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1 sistant to infection with the rodent malaria Plasmodium chabaudi.
2 wise lethal blood-stage malaria of the genus Plasmodium chabaudi.
3 ses in mice infected with erythrocytic-stage Plasmodium chabaudi.
4 re we show that after infection of mice with Plasmodium chabaudi, a c-Kit(hi) progenitor subset posit
5 els of beta S globin are well-protected from Plasmodium chabaudi adami and partially protected agains
6                                              Plasmodium chabaudi adami causes a nonlethal infection i
7 ice were immunized with purified recombinant Plasmodium chabaudi AMA-1 (PcAMA-1) and/or the 42-kDa C-
8 ctions between two rodent malaria parasites, Plasmodium chabaudi and P. yoelii.
9 socosms'' - i.e. mice infected with malaria (Plasmodium chabaudi) and nematodes (Nippostrongylus bras
10 f infections of the rodent malaria parasite, Plasmodium chabaudi, and the red blood cells (RBCs) it t
11 latelets during the ascending parasitemia in Plasmodium chabaudi- and Plasmodium berghei-infected mic
12                       Plasmodium berghei and Plasmodium chabaudi are widely used model malaria specie
13 sed the nonlethal malaria infection model of Plasmodium chabaudi AS in combination with the gastroint
14                                              Plasmodium chabaudi AS infection during early pregnancy
15            We investigated this question for Plasmodium chabaudi AS infection in C57BL/6 mice.
16 intain long-term protective immunity against Plasmodium chabaudi AS malaria.
17  erythropoiesis, A/J mice were infected with Plasmodium chabaudi AS, treated with intravenous ferric
18 his study, the roles of IFN-gamma and TNF in Plasmodium chabaudi AS-induced fetal loss in mice were d
19 ized (gdx) C57BL/6 mice were inoculated with Plasmodium chabaudi AS-infected erythrocytes, and the re
20 ice, similar to our previous observations in Plasmodium chabaudi AS-infected mice.
21 verity and parasitemia in mice infected with Plasmodium chabaudi AS.
22 enesis of malaria during pregnancy using the Plasmodium chabaudi AS/C57BL/6 mouse model.
23 etitive suppression of an avirulent clone of Plasmodium chabaudi (AS) by a virulent clone (AJ) in imm
24  strategies for the rodent malaria parasite, Plasmodium chabaudi, as a function of the time since inf
25                            Immunization with Plasmodium chabaudi by mosquito bite under chloroquine c
26 oplast genome of the rodent malaria parasite Plasmodium chabaudi chabaudi (Pcc) isolate CB was charac
27                           When infected with Plasmodium chabaudi chabaudi AS in early gestation, seve
28                                  Blood-stage Plasmodium chabaudi chabaudi AS infection requires cell-
29 t more severe disease in a model of malaria (Plasmodium chabaudi chabaudi AS).
30 rus (HIV) transgenic mice were infected with Plasmodium chabaudi chabaudi clone AS.
31 ned "strains" of the rodent malaria parasite Plasmodium chabaudi chabaudi in mice.
32                  The rodent malaria parasite Plasmodium chabaudi chabaudi shares many features with h
33  virus Ankara vectors encoding AMA1 from the Plasmodium chabaudi chabaudi strain AS.
34 ransmission of serially blood passaged (SBP) Plasmodium chabaudi chabaudi to interrogate regulation o
35 gmodontis) and noncerebral malaria (clone AS Plasmodium chabaudi chabaudi), we quantified disease sev
36 to clear a primary erythrocytic infection of Plasmodium chabaudi chabaudi.
37  acid synthesis (FAS) in Tmem development in Plasmodium chabaudi chronic mouse malaria infection.
38 ed at peak parasitemia during infection with Plasmodium chabaudi Concentrations of the closely relate
39 hly purified antigen on the malaria parasite Plasmodium chabaudi evolving in laboratory mice.
40 with experimental data in mice infected with Plasmodium chabaudi, finding that dying mice trace a lar
41 alaria parasites, we reveal that: (i) 57% of Plasmodium chabaudi genes exhibit daily rhythms in trans
42 ng early infection with the malaria parasite Plasmodium chabaudi, implying that another MYD88-depende
43                We examine the rodent malaria Plasmodium chabaudi in laboratory mice, a parasite-host
44 ocytic stages of the rodent-malaria parasite Plasmodium chabaudi in vivo.
45          Similar to patients, infection with Plasmodium chabaudi induced acute liver damage as determ
46 f systemic inflammation, septic shock in the Plasmodium chabaudi-infected mice and the P. berghei-ind
47 these CD4 T cell responses in the spleens of Plasmodium chabaudi-infected mice.
48 h, to experimental time-series data of acute Plasmodium chabaudi infection across doses spanning seve
49 nd spleen over 12 days of erythrocytic stage Plasmodium chabaudi infection in C57BL/6 mice.
50                              An eCM model of Plasmodium chabaudi infection in mice deficient in the r
51                                       During Plasmodium chabaudi infection in mice, a population of C
52                                  Blood-stage Plasmodium chabaudi infections are suppressed by antibod
53 uppress the parasitemia of acute blood-stage Plasmodium chabaudi malaria by an antibody- or T-cell-de
54 of splenic gammadelta T cells during chronic Plasmodium chabaudi malaria.
55    We outline here the parallels between the Plasmodium chabaudi mouse model of malaria and human mal
56                                    Using the Plasmodium chabaudi mouse model of malaria and immunizat
57        Artemisinin- and artesunate-resistant Plasmodium chabaudi mutants, AS-ART and AS-ATN, were pre
58 viously that chronic exposure to blood-stage Plasmodium chabaudi offers the best protection from para
59 e now report that the time course shows that Plasmodium chabaudi parasitemia in NADPH oxidase KO (p47
60       The production of NO during descending Plasmodium chabaudi parasitemia, a period when parasites
61 s an important fitness-determining trait and Plasmodium chabaudi parasites adjust their sex allocatio
62 ng infection of the HIV-transgenic mice with PLASMODIUM: chabaudi parasites.
63 rasitic disease and lymphomagenesis, we used Plasmodium chabaudi (Pc) to produce chronic malaria infe
64 e have reported that isolated, permeabilized Plasmodium chabaudi, releases Ca(2+) upon addition of ex
65          Infection of MIF knockout mice with Plasmodium chabaudi resulted in less severe anemia, impr
66 ne expression profiles in mice infected with Plasmodium chabaudi revealed and validated similar respo
67  infection with the murine malaria parasite, Plasmodium chabaudi, shapes sickness in terms of parasit
68 o real data from mice infected with a single Plasmodium chabaudi strain, we estimate that transmissio
69 f two strains of the rodent malaria parasite Plasmodium chabaudi that differ in virulence.
70 for the merozoite surface protein (MSP)-1 of Plasmodium chabaudi to show that activated T cells gener
71 ized that an intrinsic clock in the parasite Plasmodium chabaudi underlies the 24-hour-based rhythms
72 re of the extracellular domain of a PIR from Plasmodium chabaudi We use structure-guided sequence ana
73                                        Using Plasmodium chabaudi, we show that a single malaria episo
74           Using the rodent model of malaria, Plasmodium chabaudi, we show that individual CIR protein
75 -term artificial selection for resistance in Plasmodium chabaudi, we tested resilience of WP against
76 zed red blood cells from the rodent parasite Plasmodium chabaudi with seco-cyclopropyl pyrrolo indole