PI3K)/Akt signaling increased E. coli O157:H7 adherence to HT-29 cells.

41 Similarly, E. coli O157:H7 adhesion to cattle colonic explants was reduced

42 inflammatory response to attenuated E. coli O157:H7 adhesion, mucin 2 (MUC2) expression was analyzed

43 units (cfu)/mL, or approximately 500 E. coli O157:H7 and approximately 500 E. coli K12 cfu/mL.

44 hip with a case testing positive for E. coli O157:H7 and coincident diarrheal illness.

45 actively transposes and proliferates in EHEC O157:H7 and enterotoxigenic E. coli (ETEC) O139 and O149

46 d to a reduction in the transport of E. coli O157:H7 and K12 from 98 to 10% and from 95 to 70%, respe

47 li strains to be correctly classified to the O157:H7 and K12 sub-species clades.

48 anise oil reduced the population of E. coli O157:H7 and L. monocytogenes by 1.48 and 0.47 log cfu/ml

49 nise oil nanoemulsion (AO75) reduced E. coli O157:H7 and L. monocytogenes count by 2.51 and 1.64 log

50 We found espW in the sequenced O157:H7 and non-O157 EHEC strains as well as in Shigella

51 the transport of Escherichia coli pathogenic O157:H7 and nonpathogenic K12 strains in water-saturated

52 ulated (Mat) fimbriae) for E. coli serotypes O157:H7 and O18:K1:H7.

53 7BL/6 mice were also inoculated with E. coli O157:H7 and only 1 of 14 developed disease, whereas 10 o

54 cial to reduce the further spread of E. coli O157:H7 and other (emerging) STEC strains globally.

55 s testing of the foodborne pathogens E. coli O157:H7 and Salmonella enterica, in detail a nucleic aci

56 ow as 20 nm was capable of detecting E. coli O157:H7 and Salmonella sp. in complex hamburger and cucu

57 dicating different concentrations of E. coli O157:H7 and smart phone imaging APP for monitoring color

58 covalently grafting an anti-Escherichia coli O157:H7 antibody onto SAM-modified gold electrodes.

59 e feasibility was demonstrated using E. coli O157:H7 as a model analyte, this approach could be easil

60 d decimal reduction times of Escherichiacoli O157:H7 at different heating temperatures were used in e

61 nsor was able to specifically detect E. coli O157:H7 at the low concentration within 10 min in pure c

62 he complete assembly of the Escherichia coli O157:H7 autotransporter EspP in vitro.

63 nalysis of mutations in the Escherichia coli O157:H7 autotransporter EspP.

64 nhanced the sensitivity for Escherichia coli O157:H7 bacteria detection.

65 ghly sensitive detection of Escherichia coli O157:H7 bacteria.

66 The Escherichia coli O157:H7 bacteriophage CBA120 genome encodes four distinc

67 The E. coli O157:H7 bacteriophage PhiV10 was modified to express Nan

68 The genome of Escherichia coli O157:H7 bacteriophage vB_EcoM_CBA120 encodes four distin

69 ic substrates were anti-adhesive for E. coli O157:H7 binding to human HT29 cells.

70 tation and genome diversification in E. coli O157:H7 but also contributing to the development of path

71 QS columns enhanced the transport of E. coli O157:H7 by 3.1 fold compared to the unoxidized counterpa

72 , we reported the rapid detection of E. coli O157:H7 by using calcium signaling of the B cell upon ce

73 E. coli O157:H7 can cause bloody diarrhea, hemolytic uremic synd

74 rical properties once the pathogenic E. coli O157:H7 captured on the sensor surface.

75 Enterohemorrhagic Escherichia coli (EHEC) O157:H7 causes hemorrhagic diarrhea and potentially fata

76 s of antibodies with single Escherichia coli O157:H7 cells and demonstrated a capability of determini

77 rotect host epithelial cells against E. coli O157:H7 colonization, at least in part, by promoting muc

78 lular immune responses of cattle during EHEC O157:H7 colonization.

79 157:H7 from cultures containing 1000 E. coli O157:H7 colony-forming units (cfu)/mL, or approximately

80 ion of color was correlated with the E. coli O157:H7 concentration.

81 All samples testing positive for E. coli O157:H7 contained deer feces, and 5 tested farm fields h

82 munosensor for specific detection of E. coli O157:H7 contamination with the use of sandwich assay eva

83 ed out in a single step for Escherichia coli O157:H7 detection.

84 tphone based fluorescence device for E. coli O157:H7 detection.

85 ction experiments showed that 73% of E. coli O157:H7 died within 2 h with a disinfection rate constan

86 lity that E. coli Nissle 1917 can starve the O157:H7 E. coli strain EDL933 of gluconeogenic nutrients

87 157:H7 when tested against a panel of 15 non-O157:H7 E. coli.

88 cfp) and Stx1 production (stx1::yfp) in EHEC O157:H7 EDL933.

89 Tir from enterohemorrhagic Escherichia coli O157:H7 expressed in epithelial cells induced a loss of

90 min and subsequently incubated with E. coli O157:H7 for 30 min to achieve a 3.2 +/- 0.2 log CFU/mL r

91 Concentrations of E. coli O157:H7 from 3x10(1) to 3x10(7)cfu/mL could be detected.

92 ction and identification of Escherichia coli O157:H7 from colony isolates in a colorimetric multiplex

93 tely 95%) in isolating live Escherichia coli O157:H7 from cultures containing 1000 E. coli O157:H7 co

94 sensitive tools for the detection of E. coli O157:H7 from food as they are host-specific and able to

95 assay shows promise for detection of E. coli O157:H7 from food in a simple, fast and sensitive manner

96 ar epidemiology data was applied on the EHEC O157:H7 genome to select new potential vaccine candidate

97 w niches, interrogation of sequenced E. coli O157:H7 genomes showed a high level of CycA conservation

98 Enrichment assays using E. coli O157:H7 grown in LB broth with a reporter phage concentr

99 t is critical to eliminate or reduce E. coli O157:H7 gut colonization.

100 Escherichia coli O157:H7 has been shown to express heme uptake and transp

101 ted with different concentrations of E. coli O157:H7 have been tested using anti-E. coli O157-magneti

102 A specific DNA sequence from E. coli O157:H7 having 22 mers as an amine-terminated probe ssDN

103 demonstrate that recognition of the E. coli O157:H7 host by CBA120 involves binding to and digesting

104 upon cellular membrane anchors anti-E. coli O157:H7 IgM.

105 ellular oxidative stress detected in E. coli O157:H7 illustrated that ROS also played a role in the a

106 Presence of higher than 1cfu E.coli O157:H7 in 25g of food has been considered as a dangerou

107 nd quantum dots for determination of E. coli O157:H7 in beef and river water.

108 could detect as low as 100 CFU/ml of E. coli O157:H7 in buffer and 600 CFU/ml E. coli O157:H7 in liqu

109 ity and sensitivity for detection of E. coli O157:H7 in chicken samples with a lower detection limit

110 id method which can detect low level E. coli O157:H7 in foods at real-time.

111 The detection of E. coli O157:H7 in foods has held the attention of many research

112 sensor was used for the detection of E. coli O157:H7 in ground beef samples.

113 oli O157:H7 in buffer and 600 CFU/ml E. coli O157:H7 in liquid food systems.

114 ction and quantification of Escherichia coli O157:H7 in meat and water samples based on the electroca

115 complete inhibition of the growth of E. coli O157:H7 in the later 24 h irradiation.

116 rating the assay's ability to detect E. coli O157:H7 in the presence of high levels of background DNA

117 Limits of detection of Escherichia coli O157:H7 in undiluted milk were determined to be 6x10(4),

118 ppaB inflammatory signaling, whereas E. coli O157:H7 infection suppressed this pathway by inhibiting

119 strawberries as a novel vehicle for E. coli O157:H7 infection, implicated deer feces as the source o

120 tect consumers from deadly foodborne E. coli O157:H7 infection, it is vital to develop a simple, reli

121 nt complex, was protective in murine E. coli O157:H7 infection.

122 reased by TNF-alpha treatment and by E. coli O157:H7 infection.

123 was investigated in a mouse model of E. coli O157:H7 infection.

124 In a recent cluster of Escherichia coli O157:H7 infections attributed to salad bar exposures and

125 This multistate outbreak of STEC O157:H7 infections was associated with consumption of ro

126 dates potentially useful for preventing EHEC O157:H7 infections.

127 nflammatory response acts to perturb E. coli O157:H7 intestinal colonization.

128 Escherichia coli O157:H7 is a notorious foodborne pathogen due to its low

129 Shiga toxin-producing Escherchia coli (STEC) O157:H7 is a zoonotic pathogen that causes numerous food

130 E. coli O157:H7 is an enterohemorrhagic bacteria responsible for

131 Enterohemorrhagic Escherichia coli (EHEC) O157:H7 is an important food-borne pathogen responsible

132 The pathogenicity of E. coli O157:H7 is mainly caused by the expression of Shiga-like

133 n of the foodborne pathogen Escherichia coli O157:H7 is of vital importance for public health worldwi

134 n of food contaminated with Escherichia coli O157:H7 is one of the major concerns in ensuring food sa

135 Escherichia coli O157:H7 is one of the most notorious foodborne pathogens

136 Enterohemorrhagic Escherichia coli O157:H7 is responsible for many outbreaks of gastrointes

137 red the virulence of K3995 to those of other O157:H7 isolates and an isogenic Stx2 mutant in rabbits

138 site platform was modified with anti-E. coli O157:H7 monoclonal antibody.

139 Otherwise, E. coli O157:H7 only growth 50% when PBE was added to the cultur

140 he 22 identified outbreak-associated E. coli O157:H7 or E. coli O61 pulsed-field gel electrophoresis

141 he 22 identified outbreak-associated E. coli O157:H7 or E. coli O61 pulsed-field gel electrophoresis

142 In 2006, a deadly Escherichia coli O157:H7 outbreak in bagged spinach was traced to Califor

143 Considering the health risks of E. coli O157:H7 presence in food and water, an affordable and hi

144 Shiga toxin-producing Escherichia coli O157:H7 primarily resides in cattle asymptomatically, an

145 fected by the PhiV10 reporter phage, E. coli O157:H7 produces a strong bioluminescent signal upon add

146 Given that E. coli O157:H7 produces effectors that attenuate inflammatory s

147 as developed based on the ability of E. coli O157:H7 proteases to change the optical response of a su

148 lting from contamination of Escherichia coli O157:H7 remain a serious concern in food safety.

149 57 STEC in clinical samples and that E. coli O157:H7 remains the predominant cause of HUS in our inst

150 A 2006 spinach-associated outbreak of STEC O157:H7 resulted in higher hospitalization and HUS rates

151 n strategy for pathogenic strains of E. coli O157:H7 serotype based on a conserved signature insertio

152 ory effect of C70-TiO2 thin films on E. coli O157:H7 showed a decrease of the bacterial concentration

153 ors that were biofunctionalized with E. coli O157:H7 specific antibodies for sensitive pathogenic bac

154 r detecting and quantifying Escherichia coli O157:H7 specific eaeA gene.

155 a lineage distinct from previously reported O157:H7 ST11 EHEC and was not a member of the hypervirul

156 E. coli microdiversity analysis yielded one (O157:H7 str.

157 infection, while none of those infected with O157:H7 strain 2812 (Stx1a(+) Stx2a(+)) died or showed p

158 w (2%) fiber diets and infected with E. coli O157:H7 strain 86-24 (Stx2+).

159 with K3995 died than did those infected with O157:H7 strain 86-24 (Stx2a(+)).

160 We previously showed that enterohemorrhagic O157:H7 strain E. coli EDL933 colonizes a niche in the s

161 as able to reduce the bacterial load of EHEC O157:H7 strain in feces, colon and caecum tissues after

162 ne based capacitors were specific to E. coli O157:H7 strain with a sensitivity as low as 10-100 cells

163 oroge et al. reported that E. coli 86-24, an O157:H7 strain, activates the expression of virulence ge

164 This study characterized an E. coli O157:H7 strain, designated PA2, that belongs to the hype

165 hen it is confronted with E. coli EDL933, an O157:H7 strain.

166 Enterohemorrhagic Escherichia coli (EHEC) O157:H7 strains are major human food-borne pathogens, re

167 a probiotic E. coli strain that outcompetes O157:H7 strains for gluconeogenic nutrients could render

168 greater virulence of K3995 compared to other O157:H7 strains in rabbits and mice.

169 was less effective against Escherichia coli O157:H7 than free LAE, which was correlated with the ava

170 cision enhancer (IEE) was discovered in EHEC O157:H7 that promoted the excision of IS3 family members

171 The binding of E. coli O157:H7 to the IgM on B cell surface activates the B cel

172 p and tested for immunogenicity against EHEC O157:H7 using a murine model of gastrointestinal infecti

173 id technique to detect low levels of E. coli O157:H7 using membrane filtration and silver intensifica

174 (2) The retention of E. coli O157:H7 was 3.3 fold higher than that of E. coli K12 in

175 tocatalytic disinfection of Escherichia coli O157:H7 was achieved by using a C70 modified TiO2 (C70-T

176 sed Stx2 accumulation has been reported when O157:H7 was cocultured with phage-susceptible nonpathoge

177 or the common food pathogen Escherichia coli O157:H7 was developed.

178 An outbreak of Escherichia coli O157:H7 was identified in Oregon through an increase in

179 s with 10(3), 10(4) and 10(5) CFU/mL E. coli O157:H7 were 106.98, 96.52 and 102.65 (in yogurt) and 10

180 sence of UV-A light against Escherichia coli O157:H7 were investigated.

181 O157:H7, the sensitive detection of E. coli O157:H7 were realized both in standard samples and real

182 .0 x 10(7) genome copies and was specific to O157:H7 when tested against a panel of 15 non-O157:H7 E.

183 ducing, food-borne pathogen Escherichia coli O157:H7 will develop a life-threatening sequela called t

184 confirmed cases as persons from whom E. coli O157:H7 with the outbreak PFGE pattern was cultured duri

185 and thus, enable visual detection of E. coli O157:H7 without instrumentation.

186 e assay truly discriminated Escherichia coli O157:H7's 16s rRNA from closely related bacteria with a

187 ontaminant Escherichia coli O157:H7 (E. coli O157:H7) in complex food products due to the recent outb

188 Whole bacteria (Escherichia coli O157:H7) were assayed in buffer as well as 5% diluted hu

189 pathogenic Escherichia coli O157:H7 (E.coli O157:H7), a dangerous strain among 225 E. coli unique se

190 (Listeria monocytogenesis 19115 and E. coli O157:H7), clinical isolates (methicillin-resistant S. au

191 the gram-negative organism (Escherichia coli O157:H7), preventing unbiased detection and quantitation

192 ion of pathogenic bacteria (Escherichia coli O157:H7).

193 (Salmonella enteritidis and Escherichia coli O157:H7).

194 Escherichia coli O157:H7, a major Shiga toxin-producing pathogen, has a l

195 CFT073 and UTI89, enterohemorrhagic E. coli O157:H7, and enterotoxigenic E. coli O78:H11, compared t

196 urrent international distribution of E. coli O157:H7, and it is likely that these events were facilit

197 uding Campylobacter jejuni, Escherichia coli O157:H7, and multidrug resistant Klebsiella pneumoniae.

198 f cattle is the primary reservoir of E. coli O157:H7, and thus, it is critical to eliminate or reduce

199 teraction between AuNPs and Escherichia coli O157:H7, AuNPs attached to the surface of the bacteria a

200 experiments using meats spiked with E. coli O157:H7, colicins efficiently reduced the population of

201 In enterohemorrhagic Escherichia coli (EHEC) O157:H7, EutR responds to ethanolamine to activate expre

202 gens (Campylobacter jejuni, Escherichia coli O157:H7, non-O157 E. coli, Listeria monocytogenes, and S

203 desirable for specific detection of E. coli O157:H7, one of the leading bacterial pathogens causing

204 biota could enhance the virulence of E. coli O157:H7, particularly a subset of clade 8 strains.

205 us aureus, Bacillus cereus, Escherichia coli O157:H7, Pseudomonas aeruginosa and Salmonella typhimuri

206 ilk), the LOD was under 5 CFU/mL for E. coli O157:H7, S. typhimurium and L. monocytogenes.

207 ng dose-dependent bacteria (Escherichia coli O157:H7, Salmonella enterica serovar Typhimurium and S.

208 ccus faecalis, Escherichia coli K12, E. coli O157:H7, Salmonella enterica serovar Typhimurium LT2, St

209 mL of the tested pathogens (Escherichia coli O157:H7, Salmonella typhimurium, and Listeria monocytoge

210 specific recognition of antibody to E. coli O157:H7, the sensitive detection of E. coli O157:H7 were

211 mune responses during colonization with EHEC O157:H7, the temporality of which is strain dependent, w

212 Irrespective of the presence of E. coli O157:H7, TNF-alpha enhanced activation of p65, the key m

213 Three strains of E. coli, BL 21, ATCC8739, O157:H7, when spiked into UHT milk and fermented palm ju

214 tes the first reported human case of E. coli O157:H7, which was in 1975 from the United States.

215 1,25(OH)2D3 altered E. coli O157:H7-induced reductions in transepithelial electrical

216 ly associated with enterohemorrhagic E. coli O157:H7-mediated hemorrhagic colitis that sometimes prog

217 Listeria monocytogenes and Escherichia coli O157:H7.

218 n even in low concentration range of E. coli O157:H7.

219 visual and quantitative detection of E. coli O157:H7.

220 terica serovar Bareilly and Escherichia coli O157:H7.

221 LT-1 from the complex cell lysate of E. coli O157:H7.

222 ains, including the enterohemorragic E. coli O157:H7.

223 nged with enterohemorrhagic Escherichia coli O157:H7.

224 hanced colorimetric signal to detect E. coli O157:H7.

225 nd resulted in decreased adhesion of E. coli O157:H7.

226 toxin-antitoxin system from Escherichia coli O157:H7.

227 toxin-antitoxin module from Escherichia coli O157:H7.

228 ing the imbalance of redox status of E. coli O157:H7.

229 at confers phage specificity towards E. coli O157:H7.

230 is gram negative pathogenic species E. coli O157:H7.

231 cause injury to the cell membrane of E. coli O157:H7.

232 stolytica, Cryptosporidium spp., and E. coli O157:H7; 95% for Giardia lamblia; 94% for ETEC and STEC;

233 te stand-alone test for detection of E. coli O157 in clinical samples.

234 ng enterohemorrhagic Escherichia coli (EHEC) O157 in seeded stool samples.

235 tool to inform national surveillance of STEC O157 in terms of identifying linked cases and clusters a

236 ylvania officials reported a cluster of STEC O157 infections associated with multiple locations of a

237 we have determined that a subset of E. coli O157 infections will not be detected if an agar-based me

238 oratory characterization of Escherichia coli O157 is essential to inform epidemiological investigatio

239 toxin-producing Escherichia coli O157 (STEC O157), is key to rapidly identifying linked cases in the

240 rt vector machine analysis of bovine E. coli O157 isolate sequences can be applied to predict their z

241 nor subset (less than 10%) of bovine E. coli O157 isolates analyzed in our datasets were predicted to

242 E. coli O157 isolates clearly segregated into SNP lineages that

243 orphism (SNP) assay to segregate 148 E. coli O157 isolates from Australia, Argentina, and the United

244 enome sequencing was used to compare E. coli O157 isolates from host reservoirs (cattle and sheep) fr

245 olution of the relationships between E. coli O157 isolates than that provided by MLVA.

246 differences between human and bovine E. coli O157 isolates were due to the relative abundances of hun

247 Among E. coli O157 isolates, 57 (88%) were identified by both SMAC aga

248 comparison of human PT8 and animal PT21 VTEC O157 isolates.

249 tool to inform national surveillance of STEC O157; it can be used in real time to provide the highest

250 tudies of the global distribution of E. coli O157 lineages and the impacts of regionally predominant

251 he impacts of regionally predominant E. coli O157 lineages on the prevalence and severity of disease.

252 ecies of pathogens, such as Escherichia coli O157, Listeria innocua, Staphylococcus aureus, Enterococ

253 O157:H7 have been tested using anti-E. coli O157-magnetic beads conjugate (MBs-pECAb) as a capture p

254 ults prompted successful recovery of E. coli O157 (n = 25) and non-O157 STEC (n = 8) isolates, althou

255 CBA120 involves binding to and digesting the O157 O-antigen by TSP2.

256 TSP2 in complex with a repeating unit of the O157 O-antigen.

257 nzymes in the E. coli serotype O157 (E. coli O157) O-PS biosynthetic pathway.

258 Escherichia coli serogroups O157, O26, O45, O103, O111, O121, and O145, when carryin

259 TSP3 and TSP4 are hydrolases that digest the O157, O77, and O78 Escherichia coli O-antigens, respecti

260 2, TSP3, and TSP4, CBA120 can infect E. coli O157, O77, and O78, respectively.

261 curred in 12 patients (10 infected with STEC O157, one infected with STEC O125ac, and one with PCR ev

262 : Aeromonas, Campylobacter, Escherichia coli O157, other Shiga toxin-producing E. coli, enterotoxigen

263 largest multistate leafy greens-linked STEC O157 outbreak in several decades.

264 e largest multistate leafy green-linked STEC O157 outbreak in several decades.

265 timates of linked clusters representing STEC O157 outbreaks in England and Wales increased by 2-fold

266 ocytotoxin-producing Escherichia coli (VTEC) O157 phage types (PTs), such as PT8 and PT2, are associa

267 ter-positive specimens, 0/4 Escherichia coli O157-positive specimens, 0/9 Salmonella-positive specime

268 hi8-positive strains were compared with VTEC O157 possessing BP933W.

269 s randomly sampled from 1002 strains of STEC O157 received by the Gastrointestinal Bacteria Reference

270 atic synthesis of Escherichia coli (serotype O157) RU-PP-Und.

271 iarrhea for non-O157 STEC in addition to the O157 serotype by using a sensitive assay.

272 Parenteral vaccination can reduce O157 shedding in cattle after challenge and limit zoonot

273 ighly pathogenic serotypes (O157 and top non-O157 Shiga toxin-producing Escherichia coli [STEC]) impl

274 x2) (including specific detection of E. coli O157), Shigella spp./enteroinvasive E. coli, Cryptospori

275 ivity and reliably detected Salmonella, EHEC O157, Shigella, and Campylobacter at concentrations 1- t

276 ated whole-cell vaccines for O157 can induce O157-specific cellular and mucosal immune responses that

277 in Adj-Vac group had a higher percentage of O157-specific IFNgamma producing CD4(+) and gammadelta(+

278 Furthermore, O157-specific IgA levels detected in feces of the Adj-Va

279 ul recovery of E. coli O157 (n = 25) and non-O157 STEC (n = 8) isolates, although the primary culture

280 th acute community-acquired diarrhea for non-O157 STEC in addition to the O157 serotype by using a se

281 lture and distinguishes between O157 and non-O157 STEC in clinical samples and that E. coli O157:H7 r

282 e IEE sequences from O157 and the top 10 non-O157 STEC serotypes fell into clusters I and II, while l

283 ta showed an incidence rate of 51.2% for non-O157 STEC strains, with 5.8% of patients (1/17) with non

284 E. coli O157 and non-O157 STEC were detected in 35 and 18 cases, respectively

285 red in attempts to recover both O157 and non-O157 STEC.

286 li O157 and 3 (10%) were attributable to non-O157 STEC.

287 ch as Shiga toxin-producing Escherichia coli O157 (STEC O157), is key to rapidly identifying linked c

288 with non-O157 strains and 42.9% (6/14) with O157 strains (P = 0.03) developing hemolytic-uremic synd

289 rains, with 5.8% of patients (1/17) with non-O157 strains and 42.9% (6/14) with O157 strains (P = 0.0

290 were attributable to six "atypical" E. coli O157 strains and included recombinant regions.

291 esence of PT8-like phages in a panel of VTEC O157 strains belonging to different PTs and determined t

292 ich we named Phi8, was more frequent in VTEC O157 strains from human disease than in bovine strains.

293 One hundred five E. coli O157 strains isolated over a 5-year period from human fe

294 pattern combinations, or with a strain STEC O157 that was closely related to the main outbreak strai

295 pattern combinations, or with a strain STEC O157 that was closely related to the main outbreak strai

296 Citrobacter rodentium and Escherichia coli O157 triggered similar Th17 responses, whereas adhesion-

297 Subtilase cytotoxin (SubAB), produced by non-O157 type Shiga-toxigenic Escherichia coli (STEC), is an

298 LD-PCR differentiated STEC O157 from non-O157 using rfbEO157, and LD-PCR results prompted success

299 r characterize the cattle immune response to O157 vaccination, cattle were vaccinated with either wat

300 Cattle are the primary reservoir for O157, which colonizes the intestinal tract without induc