ライフサイエンスコーパス: Plasmodium falciparum

1 st severe form of human malaria is caused by Plasmodium falciparum.

2 ic screen against the human malaria parasite Plasmodium falciparum.

3 is essential for the survival of blood stage Plasmodium falciparum.

4 phylaxis prior to challenge with blood stage Plasmodium falciparum.

5 inst liver and sexual transmission stages of Plasmodium falciparum.

6 genes required for apicoplast biogenesis in Plasmodium falciparum.

7 mania donovani and nanomolar potency against Plasmodium falciparum.

8 e eradication of the human malaria parasite, Plasmodium falciparum.

9 needed by the lethal human malaria parasite Plasmodium falciparum.

10 cellular development of the malaria parasite Plasmodium falciparum.

11 rythrocytes infected with the human parasite Plasmodium falciparum.

12 source of human-to-mosquito transmission of Plasmodium falciparum.

13 genetic underpinnings of drug resistance in Plasmodium falciparum.

14 itoes to transmit the human malaria parasite Plasmodium falciparum.

15 erythrocyte signaling during infection with Plasmodium falciparum.

16 24 participants were infected by bites from Plasmodium falciparum 3D7-infected mosquitoes (MB, n=12)

17 nd Jensen established a method for culturing Plasmodium falciparum, a breakthrough for malaria resear

18 In the progression of the life cycle of Plasmodium falciparum, a small proportion of asexual par

19 In the human malaria parasite Plasmodium falciparum, a unicellular eukaryotic pathogen

20 s and associated reader protein complexes of Plasmodium falciparum, a unicellular parasite causing ma

21 enomic variation and population structure of Plasmodium falciparum across Africa is necessary to sust

22 ddition to its role in erythrocyte invasion, Plasmodium falciparum actin is implicated in endocytosis

23 his approach to the malaria-causing parasite Plasmodium falciparum, an organism that has resisted con

24 ria infections, including a large cluster of Plasmodium falciparum and 3 P. knowlesi infections.

25 n-binding chromobodies as F-actin-sensors in Plasmodium falciparum and characterised in-vivo actin dy

26 rin is active against blood- and liver-stage Plasmodium falciparum and Cryptosporidium parvum in cell

27 to the AMA-1 protein of the malaria parasite Plasmodium falciparum and find evidence of epitopes amon

28 tems, in vitro culture of the human parasite Plasmodium falciparum and in vivo infections of laborato

29 of these processes in experimentally induced Plasmodium falciparum and P. vivax infection.

30 In Adama district, Ethiopia, Plasmodium falciparum and P. vivax malaria patients and

31 mammalian enzymes and equivalent activity on Plasmodium falciparum and Plasmodium vivax DHODH.

32 However, while cases of Plasmodium falciparum and Plasmodium vivax have decrease

33 Plasmodium falciparum and Plasmodium vivax infections ar

34 ne responses during natural and experimental Plasmodium falciparum and Plasmodium vivax infections as

35 free crystallographic structures of PKG from Plasmodium falciparum and Plasmodium vivax, revealing ho

36 creases were observed in the transmission of Plasmodium falciparum and Plasmodium vivax.

37 compounds that target malaria caused by both Plasmodium falciparum and Plasmodium vivax.

38 assessed against the asexual blood stages of Plasmodium falciparum and revealed cladophorols A (4) an

39 For instance, the malarial parasite Plasmodium falciparum and the Lyme disease spirochete Bo

40 d their detection in the protozoan parasites Plasmodium falciparum and Trypanosoma brucei by microsco

41 of malaria caused by the protozoan parasite Plasmodium falciparum and underscores the urgent need fo

42 ocking intervention (TBI) candidates against Plasmodium falciparum and vivax.

43 ch we demonstrate using the malaria parasite Plasmodium falciparum, and non-canonical databases, whic

44 ite stimulation, as a rosette-stimulator for Plasmodium falciparum- and P. vivax-IRBC.

45 pid diagnostic tests (RDTs) primarily detect Plasmodium falciparum antigen histidine-rich protein 2 (

46 Extreme diversity of the major Plasmodium falciparum antigen, PfEMP1, poses a barrier t

47 Vaccines based on Plasmodium falciparum apical membrane antigen 1 (AMA1) h

48 Artemisinin and partner-drug resistance in Plasmodium falciparum are major threats to malaria contr

49 he surface of sexual stages and ookinetes of Plasmodium falciparum, as a malaria transmission-blockin

50 ium vivax bench research greatly lags behind Plasmodium falciparum because of an inability to culture

51 ystems: (1) picomolar inhibition of in vitro Plasmodium falciparum blood-stage growth against multidr

52 This distinction can be achieved for Plasmodium falciparum by comparing parasite genotypes ob

53 ntibiotic actinonin kills malaria parasites (Plasmodium falciparum) by interfering with apicoplast fu

54 bia and determining transmission dynamics of Plasmodium falciparum can help targeting control interve

55 PV5 inactivation in the human malaria agent Plasmodium falciparum causes excessive multidirectional

56 Plasmodium falciparum causes the severe form of malaria

57 The Plasmodium falciparum chloroquine resistance transporter

58 ently, the moderate clinical efficacy of the Plasmodium falciparum circumsporozoite protein (CSP)-bas

59 The Plasmodium falciparum circumsporozoite protein (PfCSP) i

60 n monoclonal antibodies (mAbs) targeting the Plasmodium falciparum circumsporozoite protein (PfCSP) o

61 rs to a key target, the repeat region of the Plasmodium falciparum circumsporozoite protein (PfCSP),

62 exhibited improved in vitro potency against Plasmodium falciparum compared to the parent phenyl comp

63 nsporter PfCRT of the human malaria parasite Plasmodium falciparum confers resistance to the former f

64 The human malaria parasite, Plasmodium falciparum, contains an essential plastid cal

65 Malaria infection by Plasmodium falciparum continues to afflict millions of p

66 Malaria caused by the protozoan parasite Plasmodium falciparum continues to impose significant mo

67 tidylinositol 3-phosphate (PI(3)P) levels in Plasmodium falciparum correlate with tolerance to cellul

68 nanomolar antiplasmodial compounds against a Plasmodium falciparum CQ-resistant Dd2 strain, with exce

69 mosome 1 including pvcrt, an ortholog of the Plasmodium falciparum CQR transporter gene.

70 and massively expanded upon activation with Plasmodium falciparum culture supernatant.

71 hibit potent inhibitory activity against the Plasmodium falciparum cysteine proteases falcipain 2 and

72 Here we show that Plasmodium falciparum development is intimately but not

73 ich protein 2 (HRP2) are important tools for Plasmodium falciparum diagnosis.

74 It is now well established that Plasmodium falciparum emerged following the transmission

75 evidence suggests that the malaria parasite Plasmodium falciparum employs a broad range of mechanism

76 The malaria parasite Plasmodium falciparum encodes a cGMP-dependent protein k

77 Vaccine trials and cohort studies in Plasmodium falciparum endemic areas indicate that natura

78 th putative cyclic nucleotide binding sites, Plasmodium falciparum EPAC (PfEpac), does not play an es

79 specific member of the antigenically variant Plasmodium falciparum erythrocyte membrane protein 1 (Pf

80 Plasmodium falciparum erythrocyte-binding antigen 140 (E

81 Phosphoproteomic studies in Plasmodium falciparum erythrocytic stages and Plasmodium

82 age of the infection of the malaria parasite Plasmodium falciparum exhibits a 48-hour developmental c

83 ovo mutation events in 119 progeny from four Plasmodium falciparum experimental crosses, using long-r

84 Recent concepts suggest that both Plasmodium falciparum factors and coagulation contribute

85 The Plasmodium falciparum gametocyte surface protein, Pfs48/

86 Here we study the in vivo dynamics of Plasmodium falciparum gametocytes by establishing a fram

87 when present in infected humans, developing Plasmodium falciparum gametocytes may express proteins o

88 Plasmodium falciparum gametocytes, the sexual stage resp

89 However, with Plasmodium falciparum, gametocytes can only be detected

90 -of-synchrony with host rhythms; (iii) 6% of Plasmodium falciparum genes show 24 h rhythms in express

91 tand transmission dynamics, we characterized Plasmodium falciparum genetic diversity in Eswatini, whe

92 Plasmodium lactate dehydrogenase ( PLDH) and Plasmodium falciparum glutamate dehydrogenase ( PfGDH),

93 ) for the detection of the malaria biomarker Plasmodium falciparum glutamate dehydrogenase (PfGDH) in

94 f multiple antimalarial-resistant strains of Plasmodium falciparum (half maximal inhibitory concentra

95 Malaria caused by the apicomplexan parasite Plasmodium falciparum has served as a strong evolutionar

96 The intricate interactions the parasite Plasmodium falciparum has with its host allows it to gro

97 eased in the blood of patients infected with Plasmodium falciparum have been extensively studied, oth

98 we have utilized three targets of interest (Plasmodium falciparum, Hepatitis C virus and T-cells) to

99 We carried out a functional analysis of the Plasmodium falciparum homolog of Protein Phosphatase 1 (

100 entially expressed between the isolates lack Plasmodium falciparum homologs and are predicted to be i

101 household SEP and individual infection with Plasmodium falciparum, hookworm (Ancylostoma duodenale a

102 The Plasmodium falciparum Hsp70-x chaperone assists the heat

103 The emergence and spread of drug-resistant Plasmodium falciparum impedes global efforts to control

104 lawi, with an estimated 18-19% prevalence of Plasmodium falciparum in children 2-10 years in 2015-201

105 ed rapid diagnostic tests (RDTs) identifying Plasmodium falciparum in clinical and community settings

106 The growth of the malaria parasite Plasmodium falciparum in human blood causes all the symp

107 factors underlying the relationship between Plasmodium falciparum in pregnancy and in the community,

108 Plasmodium falciparum in pregnancy is a major cause of a

109 g in Chennai (70.8%) and Nadiad (67.9%), and Plasmodium falciparum in Rourkela (77.3%).

110 emergence and spread of multidrug-resistant Plasmodium falciparum in the Greater Mekong Subregion (G

111 f artemisinin and partner drug resistance in Plasmodium falciparum in the Greater Mekong Subregion (G

112 ost active derivative inhibits the growth of Plasmodium falciparum in vitro in the nanomolar range (I

113 he recurrent emergence of drug resistance in Plasmodium falciparum increases the urgency to genetical

114 en-label volunteer infection study using the Plasmodium falciparum induced blood-stage malaria model

115 ine the responses of the olfactory system in Plasmodium falciparum infected Anopheles gambiae, Plasmo

116 During pregnancy, Plasmodium falciparum-infected erythrocytes (IE) accumul

117 Sequestration of Plasmodium falciparum-infected erythrocytes (IEs) in the

118 dhesive proteins expressed on the surface of Plasmodium falciparum-infected erythrocytes (IEs), where

119 Plasmodium falciparum-infected erythrocytes bind to spec

120 It is expressed on the surface of Plasmodium falciparum-infected host red blood cells and

121 Microvascular lesions caused by Plasmodium falciparum-infected human erythrocytes/red bl

122 unvaccinated control participants underwent Plasmodium falciparum-infected mosquito challenge (contr

123 og of the drug chloroquine in rapidly frozen Plasmodium falciparum-infected red blood cells.

124 SMA (n = 17) and community children without Plasmodium falciparum infection (n = 12) and determined

125 l malaria is a common presentation of severe Plasmodium falciparum infection and remains an important

126 e was high; 70% (n = 1,138) had PCR-positive Plasmodium falciparum infection at least once over the c

127 Mosquito-transmitted Plasmodium falciparum infection can cause human cerebral

128 -RDT) has been developed for improved active Plasmodium falciparum infection detection.

129 Plasmodium falciparum infection during pregnancy is a ma

130 most common life-threatening complication of Plasmodium falciparum infection in African children.

131 relationship between placental pathology and Plasmodium falciparum infection in the placenta with PE

132 Upon Plasmodium falciparum infection of the red blood cells (

133 How antibodies naturally acquired during Plasmodium falciparum infection provide clinical immunit

134 Plasmodium falciparum infection was confirmed by 18S rDN

135 udy involved 226 patients with uncomplicated Plasmodium falciparum infection who had successfully com

136 parity, Plasmodium falciparum infection, and molecular insectici

137 CM), a severe encephalopathy associated with Plasmodium falciparum infection, has a 20-30% mortality

138 ortunity to study the immune response during Plasmodium falciparum infection.

139 Asymptomatic Plasmodium falciparum infections are common in Malawi; h

140 The effect of timing of exposure to first Plasmodium falciparum infections during early childhood

141 etocytes were quantified in 161 PCR-positive Plasmodium falciparum infections from a cross-sectional

142 emiology of malaria, we intensively followed Plasmodium falciparum infections in a cohort in a malari

143 xist on the incidence or duration of natural Plasmodium falciparum infections in high-transmission se

144 Plasmodium falciparum infections lead to febrile illness

145 The majority of Plasmodium falciparum infections, constituting the reser

146 We sequence 2537 Plasmodium falciparum infections, including a nationally

147 h genomic regions to characterize polyclonal Plasmodium falciparum infections.

148 nancy, mainly with respect to submicroscopic Plasmodium falciparum infections.

149 al attention is being paid to submicroscopic Plasmodium falciparum infections.

150 ythrocytic phase of the parasite life cycle, Plasmodium falciparum invades red blood cells, where it

151 Transmission of Plasmodium falciparum involves a complex process that st

152 Plasmodium falciparum is a causative agent of human mala

153 olipid biosynthesis of the malaria parasite, Plasmodium falciparum is a key process for its survival

154 ekong Subregion, where artemisinin-resistant Plasmodium falciparum is now widespread, MDA has been pr

155 The human parasite Plasmodium falciparum is responsible for the majority of

156 minant of pathogenicity in malaria caused by Plasmodium falciparum is the adhesion of parasite-infect

157 Plasmodium falciparum is the causative agent of the dead

158 Plasmodium falciparum is the most lethal of human-infect

159 ether with the spread of multidrug-resistant Plasmodium falciparum, is a major challenge for malaria

160 Plasmodium falciparum isolates (n = 914) from 2 randomiz

161 y was designed to determine the frequency of Plasmodium falciparum isolates with histidine-rich prote

162 deaths are caused by the protozoan parasite Plasmodium falciparum Its life cycle is regulated by a c

163 Numerous mutations in the Plasmodium falciparum Kelch13 (K13) protein confer resis

164 Infection with Plasmodium falciparum leads to severe malaria and death

165 the APN homologue from the malarial parasite Plasmodium falciparum M1 aminopeptidase (PfA-M1).

166 associated with Epstein-Barr virus (EBV) and Plasmodium falciparum malaria coinfections.

167 09, 687 of 2885 patients (23.8%) treated for Plasmodium falciparum malaria in clinical studies in Mya

168 s have successfully reduced the incidence of Plasmodium falciparum malaria in many areas, there has b

169 The RTS,S/AS01 vaccine against Plasmodium falciparum malaria infection completed phase

170 Best-fit models of Plasmodium falciparum malaria infection prevalence among

171 l parasite load and the clinical severity of Plasmodium falciparum malaria infections.

172 Acidosis in severe Plasmodium falciparum malaria is associated with high mo

173 Plasmodium falciparum malaria is widespread in the tropi

174 and anti-malarial resistance has enabled the Plasmodium falciparum malaria parasite to inflict high m

175 Accordingly, Plasmodium falciparum malaria parasites can be killed by

176 Transmission of Plasmodium falciparum malaria parasites occurs when noct

177 ally conducted to identify studies on severe Plasmodium falciparum malaria that included information

178 The emergence and spread of resistance in Plasmodium falciparum malaria to artemisinin combination

179 sed malaria elimination project to interrupt Plasmodium falciparum malaria transmission in a rural di

180 Zambia has experienced a dramatic decline in Plasmodium falciparum malaria transmission in the past d

181 l bloodstream infection and 35 children with Plasmodium falciparum malaria were analyzed using protei

182 A multidrug-resistant co-lineage of Plasmodium falciparum malaria, named KEL1/PLA1, spread a

183 te load in individual subjects infected with Plasmodium falciparum malaria, using only data obtained

184 endemic areas, children suffer the most from Plasmodium falciparum malaria, yet newborns and young in

185 are the population most severely affected by Plasmodium falciparum malaria.

186 re important mediators of protection against Plasmodium falciparum malaria.

187 000s have dramatically reduced the burden of Plasmodium falciparum malaria.

188 sis before, during, and after infection with Plasmodium falciparum malaria.

189 isinins have revolutionized the treatment of Plasmodium falciparum malaria; however, resistance threa

190 Reads from Plasmodium falciparum (malaria) were detected in 21 pati

191 Malaria caused by Plasmodium falciparum manifests in many organ-specific f

192 ) subclasses and C1q fixation in response to Plasmodium falciparum merozoite antigens (erythrocyte-bi

193 fic targets of functional antibodies against Plasmodium falciparum merozoites remain largely unexplor

194 being found diversely in male gametes (e.g., Plasmodium falciparum microgametocytes and human and Dro

195 Estimates of Plasmodium falciparum migration may inform strategies fo

196 donesia, we enrolled adults with symptomatic Plasmodium falciparum (n = 47 uncomplicated, n = 8 sever

197 ites with resistance-conferring mutations in Plasmodium falciparum NCR1 (PfNCR1) during selections wi

198 We assessed the impact of exposure to Plasmodium falciparum on parasite kinetics, clinical sym

199 ngenital CMV or mothers with prenatal HIV or Plasmodium falciparum or no identified infectious exposu

200 ties and further tested against P. vivax and Plasmodium falciparum (P. falciparum) NMTs.

201 Plasmodium falciparum, P. malariae and P. ovale sporozoi

202 isen through convergent de novo mutations in Plasmodium falciparum parasite populations in Southeast

203 Plasmodium falciparum parasitemia was assessed using mic

204 In pregnancy, Plasmodium falciparum parasites express the surface anti

205 A TORCA assay was developed for detection of Plasmodium falciparum parasites in blood.

206 A previous analysis of 1602 genotyped Plasmodium falciparum parasites in Kilifi, Kenya collect

207 The dry season is a major challenge for Plasmodium falciparum parasites in many malaria endemic

208 The apicoplast of Plasmodium falciparum parasites is believed to rely on t

209 ting reliable and effective immunity against Plasmodium falciparum parasites remains an elusive goal

210 Plasmodium falciparum parasites resistant to chloroquine

211 Bites of Anopheles mosquitoes transmit Plasmodium falciparum parasites that cause malaria, whic

212 The surface protein Pfs47 allows Plasmodium falciparum parasites to survive and be transm

213 modium yoelii parasites and human-infectious Plasmodium falciparum parasites.

214 In Plasmodium falciparum patients with anemia, we show that

215 Plasmodium falciparum (Pf) 4-nitrophenylphosphatase has

216 g of the protective humoral response against Plasmodium falciparum (Pf) circumsporozoite protein (PfC

217 ia vaccine inducing immune responses against Plasmodium falciparum (Pf) merozoite surface protein-3 a

218 Plasmodium falciparum (Pf) relies solely on the salvage

219 genetic deletion of the PA28 regulator from Plasmodium falciparum (Pf) renders malaria parasites mor

220 A live-attenuated Plasmodium falciparum (Pf) sporozoite (SPZ) vaccine (PfS

221 attenuated, aseptic, purified, cryopreserved Plasmodium falciparum (Pf) sporozoites (PfSPZ Vaccine) a

222 103 aseptic, purified, cryopreserved, vialed Plasmodium falciparum (Pf) strain NF54 sporozoites, PfSP

223 t cell invasion by the apicomplexan parasite Plasmodium falciparum (Pf), the causative agent of malar

224 Prx1m from the apicomplexan malaria parasite Plasmodium falciparum (Pf).

225 oinfected with different species of malaria (Plasmodium falciparum [Pf] and Plasmodium vivax [Pv]) as

226 attenuated, aseptic, purified, cryopreserved Plasmodium falciparum [Pf] sporozoites [SPZ]) has been w

227 attenuated, aseptic, purified, cryopreserved Plasmodium falciparum [Pf] sporozoites [SPZ]) has been w

228 The M17 aminopeptidases from Plasmodium falciparum (PfA-M17) and Plasmodium vivax (Pv

229 Here we demonstrate that myosin A from Plasmodium falciparum (PfMyoA) is critical for red blood

230 Here, we demonstrate that Plasmodium falciparum phosphodiesterase beta (PDEbeta) h

231 rasensitive detection and differentiation of Plasmodium falciparum, Plasmodium vivax, Plasmodium oval

232 carried out with the human malaria parasite Plasmodium falciparum played a key role in determining t

233 sing in Southeast Asia and the prevalence of Plasmodium falciparum polymorphisms associated with resi

234 PIMMS43 genetic structure across African Plasmodium falciparum populations indicates allelic adap

235 ral antibody response profile of seven novel Plasmodium falciparum pre-erythrocytic antigens and thei

236 movement patterns and their relationship to Plasmodium falciparum prevalence.

237 We identified a selective inhibitor of the Plasmodium falciparum protein kinase PfCLK3, which we us

238 ed on a recombinant fragment of VAR2CSA, the Plasmodium falciparum protein responsible for binding to

239 d nucleotide derivatives designed to inhibit Plasmodium falciparum purine metabolism.

240 infectivity, and ultimately pathogenesis of Plasmodium falciparum rely on a macromolecular complex,

241 Plasmodium falciparum remains a serious public health pr

242 efforts, a highly effective vaccine against Plasmodium falciparum remains elusive.

243 Plasmodium falciparum remains responsible for most impor

244 Malaria caused by Plasmodium falciparum remains the leading single-agent c

245 ears, an efficacious subunit vaccine against Plasmodium falciparum remains to be licensed and deploye

246 Infection with Plasmodium falciparum results in immune dysfunction char

247 ng vaccination of non-human primates against Plasmodium falciparum reticulocyte binding protein homol

248 The Plasmodium falciparum reticulocyte-binding protein homol

249 ro cultivation of the erythrocytic stages of Plasmodium falciparum revolutionized research on this im

250 f the most deadly form of malaria in humans, Plasmodium falciparum, RIFINs form the largest family of

251 collected cord blood, measured antibodies to Plasmodium falciparum Schizont Egress Antigen-1 (PfSEA-1

252 himpanzee parasite Plasmodium reichenowi and Plasmodium falciparum sequences.

253 In this study, we show that Plasmodium falciparum skewed CD16+ DC cytokine responses

254 apid diagnostic tests (RDTs) that detect the Plasmodium falciparum-specific histidine-rich protein 2

255 llance every 1-3 months using microscopy and Plasmodium falciparum-specific loop-mediated isothermal

256 For Plasmodium falciparum specimens only, the sensitivity fo

257 vectors of the disease, ivermectin inhibits Plasmodium falciparum sporogonic and blood stage develop

258 uine induces protection against a homologous Plasmodium falciparum sporozoite (PfSPZ) challenge, but

259 The whole Plasmodium falciparum sporozoite (PfSPZ) vaccine is bein

260 RTS,S is based on the major Plasmodium falciparum sporozoite surface antigen, circum

261 after repeated administration of attenuated Plasmodium falciparum sporozoite vaccine.

262 ntrolled human malaria infection (CHMI) with Plasmodium falciparum sporozoites.

263 Longitudinal tracking of individual Plasmodium falciparum strains in multi-clonal infections

264 s of both sensitive and multi-drug-resistant Plasmodium falciparum strains.

265 tral role that erythrocyte invasion plays in Plasmodium falciparum survival and reproduction makes th

266 ne and identify that clemastine binds to the Plasmodium falciparum TCP-1 ring complex or chaperonin c

267 have a disproportionately greater impact on Plasmodium falciparum than on P. vivax in areas where bo

268 ids are more diverse in the malaria parasite Plasmodium falciparum than previously postulated as we u

269 In the human malaria parasite Plasmodium falciparum, the clustering of a family of vir

270 series of pyrazolo[3,4-b]pyridines targeting Plasmodium falciparum, the deadliest species of the mala

271 Plasmodium falciparum, the etiological agent of severe h

272 For the causative agent of malaria, Plasmodium falciparum, the high recombination rates and

273 A contributing to antimalarial resistance in Plasmodium falciparum, the most virulent human malaria p

274 For Plasmodium falciparum, the most virulent of the human ma

275 antitative and fully-electronic detection of Plasmodium falciparum, the predominant malaria-causing p

276 In the malaria parasite Plasmodium falciparum, the switch from asexual multiplic

277 For Plasmodium falciparum, these reports are scarce, and the

278 es, attributed to the increased tolerance of Plasmodium falciparum to artemisinin.

279 f the merozoite form of the malaria parasite Plasmodium falciparum to invade red blood cells (RBCs).

280 Resistance of Plasmodium falciparum to sulfadoxine-pyrimethamine threa

281 le stages prompt the human malaria parasite, Plasmodium falciparum, to acquire sophisticated molecula

282 o determine their role in cytoadherence, two Plasmodium falciparum transgenic lines expressing two va

283 Plasmodium falciparum transmission depends on mature gam

284 te, characterization of humoral responses to Plasmodium falciparum transmission-blocking vaccine cand

285 n unstructured 104mer synthetic peptide from Plasmodium falciparum trophozoite exported protein 1 (TE

286 Multidrug-resistant Plasmodium falciparum undermines the efficacy of current

287 s that the most lethal of malaria parasites, Plasmodium falciparum, uses to sense nutrient levels and

288 he parasite growth rate of the 3D7 strain of Plasmodium falciparum using data from 177 subjects from

289 study this part of the malaria life cycle in Plasmodium falciparum using PfAP2-G, the master regulato

290 Plasmodium falciparum vaccine RTS,S/AS01 is based on the

291 superfamily, and whether the family includes Plasmodium falciparum variant surface proteins, such as

292 nostic tests (mRDTs), which generally detect Plasmodium falciparum via its abundant histidine-rich pr

293 e responses, we recombined vectors to encode Plasmodium falciparum virulence factors: two cysteine-ri

294 eholds had at least one member infected with Plasmodium falciparum, vivax, and/or ovale spp. 47% of c

295 ead to the death of the unicellular parasite Plasmodium falciparum, we investigated its recombinase,

296 k of P vivax parasitaemia after treatment of Plasmodium falciparum with commonly used antimalarial dr

297 practice, we combined predicted surfaces of Plasmodium falciparum with country-specific ratios of P

298 ria infections can carry multiple strains of Plasmodium falciparum with varying levels of relatedness

299 The spreading of malaria parasites, Plasmodium falciparum, with resistance to all known drug

300 th, proliferation, and egress of blood-stage Plasmodium falciparum, yet our understanding of Ca(2+) s