ライフサイエンスコーパス: PBL

1 PBL accounts for 0.4% of breast malignancies and 2% of e

2 PBL CD4+CD25+high percentages were lower, but DC2:DC1 va

3 PBL DNA methylation and concentrations of plasma folate,

4 PBL gene expression was assessed using Affymetrix microa

5 PBL is enriched in the Tm cDNA of these Igs.

6 PBLs from normal donors showed that HLA-A and HLA-B prot

7 PBLs from patients with symptomatic knee OA display a ch

8 PBLs were incubated with (activated) or without (control

9 among participants with PVL >= 1000 per 10 5 PBL were 7.08 (2.67, 18.74; p<0.001), 9.81 (3.52, 27.35;

10 e responses of 3,000 to 3,500 ELISPOTS/10(6) PBL were detected in macaques that were primed with rMuV

11 ng interactions, increasing the likelihood a PBL would remain interacting with the surface.

12 both in vitro using PHA plus IL-2 activated PBL and in vivo using the human PBL-SCID mouse.

13 lectively labeled noxious-stimulus-activated PBL neurons and performed comprehensive anatomical input

14 CC is the ratio of PCC activity in activated PBLs to that in control PBLs.

15 8 T cells exceeding a threshold of 1% of all PBLs.

16 Using IFN-gamma and IL-5 ELISPOT assays and PBL from patients with NY-ESO-1-expressing tumors, we ob

17 d MHC-matched clonal T cells (G14D-CCV), and PBL were collected at various times after immunization f

18 Three cell lines (MJ, Hut78, and HH) and PBL from 11 patients and three healthy donors were treat

19 ed with normal tissue T-cell infiltrates and PBL, and PD-1 expression correlated with an exhausted ph

20 Strikingly, DP8alpha LPL and PBL exhibited a highly skewed repertoire toward the reco

21 The SIM and PBL groups had similar mean (PBL 0.44, SIM 0.47, p = .64

22 list scores and their change for the SIM and PBL groups were compared using the Student's t-test.

23 L was impaired compared with PD-1(-) TIL and PBL.

24 -) TIL and T cells in the normal tissues and PBL.

25 derived from the lymphoid kidney tissues and PBL.

26 popolysaccharide in synovial fibroblasts and PBLs.

27 The impact of TBI on LTs and PBLs is discordant, in which as few as 32.4% of CD4(+) c

28 gp52 and further examined tissue samples and PBLs for specific MMTV genome sequences.

29 microRNAs (miRNAs) using colonic tissues and PBLs from animals having either mild inflammation or sev

30 of tumor histologies will receive autologous PBLs that have been transduced with this optimized anti-

31 beta cell autoimmunity was detected in both PBL and islets in NOD female mice.

32 90) stimulated epitope-specific CTLs in both PBLs and spleen cells of transgenic rabbits.

33 ponse XlAID was also noticeably expressed by PBLs, suggesting that XlAID remains active in a subset o

34 from MAGE-A2 and MAGE-A6) were recognized by PBLs engineered to express this TCR.

35 Notably, the VLA-1+ cells in fresh CD4+ PBLs are composed of resting CD45RO+/RA-, CCR7-, CD62L+,

36 all fraction of VLA-1+ cells present in CD4+ PBLs prior to stimulation significantly abrogated the pr

37 kewise expressed on all IL-17-producing CD8+ PBL.

38 )-treated peripheral blood mononuclear cell (PBL) cultures from healthy volunteers were stimulated wi

39 Telomere length in prospectively collected PBLs was measured in incident melanoma cases and age-mat

40 ctivity in activated PBLs to that in control PBLs.

41 otal RNA was isolated from monocyte-depleted PBL and analyzed using cDNA microarrays containing probe

42 st overall agreement for the fully developed PBL had R(2) = 0.72 and a bias of only 0.128 km.

43 m normal lymph nodes as well as normal donor PBL, regardless of very robust stimulation of the target

44 Furthermore, the frequencies of DP8alpha PBL and colonic LPL were lower in patients with IBD than

45 Adoptive immunotherapy using TCR-engineered PBLs against melanocyte differentiation Ags mediates obj

46 e underlying mechanisms to be the following: PBLs up-regulated the protein levels of the class A and

47 (original R(2) between tissues) to 0.89 for PBL-to-artery prediction; from 0.39 to 0.95 for PBL-to-a

48 -to-artery prediction; from 0.39 to 0.95 for PBL-to-atrium; and from 0.81 to 0.98 for lymphoblastoid

49 and diverse TCR repertoire was detected for PBL-derived g7-mBDC(+) T cells.

50 l loss of transgene expression was noted for PBLs transduced with this 4D5 CAR.

51 ted EM TIL clones, when reexpressed in fresh PBL, recognized an MHC-class II or MHC-class I-restricte

52 d to identify or isolate CD4(+) T cells from PBL and the islets of NOD mice.

53 site as oligoclonal TCRs were distinct from PBL TCRs from the same patient.

54 ing beta cell-specific T cells isolated from PBL, but whether these T cells accurately reflect the re

55 et-infiltrating T cells can be obtained from PBL.

56 ited specific CD4(+) and CD8(+) T cells from PBLs of cancer patients.

57 DNA and RNA were extracted from PBLs collected 20 to 28 h post-drug infusion.

58 LAGE-1-specific CD4(+) T cells isolated from PBLs of patients with advanced LAGE-1(+)/NY-ESO-1(+) mel

59 stained the same leukocyte populations from PBLs.

60 No virus was recovered from PBLs.

61 Genomic PBL DNA methylation was measured by using a [(3)H]-methy

62 osure was positively associated with genomic PBL DNA methylation in a dose-dependent manner.

63 hu PBL-SCID mice injected with A/A genotype PBLs resulted in reduced LPD development and expanded hu

64 However, PBL can also be seen in patients with other immunodefici

65 mportantly, neutralization of TGF-beta in hu PBL-SCID mice injected with A/A genotype PBLs resulted i

66 s producing rapid, high-penetrance LPD in hu PBL-SCID mice, compared to PBLs producing late, low-pene

67 ons of human peripheral blood leukocytes (hu PBL-SCID [Severe Combined Immunodeficient] mice) to test

68 ge compared to control cells ex vivo In a hu-PBL mouse study, GPI-scFv X5-transduced CD4 T cells were

69 After immunization, hu-PBL-SCID mice (n = 8 in each group) were xenografted wit

70 RNA copy numbers relative to CD4 cells in hu-PBL mice compared to mice with GPI-scFv AB65-transduced

71 induce virus-specific immune responses in hu-PBL-NOD/SCID mice.

72 ization inhibited breast cancer growth in hu-PBL-SCID mice.

73 In HIV-infected Hu-PBL mice, treatment with anti-CCR5 (viral coreceptor) an

74 nized [human peripheral blood lymphocyte (Hu-PBL)] mice by completely suppressing viral loads and pre

75 ice reconstituted with human lymphocytes (Hu-PBL) or CD34+ hematopoietic stem cells (Hu-HSC).

76 with human peripheral blood lymphocytes (hu-PBL-NOD/SCID mice).

77 ts of human peripheral-blood lymphocytes (hu-PBL-SCID).

78 Treatment of hu-PBL-SCID mice with IL-15 resulted in rapid fatality, lym

79 V-1 gp120 and were then injected into the hu-PBL-NOD/SCID mice.

80 Here we use the hu-PBL-SCID mouse model of Epstein-Barr virus (EBV)-associa

81 tion ex vivo In a preclinical study using hu-PBL mice, we show that CD4 T cells were protected and th

82 on VLA-4-mediated lymphocyte adhesion, human PBL were flowed over VCAM-1 substrates in a parallel pla

83 r expression in a T cell hybridoma and human PBL.

84 fic CAR for expression and function in human PBL, these gene-modified T cells secreted cytokines, wer

85 model of vaginal HIV-1 transmission in human PBL-SCID mice, anti-murine ICAM-1 Abs (0.4 microg/10 mic

86 CD3/CD28 costimulation of human PBL elevated cytoplasmic and mitochondrial Ca(2+) levels

87 -2 activated PBL and in vivo using the human PBL-SCID mouse.

88 etic SCID mice were reconstituted with human PBL and a focal encephalitis induced by intracranial inj

89 Human PBLs expressing the 4D5 CAR demonstrated Ag-specific act

90 Expression of both TCRs in human PBLs demonstrated Ag-specific reactivity against a range

91 mple microfluidic device, we show that human PBLs migrate toward the cathode in physiologically relev

92 und to be differentially expressed in immune PBLs.

93 cell Ag immunization expanded immunodominant PBL clonotypes present in the islets and PLN.

94 More importantly, PBLs expressing this new version of the 4D5 CAR could no

95 In PBL, the imprinted CpG island (P1) is differentially met

96 rated that the same repertoire of T cells in PBL was detected in the islets and PLN, although the fre

97 In contrast to T cells in PBL, however, the majority of islet g7-mBDC(+) T cells e

98 fat) were correlated with LOI expression in PBL.

99 ere prevalent in the islets but not found in PBL were also detected.

100 3.6) increased risk of having LOI of IGF2 in PBL compared with those without CRN.

101 lls was examined at the single-cell level in PBL, pancreatic lymph nodes (PLN), and the islets of ind

102 es of differentiation (with complete loss in PBL).

103 tive atrial fibrillation with methylation in PBL, atrium and artery.

104 unlike IL-10(-/-) mice, changes in miRNAs in PBL of dextran sulfate sodium-treated mice were minimal

105 n of CD4 and up-regulation of HRES-1/Rab4 in PBL.

106 e of Ca-independent inducible NO synthase in PBL.

107 were significantly higher in marrow than in PBL, supporting our previous reports of ex vivo DBMC imm

108 In PBLs purified from multiple donors, direct inhibition of

109 In PBLs, the block occurs at the level of reverse transcrip

110 s) from SS patients and its total absence in PBLs from patients with mycosis fungoides, inflammatory

111 chondrial dysfunction and mTOR activation in PBLs relative to the Systemic Lupus Erythematosus Diseas

112 reactive tumor Ag-specific CD4(+) T cells in PBLs of cancer patients required the presence of tumor A

113 TGF-beta) inhibited restimulation of CTLs in PBLs with adenosine at IFNG base + 874, but not in PBLs

114 expression of dopamine receptor D3 (D3R) in PBLs from PD patients has been correlated with disease s

115 itive patients, they were only detectable in PBLs following in vitro stimulation.

116 CD4+CD25- T cells and were not detectable in PBLs of other melanoma patients and of healthy donors, s

117 nses to the tetanus peptide were detected in PBLs after vaccination and correlated with T-cell reacti

118 of these miRNAs plus miR-375 was elevated in PBLs of IL-10(-/-) mice at a time when colonic inflammat

119 rosclerosis indicate that CD69 expression in PBLs inversely correlates with the presence of disease.

120 ERCC1 mRNA expression in PBLs was not associated with any clinical end point meas

121 A adducts, but not ERCC1 mRNA expression, in PBLs was associated with better survival, but was not an

122 analyzed the kinetics of gamma-H2AX foci in PBLs of 11 patients undergoing PRRT.

123 ssociated with global DNA hypomethylation in PBLs.

124 lymphocytic leukemia (CLL), the increment in PBLs is slower than the expected increment calculated fr

125 of the trout C5a receptor (TC5aR) message in PBLs and kidney.

126 ith adenosine at IFNG base + 874, but not in PBLs homozygous for thymidine.

127 Increased Fli-1 mRNA is present in PBLs from systemic lupus erythematosus patients, and tra

128 ma) gene was significantly more prevalent in PBLs producing rapid, high-penetrance LPD in hu PBL-SCID

129 l variables, and gene expression profiles in PBLs associated to activation (CD4, CD8, CD25, CD62L and

130 dence for functional PSGL-1 up-regulation in PBLs during acute inflammation.

131 in synthesis between uninfected and infected PBL.

132 Since conditioned medium from HIV-infected PBL also increased fibronectin promoter activity, we hyp

133 in fibroblasts cocultured with HIV-infected PBL by real-time PCR.

134 We confirmed that HIV-infected PBL produced increased TGFbeta1 by ELISA.

135 Furthermore, when HIV-infected PBL were added to reporter fibroblasts stably transfecte

136 retreatment of supernatant from HIV-infected PBL with a neutralizing Ab to TGFbeta1 abrogated the inc

137 on fibroblasts cocultured with HIV-infected PBL.

138 tive TGFbeta in supernatants of HIV-infected PBL.

139 of virus from extraocular sites via infected PBLs.

140 Ag specificity by transfer of TCR genes into PBLs is an attractive method to generate large numbers o

141 usion of EBV-transformed or freshly isolated PBLs from six PV patients with mouse myeloma cells.

142 ir temperature and planetary boundary layer (PBL) height.

143 Simulation of the planetary boundary layer (PBL) is key for forecasting air quality and estimating g

144 ulate in a shallow planetary boundary layer (PBL).

145 ng the atmospheric planetary boundary layer (PBL).

146 Piper betel leaf (PBL) has the biological capabilities of detoxification a

147 grated group-centred problem-based learning (PBL) using real clinical case studies worked on during e

148 tivity of the extract of Piper betel leaves (PBLs) on the basis of Cu(2+)-mediated oxidation, and its

149 noted in human peripheral blood leucocytes (PBL).

150 pomethylation of peripheral blood leukocyte (PBL) DNA in Bangladeshi adults who are chronically expos

151 lockade, a large peripheral blood leukocyte (PBL) population showed elevated PSGL-1, which could acco

152 of ASM in total peripheral-blood leukocytes (PBL) and buccal cells from a series of monozygotic twin

153 d directly with peripheral blood leukocytes (PBL) and enhanced the respiratory burst, acid phosphatas

154 easured in both peripheral blood leukocytes (PBL) and lymphoblastoid cell lines; and a study of posto

155 e expression in peripheral blood leukocytes (PBL) from normal individuals sampled multiple times over

156 omere length in peripheral blood leukocytes (PBL) was associated with a decreased risk of cutaneous m

157 1 (ERCC1) from peripheral blood leukocytes (PBL) were associated with clinical outcome in women with

158 methylation of peripheral blood leukocytes (PBLs) and the relation to plasma EPA and DHA concentrati

159 termination and peripheral blood leukocytes (PBLs) isolation.

160 to splenocytes, peripheral blood leukocytes (PBLs) represent a means to bridge vaccine efficacy in an

161 ondrocytes, and peripheral blood leukocytes (PBLs) was analyzed by semiquantitative and real-time pol

162 injected eyes, peripheral blood leukocytes (PBLs), and extraocular tissues by plaque assay and by st

163 mographic imaging of periodontal bone level (PBL) loss and histomorphometry for inflammatory cell inf

164 y (GWAS) on a set of 179 pre-breeding lines (PBLs).

165 e sequencing in peripheral blood lymphocyte (PBL) and hair follicle DNA from two Caucasian adults.

166 and 3) GalT-KO peripheral blood lymphocytes (PBL) and cultured endothelial cells.

167 Ispot assay in peripheral blood lymphocytes (PBL) and in a lymph node draining a vaccination site (se

168 eshly isolated peripheral blood lymphocytes (PBL) from CTCL patients with high percentage of circulat

169 ytes (LPL) and peripheral blood lymphocytes (PBL) from healthy individuals, and those with colon canc

170 eshly isolated peripheral blood lymphocytes (PBL) from SS patients with circulating atypical T cells

171 IGF2 on normal peripheral blood lymphocytes (PBL) of individuals.

172 esh RCC TIL to peripheral blood lymphocytes (PBL) or melanoma TIL.

173 ith autologous peripheral blood lymphocytes (PBL), genetically modified to express human leukocyte an

174 LISPOTS)/10(6) peripheral blood lymphocytes (PBL).

175 tissues and peripheral blood T lymphocytes (PBL).

176 ls), and fresh peripheral blood lymphocytes (PBLs) (10 PBC and 10 controls).

177 in samples of peripheral blood lymphocytes (PBLs) (n = 60) by competitive PCR (cPCR).

178 1 infection in peripheral blood lymphocytes (PBLs) and a B lymphocytic cell line (B-LCL).

179 ville, CA), in peripheral-blood lymphocytes (PBLs) and in a lymph node draining a vaccine site (senti

180 (HA-dnCDK9) in peripheral blood lymphocytes (PBLs) and other cells.

181 ERK (pERK) in peripheral-blood lymphocytes (PBLs) before and after 1 month of treatment.

182 CD8+) in human peripheral blood lymphocytes (PBLs) can respond directly to beta-agonist, with effects

183 ction of human peripheral blood lymphocytes (PBLs) derived from healthy donors as well as chronically

184 f T-plastin in peripheral blood lymphocytes (PBLs) from SS patients and its total absence in PBLs fro

185 C) activity in peripheral blood lymphocytes (PBLs) is a sensitive indicator of biotin status.

186 In the peripheral blood lymphocytes (PBLs) of a separate group of HAA patients prior to treat

187 e performed on peripheral blood lymphocytes (PBLs) vs. marrow in normal laboratory volunteers of CD4+

188 cells, primary peripheral blood lymphocytes (PBLs) were nucleofected with SB vectors carrying a DsRed

189 gainst patient peripheral blood lymphocytes (PBLs) with those detected in vivo posttransplantation.

190 ver, for human peripheral blood lymphocytes (PBLs), this is generally regarded as a technically deman

191 s derived from peripheral blood lymphocytes (PBLs), tumor-infiltrating lymphocytes (TILs), and tumor-

192 nduced DSBs in peripheral blood lymphocytes (PBLs).

193 ein) and fresh peripheral blood lymphocytes (PBLs).

194 recovery, peripheral blood (PB) lymphocytes (PBLs) have been used for analysis, but they represent <2

195 Plasmablastic lymphoma (PBL) is an aggressive lymphoma commonly associated with

196 markers in DLBCL and plasmablastic lymphoma (PBL).

197 sponsible for the blocks present in marmoset PBLs and B-LCLs are different.

198 The SIM and PBL groups had similar mean (PBL 0.44, SIM 0.47, p = .64) initial assessment scores (

199 the PBL group on the final assessment (mean, PBL 0.53, SIM 0.72, p < .0001).

200 or found on approximately 50% of CD4+ memory PBL.

201 Moreover, PBL and LPL from most patients with active IBD failed to

202 nally, expression of selected genes in mouse PBLs obtained shortly after vaccination, without ex vivo

203 Our studies demonstrate that murine PBLs can be used productively to identify potential corr

204 T-plastin synthesis was induced in negative PBLs from SS patients, other studied patients, and healt

205 expression was inducible, T-plastin-negative PBLs were stimulated by phorbol 12-myristate 13-acetate

206 -specific TCRs can be transduced into normal PBLs, which persist after transfer in approximately 30%

207 restingly, the lateral parabrachial nucleus (PBL), a critical node in the affective pain circuit, is

208 nd the pontine lateral parabrachial nucleus (PBL; an important component of ascending viscerosensenso

209 or down-regulated (>/=1.5-fold change) in OA PBLs, at a false discovery rate of 5%.

210 he melanoma peptides were observed in 37% of PBL and 38% of SINs in group 1, and in 53% of PBL and 83

211 BL and 38% of SINs in group 1, and in 53% of PBL and 83% of SINs in group 2.

212 Chemokine activation of PBL firm adhesion on VCAM-1 depended on induction of hig

213 ploited in the LuTate PRRT as a biomarker of PBL cytotoxicity.

214 sults in (3) further dimming and decrease of PBL height, and thus further depressing of aerosol and w

215 measure the variations in gene expression of PBL from different individuals in response to environmen

216 chemotactic activity, and gene expression of PBL.

217 e to tumor and bone marrow and the extent of PBL reduction.

218 tabilized HIV and increased HIV infection of PBL.

219 ositively associated with the methylation of PBL DNA (P = 0.009, 0.03, and 0.03, respectively).

220 between arsenic exposure and methylation of PBL DNA were restricted to persons with folate concentra

221 lized SDF-1alpha also increased the ratio of PBL firm adhesion to rolling.

222 the first long-term and continuous study of PBL heights (PBLHs) in Boston, MA, using a compact lidar

223 Following transduction of PBL, specific reactivity was confirmed by cytokine produ

224 A standard consensus treatment of PBL is not available.

225 ectomy offers no benefit in the treatment of PBL.

226 to melanoma peptides were observed in 42% of PBLs and 80% of SINs, but in patients vaccinated with DC

227 ces activation of caspase 8 and apoptosis of PBLs and Jurkat cells, which are partially blocked by ad

228 portantly, these Abs inhibited chemotaxis of PBLs toward a chemoattractant fraction purified from com

229 s both ex vivo and after in vitro culture of PBLs </=20 y after diagnosis.

230 The in vitro functions of PBLs, particularly the control of intramacrophage LVS re

231 starting from a mixed naive T/T(SCM) pool of PBLs.

232 lotype, cannot be detected on the surface of PBLs using the KIR3DL1-specific Ab DX9.

233 R is an acceptable alternative to testing of PBLs by cPCR for quantifying EBV DNA load.

234 We report that treatment of PBLs with two mitogens, PHA and PMA, results in accumula

235 Similar studies were performed on PBL of the majority of the 10-year patients remaining wi

236 ls derived from either normal lymph nodes or PBL are not capable of suppressing allogeneic CD8+CD25-

237 tudents were randomized to either the SIM or PBL group.

238 apidly internalized by B cells, T2 cells, or PBLs and submitted to cellular proteolytic action to yie

239 for MMTV was found in the liver specimens or PBLs.

240 lta isoform is enhanced in human SLE patient PBLs.

241 osis in three MF/SS cell lines and patients' PBL compared to vehicle controls.

242 8 h also induced more apoptosis of patients' PBL than healthy donors' (15%-32%versus 6%-13%, p < 0.05

243 In the induction phase, PBL and inflammatory cell infiltration were significantl

244 In the recovery phase, PBL and inflammatory cell infiltration were significantl

245 patients with newly diagnosed pineoblastoma (PBL).

246 confirmed by RNA electroporation of primary PBL.

247 ediate stable gene transfer in human primary PBLs, which may be advantageous for T-cell-based gene th

248 ivation markers and DNA synthesis in primary PBLs at the minimal concentrations required to inhibit H

249 We reviewed all published PBL reports from June 1972 to March 2005.

250 organs than did the control groups receiving PBLs or saline.

251 erentially binds high-affinity VLA-4 reduced PBL firm adhesion to VCAM-1 by 90%.

252 mory (EM) T cells than melanoma TIL or renal PBL.

253 pses and subsequent chemotherapy resistance, PBL can represent a diagnostic and therapeutic challenge

254 A pedigree analysis of confirmed resistant PBLs revealed that Aegilops species is one of the parent

255 contributed the D genome in these resistant PBLs.

256 a 4-fold increase in the fraction of rolling PBL.

257 f water vapour is constrained by the shallow PBL, leading to an increase in relative humidity (RH); (

258 f aerosol and water vapour in a very shallow PBL.

259 The shallower PBL and accelerated cloud chemistry due to larger LWP in

260 Analyses of splenocytes, PBLs, and graft-infiltrating cells revealed increased al

261 BL group learned about dyspnea in a standard PBL format.

262 In other studies, PBLs were successfully transfected using the SB transpos

263 , bone marrow, and tumor and with subsequent PBL reduction.

264 These results demonstrate that PBL and islet CD4(+) T cells specific for a given beta c

265 Finally, we show that PBL is negative for MHC II.

266 Our data demonstrated that PBLs were able to inhibit LDL oxidation in vitro and are

267 The results suggested that PBLs activated the reverse cholesterol transport mechani

268 The PBL provides a major LPS-activated input to the BST and

269 tion between cranial sensory neurons and the PBL-nociceptive neurons.

270 CC41(+) cells magnetically isolated from the PBL specifically killed CCV-infected targets as measured

271 ted CD4(+) CD25(+) regulatory T cells in the PBL of tumor-bearing animals suggested that IFN-gamma-pr

272 with higher magnitude, than responses in the PBL.

273 easurements and evaluated simulations of the PBL from seven numerical weather prediction (NWP) model

274 Subsequently, the PBL group learned about dyspnea in a standard PBL format

275 The SIM group performed better than the PBL group on the final assessment (mean, PBL 0.53, SIM 0

276 To equalize simulator education time, the PBL group learned about acute abdominal pain on the simu

277 tly non-catecholaminergic projections to the PBL.

278 he simulator, whereas the SIM group used the PBL format.

279 of Ig secretion was only observed within the PBL.

280 fish have <5% IgM(-)/IgD(+) B cells in their PBLs, whereas in others the IgM(-)/IgD(+) B cell populat

281 oved tumor reactivity that was comparable to PBL transduced with either anti-melanoma Ag recognized b

282 to respond to F. prausnitzii in contrast to PBL and LPL from patients in remission and/or healthy do

283 Such analyses, however, are limited to PBL because T cells infiltrating the pancreatic islets a

284 etrance LPD in hu PBL-SCID mice, compared to PBLs producing late, low-penetrance LPD or no LPD.

285 ivation of this monosynaptic craniofacial-to-PBL projection induced robust escape and avoidance behav

286 0.81 to 0.98 for lymphoblastoid cell line-to-PBL based on cross-validation, and confirmed using cross

287 BL and also to increase the awareness toward PBL in the medical community.

288 transgene persistence in 4D5 CAR-transduced PBLs.

289 ockade and CD4/CD8 sorting of the transduced PBLs demonstrated that this antityrosinase TCR was CD4/C

290 sequencing data was performed on Tat-treated PBLs of seven donors using provirus-specific primers and

291 alyses indicated that pERK in post-treatment PBL specimens was associated with PFS.

292 Using PBLs from mice vaccinated with F. tularensis Live Vaccin

293 an improvement, SIM 25 percentage points vs. PBL 8 percentage points, p < .04)

294 nomewide screen for genomic regions at which PBL DNA methylation is affected by season of conception

295 I(-) DLBCL immunophenotypically overlap with PBL and demonstrate an inverse correlation between MHC I

296 ovel therapeutic approaches in patients with PBL and also to increase the awareness toward PBL in the

297 ated and aggressive lymphomas, patients with PBL for the most part have poor outcomes.

298 Twelve patients with PBL were initially treated with surgery and induction ch

299 Compared with PBLs, tumor-derived NY-ESO-1-specific CD8(+) T cells dem

300 rrelated with the age of the individual with PBLs of donors older than 60 y yielding low numbers of F