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1 tion with SbGP/MPV and aster yellows (16SrI) phytoplasma.
2 ted molecular diagnostic assays for SbGP/MPV phytoplasma.
3 ts infected with a cell wall-less bacterium, phytoplasma.
4 So far, these PMUs appear to be unique to phytoplasmas.
5 ar methods to detect, identify, and classify phytoplasmas.
6 infection by Paulownia witches' broom (PaWB) phytoplasmas.
7 h focuses on the jujube witches' broom (JWB) phytoplasma and investigates the host-manipulating activ
9 and glnQ genes are syntenic between the two phytoplasmas and contain the majority of the metabolic g
12 tribution, and phylogenetic relationships of phytoplasmas, and a taxonomic system has emerged in whic
13 lant pathogens, including viruses, bacteria, phytoplasmas, and fungi depends upon the abundance and b
16 xonomic system has emerged in which distinct phytoplasmas are named as separate "Candidatus phytoplas
21 nd raise a tantalizing possibility for using phytoplasma as a tool to dissect the course of normal pl
22 s the first reported example of a pathogenic phytoplasma as the causal agent of a desirable and econo
23 ercentages of the chromosomes of 'Candidatus Phytoplasma asteris'-related strains OYM and AYWB, occup
24 sitive bacterial genome to be sequenced; and Phytoplasma asteris, the small genome that lacks importa
25 HYL1) effector of PnWB from other species of phytoplasma can trigger the proteasomal degradation of s
27 ecently begun on the phytoplasma genome, how phytoplasmas cause disease, the role of mixed phytoplasm
32 essfully differentiating it from other known phytoplasma cpn60 UT sequences, while identifying a doub
37 s studies have characterized a few different phytoplasma effector proteins that destabilize specific
38 to an improved understanding of the role of phytoplasma effector SAP11 and provide new insights for
40 ur research underscores the dual role of the phytoplasma effector SAP54 in host development alteratio
41 n interaction network between a broad set of phytoplasma effectors and a large, unbiased collection o
42 despread, but specific, interactions between phytoplasma effectors and host transcription factors, es
43 ogen protein interaction networks shows that phytoplasma effectors have unusual targets, indicating t
44 d to the hemocoel at 14-21 daas; finally, OY phytoplasmas entered into type III cells of salivary gla
46 o SVM formation occurred after divergence of phytoplasmas from acholeplasmas, triggering evolution of
48 ens in HTS data, such as bacteria (including phytoplasmas), fungi, and oomycetes, and this tool shoul
51 ytoplasma pathogenicity, organization of the phytoplasma genome, evolution of new phytoplasma strains
52 continue, research has recently begun on the phytoplasma genome, how phytoplasmas cause disease, the
53 Genome sequencing has revealed that many phytoplasma genomes appear to contain repeated genes org
55 ectors have unusual targets, indicating that phytoplasmas have evolved a unique and unusual infection
59 ansmissible agents, particularly viruses and phytoplasmas, have advanced substantially over the past
60 1 providing evidence that PMUs contribute to phytoplasma host adaptation and have integrated into the
61 igned that was capable of detecting SbGP/MPV phytoplasma in infected plant tissues, successfully diff
62 patiotemporal dynamics of onion yellows (OY) phytoplasma in its vector Macrosteles striifrons were in
65 ecture, similarly to the disease symptoms of phytoplasma-infected plants, by forming hairy roots.
69 tissues in the presence of SAP54 and during phytoplasma infection, emphasizing the importance of RAD
75 st and it was concluded that an unculturable phytoplasma is the cause of free-branching in poinsettia
76 ation of these assays revealed that SbGP/MPV phytoplasma is widely distributed in Central Mexico, wit
78 wers are more attractive for colonization by phytoplasma leafhopper vectors and this colonization pre
80 d DNA modification functions, and therefore, phytoplasmas may use different mechanisms of recombinati
81 fore, the molecular mechanisms through which phytoplasmas modulate their hosts require further invest
83 nostics did not identify an association with phytoplasma or reovirids-pathogens historically reported
84 s employ obligate pathogens such as viruses, phytoplasma, or symbiotic bacteria to intervene with phy
85 progress in understanding the mechanisms of phytoplasma pathogenicity, organization of the phytoplas
88 s to leafhopper vectors helping the obligate phytoplasmas reproduce and propagate (zombie plants).
89 avastanoi, Pantoea agglomerans, 'Candidatus' phytoplasma, rust fungi, Ustilago smuts, root knot and c
90 ytoplasmas are named as separate "Candidatus phytoplasma species." In large part, this progress has r
92 is independent of the presence of Candidatus Phytoplasma spp. and is not associated with detectable c
93 uorescence staining further revealed that OY phytoplasmas spread along the actin-based muscle fibers
95 a) virulence effector SAP11 of Aster Yellows phytoplasma strain Witches' Broom (AY-WB) binds and dest
96 s report that one PMU from the aster yellows phytoplasma strain Witches' Broom (AY-WB) can exist as b
97 hromosome and four plasmids of aster yellows phytoplasma strain witches' broom (AY-WB) were sequenced
99 liana) expressing the secreted Aster Yellows phytoplasma strain Witches' Broom protein11 shows an alt
100 ponses, we found that secreted Aster Yellows phytoplasma strain Witches' Broom protein11 suppresses s
102 of the phytoplasma genome, evolution of new phytoplasma strains and emergence of new diseases, bases
104 ectors from insect-vectored plant pathogenic phytoplasmas take control of several plant developmental
105 fraction of the potential effectors used by phytoplasmas; therefore, the molecular mechanisms throug
107 s, for the creation of variability, allowing phytoplasmas to adjust to the diverse environments of pl
108 effector protein enables obligate parasitic phytoplasmas to induce a plethora of developmental pheno