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1                                              ORD inhibition of 3,3'-T2 formation from rT3 was also ob
2                                              ORD-loaded SNEDDS formulations could be a potential oral
3                                              ORD@SHp@ANG NPs effectively crossed the BBB by ANG pepti
4 posite microbiological score (n = 80 TB, 111 ORD), the TB score was able to discriminate between TB a
5     When data from all sites (n = 75 TB, 120 ORD) were analyzed, the TB score discriminated between T
6 th many environmental challenges to address, ORD was eliminated by the Trump administration in July.
7 port (MDW) and O'Hare International Airport (ORD).
8                                      ECD and ORD spectra are used to investigate the complementarity
9  mechanical predictions of the VCD, ECD, and ORD for monomeric tert-butylphenylphosphinoamidate were
10 ed by the chromosome axis proteins C(2)M and ORD.
11 use C(2)M encodes a Kleisin-like protein and ORD is required for sister-chromatid cohesion, we tested
12 on meiosis-specific proteins SOLO, SUNN, and ORD is required for sister-chromatid cohesion, localizes
13 d, the TB score discriminated between TB and ORD with an area under the curve (AUC) of 0.94 (95% conf
14 core was able to discriminate between TB and ORD with an AUC of 0.88 (95% CI, .83-.94), 80% sensitivi
15 ion between cone photoreceptor terminals and ORDs suggests a novel photoreceptor to ganglion cell con
16                        Second, we calculated ORD and CD spectra of the structural models and compared
17 ole for biplexiform melanopsin ganglion cell ORDs.
18                          After condensation, ORD remains bound at the centromeres of wild-type sperma
19       We present the Open Reaction Database (ORD), an open-access schema and infrastructure for struc
20                     Outer Retinal Dendrites (ORDs) either ramify within the outer plexiform layer (OP
21 oximately 40% decrease in the time-dependent ORD signal at 230 nm that is best fit to a single-expone
22 nce, its Office of Research and Development (ORD) has since developed and translated science to infor
23 thin its Office of Research and Development (ORD).
24  as having TB or other respiratory diseases (ORD).
25 ry TB (ATB) from other respiratory diseases (ORDs), and latent TB infection (LTBI) from healthy contr
26 nd experimental optical rotatory dispersion (ORD) data provides the most straightforward way to assig
27 oism (ECD), and optical rotatory dispersion (ORD) spectroscopy.
28 ination of NMR, optical rotatory dispersion (ORD), and circular dichroism (CD) spectroscopy together
29 oism (ECD), and optical rotatory dispersion (ORD).
30 ectroscopy, and optical rotatory dispersion (ORD).
31 so supported by optical rotatory dispersion (ORD).
32 d time-resolved optical rotatory dispersion (ORD).
33 tem to study the oligopeptide repeat domain (ORD) expansions of the prion protein, PrP, and their eff
34                  Their OSBP-related domains (ORDs) harbored either PI4P or phosphatidylserine (PS) an
35 Sup35p with that from wild type and expanded ORDs of PrP and compared their biochemical properties in
36 normal meiotic chromosome segregation, extra ORD+ protein in mei-S332 mutant males enhances nondisjun
37 rotein-protein interactions are critical for ORD function.
38                       Additionally, we found ORDs asymmetrically distributed to the dorsal retina.
39 e for diagnosing and distinguishing ATB from ORDs and LTBI from HC.
40 ed high differentiative ability for ATB from ORDs, LTBI or HC while KLF2, PTPRC, NEMF, ASUN, and ZNF2
41 quired for ATB and LTBI differentiation from ORDs or HC respectively and demonstrate the feasibility
42                                 Furthermore, ORD@SHp@ANG NPs showed excellent biocompatibility.
43 e with lower droplet sizes and showed higher ORD solubility.
44 well as the paucity of information about how ORD would be absorbed into policy or political parts of
45                                           If ORD activity is more severely disrupted, achiasmate chro
46  the morphology of melanopsin-immunopositive ORDs in the OPL at different developmental time points i
47                                            L-ORD shows striking phenotypic similarities to age-relate
48 ved serine to arginine (Ser163Arg) in 7/14 L-ORD families and 0/1000 control individuals.
49 rotein 5 (C1QTNF5) has been shown to cause L-ORD in a subset of affected families.
50  of 29 patients with molecularly confirmed L-ORD were included in this prospective study.
51           Late-onset retinal degeneration (L-ORD) is a rare autosomal dominant retinal dystrophy, cha
52           Late-onset retinal degeneration (L-ORD) is an autosomal dominant disorder with striking cli
53   Late-onset retinal macular degeneration (L-ORD) is an autosomal dominant inherited disorder caused
54  dominant late-onset retinal degeneration (L-ORD), a retinopathy that becomes symptomatic after age 5
55 on (AMD), late-onset retinal degeneration (L-ORD), and choroideremia (CHM).
56 (LAZ) and late-onset retinal degeneration (L-ORD).
57 uggests that the heterozygous mutations in L-ORD show a dominant negative, rather than a haploinsuffi
58  also has a peripheral retinal location in L-ORD.
59 ls (ages 35-60) at a 50:50 risk to inherit L-ORD were also studied with dark adaptometry.
60 rotein may underlie the pathophysiology of L-ORD and LAZ.
61  precede symptoms and funduscopic signs of L-ORD by at least a decade.
62 new insight into the pathogenetic basis of L-ORD has implications for future therapeutic strategies s
63               Early phenotypic features of L-ORD include: dark adaptation abnormalities, nyctalopia,
64                          Here we show that L-ORD is genetically heterogeneous and that a proposed fou
65 dditional power for genetic mapping of the L-ORD locus.
66 rusen is found frequently in patients with L-ORD and at a younger age than in individuals with age-re
67                        Three families with L-ORD were included; two families had postmortem eye donor
68 germ-line mitotic divisions in flies lacking ORD activity.
69 esion is severely disrupted in flies lacking ORD protein.
70 on directly, we confirm that oocytes lacking ORD activity exhibit cohesion defects, consistent with p
71 gy and successfully prepared multifunctional ORD@SHp@ANG nanoparticles (NPs) that consist of a stroke
72      Our results suggest that association of ORD with meiotic chromosomes during mid to late G2 is re
73 ions demonstrate that the C-terminal half of ORD is essential for sister chromatid cohesion and sugge
74                The analytical performance of ORD, LCOR, and their coupling resulted from the CSI simu
75 tion provided a significantly higher rate of ORD release (98.94 +/- 0.68 in 1.0 h) compared to API.
76  prophase condensation and that retention of ORD at the centromeres after condensation ensures the ma
77                    Equilibrium solubility of ORD in the anhydrous and diluted SNEDDS was conducted to
78 elf-nanoemulsifying drug delivery systems of ORD were prepared using various oils, non-ionic surfacta
79         While it is known that the number of ORDs in the retina is developmentally regulated, little
80 he mouse retina and identified five types of ORDs originating from either M1 or M1d cells.
81 mers with very similar mass spectra based on ORD, LCOR, and their coupling were much more efficient,
82 eport here a new strategy based on ordering (ORD), linear correlation (LCOR) algorithms, and their co
83 ug delivery systems (SNEDDSs) of Ornidazole (ORD), a poorly water-soluble drug.
84   Significantly, targeting the ORP5 and ORP8 ORDs to the STIM1 ER subdomain reversed their function.
85 eak mutation in the meiotic cohesion protein ORD coupled with a reduction in centromere-proximal hete
86 tiation sites depend on the cohesion protein ORD.
87                Brain deiodinase activity (T4-ORD) was reduced by approximately 65% at both doses.
88 T3), and hepatic outer ring deiodination (T4-ORD).
89 T3 and TT4, thyroid histology and hepatic T4-ORD were determined at the final 18 day exposure.
90  no effect on plasma FT3, TT3, or hepatic T4-ORD.
91 nerally longer and more highly branched than ORDs from conventional M1 cells.
92 characterised by higher gene expression than ORDs, while LTBI was associated with lower gene expressi
93                             We conclude that ORD activity suppresses sister chromatid exchange and st
94                     We provide evidence that ORD is concentrated within the extrachromosomal domains
95 observed for ord null flies, indicating that ORD function is not essential for cohesion during somati
96                     Sequencing revealed that ORD@SHp@ANG NPs promote cell proliferation, activate imm
97                                 We show that ORD protein localizes along oocyte chromosomes during th
98                      The results showed that ORD@SHp@ANG NPs can effective at scavenging ROS, and pro
99 analysis of weaker ord alleles suggests that ORD is required for proper centromeric cohesion after ar
100                                          The ORD schema supports conventional and emerging technologi
101 e severity with respect to the length of the ORD in humans.
102 On the other hand, TDDFT calculations of the ORD of (1R,5S,8S,9S,10S)-1 and -2 over the range of 365-
103 lish a system to study the properties of the ORD of PrP both in vivo and in vitro.
104                              We replaced the ORD of the yeast prion protein Sup35p with that from wil
105                                   Therefore, ORD activity appears to promote centromeric cohesion dur
106     However, a pattern emerges within these: ORDs from M1d cells are generally longer and more highly
107 g the ORP5 and ORP8 and their lipid transfer ORD domains to PM subdomains revealed that ORP5 sets low
108 ocytes shortly after the time when wild-type ORD associates with the chromosomes.
109                   The simulation of CSI with ORD, LCOR, and their coupling of six TeCBs (IUPAC no.

 
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