ライフサイエンスコーパス: PC

1 PC contained 55.7% of proteins with a balanced amino aci

2 PC is an orphan malignancy for which diagnostic workup a

3 PC meets FAO protein quality expectations.

4 PC occurrences (N = 36,360) were classified into one or

5 PC-like WM displayed marked global hypomethylation and s

6 PC-specific deletion of AMPKalpha or PGC1alpha causes de

7 PCs are released from insect eggs, and they induce salic

8 o acids and sphingomyelins; Lyso.PC.a.C18.0, PC.ae.C34.2, C3.DC..C4.OH, glutamine and SM.C16.1, being

9 ioleoyl-sn-glycero-3-phosphocholine (DC(18:1)PC) lipid bilayer.

10 erucoyl-sn-glycero-3-phosphocholine (DC(22:1)PC) but not in thin 1,2-dioleoyl-sn-glycero-3-phosphocho

11 Here we successfully reconstitute the PC-1/PC-2 complex in the plasma membrane of mammalian cells a

12 framework for understanding the role of PC-1/PC-2 heteromers in ADPKD and suggest new therapeutic str

13 unctional understanding of the putative PC-1/PC-2 polycystin complex is lacking due to technical hurd

14 Fortunately, two of the Ir[TpCF(3)PC)]L(2) derivatives, which were tested for their abilit

15 e-crystal X-ray structures of Re[Cl(8)TpCF(3)PC](O), Re[Cl(8)TpCH(3)PC](O), and Re[Br(8)TpFPC](O) rev

16 res of Re[Cl(8)TpCF(3)PC](O), Re[Cl(8)TpCH(3)PC](O), and Re[Br(8)TpFPC](O) revealed mild saddling for

17 lus three other defining features of layer 5 PC synaptic integration: linear frequency-current relati

18 g I (h) Simulating 1000 synchronized layer 5 PCs, we reproduced the current source density patterns e

19 lementation produced an RMSE of 18.1 days, a PC of 0.41 and the EMD ranged between -31.5 and 28.7 day

20 he CB method returned an RMSE of 7.3 days, a PC of 0.93 and the EMD ranged between -6.4 and 4.1 days

21 and hedgehog pathways improves outcomes in a PC mouse model.

22 Educating hepatology providers about PC principles and developing clear prognostic criteria f

23 he activation of protein C (PC) to activated PC (aPC) on endothelial surfaces.

24 Active PCs contain primarily C16 to C18-fatty acyl chains with

25 no-systems, comment on PC signature, address PC complexity in fluids, and outline challenges associat

26 promising drug delivery system for advanced PC.

27 s, IKBKE is a therapeutic target in advanced PC suggesting repurposing of clinically tested IKBKE inh

28 or PSMA-based alpha-particle TRT in advanced PC.

29 that IKBKE drives AR signalling in advanced PC.

30 improvement of clinical manifestations after PC clone-targeted treatment.

31 conveyed the greatest relative risk for all PC subtypes combined (RR, 2.30; 95% CI, 2.22 to 2.40), f

32 tations in the polycystin proteins, PC-1 and PC-2, result in autosomal dominant polycystic kidney dis

33 He had a history of frequent bruises and PC deficiency, treated with warfarin.

34 Microbial composition and PC function were examined in mice.

35 assessment of DOX release/uptake in CT26 and PC-3 cells revealed that the use of a high lipid:DOX rat

36 For d > 14 nm, both dark and PC become electric field (E) dependent and exhibit appro

37 three-dimensional endothelial cell (EC) and PC co-culture model, transformed PCs induced thicker cap

38 B cells and reduced GC B cell expansion and PC formation.

39 Depletion of gB- and PC-specific ABs revealed that, in particular, gB-specifi

40 hylation patterns, related to normal MBC and PC profiles, and reminiscent of other memory and PC-deri

41 rofiles, and reminiscent of other memory and PC-derived malignancies.

42 Presence of teratoma in ORCH and PC-RPLND specimens was not a prognostic factor in this l

43 known to govern progenitor proliferation and PC fate choice, and PGC1alpha, which we show controls PC

44 of IgE-producing germinal center B cells and PCs subsequent to primary and secondary infection with h

45 r role and at an earlier stage than for anti-PC.

46 Both IgM anti-PC and anti-MDA increased during the first 2 y of life,

47 d during the first 2 y of life, but IgM anti-PC in contrast to IgM anti-MDA was still significantly l

48 We hypothesize that anti-PC is developed by a combination of preprogramming and e

49 ted optimized parameters were established as PC/EF molar ratio 1/15, enzyme load 30% (w/w) and incuba

50 b removal does not influence transmission at PC-to-DCN synapses, indicating that conclusions based on

51 ligands in human prostate carcinoma bearing (PC-3) mice revealed two compounds with a biodistribution

52 lic acid proved to be the most bioaccessible PC (6359 mug/g).

53 nstrated improved efficacy in eliminating BM PCs and reducing anti-HLA Abs in chronically HLA-sensiti

54 ositively with the number of VZV-specific BM PCs.

55 hosphatidylcholine-specific phospholipase C (PC-PLC) of L. monocytogenes, is a potent inhibitor of in

56 t EPCR promotes the activation of protein C (PC) to activated PC (aPC) on endothelial surfaces.

57 study, we determined the effectiveness of C-PC as an inhibitor of osteoclast differentiation, activi

58 We found that C-PC strongly inhibited the differentiation of macrophages

59 rget miR-1291-5p in human pancreatic cancer (PC) cells.

60 wiring as a necessity for pancreatic cancer (PC) growth, invasion, and chemotherapy resistance.

61 Today, pancreatic cancer (PC) remains a major health problem in the US.

62 Pancreatic cancer (PC) remains a therapeutic challenge because of its intri

63 -based TRT of patients with prostate cancer (PC) by evaluating the matching gamma-emitting surrogate,

64 Prostate cancer (PC) care is rapidly evolving, with improved treatment op

65 N-staging of patients with prostate cancer (PC) compared with morphologic imaging (CT or MRI) and va

66 nt plays a critical role in prostate cancer (PC) development and progression.

67 ) targeting therapeutics in prostate cancer (PC) is a significant clinical problem.

68 Prostate cancer (PC) is the most frequently diagnosed cancer in North Ame

69 MA-1007 PET/CT in recurrent prostate cancer (PC) patients.

70 hemical recurrence (BCR) of prostate cancer (PC).

71 therapy in a mouse model of prostate cancer (PC).

72 ically localized, high-risk prostate cancer (PC).

73 ntation and workup of parathyroid carcinoma (PC) and determine its clinical prognostic parameters.

74 Palliative care (PC) that has evolved from a focus on end-of-life care to

75 se resulting from an underlying plasma cell (PC) dyscrasia.

76 ells fated for GC expansion and plasma cell (PC) formation, so-called positively selected GC B cells.

77 tory synapses onto cerebellar Purkinje cell (PC) dendrites and trigger distinctive responses known as

78 To analyze human Purkinje cell (PC) differentiation, we optimized a protocol to generate

79 ation deficits and defects to Purkinje cell (PC) integrity.

80 ticity at parallel fibre (PF)-Purkinje cell (PC) synapses in cerebellar cortex.

81 e morphogenesis of cotyledon pavement cells (PC) in Arabidopsis.

82 mination of antibody-secreting plasma cells (PC) with a proteasome inhibitor, bortezomib, followed by

83 at protecting and regenerating Paneth cells (PCs) and intestinal stem cells (ISCs).

84 Paneth cells (PCs) are crucial for epithelial immune defense and highl

85 and increases acid-secreting parietal cells (PCs) in mice and organoids.

86 d the quantity of VZV-specific plasma cells (PCs) and CD4 T cells in the bone marrow (BM) of healthy

87 Plasma cells (PCs) are essential for protection from infection, and at

88 ensuring its production by all plasma cells (PCs).

89 viors, whereas activation of Purkinje cells (PCs) in Rcrus1 and posterior vermis improved social pref

90 e lines labeling excitatory pyramidal cells (PCs) and inhibitory PV cells.

91 need of accurate biomarkers to characterize PC, particularly to confirm the presence of intraductal

92 al transduction requires the primary cilium (PC) where major pathway components are dynamically enric

93 While most classic PCs belong to simple globular molecular systems, a vast

94 tudy were evaluated: The Prospective Cohort (PC, n=581): Patients undergoing first for cause kidney b

95 ous quantification of 32 phenolic compounds (PCs) in winemaking products.

96 ize the production of a protein concentrate (PC) from Moringa Oleifera defatted leaves (MODL) by enzy

97 hoice, and PGC1alpha, which we show controls PC maturation after their specification.

98 The proprotein convertases (PCs) form a family of nine secretory subtilisin-like ser

99 Semiconductor III-V photonic crystal (PC) laser is regarded as a promising ultra-compact light

100 nocavities inside a planar photonic crystal (PC).

101 Plastic crystals (PCs), formed by certain types of molecules or ions with

102 transfer of this labeled methyl group to D3-PC indicated that liver PEMT activity was essentially in

103 Targeting MUC1-C decreases PC self-renewal capacity and tumorigenicity, suggesting

104 n of AMPKalpha or PGC1alpha causes defective PC maturation, which could not be rescued by metformin.

105 a new potential therapy for FRMD6 deficient PC cells.

106 providers report lack of training to deliver PC along with no clear prognostic criteria on when to in

107 ociation (CID) of the resulting demethylated PC product anions allows for the determination of the li

108 upregulation of MUC1-C in androgen-dependent PC cells suppresses androgen receptor (AR) axis signalin

109 Pten repression in rat neural crest-derived PC-12 cells, and enhanced neurite outgrowth after nerve

110 ging the entire specimen's surface to detect PC tissue at the resection margin.

111 , and 81% and 82%, respectively, to diagnose PC, and of 85% and 91%, 85% and 88%, and 86% and 93%, re

112 ke other Leishmania spp., to take up diamino-PC (PC2) for red light inactivation.

113 cholesterol, and either unsaturated dioleoyl PC (DOPC) or 1-palmitoyl 2-oleoyl PC (POPC).

114 erapy retroperitoneal lymph node dissection (PC-RPLND) specimen and assess impact on prognosis.

115 The optically pumped InAs/GaAs quantum-dot PC lasers exhibit single-mode operation with an ultra-lo

116 monly referred to as nonhospice PC (or early PC).

117 hat CMV IVIG prophylaxis moderately enhances PC-entry nAB activity in D+/R- HCT recipients.

118 .705, 0.272, 0.228, and 0.147 for MBIL, FGS, PC, and CPC respectively.

119 The first PC (eigenvalue 4.2) showed one major pattern of clinical

120 on of RNT and immunotherapy combinations for PC patients.

121 five PG molecules with a K(d) ~ 0.2 muM for PC/PG 1:1 and 0.6 muM for PC/PG 7:3 liposomes.

122 K(d) ~ 0.2 muM for PC/PG 1:1 and 0.6 muM for PC/PG 7:3 liposomes.

123 mine-N-methyltransferase (PEMT) pathways for PC synthesis were assessed from incorporations of deuter

124 cyltransferase (LPCAT) cycles FA to and from PC for modification.

125 ate the highly efficient removal of HFA from PC in castor seeds.

126 total oil contents by transferring HFA from PC to diacylglycerol.

127 that RcLPCAT reduced the removal of HFA from PC, possibly by competing with the endogenous LPCAT isoz

128 cLPCAT) would preferentially remove HFA from PC, resulting in greater incorporation onto TAG.

129 ctly involved in the liberation of HFAs from PC, but the functions of their over-expression in HFA ac

130 ly lower in HH group indicating a shift from PC to PE.

131 ses, which degrade information transfer from PCs and likely contribute to ataxia in Cacna2d2 KO mice.

132 Olive fruit PCs were positively associated with Ruminococcaceae UCG-

133 erophosphocholine plus phosphocholine (GPC + PC), metabolites that are markers of neuronal viability

134 is significantly overexpressed in high-grade PC tumors (P < 0.05) and in chemotherapy-treated patient

135 is work, we propose a novel design of a GRIN-PC in which neighboring layers are separated by partitio

136 Gradient-index phononic crystals (GRIN-PC), characterized by layers with spatially changing ref

137 Each man was evaluated for FH of hereditary PC (HPC), HBOC, and Lynch syndrome (LS) and for his own

138 To explore how PC unsaturation influences the balance between these beh

139 otent stem cell-derived Purkinje cells (hPSC-PCs) that formed synapses when cultured with mouse cereb

140 done in patient-derived xenografts and human PC organoids.

141 ed higher values for two of the three immune PCs compared to invertebrate-fed bulbuls.

142 In PC-3 tumor-bearing xenograft mice, the CC-trapping, GRPR

143 4 (CXCR4) and hedgehog pathways cooperate in PC chemoresistance via bidirectional tumor-stromal cross

144 We observed a decrease in PC/crypt and lysozyme intensity in the first week after

145 This study revealed a transient fall in PC numbers in the early post-ITx period but a permanent

146 trast, other changes, such as an increase in PC recurrent axonal collateral formation and hypertrophy

147 ospholipids bearing n-3 fatty acids (most in PC and PE) was also recorded.

148 between dormancy and disease progression in PC dyscrasias.

149 Reductions in PC and OC2 drive decreases in TOC (TOC = OC1 + OC2 + OC3

150 ide a detailed description of the changes in PCs lacking alpha2delta-2, and provide a comprehensive m

151 first-ever specific genetic manipulation in PCs in vivo, across immunoglobulin isotypes.

152 g four prostate cancer cell lines, including PC-3, VCaP, DU-145, and LNCaP, obtaining high assay repr

153 her, the study suggests that alcohol-induced PC alpha-defensin dysfunction is mediated by zinc defici

154 lear prognostic criteria on when to initiate PC.

155 is against postcontrast acute kidney injury (PC-AKI) in 2018 (ESUR 10.0).

156 ostic criteria for when and how to integrate PC on the basis of individual patient needs are the init

157 dies have provided substantial insights into PC biology, thereby further enhancing our understanding

158 t linear transfer of behavior-driven/learned PC rates concurrently with background activity adaptatio

159 f new strategies for the treatment of lethal PC.

160 he STAT3-deficient prostate cancer cell line PC-3 (PC3) along with STAT3-proficient breast cancer cel

161 (phosphatidylcholine [PC] aa C36:1 and Lyso-PC a C18:1).

162 nitine, amino acids and sphingomyelins; Lyso.PC.a.C18.0, PC.ae.C34.2, C3.DC..C4.OH, glutamine and SM.

163 /inflexible behaviors, respectively, in male PC-Tsc1 mutant mice.

164 support the survival of normal and malignant PCs in the BM niche, highlighting the diverse roles of i

165 that B/B therapy selectively depleted mature PC producing donor-specific antibodies (DSA) and reduced

166 les and positive outcomes for non-metastatic PC with androgen receptor antagonists, respectively.

167 weightings for calculation of gut microbiota PC scores at the genus level, and was examined in relati

168 iTrEnDi-modified PC ([PC(Tr)](+)) and PE ([PE(Tr)](+)) showed similar sen

169 ow a positive correlation of monounsaturated PC with hallmarks of cell proliferation and hepatic carc

170 ot label quiescent cells, a property of most PCs.

171 nscribed genes in developing postnatal mouse PCs and used metagene projection to identify the most sa

172 , which replace each lost cell with multiple PCs of lower ploidy.

173 s that nonsynaptically inhibited neighboring PCs.

174 milies is commonly referred to as nonhospice PC (or early PC).

175 key structures and processes for nonhospice PC integration would allow hepatology providers to impro

176 The fields of nonhospice PC and hepatology have ample opportunities to partner cl

177 Despite the proven benefits of nonhospice PC for other serious illnesses and cancer, there are no

178 Importantly, CLR01 and CLR05 but not PC form closed inclusion complexes with lipid head group

179 reases in TOC (TOC = OC1 + OC2 + OC3 + OC4 + PC) mass concentrations.

180 In O-ATRP, the combination of PC, implementation of continuous-flow reactors, and prom

181 trates for attachment and differentiation of PC-12 neural progenitor cells.

182 from the N-terminal extracellular domain of PC-1 functions as an intrinsic agonist that is necessary

183 cer cells and of treatment with erlotinib of PC-9 non-small cell lung cancer cells.

184 o determine the potential clinical impact of PC impairment after ITx.

185 oved cisplatin efficacy in the inhibition of PC cell viability.

186 mib, followed by the sustained inhibition of PC generation with CTLA4-Ig or belatacept (B/B).

187 Also, the investigation of PC population dynamics has mostly relied on nucleotide a

188 ion to identify the most salient patterns of PC gene expression over time.

189 We performed comprehensive quantification of PC intake, together with 16S rRNA gene sequencing of the

190 The ratio of PC/PE was clearly lower in HH group indicating a shift f

191 pulation-based, family database, the risk of PC varied by cancer FH and was most strongly associated

192 stic framework for understanding the role of PC-1/PC-2 heteromers in ADPKD and suggest new therapeuti

193 and demonstrate the molecular specificity of PC synthesis by the immature preterm liver in vivo.

194 ress was enriched in the squamous subtype of PC (P < .001) but was not associated with DDR deficiency

195 ereby further enhancing our understanding of PC populations.

196 ral origins of the unique characteristics of PCs, such as exceptional plasticity, facile reversible s

197 thod was applied for the characterization of PCs of wines, bunch stems and grape canes.

198 The finding of PCs as egg-associated molecular patterns (EAMPs) illustr

199 e and blood flow, increased proliferation of PCs and ECs, and attenuated loud sound-caused loss in en

200 Whole-cell recordings of PCs from acute cerebellar slices revealed altered climbi

201 GLP-2 substitution promoted regeneration of PCs and ISCs, which enhanced production of antimicrobial

202 trolled expression of mIgE on the surface of PCs.

203 Transplantation of PCs pretransfected with adeno-associated virus 1-vascula

204 d dioleoyl PC (DOPC) or 1-palmitoyl 2-oleoyl PC (POPC).

205 surrounding various nano-systems, comment on PC signature, address PC complexity in fluids, and outli

206 In addition, the influence on PC formation of various nanoparticle parameters is summa

207 RNA sequencing conducted on PCs demonstrated that LCDD LC induces endoplasmic reticu

208 the phosphirane P-C bonds to yield PC(4) or PC(5) heterocycles.

209 s, flavanols, flavonols, stilbenes and other PCs in the matrices under study.

210 stically improve therapeutic outcomes in our PC model, supporting the evaluation of RNT and immunothe

211 ameter (17-150 um) affect neurite outgrowth (PC-12 cells and primary mouse hippocampal neurons) and t

212 lesterol-containing asymmetric SM+PC outside/PC inside vesicles, the PC-containing inner leaflet tend

213 BOC, and Lynch syndrome (LS) and for his own PC status.

214 iTrEnDi-modified PC ([PC(Tr)](+)) and PE ([PE(Tr)](+)) showed similar sensitiv

215 PF-PC LTP is associated with an increase in the size of the

216 I controls the direction of plasticity at PF-PC synapses, and demonstrates that the binding of filame

217 the beta1AR antagonist metoprolol blocked PF-PC LTP, which was also absent in Epac2 (-/-) mice.

218 ergic receptors (betaARs) is required for PF-PC LTP, although noradrenergic varicosities are apposed

219 noradrenergic varicosities are apposed in PF-PC contacts.

220 rior activation of these betaARs occluded PF-PC LTP, while the beta1AR antagonist metoprolol blocked

221 ssion (LTD) and potentiation (LTP) at the PF-PC synapse.

222 ase in the size of the RRP contributes to PF-PC LTP.SIGNIFICANCE STATEMENT G-protein-coupled receptor

223 Phosphatidylcholine (PC), phosphatidylethanolamine (PE), cardiolipin (CL) and

224 ylceramide (GlcCer) and phosphatidylcholine (PC, or their lyso-lipid derivatives), and we find that t

225 aim was to characterize phosphatidylcholine (PC) and choline metabolism in preterm infants and demons

226 e rate-limiting step in phosphatidylcholine (PC) biosynthesis.

227 e inversion reaction of phosphatidylcholine (PC) lipid cations generated from rat brain tissue via ma

228 tion, and generation of phosphatidylcholine (PC).

229 he site of synthesis on phosphatidylcholine (PC).

230 de of PDMS-g-PEO and/or phosphatidylcholine (PC).

231 E), sphingomyelin (SM), phosphatidylcholine (PC) and their molecular species were observed following

232 composed of unsaturated phosphatidylcholine (PC) and cholesterol.

233 d glycerophospholipids (phosphatidylcholine [PC] aa C36:1 and Lyso-PC a C18:1).

234 Phosphatidylcholines (PC) and sphingomyelins (SM) were the most abundant phosp

235 mass spectrometry (21 phosphatidylcholines (PCs), 7 lysophosphatidylethanolamines, 5 ceramides, 3 br

236 the identification of phosphatidylcholines (PCs).

237 catalyzes the hydrolysis of phosphocholine (PC) to choline.

238 The photocurrent (PC) response is orders of magnitude higher than the dark

239 ng with red light excitable phthalocyanines (PC) that was cationized by chemical engineering.

240 Large polyploid PCs are outnumbered by RSCs, which replace each lost cel

241 tro and in vivo models of the GRPR-positive, PC-3 human prostate cancer cell line.

242 UBM in detecting preoperative posttraumatic PC rupture.

243 However, the predicted PC scores from SP can be biased toward NULL.

244 IgE titers and accumulation of IgE-producing PCs.

245 opportunities for therapy in DDR-proficient PC and after platinum therapy.

246 Mutations in the polycystin proteins, PC-1 and PC-2, result in autosomal dominant polycystic k

247 , a functional understanding of the putative PC-1/PC-2 polycystin complex is lacking due to technical

248 Then, lesions suspected of being recurrent PC were differentiated from lesions attributed to a beni

249 ed 102 patients with biochemically recurrent PC after radical prostatectomy undergoing (18)F-PSMA-100

250 11 PET/CT because of biochemically recurrent PC.

251 c or pharmacological PGM3 inhibition reduces PC cell growth, migration, invasion, in vivo tumor growt

252 e, provides an alternative pathway to refine PC output.SIGNIFICANCE STATEMENT The brain can adapt to

253 owever, long-lived bone marrow (BM) resident PCs (LLPCs) demonstrate therapeutic resistance, particul

254 rs could be beneficial to castrate resistant PC patients.

255 ethods: Data from 58 patients with high-risk PC (according to the D'Amico criteria) who were staged w

256 atients with clinically localized, high-risk PC at this time.

257 to reinforce the identification of high-risk PC patients and lead to more precise diagnosis of IDC-P.

258 Men with clinically localized, high-risk PC were assigned to RP alone or neoadjuvant CHT with and

259 of biopsy-proven intermediate- or high-risk PC, and 57 patients (mean age, 70.5 y old) who underwent

260 ted that miR-1291 was effective to sensitize PC cells to arginine deprivation treatment and chemother

261 Serum-PC ALA was inversely related to all-cause mortality (HR:

262 We determined serum-PC fatty acids with gas chromatography.

263 known clinical predictors showed that serum-PC EPA at the time of STEMI was inversely associated wit

264 ing with Ca2+ leads to substantially shorter PC lifetimes, analysis of the complete lifetime distribu

265 In cholesterol-containing asymmetric SM+PC outside/PC inside vesicles, the PC-containing inner l

266 stabilize ordered domain formation in the SM+PC-containing outer leaflet relative to ordered domain s

267 ility in cholesterol-containing symmetric SM/PC vesicles.

268 cts highlight the potential role of specific PCs as gut microbiome modulators in either the pathogene

269 By co-staining PC-3 cells with a Golgi staining reagent, NBD C(6) -cera

270 hosphaallenes, 10, which features a strained PC(2) heterocycle.

271 rb GlcCer transport to a greater extent than PC transport.

272 There was no clear evidence that PCs were affected throughout rejection.

273 The PC and GM groups showed favorable root development witho

274 The PC- and gB-specific ABs were assessed by enzyme-linked i

275 ine challenges associated with analysing the PC.

276 ion, Hh-independent GLI2 accumulation at the PC tip in cells from the affected individuals suggests a

277 stem reveals that the process of exiting the PC involves unidentified molecular details whose involve

278 both 50-MHz UBM and SS-OCT evaluation of the PC by different operators.

279 We also summarize the evolution of the PC surrounding various nano-systems, comment on PC signa

280 nough to prevent slit lamp evaluation of the PC, were included in a simple sequence without randomiza

281 Here we successfully reconstitute the PC-1/PC-2 complex in the plasma membrane of mammalian ce

282 was significantly higher in the CSC than the PC; time from transplant to indication biopsy was associ

283 Follow-up biopsies showed that the PC number recovered, whereas lysozyme intensity remained

284 metric SM+PC outside/PC inside vesicles, the PC-containing inner leaflet tended to destabilize ordere

285 degrees C yielded powders with similar total PC contents, but with distinct profile, the former riche

286 ll (EC) and PC co-culture model, transformed PCs induced thicker capillary-like branches in ECs and i

287 r results indicate that loud sound-triggered PC transformation contributes to capillary wall thickeni

288 adaptation and suitable for flexibly tuning PCs output via background activity modulation.

289 y an opposite leaflet containing unsaturated PC and cholesterol.

290 Here, we demonstrate ultrasmall III-V PC membrane lasers monolithically grown on CMOS-compatib

291 We show that when PC cells are co-cultured with pancreatic stellate cells

292 2), and thrombocytopenia (P = .004), whereas PC-like WM harbored more deletion 6q (P = .012), gain 6p

293 is highly expressed in human islets, whereas PCs that potentially could provide redundancy are expres

294 s the diversity of scientific areas in which PCs are gaining attention but also the opportunities one

295 bicyclic 1-benzo-dihydrophosphetes (2) with PC(3) heterocycles.

296 major challenging phenomenon associated with PC metastasis and limiting therapy efficacy.

297 phospholipids in the RBCs outer leaflet with PC 34:1 and SM 34:1 being the most abundant species.

298 ablished prognostic marker for patients with PC, it is unclear how CA19-9 monitoring should be used t

299 of one of the phosphirane P-C bonds to yield PC(4) or PC(5) heterocycles.

300 ry wall thickening and regression, and young PC transplantation effectively rehabilitates the vascula