ライフサイエンスコーパス: PUD

1 n of H. pylori genotype with esophagitis and PUD.

2 The associations between WBGT and PUD differed across the geographical regions of Taiwan.

3 was a reverse relationship between WBGT and PUD during the noon and working periods in eastern Taiwa

4 no significant associations between WBGT and PUD in central Taiwan.

5 The association between WBGT and PUD was examined with logistic regression analysis.

6 etween wet-bulb globe temperature (WBGT) and PUD.

7 g Chinese H. pylori, most isolates from both PUD and gastritis patients were toxigenic (35/46 and 29/

8 o evaluate select eligible patients with CHD-PUD from 2004 to 2015.

9 es reporting the relative risk of developing PUD, dyspepsia, or gastric lymphomas due to H pylori to

10 h an increased risk of peptic ulcer disease (PUD) (P = 0.003).

11 Perforated peptic ulcer disease (PUD) affects 4 million people annually worldwide, with a

12 biopsy specimens of 68 peptic ulcer disease (PUD) and 327 chronic gastritis (CG) patients with a posi

13 ldwide and can lead to peptic ulcer disease (PUD) and gastric cancer.

14 nized to contribute to peptic ulcer disease (PUD) and other gastrointestinal diseases, such as gastro

15 risk of uncomplicated peptic ulcer disease (PUD) in a cohort of new users of low-dose acetylsalicyli

16 Peptic ulcer disease (PUD) is a common and important cause of morbidity worldw

17 agnosis and treatment, peptic ulcer disease (PUD) remains a common reason for hospitalization and ope

18 Peptic ulcer disease (PUD) remains a significant yet poorly understood public

19 ther diseases, such as peptic ulcer disease (PUD), dyspepsia, and gastric lymphomas, is often overloo

20 ines for management of peptic ulcer disease (PUD), trends in physician practice and outcomes related

21 ng (UGIB) secondary to peptic ulcer disease (PUD).

22 ils is associated with peptic ulcer disease (PUD).

23 nderlying vacA gene to peptic ulcer disease (PUD).

24 flammation, leading to peptic ulcer disease (PUD).

25 e (CHD) and subsequent peptic ulcer disease (PUD).

26 n increased risk of developing CFS following PUD, especially in females and the aging population.

27 Post-GWAS analyses for PUD, GORD, IBS and IBD add insights into relationships b

28 th a reduction in repeat hospitalization for PUD or subsequent mortality, whereas counseling about th

29 Hospitalizations for PUD decreased in the United States from 1993 to 2006, su

30 he national estimate of hospitalizations for PUD decreased significantly from 222,601 in 1993 to 156,

31 omparison to 1993, patients hospitalized for PUD in 2006 more frequently had endoscopic treatment to

32 ntify 8 independent and significant loci for PUD at, or near, genes MUC1, MUC6, FUT2, PSCA, ABO, CDX2

33 rends of hospitalizations and operations for PUD in the United States (US) since 1993.

34 ease in risk of 1-year rehospitalization for PUD (adjusted OR, 0.47; 95% CI, 0.22-0.99) and risk of a

35 ntable case numbers remained substantial for PUD (134,000 [93,000-177,000]) and dyspepsia (860,000 [1

36 es, there has been a significant decrease in PUD mortality, a significant increase in the use of ther

37 polymorphic H. pylori genes are important in PUD.

38 as higher in the PUD cohort than in the non- PUD cohort (HR = 2.01, 95% CI = 1.75-2.30), with the sam

39 The sex-specific PUD cohort to the non-PUD cohort relative risk of CFS was significant in both

40 However, the overall burden of PUD in Africa, its patterns (duodenal ulcers, gastric ul

41 he analysis revealed a significant burden of PUD in Africa, with DU being more common than GU.

42 ere to be associated with the development of PUD and was a characteristic more frequently identified

43 Individuals with a history of PUD were more likely to develop uncomplicated PUD than t

44 res were used to ascertain the occurrence of PUD.

45 ies reporting the prevalence and patterns of PUD among the African population were included.

46 nalyze the pooled prevalence and patterns of PUD in Africa through a systematic review and meta-analy

47 icantly associated with a high prevalence of PUD (OR, 1.049, 95% CI, 1.003-1.097; OR, 1.047, 95% CI,

48 f 58 studies revealed a pooled prevalence of PUD in Africa at 15.2%.

49 ficantly associated with a low prevalence of PUD in northern Taiwan (odds ratio [OR], 0.960, 95% conf

50 Furthermore, the pooled prevalence of PUD was 14% before 2010 and 15% in 2011 and later.

51 ficantly associated with a low prevalence of PUD.

52 ficantly associated with a low prevalence of PUD.

53 The proportions of PUD, dyspepsia, and gastric lymphoma attributable to H p

54 The inpatient mortality rate of PUD decreased from 3.8% to 2.7% during 1993 to 2006 (P <

55 Omitting PUD and dyspepsia as outcomes in studies on H pylori scr

56 e needed to elucidate the effects of WBGT on PUD.

57 -specific estimates varied with lowest PAFs (PUD, dyspepsia, gastric lymphomas) observed in the Unite

58 gery is the preferred approach in perforated PUD, with improved outcomes compared with open technique

59 current evidence on management of perforated PUD, including management of failed repairs.

60 nagement and surgical techniques, perforated PUD continues to have a relatively high rate of morbidit

61 the understanding and treatment, perforated PUD continues to have a high rate of morbidity (48.5%) a

62 The sex-specific PUD cohort to the non-PUD cohort relative risk of CFS wa

63 e in patients with stable CHD and subsequent PUD.

64 e overall incidence of CFS was higher in the PUD cohort than in the non- PUD cohort (HR = 2.01, 95% C

65 hospitals with their first UGIB secondary to PUD from 2004-2010 were identified using administrative

66 increase the incidence of UGIB secondary to PUD.

67 UD were more likely to develop uncomplicated PUD than those without such a history (hazard ratio [HR]

68 er risk factors for developing uncomplicated PUD included smoking, stress, depression, anaemia and so

69 ase the risk of development of uncomplicated PUD in new users of low-dose ASA.

70 The crude incidence of uncomplicated PUD was 1.41 per 1000 person-years (95% confidence inter

71 -control analyses, the risk of uncomplicated PUD was associated with current use of non-steroidal ant

72 Incidence of uncomplicated PUD was calculated and a nested case-control analysis ad

73 idered to be incident cases of uncomplicated PUD.

74 of potential risk factors with uncomplicated PUD.

75 ce reaching 76.4% among those diagnosed with PUD.

76 It was used to study hospitalizations with PUD as the principal diagnosis during 1993 to 2006, incl

77 mprovement program for elderly patients with PUD resulted in increased screening for H pylori and inc

78 evelopment between patients with and without PUD.

79 t study identifying patients with or without PUD respectively by analyzing the Longitudinal Health In