ライフサイエンスコーパス: O2

1 O2 binding to the Fe(II) heme complex of its N-terminal

2 O2 plasma etching increased the sensitivity due to incre

3 O2 plasma etching was performed by a microwave plasma sy

4 e cells adapted to a hypoxic environment (1% O2), those cultured in 5% O2 still mobilized sufficient

5 dothelial cells into a hypoxic incubator (1% O2) for 24 hours, with reoxygenation for 6 hours.

6 Our data support a new model in which a (1) O2 retrograde signal, generated by chlorophyll precursor

7 ncer cells, whereas provides an elevated (1) O2 environment in the mitochondria.

8 luorescent nanoprobe for singlet oxygen ((1) O2 ) detection in biological systems was designed, synth

9 t enables tumor-specific singlet oxygen ((1) O2 ) generation for cancer therapy, based on a Fenton-li

10 t specifically generates singlet oxygen ((1) O2 ) in mitochondria under white light irradiation that

11 vitro and in vivo through tumor-specific (1) O2 generation and subsequent ROS mediated mechanism.

12 (1)O2 generation by BADs in living cells enables visualizat

13 M generated similar levels of (3)DOM* and (1)O2, enhancing degradation of CTC, ROX, and SMX.

14 Finally, PhiRI for (1)O2 and (3)DOM* correlated negatively with antioxidant ac

15 Imidazole, a well-known (1)O2 scavenger, was incorporated in the hydrophobic core o

16 in the BODIPY subunit defines the site of (1)O2 addition.

17 enhanced fluorescence and singlet oxygen ((1)O2) production upon laser irradiation.

18 yer via the production of singlet oxygen ((1)O2).

19 mited diffusion radius of singlet oxygen ((1)O2).

20 s a second O2 to form trans-[Pd(IPr)2(eta(1)-O2)2].

21 zed and converted into their endoperoxides 1-O2 upon oxidation with singlet oxygen.

22 min after three 5 min hypoxic episodes (11% O2, H1-H3).

23 d that continuously breathing normobaric 11% O2 from an early age prevents neurological disease and d

24 nally, we show that breathing normobaric 11% O2 in mice with late-stage encephalopathy reverses their

25 tion reactions carried out with labeled (18) O2 unambiguously show that the oxygen atoms present in t

26 e of an unusual (S)P species [(MeAN)2Cu(II)2(O2(2-))](2+) ((S)P(MeAN), MeAN: N-methyl-N,N-bis[3-(dime

27 similar -40 degrees C, cis-[Pd(IPr)2(eta(2)-O2)] adds a second O2 to form trans-[Pd(IPr)2(eta(1)-O2)

28 on to reaction with O2, cis-[Pd(IPr)2(eta(2)-O2)] reacts at low temperature with H2O in methanol/ethe

29 ing of the NHC plane in cis-[Pd(IPr)2(eta(2)-O2)].

30 e to further stabilize cis-[Pd(IMes)2(eta(2)-O2)].

31 ia formation of a transient [Cu(I)(NH3)2](+)-O2-[Cu(I)(NH3)2](+) intermediate.

32 engthy adaptation to atmospheric oxygen (20% O2) in cell culture, limiting pre-clinical studies of pa

33 promising cathode materials, Na(Li1/3 M2/3 )O2 (M: transition metals featuring stabilized M(4+) ), f

34 tion mechanism in Li2 MnO3 , Na(Li1/3 Mn2/3 )O2 is designed as an example of a new class of promising

35 Human MSCs, grown chronically at 5% O2, were administered intravenously.

36 ellular matrix and physiological hypoxia (5% O2).

37 Culture of cells in 5% O2 (>5 d) decreased histamine- but not shear stress-stim

38 ic environment (1% O2), those cultured in 5% O2 still mobilized sufficient Ca(2+) to activate AMPK.

39 r, increased dephosphorylation of eNOS in 5% O2 was Ca(2+)-sensitive and reversed by okadaic acid or

40 brane targeting of PP2A-C was observed in 5% O2, resulting in greater interaction with eNOS in respon

41 and biofilm formation under microaerobic (5% O2) conditions.

42 ban was increased in cells cultured under 5% O2.

43 dothelial cells cultured in physiologic (5%) O2 and stimulated with histamine or shear stress.

44 Simulation revealed that 12 degrees C of 50% O2 PIM-R culture supplied O2 effectively into the islet

45 re chopped, modified atmosphere packaged (70%O2/30%CO2) and maintained under retail conditions (4+/-0

46 device based on the model system Gd0.1 Ce0.9 O2-delta /Er2 O3 to set and tune the property of "memris

47 ped SWCNTs with ionic strength, pH, adsorbed O2, and ascorbic acid.

48 of O2 with Al2O3 also activates the adsorbed O2 for receiving electrons from the photoexcited dyes.

49 of the Ru was still in molecular form after O2 evolution.

50 in experimental models of IH patterned after O2 profiles seen in OSA.

51 and the relatively hydrophilic surface after O2 plasma etching provided better resistance to fouling

52 in-between NIV sessions, was superior to air/O2 in reducing NIV failure (25-15%) in severe hypercapni

53 he decrease in matrix [dicarboxylate] allows O2 access to reduced site IIf, thereby making electron d

54 noxygenic phototrophic bacteria use BchE, an O2-sensitive [4Fe-4S] cluster protein, whereas plants, c

55 from Toxoplasma gondii is hydroxylated by an O2-dependent prolyl-4-hydroxylase (PhyA), and the result

56 e of NO consumption by metabolizing NO in an O2-dependent manner with decreased NO consumption in phy

57 a, and some phototrophic bacteria possess an O2-dependent enzyme, the major catalytic component of wh

58 grees C, 22 degrees C, and 37 degrees C) and O2 concentration (21% and 50%).

59 ligated (deoxy), CO-inhibited (carboxy), and O2-bound (oxy) hemes in myoglobin (MB) and hemoglobin (H

60 treatment as it biologically couples CO2 and O2 fluxes.

61 l example, Schonecker showed that copper and O2 promoted the hydroxylation of steroid-containing liga

62 flue gas components (SO2, NO, NO2, H2O, and O2) on vanadium at 500-600 degrees C were investigated b

63 nability, relying on green oxidants H2O2 and O2 as the ultimate oxygen source.

64 ve activation of peroxide intermediates, and O2 evolution.

65 s on electronic charge of both heme iron and O2 , resulting in increased O2 dissociation and reduced

66 ng gradual depletion of TAN (NH3 + NH4+) and O2 .

67 monooxygenase, all of which can bind NO and O2.

68 catalyze OH radical formation from H2 O and O2 at high temperatures.

69 uring simultaneously high CO2 resistance and O2 permeability and the exploitation of phase reactions

70 By optimizing temperature and O2 concentration, we cultured human islets for 2 weeks w

71 lipid peroxidation and of superoxide anion (O2(* horizontal line )) were higher in Prdx6 (-/-) than

72 the transformation of the superoxide anion (O2*-) into hydrogen peroxide.

73 actions conducted with air rather than 1 atm O2.

74 thology and the concentration of atmospheric O2 Future work on glaciation-weathering-carbon cycle fee

75 ither assembling into spoked wheels, 1-5 bar O2, or closely packed in parallel lines, above 2.2 bar.

76 to use our previously reported gelatin-based O2-controllable hydrogels that can provide hypoxic micro

77 elivery facilitated by hemoglobin (Hb)-based O2 carriers (HBOCs) is a promising strategy to increase

78 hniques, reflecting a tight coupling between O2 production and consumption by aerobic processes under

79 sed for detection and discrimination between O2 and H2O at low temperature.

80 Blood O2 saturation (r(2) = 0.80, P < 0.0001) and plasma gluco

81 ddition to -C( identical withN), followed by O2 addition to -C(OH) horizontal lineN., internal H-shif

82 However, inactivation by O2 remains an obstacle that prevents them being used in

83 roduct resulting from the oxidation of NO by O2 under aerobic conditions.

84 Abiotic Fe(II) oxidation by O2 commonly occurs in the presence of mineral sorbents a

85 alkyl halides, as well as rapid oxidation by O2, to generate detectable Ni(III) and/or Ni(IV) interme

86 Therefore, cardiac O2 consumption is controlled by endothelial NO in a para

87 ystins in sensing and responding to cellular O2 levels.

88 with molecular oxygen: for instance, CH2I + O2 --> CH2OO + I.

89 bon nanotube yarn microelectrodes (CNTYMEs): O2 plasma etching and antistatic gun treatment.

90 The futile redox cycle rapidly consumes O2, rendering standard assays of Krebs cycle turnover un

91 diments to artificial groundwater containing O2 or NO3(-) under diffusion-limited conditions.

92 d that hyen D, E, L, and M and cycloviolacin O2 preferentially interact with model lipid membranes th

93 lotides and 1 known cyclotide (cycloviolacin O2) from H. enneaspermus and used tandem MS to determine

94 ding antibacterial activity of cycloviolacin O2 against A. baumannii.

95 activity comparable to that of cycloviolacin O2, one of the most cytotoxic known cyclotides.

96 uced arterial hypoxaemia (EIAH) can decrease O2 delivery and exacerbate exercise-induced quadriceps f

97 y inhibits mitochondrial complex I-dependent O2 consumption and reverse electron transfer-mediated re

98 of transition-metal complexes with dioxygen (O2 ) is important for understanding oxidation in biologi

99 Direct O2 utilization suffers from intrinsic challenges imposed

100 eal reaction mechanism, which permits direct O2 formation in single collisions of energetic water ion

101 d on islet O2 consumption rate and dissolved O2 in the medium.

102 indow of pH and in the presence of dissolved O2, but occurs independently of this optical quenching.

103 theory (NRT) analyses identified a dominant O2 imidate resonance form for residue bound 1-methyl-ura

104 antified the functional significance of each O2 pathway defect by calculating the improvement in exer

105 en envisioned because they provide efficient O2 reduction with almost no overpotential.

106 parate electron inventories of four-electron O2 reduction and two-electron substrate oxidation.

107 eeting existence of reversible four-electron O2 reduction and water oxidation by 'blue' Cu oxidases,

108 Finally, elevated O2 pressures are shown to contribute to background oxida

109 ell as in situ collection of locally evolved O2 by photosystem 2 using a positioned scanning electroc

110 ut) and functional capacity by peak exercise O2 consumption.

111 An extracellular O2 concentration of approximately 0.007 mm could supply

112 ential EPTM, 2-chloro-H2Q, allows for faster O2 reduction rates at higher applied potential.

113 The emerging Fe-O2 bonding situation includes in essence a ferrous iron

114 oproteins and are also used as models for Fe-O2 systems.

115 During cognitive testing at 100% FI,O2 , alpha activity was less desynchronized within the t

116 2 ) followed by a 30-min exposure to 100% FI,O2 .

117 measured during exposure to 21% and 100% FI,O2 .

118 x did not influence gCBF response to 100% FI,O2 .

119 We found that exposure to 100% FI,O2 reduced gCBF to 63% of baseline values across all par

120 d within the temporal regions than at 21% FI,O2 .

121 Cognitive performance testing at 21% FI,O2 was accompanied by increased theta and beta power wit

122 rred through fractional inspired oxygen ( FI,O2 ) concentrations of 60-100% (hyperoxia).

123 during exposure to 21% inspired oxygen ( FI,O2 ) followed by a 30-min exposure to 100% FI,O2 .

124 te >22/min, abnormal chest imaging findings, O2 saturation lower than 90%, and aspartate aminotransfe

125 the development of efficient biocathodes for O2 reduction relying both on direct and mediated electro

126 ip surpass the 2008 Robeson upper bounds for O2/N2, H2/N2, CO2/N2, H2/CH4 and CO2/CH4, with the poten

127 We determined the optimal conditions for O2 distribution and volume maintenance in a 2-week cultu

128 Free O2 levels in this layer were, however, undetectable by c

129 present in the Mn(IV) dimers originate from O2 .

130 The rates of gross O2 production and carbon fixation in the SCM were found

131 The molecular basis for the O1 --> O2 transition and how ChR2 modulates selectivity between

132 n ordered Na(+) /vacancy arrangement and P2->O2/OP4 phase transitions, leading them to exhibit multip

133 Furthermore, the generated acidic H2 O2 can oxidize l-Arginine (l-Arg) into NO for enhanced g

134 124 promoted anthocyanin accumulation and H2 O2 detoxification in response to cold.

135 ddition of ADMA reduced NOx and increased H2 O2 levels (p<0.001).

136 bsequently maintaining hydrogen peroxide (H2 O2 ) homeostasis in Arabidopsis.

137 -CoAs into trans-2-enoyl-CoA and produced H2 O2 .

138 nal excitation, which yields a remarkable H2 O2 -NO cooperative anticancer effect with minimal advers

139 n of glucose into gluconic acid and toxic H2 O2 , a novel treatment paradigm of starving-like therapy

140 plateaued to a rate similar to the U + H2O + O2 reaction.

141 (MAP), in our case, aerobic, vacuum or high O2, to extend the shelf life of beef.

142 that the best packaging conditions were high-O2 atmosphere in combination with REO.

143 The Mo layer likely hinders O2 gas permeation, impeding contact with active Pt.

144 of adenoside triphosphate in the cortex HMP/O2 versus HMP/Air kidneys (19.8 mmol/mg protein vs 2.8 m

145 tructural features were also observed in HMP/O2 kidneys.

146 centrations were higher in the cortex of HMP/O2 kidneys inferring relative increases in tricarboxylic

147 underwent 18 hours of either oxygenated (HMP/O2) or aerated (HMP/Air) HMP in a paired donation after

148 lactate in the cortex of HMP/Air versus HMP/O2 kidneys (0.056 mM vs 0.026 mM, P < 0.05).

149 stress responses and thermogenesis, and how O2 deficiency leads to metabolic reprograming in cancer.

150 HIm)4](+), and LS-3DCHIm, [(DCHIm)F8Fe(III)-(O2(2-))-Cu(II)(DCHIm)3](+) (F8 = tetrakis(2,6-difluoroph

151 -Cu complexes, LS-4DCHIm, [(DCHIm)F8Fe(III)-(O2(2-))-Cu(II)(DCHIm)4](+), and LS-3DCHIm, [(DCHIm)F8Fe(

152 ction, as indicated by the level of impaired O2 extraction from arterial blood during peak exercise.

153 stive and capacitive responses to changes in O2 and H2O.

154 extent to which this reflects differences in O2 storage fluctuations and/or contributions from oxidat

155 -dependent structural dynamics of FeO NSs in O2.

156 the hypothesis that age-associated increased O2(*-) and resulting DNA damage mediate the increased su

157 th heme iron and O2 , resulting in increased O2 dissociation and reduced O2 affinity at high E degree

158 the O2 distribution in islets based on islet O2 consumption rate and dissolved O2 in the medium.

159 The lithium-air (Li-O2 ) battery has been deemed one of the most promising n

160 ncompetitive transport, the textile-based Li-O2 cathode exhibits a high discharge capacity of 8.6 mAh

161 ns of cathodic reactions in a liquid-cell Li-O2 microbattery in the presence of the redox mediator te

162 ies or a replacement for lithium metal in Li-O2 and Li-S batteries.

163 y of the discharging/charging products in Li-O2 cells.

164 charge potentials mainly takes place at Li2 O2 /electrolyte interfaces and has obvious correspondenc

165 otonation of the base and protonation of Li2 O2 .

166 Discharge in the lithium-O2 battery is known to occur either by a solution mechan

167 +), whereas nitrifier denitrification at low O2 levels was stimulated by NO2(-) at levels as low as 0

168 h both production pathways stimulated by low O2, independent of NO2(-) concentrations.

169 Hypoxia (low O2) is a fundamental microenvironmental determinant of b

170 How cancer cells adapt to low O2 has been illuminated by numerous studies, with "repro

171 fter purification from cells grown under low O2 and high Fe2+ and (iv) a small fraction of Fe2+ that

172 es bacteria possibly contributed to lowering O2 levels in leaf pockets but did not release detectable

173 easonable faradaic efficiencies for measured O2 production.

174 Enhanced current densities for mediated O2 reduction are observed with the redox nanoparticle sy

175 ucts stimulated significantly more microbial O2 consumption (113 +/- 4 muM) than either the dark (78

176 total electron flow), even at sub-micromolar O2 concentrations.

177 CPC inhibited both mitochondrial O2 consumption [half maximal inhibitory concentration (I

178 sed SOD2 acetylation, elevated mitochondrial O2(. -), and diminished endothelial nitric oxide.

179 ored, demonstrating a role for mitochondrial O2(*-) in these effects.

180 METHOD: Mitochondrial O2 consumption and adenosine triphosphate (ATP) synthesi

181 anism such as the incorporation of molecular O2 are poorly understood.

182 of phoQ Salmonella to RNS requires molecular O2 and coincides with the nitrotyrosine formation, the o

183 ases with a precision better than 1% for N2, O2, CO2, He, Ar, 2% for Kr, 8% for Xe, and 3% for CH4, N

184 al but inert molecules such as H2, COx, N2O, O2, H2O, NH3, C2H4 and E4 (E = P, As).

185 Small molecules such as H2, N2, CO, NH3, O2 are ubiquitous stable species and their activation an

186 uaternary state (R-State) PolyhHb, and a non O2 carrying control.

187 ntigens from Escherichia coli serotypes O1A, O2, O6A, and O25B, developed for the prevention of invas

188 eceive a single dose of ExPEC4V (antigen O1A:O2:O6A:O25B content 4:4:4:4 mug [group 1]; 4:4:4:8 mug [

189 both the presence of H2O2 and the absence of O2 Experiments show that Ccp lacks enough activity to sh

190 The activation of O2 on metal surfaces is a critical process for heterogen

191 l overlayer involves the mixed adsorption of O2 and H2O on a partially defected surface.

192 permeability that limit the applicability of O2 carriers during infusion.

193 The surface complexation of O2 with Al2O3 also activates the adsorbed O2 for receivi

194 of conditions with varying concentrations of O2, NH4(+), and NO2(-).

195 ed models are not a definitive descriptor of O2 carrier interaction in tumor capillary networks, we a

196 ion cohort using ambulatory determination of O2 requirements.

197 and chemical absorption and dissociation of O2 , especially at tellurium vacancy sites.

198 The mechanism and dynamics of O2 dissociation are also reviewed, including the importa

199 experimental and computational enthalpies of O2 binding.

200 m characterized by physiological extremes of O2 tension and blood flow.

201 asis for the various biological functions of O2-utilizing metalloproteins.

202 n together, we propose that the influence of O2 availability on the levels of active Fur adds a previ

203 ur results suggest that seasonal influxes of O2 and NO3(-) may cause only localized mobilization of U

204 en shown that LPMOs can use H2O2, instead of O2, as a cosubstrate.

205 Resting levels of O2 in the rodent brain varied between 6.6 +/- 0.7 muM in

206 er conditions of variable cellular levels of O2.

207 Molecular dynamic simulations (MD) of O2 and H2O adsorption energy on ZnO surfaces were perfor

208 The dominant mechanism of O2 transport into silage remains unresolved.

209 vide support for the postulated mechanism of O2(.-) activation at class I b Mn2 RNRs.

210 in the junctions, induced by the presence of O2 and H2 molecules, respectively.

211 the conversion are formed in the presence of O2 and that high temperature together with prolonged act

212 well as terminal oxidant in the presence of O2 as an external oxidant.

213 ngly, the decrease of GSH, the production of O2 , and the formation of nanoDVD are shown to be synerg

214 In this review, we survey the range of O2 activation processes mediated by heme proteins and mo

215 e hypoxia and modulate the metabolic rate of O2 consumption.

216 X4) enzyme, which catalyzes the reduction of O2 to hydrogen peroxide (H2O2), has been implicated in t

217 The 4H(+)/4e(-) reduction of O2 to water, a key fuel-cell reaction also carried out i

218 that serves as the main in vivo regulator of O2-dependent NO degradation in smooth muscle remains elu

219 The release of O2 creates a hollow nanostructure with Li2O outer-shell

220 enges imposed by the triplet ground state of O2 and the disparate electron inventories of four-electr

221 ce on [Co] and [AcOH], but no dependence on [O2] or [Fc*].

222 o identify residues that contribute to O1 or O2 selectivity and gating to minimize undesirable effect

223 ndrial energy metabolism (glucose oxidation, O2 consumption, and ATP production), insulin secretion w

224 Oxygen (O2) delivery facilitated by hemoglobin (Hb)-based O2 car

225 Oxygen (O2) depletion in the islet core, which leads to central

226 ene by using a controlled low energy oxygen (O2(+)/O(+))-ion for chemical adsorption and a low energy

227 Then, using isotopically labeled oxygen (O2) as an oxidant in the presence of hydrogen peroxide (

228 -type exoplanets including molecular oxygen (O2), ozone (O3), water vapor (H2O), carbon dioxide (CO2)

229 e eighth key metabolite is molecular oxygen (O2), thermodynamically activated for reduction by one el

230 However, LCHF also increased the oxygen (O2 ) cost of race walking at velocities relevant to real

231 s underwent normoxia or hypoxia (10% oxygen [O2]) +/- vitamin C treatment (maternal 200 mg.kg-1 IV da

232 Mixed venous P(O2) decreased during the initial portion of flights in h

233 ng and the elimination of the detrimental P2-O2 phase transition, revealed by ex situ and in situ X-r

234 are attributed to the elimination of the P2-O2 phase transition upon cycling to 4.5 V.

235 k cardiac power output and 69% achieved peak O2 consumption within the ranges of healthy controls.

236 ties relevant to real-life race performance: O2 uptake (expressed as a percentage of new VO2 peak ) a

237 ised order at sea level; acute hypoxia ( PET,O2 = 50 mm Hg) was imposed at baseline and at each therm

238 ed a substantial fraction (70%) of gas-phase O2 More oxygenated products were formed than the amount

239 nce, photosynthetic CO2 and photorespiratory O2 fixation, and starch synthesis in response to changes

240 timuli that are apparent only in physiologic O2 levels.

241 nd mixtures, with and without physiological [O2].

242 espiration (9.7 +/- 7.7 vs 13.7 +/- 4.1 pmol O2/s/10 cells; p = 0.02) and spare respiratory capacity

243 recursor state, which dissociates to produce O2(-).

244 ost of these structures also develop in pure O2 and are identified as (surface) oxides.

245 ing in increased O2 dissociation and reduced O2 affinity at high E degrees ' values.

246 First, reductive O2 activation induces selective oxidative cleavage, reve

247 show that, in general, the way the released O2 is accommodated is linked to lithium-ion diffusion an

248 By removing O2 in electrolyte, a dramatic decrease in Tafel slope of

249 Assembly of this cofactor requires O2, Fe(II), and a reducing equivalent.

250 rporating the total number of days requiring O2 (without restricting at 36 weeks PMA) improved the pr

251 HAH subjects' oxyhaemoglobin saturation ( Sa,O2 ) was ~7% higher.

252 s C, cis-[Pd(IPr)2(eta(2)-O2)] adds a second O2 to form trans-[Pd(IPr)2(eta(1)-O2)2].

253 is was put forward after discounting several O2 production mechanisms in comets, including photolysis

254 Steady-state O2-evolution activity assays revealed that substitution

255 he metal center for reduction and subsequent O2 binding.

256 IV) with prolonged exposure to supplemental O2 (>=120 days) that has the highest risk of respiratory

257 y levels using the duration of supplementary O2 as predictor and respiratory hospitalization after di

258 2 degrees C of 50% O2 PIM-R culture supplied O2 effectively into the islet core.

259 nd maximal oxygen uptake ([Formula: see text]O2 max) were determined with the use of indirect calorim

260 iability explained by the [Formula: see text]O2 max, sex, and SRPAL; dietary carbohydrate and fat int

261 We found that O2 levels regulate the subcellular localization and chan

262 Our studies reveal that O2 reduces hydroxyl ion density at catalyst interface, r

263 The O2 concentration exerted the strongest control on net N2

264 neral acid of the hammerhead ribozyme is the O2' of G8, while that of the pistol ribozyme is a hydrat

265 Systematic analysis of the O2 pathway in HFpEF showed that exercise capacity was un

266 Examination of the O2 requirements of the CO release step revealed that the

267 active site ensures an in-line attack of the O2' nucleophile, and the conformation at the scissile ph

268 rophic bacteria reveals three classes of the O2-dependent enzyme.

269 ermore, both oxides are unstable outside the O2 atmosphere, indicating the presence of active O atoms

270 n species, which damages DNA and reduces the O2 level; (2) decreased cross-membrane proton gradient f

271 We simulated the O2 distribution in islets based on islet O2 consumption

272 rovide direct experimental evidence that the O2 generated during the OER on some highly active oxides

273 eps of oxygen transport and utilization (the O2 pathway) in each patient with HFpEF, identifying the

274 tin complex through its interaction with the O2-sensing prolyl hydroxylase domain containing protein

275 s via their role in Fe(II) oxidation through O2 production, the capacity of their cell surfaces to so

276 ite IIf, thereby making electron donation to O2 possible, explaining the rapid increase in ROS produc

277 How to efficiently oxidize H2O to O2 (oxygen evolution reaction, OER) in photoelectrochemi

278 ), reducing CO2 into CO and oxidizing H2O to O2 with a 64% electricity-to-chemical-fuel efficiency.

279 h lower transport efficiency with respect to O2 consumption.

280 PFM), demonstrate differences in response to O2 and H2O, confirming that different adsorption mechani

281 We propose that the distinctive responses to O2 and H2O adsorption on ZnO could be utilized to statis

282 MoTe2 is found to be ultrasensitive to O2 at elevated temperatures (250 degrees C).

283 Here, multiple sensors tracked O2 and CO2, gas pressure (DeltaP) between internal silag

284 ion of the two unpaired electrons in triplet O2, relative to the unpaired electrons in two hydroxyl r

285 r propane ODH after thermal activation under O2 to open a cobalt coordination site and to oxidize Co(

286 Here we combine high-resolution underway O2/Ar, which provides an estimate of net community produ

287 single, steady-state process at 723 K using O2 as an abundantly available oxidant.

288 larly in developing systems that can utilize O2, will be required to develop a practical process that

289 reported can directly or indirectly utilize O2 as the only net coreactant based only on thermodynami

290 xcludes the intermediacy of mer-(ONO(Q))Re(V)O2(IMes) in this oxygen atom transfer reaction.

291 a: -0.08 +/- 0.02; P = 0.0009) and p$\dot{V}$O2 max (beta: -0.02 +/- 0.01; P = 0.047); p$\dot{V}$O2 m

292 (beta: -0.02 +/- 0.01; P = 0.047); p$\dot{V}$O2 max was 7 mL . kg-1 . min-1 higher in low- vs. high-r

293 imal volume of oxygen consumption (p$\dot{V}$O2 max), n = 47] and 24-h TEE (WBCU, n = 43) were assess

294 Whereas amine donors react with O2-chemisorbed AC and nucleophiles to give dehydrogenati

295 1 was reacted with O2(.-) at -40 degrees C resulting in the formation of a

296 the sensory module, undergoing reaction with O2 that leads to conversion to a [2Fe-2S] form with loss

297 In addition to reaction with O2, cis-[Pd(IPr)2(eta(2)-O2)] reacts at low temperature

298 chemistry at mild potentials and reacts with O2, CO2, and ethylene via formal [4+2] cycloaddition to

299 superconductivity is satisfied in O-doped Y2 O2 Bi.

300 Several Bi 6p x/y bands of Y2 O2 Bi are raised in energy by oxygen doping because the