ライフサイエンスコーパス: PTB

1 PTB and SGA may play substantial roles in the relationsh

2 PTB and SGA together contributed more to the association

3 PTB has multiple appalling sequelae, which require due a

4 PTB individuals exhibited significantly higher levels of

5 PTB individuals with bilateral or cavitary disease displ

6 PTB rates were low and not significantly different with

7 tion (OR 1.08; 95% CI 1.06-1.09; P < 0.001), PTB (1.02; 1.01-1.03; P < 0.001), macrosomia (1.07; 1.06

8 this analysis (N = 174,186; including 15,134 PTBs).

9 (RNA-seq), and DNA methylation data for 270 PTB and 521 control families.

10 median age, 2.8 [min-max 0.8-8] years) and 9 PTB controls (median age, 3.7 [min-max 1.3-11.8] years).

11 an MDD decrease of 5 cases/1000 people and a PTB decrease of 15 cases/1000 livebirths.

12 n cervical cells of women destined to have a PTB.

13 al amputees who utilized a prosthesis with a PTB feature and 15 age- and sex- matched controls partic

14 suggest that the use of a prosthesis with a PTB feature contributes to morphological changes in bila

15 between FPG levels and spontaneous abortion, PTB, macrosomia, SGA, and perinatal infant death (P for

16 cy outcomes, including spontaneous abortion, PTB, macrosomia, SGA, and perinatal infant death.

17 e culture test results, 106 cases had active PTB, the remaining cases were culture negative for PTB.

18 108 cases presented HRCT features of active PTB and the remaining cases were negative but had presen

19 Features of active PTB were centrilobular nodules, 'tree-in-bud' pattern de

20 In addition, PTB individuals with slower culture conversion displayed

21 ish an etiologic link between bile acids and PTB, and open an avenue for developing etiology-based th

22 )/IL10(-)) in the blood and lung of EPTB and PTB subjects respectively.

23 EPTB and PTB were associated with higher frequencies of CD4+T cel

24 EPTB and PTB were associated with higher frequencies of CD4+T-cel

25 to clearly differentiate LTBI from EPTB and PTB.

26 posure windows for weekly PM2.5 exposure and PTB in California using California birth cohort data fro

27 ion surveillance and prevention of LRTIs and PTB in HIV+ adolescents.

28 lung immune landscape in NHPs with LTBI and PTB using high-throughput technologies.

29 Penalized splines indicated that MDD and PTB rates decreased linearly with increasing LC omega-3

30 and its precursors may be elevating MDD and PTB rates in 85% of the countries studied.

31 3 intake would associate with higher MDD and PTB rates on the country-level.

32 sociation between the vaginal microbiota and PTB, demonstrate the benefits of high-resolution statist

33 ening, eliciting abruption-related PPROM and PTB.

34 ro studies suggested that binding of SH2 and PTB domains can enhance protein phosphorylation by prote

35 Adverse PM2.5-induced outcomes such as PTB and LBW are dependent upon the periods of maternal e

36 (P = .005) and CST-IVB (P = .018) as well as PTB (P = .049).

37 a LPS-induced model of infection-associated PTB, 101.10 prevented PTB, neonatal mortality, and fetal

38 ole IRAK1 in IUI and inflammation-associated PTB and suggest it as potential therapeutic target in IU

39 PHOSPHATE TRANSPORTER B (PTB) proteins are hypothesized to be the Na(+) /Pi sympo

40 A patellar-tendon-bearing (PTB) bar is a common design feature used in the socket o

41 Previously proposed associations between PTB and lower Lactobacillus and higher Gardnerella abund

42 discovered a unique phosphotyrosine binding (PTB) domain, namely atypical phosphotyrosine binding (aP

43 with TBC1D1 via its phosphotyrosine binding (PTB) domains and their interactions with TBC1D1 were una

44 y 2 domain (SH2) or phosphotyrosine-binding (PTB) domain deletion mutants by biolayer interferometry,

45 edominantly via its phosphotyrosine-binding (PTB) domain.

46 omology 2 (SH2) and phosphotyrosine-binding (PTB) domains, which recognize phosphotyrosine-containing

47 omology 2 (SH2) and phosphotyrosine-binding (PTB) domains.

48 ciated with increased risk of preterm birth (PTB) (31% versus 15.3%; P = .066).

49 Preterm birth (PTB) affects approximately 1 in 10 pregnancies and contr

50 Preterm birth (PTB) affects nearly 15 million infants each year.

51 iated with increased rates of preterm birth (PTB) among pregnant mothers living downwind of Los Angel

52 intervention shown to reduce preterm birth (PTB) and improve perinatal survival, but no trial eviden

53 (PM2.5) during pregnancy with preterm birth (PTB) and low birth weight (LBW) but disagree over which

54 ether and the extent to which preterm birth (PTB) and small for gestational age (SGA) at birth mediat

55 (PE), caesarean section (CS), preterm birth (PTB) and small for gestational age (SGA).

56 Preterm birth (PTB) complications are the leading cause of long-term mo

57 Preterm birth (PTB) contributes significantly to infant mortality and m

58 aded to the normal depth from preterm birth (PTB) deliveries.

59 Preterm birth (PTB) is a leading cause of neonatal death worldwide.

60 Preterm birth (PTB) is a major cause of neonatal mortality and morbidit

61 Preterm birth (PTB) is a significant global problem, but few therapeuti

62 Preterm birth (PTB) is commonly accompanied by in utero fetal inflammat

63 Preterm birth (PTB) is the leading cause of infant death and disability

64 Preterm birth (PTB) is the leading cause of neonatal morbidity and mort

65 Preterm birth (PTB) is the leading cause of neonatal mortality and morb

66 Preterm birth (PTB) is the leading cause of neonatal mortality, and sur

67 Preterm birth (PTB) is the leading cause of perinatal mortality and new

68 to understand disparities in preterm birth (PTB) prevalence between births of non-Hispanic black ind

69 pregnancy is associated with preterm birth (PTB), a leading cause of infant morbidity and mortality.

70 h potential for prediction of preterm birth (PTB), a problem affecting 15 million newborns annually.

71 depressive disorder (MDD) and preterm birth (PTB), and prenatal depression associates with PTB.

72 tBW), low birth weight (LBW), preterm birth (PTB), and small for gestational age birth (SGA).

73 cluding spontaneous abortion, preterm birth (PTB), macrosomia, small for gestational age infant (SGA)

74 Preterm birth (PTB), small for gestational age (SGA), and low birth wei

75 he risk of preeclampsia (PE), preterm birth (PTB), small for gestational age (SGA), and neonatal inte

76 y changes in the incidence of preterm birth (PTB), term low birth weight (TLBW), autism spectrum diso

77 Preterm birth (PTB), the leading cause of neonatal morbidity and mortal

78 lecular mechanisms regulating preterm birth (PTB)-associated cervical remodeling remain unclear.

79 iomarkers related to risk for preterm birth (PTB).

80 pts these pathways leading to preterm birth (PTB).

81 or gestational age (SGA), and preterm birth (PTB).

82 pregnancy sequelae, including preterm birth (PTB); yet, root planing and scaling in pregnancy has not

83 ll for gestational age (SGA), preterm birth (PTB)].In an observational study in 987 newborns from 104

84 tal POA exposure and risk for preterm birth (PTB; <37 gestational weeks) and small for gestational ag

85 rom term (n = 10), idiopathic preterm birth (PTB; n = 8), and abruption-complicated pregnancies (n =

86 tic accuracy of Xpert MTB/RIF assay for both PTB and EPTB.

87 ase observed in humans and recapitulate both PTB and LTBI.

88 f life around the Permian-Triassic boundary (PTB) remain controversial.

89 t with the marine Permian-Triassic boundary (PTB).

90 postneonatal mortality not accounted for by PTB or SGA could reflect unaddressed educational dispari

91 rtion eliminated by eliminating mediation by PTB and SGA was reported if the mortality rate ratios (M

92 eliminated (PE) by eliminating mediation by PTB and SGA.

93 ed PTB patients, microbiologically confirmed PTB cases, non-TB disease controls, and healthy controls

94 ) in identifying microbiologically confirmed PTB patients.

95 Core PTB proteins are present at the plasma membrane of the l

96 The expression of M. polymorpha core PTB proteins in the Saccharomyces cerevisiae pho2 mutant

97 , an IRAK1 inhibitor significantly decreased PTB and increased live birth in a dose-dependent manner.

98 etiology-based therapies to prevent or delay PTB.

99 bility of Th1 and Th2 cytokines to determine PTB status in AFB microscopy smear negative patients co-

100 0.82 in differentiating clinically diagnosed PTB cases from non-TB disease controls of the validation

101 lth records (EHRs) from clinically diagnosed PTB patients and non-TB disease controls in the selectio

102 764 subjects, including clinically diagnosed PTB patients, microbiologically confirmed PTB cases, non

103 in culture-negative but clinically diagnosed PTB was 37.8% and 83.8%, respectively.

104 activation markers in blood to discriminate PTB and EPTB from latent TB infection (LTBI) as well as

105 mmation is an etiological factor that drives PTB, and oxidative stress is associated with PTB.

106 o completed a standard regimen of ATT for DS-PTB and were declared cured based on a negative clinical

107 or drug-sensitive pulmonary tuberculosis (DS-PTB).

108 h classes of PTB domains, making them a dual PTB domain-containing protein.

109 identify variants associated with very early PTB (VEPTB) as well as other subcategories of disease th

110 The proportions eliminated by eliminating PTB and SGA separately were, respectively, 46% and 11% f

111 lifornia spanning 2005 to 2010 and estimated PTBs and other adverse birth outcomes for infants borne

112 < 0.001) in women subsequently experiencing PTB or cervical shortening.

113 ctive value (NPV) of Xpert MTB/RIF assay for PTB were found to be 95.5%, 96.7%, 83.8%, and 99.1% resp

114 00 mg/day for MDD and up to ~ 550 mg/day for PTB.

115 icrofluidic devices were also determined for PTB biomarkers to be in the high picomolar to low nanomo

116 biocompatibility could be more effective for PTB prevention.

117 s for women with identified risk factors for PTB.

118 ical condition and multiple risk factors for PTB.

119 included in the logistic regression fit for PTB outcome, the predictive power of discriminating betw

120 he remaining cases were culture negative for PTB.

121 P exposure to the first quartile, the OR for PTB was 1.14 (95% CI: 1.08, 1.20), adjusting for materna

122 during pregnancy were at increased risk for PTB compared with unexposed infants (6.4% exposed versus

123 ominantly Caucasian (n = 39) at low risk for PTB, the second predominantly African American and at hi

124 accurately identify women at higher risk for PTB, to promote evidenced-based decision in preterm and

125 ion, utilizing a population at high risk for PTB, we have identified a role for ADAMTS gene methylati

126 gs were investigated to predict the risk for PTB.

127 c for women considered at increased risk for PTB.

128 rising 124 patients clinically suspected for PTB with smear and culture reports, were analysed for se

129 D3 on response to antimicrobial therapy for PTB and to evaluate the influence of single-nucleotide p

130 roach to developing effective tocolytics for PTB management.

131 ent TB infection (LTBI) as well as EPTB from PTB in 270 Brazilian individuals.

132 s distinguished ATB from LTBI, and EPTB from PTB, regardless of HIV infection status.

133 ls distinguished ATB from LTBI and EPTB from PTB, regardless of HIV infection status.

134 01) cells accurately distinguished EPTB from PTB.

135 rs to distinguish ATB from LTBI or EPTB from PTB.

136 01) cells accurately distinguished EPTB from PTB.

137 xposure over the entire period of gestation, PTB risk increased by 11% (hazard ratio = 1.11, 95% conf

138 We identified PTB sequences in streptophyte genomes.

139 igher in abruption-complicated or idiopathic PTB specimens versus normal term specimens (P < 0.001).

140 (OR, 5.97; 95% CI, 3.03-12.09; P < .0001) in PTB individuals.

141 (OR, 7.57; 95% CI, 4.18-14.05; P < .0001) in PTB individuals.

142 proteinases ([MMP]-1, -2, -7, -8, and -9) in PTB individuals.

143 ease severity and higher bacterial burden in PTB.

144 isease severity and mycobacterial burdens in PTB.

145 We also examined the chemokines in PTB individuals at the end of anti-tuberculous chemother

146 ial burden and delayed culture conversion in PTB.

147 nanosuspension led to increased efficacy in PTB prevention.

148 4 (P = 0.009) median levels were elevated in PTB culture-positive (AFB microscopy smear negative) as

149 an important driver of neonatal morbidity in PTB and identify 101.10 as a safe and effective candidat

150 markers of unfavorable treatment outcomes in PTB is not known.

151 for predicting adverse treatment outcomes in PTB.

152 eta, IL-6, IL-8, and PGF2alpha, resulting in PTB and marked neonatal mortality.

153 ing discrete periods of pregnancy results in PTB and LBW.

154 ther chemokines can perform the same role in PTB is not known.

155 e TLR/IL-1 pathway, plays a critical role in PTB.

156 ions from aircraft play an etiologic role in PTBs, independent of noise and traffic-related air pollu

157 ortantly, bile acids dose-dependently induce PTB with minimal hepatotoxicity.

158 e model of intrauterine inflammation-induced PTB to determine whether Nrf2 is a modifier of susceptib

159 s for the prevention of inflammation-induced PTB.

160 Mechanistically, IRAK1 induced PTB in the mouse model of IUI by upregulating expression

161 potential therapeutic target in IUI-induced PTB.

162 PTB in a validated intrauterine LPS-induced PTB mouse model, decreased uterine proinflammatory mRNA

163 nockout mice were protected from LPS-induced PTB, which was seen in wild-type controls.

164 r receptor-bound protein 2) and the isolated PTB domain of Shc (SHC adaptor protein) to the EGF recep

165 s to identify overlaps between new and known PTB candidate gene systems.

166 y, cerclage, premature rupture of membranes, PTB, or late miscarriage; previous short cervix or short

167 e but had presented a few features mimicking PTB.

168 ent LPS doses, DDI consistently induced more PTB than SPI, and DDI showed a linear dose-response, whe

169 ise in [Ca(2+)]i Finally, ChQ prevents mouse PTBs induced by bacterial endotoxin LPS or progesterone

170 sensitivities obtained for culture-negative PTB (82.4%) and EPTB (75.0%) in HIV-positive patients si

171 ne response in AFB microscopy smear negative PTB-HIV co-infected patients.

172 .02) and SGA (aRR = 4.24, P < .001) but not PTB (aRR = 0.88, P = .87).

173 Notably, PTB-associated genes RAB31 and RBPJ were identified by a

174 tropolymerization of poly(toluidine blue O) (PTB) and glucose oxidase (GOx) with an electroanalytical

175 ay provide new insight into the aetiology of PTB and improve our ability to predict and prevent PTB.

176 ere we conduct genome-wide G x E analyses of PTB in 1,733 African-American women (698 mothers of PTB;

177 CM2 isoform proteins contain both classes of PTB domains, making them a dual PTB domain-containing pr

178 dy period and were analyzed for detection of PTB and EPTB by both Xpert MTB/RIF assay and standard co

179 were AFB smear negative in the diagnosis of PTB was good with cross validated area under the curve (

180 From 355 cases with final diagnosis of PTB, 263 (71.8%) had definite diagnosis and 92 cases had

181 ography (HRCT) modality for the diagnosis of PTB, in comparison to culture test.

182 rd conventional methods for the diagnosis of PTB.

183 , little is understood about the etiology of PTB, likely due to the multifactorial nature of the dise

184 ss socially related prenatal risk factors of PTB and impaired fetal growth.

185 Few robust genetic factors of PTB have been identified.

186 Uterine contraction is a central feature of PTB, so gaining new insights into the mechanisms of this

187 Three defining features of PTB in macaque lungs include the influx of plasmacytoid

188 could facilitate the early identification of PTB cases among suspected patients with negative M. tube

189 od in pregnancy when early identification of PTB risk allows time to deliver outcome-modifying interv

190 ow that simvastatin reduced the incidence of PTB in a validated intrauterine LPS-induced PTB mouse mo

191 essential for more successful management of PTB.

192 1,733 African-American women (698 mothers of PTB; 1,035 of term birth) from the Boston Birth Cohort.

193 loping GH but a 33% reduction in the odds of PTB.

194 tion during pregnancy postpones the onset of PTB, acts to increase the liveborn rate and survival tim

195 obiota contributes to the pathophysiology of PTB, but conflicting results in recent years have raised

196 The predicted differences in probability of PTB between black and white infants was 0.056 (95% CI: 0

197 fore 24 weeks gestation had a higher risk of PTB (24% versus 18%, p = 0.005; adjusted relative risk [

198 y before week 24 had an even greater risk of PTB (28% versus 17%, p = 0.02; with an aRR of 1.67, 95%

199 igher PDQS was associated with lower risk of PTB (RR highest compared with lowest quintile: 0.55; 95%

200 IVF increases the risk of PTB and causes epigenetic change in the placenta and fet

201 between POA use during pregnancy and risk of PTB and SGA were largely due to unmeasured confounding f

202 bstetric care for women at increased risk of PTB in an inner-city maternity service in London (UK), b

203 egnant women identified at increased risk of PTB were randomly assigned (1:1) to either midwifery con

204 gnancy, was associated with a higher risk of PTB, SGA, and NICUa.

205 m, and inflammation and an increased risk of PTB.

206 ria based on the results to rank the risk of PTB.

207 een domestic or commuting PA and the risk of PTB.

208 icantly associated with an increased risk of PTB.

209 antly associated with an increase of risk of PTB.

210 cy was inversely associated with the risk of PTB.

211 significant beneficial effect on the risk of PTB.

212 The mediating role of PTB appeared to be much stronger than that of SGA.

213 pregnancy, the pooled relative risk (RR) of PTB was 0.83 [95% confidence interval (CI) = 0.74-0.93]

214 eural, mediastinal, and vascular sequelae of PTB.

215 We conducted a study of PTB compared with term birth in two cohorts of pregnant

216 f CCL1, CCL3, CXCL1 and CXCL9 at the time of PTB diagnosis and prior to ATT.

217 tients with nontuberculous mycobacteria, old PTB scar, and immune reconstitution syndrome.

218 maternal pre-pregnancy overweight/obesity on PTB risk, with rs11161721 (PG x E=1.8 x 10(-8); empirica

219 as significantly higher in LTBI than EPTB or PTB subjects.

220 pi-PTB (RR(adj) 8.12, 95% CI [2.54-25.93], p-value 0.007),

221 lable, from which 6.7% had sPTB, 4.0% had pi-PTB and 89.3% had a term birth.

222 pontaneous (sPTB) and provider-initiated (pi-PTB) preterm birth were compared to those who had term b

223 atal outcomes, as well as late sPTB, late pi-PTB and early term neonates.

224 rtions of participants with culture-positive PTB initiated on appropriate TB treatment within 30 days

225 m), 159 (12.4%) of whom had culture-positive PTB.

226 ntaneous preterm labor (PTL), which precedes PTB.

227 ma miRNAs in the first trimester can predict PTB and cervical shortening in women at risk of preterm

228 -150-5p had the strongest ability to predict PTB (AUC = 0.8725) and cervical shortening (AUC = 0.8514

229 In hospitalized patients with presumptive PTB in a low-burden setting, NAAT can reduce AII and is

230 Among the 318 admissions with presumptive PTB, 20 (6.3%) were culture-positive for Mycobacterium t

231 stimates for omega-3 intake, MDD prevalence, PTB rate, and per capita income for 184 countries in 201

232 d improve our ability to predict and prevent PTB.

233 f infection-associated PTB, 101.10 prevented PTB, neonatal mortality, and fetal brain inflammation.

234 neurons by depleting the RNA-binding protein PTB (also known as PTBP1).

235 uish subjects with latent (LTBI), pulmonary (PTB) or extrapulmonary (EPTB) tuberculosis remains uncle

236 plementation has been demonstrated to reduce PTB rates in women with a sonographic short cervix, yet

237 esoid X receptor activation markedly reduces PTB and dramatically improves newborn survival rates.

238 Thus, simvastatin reduces PTB incidence in mice, inhibits myometrial contractions,

239 se total associations eliminated by reducing PTB and SGA together were 55% (MRRPE = 1.27, 95% CI 1.15

240 The more reliable PTB induction and uniform uterine exposure provided by t

241 sitive adult individuals with drug-sensitive PTB were recruited under the Effect of diabetes on Tuber

242 models for ligand-induced recruitment of SH2/PTB domain-containing proteins to autophosphorylation si

243 s of the adapter proteins outside of the SH2/PTB domains are important for stabilizing the binding of

244 Spontaneous PTB, resulting from preterm labor, is commonly caused by

245 expressed miRNAs associated with spontaneous PTB and/or cervical shortening (n = 16 term no short, n

246 With these data, and by stratifying PTB by subphenotype, we have identified associations bet

247 nse oligonucleotides to transiently suppress PTB.

248 Patients who had suspected PTB were enrolled into the study.

249 ove diagnosis of both EPTB and pulmonary TB (PTB) and timely initiation of anti-TB therapy.

250 isease severity in a cohort of pulmonary TB (PTB) individuals with (Ss+) or without (Ss-) seropositiv

251 n from latent (LTBI) to active pulmonary TB (PTB) remain poorly defined.

252 ove diagnosis of both EPTB and pulmonary TB (PTB), and timely initiation of anti-TB therapy.

253 for cases of culture-positive pulmonary TB (PTB; 91.3%) and extrapulmonary TB (EPTB; 92.3%), and the

254 ide further support for a role of the TBC1D1 PTB domains as a scaffold for a range of Rab regulators,

255 Our data demonstrate that PTB is associated with elevated levels of chemokines, wh

256 ditions) and climate fluctuations across the PTB.

257 ferometry, revealing the role of SH2 and the PTB domains.

258 d two Pi uptake mechanisms - mediated by the PTB and PHT1 proteins, respectively - from their strepto

259 erentially expressed in blood samples of the PTB and healthy control groups were identified by microa

260 ieved through independent mutagenesis of the PTB domain and the CH1 tyrosine residues, and successive

261 vent began at 251.941 +/- 0.037 Ma, with the PTB placed at 251.902 +/- 0.024 Ma (2sigma).

262 bile acid levels directly correlate with the PTB rates regardless of the characteristics of the subje

263 Of these PTBs, >25% are a result of inflammation or infection.

264 ctrophoresis in a 3D printed device of three PTB biomarkers, including peptides and a protein, with s

265 FB microscopy smear negative) as compared to PTB culture-negative (AFB microscopy smear negative) par

266 categories of disease that may contribute to PTB.

267 r understanding of the mechanisms leading to PTB.

268 es but have not been etiologically linked to PTB.

269 ther Nrf2 is a modifier of susceptibility to PTB and prematurity-related morbidity and mortality in t

270 ing early placentation and susceptibility to PTB.

271 actions that may determine susceptibility to PTB.

272 rt MTB/RIF assay for pulmonary tuberculosis (PTB) and extrapulmonary TB (EPTB) has not been investiga

273 als co-infected with pulmonary tuberculosis (PTB) and HIV, primarily upon antiretroviral therapy (ART

274 in the treatment of pulmonary tuberculosis (PTB) are variously limited by small sample sizes, inadeq

275 Pulmonary tuberculosis (PTB) has clinically significant sequelae, even after rec

276 nostic technique for Pulmonary Tuberculosis (PTB) in low-and-middle income countries.

277 ith culture-positive pulmonary tuberculosis (PTB) initiated on appropriate TB treatment within 30 day

278 Pulmonary tuberculosis (PTB) is one of the major health problems in the elderly

279 Previous pulmonary tuberculosis (PTB) or lower respiratory tract infection (LRTI) was sig

280 Clinically diagnosed pulmonary tuberculosis (PTB) patients lack microbiological evidence of Mycobacte

281 obacterial burden in pulmonary tuberculosis (PTB).

282 nts with presumptive pulmonary tuberculosis (PTB).

283 pinal cord); and (2) pulmonary tuberculosis (PTB).

284 (HIV) infection and pulmonary tuberculosis (PTB).

285 ulture conversion in pulmonary tuberculosis (PTB).

286 In a C57BL/6J wild-type PTB mouse model of IUI given intrauterine LPS, an IRAK1

287 e has been a strong association of IL-1 with PTB.

288 onducted a clinical trial in 390 adults with PTB in Ulaanbaatar, Mongolia, who were randomized to rec

289 to identify genomic variants associated with PTB and secondary analyses to identify variants associat

290 PDQS was inversely associated with PTB, LBW, and fetal loss, and DDS was inversely associat

291 significant genomic variants associated with PTB-related phenotypes.

292 g genes and those previously associated with PTB.

293 PTB, and oxidative stress is associated with PTB.

294 remature cervical remodeling associated with PTB.

295 t-origin UFPs was positively associated with PTB.

296 TB), and prenatal depression associates with PTB.

297 sma levels of chemokines in individuals with PTB, latent TB (LTB) or healthy controls (HC) and their

298 = -36 grams, 95% CI: -54, -17), but not with PTB (OR = 1.03, 95% CI: 0.91, 1.18).

299 y, we followed 48 HIV-positive patients with PTB from South India before and after ART initiation, ex

300 to assess associations of DDS and PDQS with PTB, SGA, LBW, and fetal loss.

301 stent with the findings from pregnant women, PTB is successfully reproduced in mice with liver injuri