ライフサイエンスコーパス: Porphyromonas gingivalis

1 bacterial species, Streptococcus mutans and Porphyromonas gingivalis.

2 clearance by opportunistic pathogens such as Porphyromonas gingivalis.

3 ontal disease, whose main infective agent is Porphyromonas gingivalis.

4 of F. alocis are enhanced in coculture with Porphyromonas gingivalis.

5 roded titanium surfaces on the attachment of Porphyromonas gingivalis.

6 etween HIV-1 and an invasive oral bacterium, Porphyromonas gingivalis.

7 obulin (TG), Escherichia coli LPS, or intact Porphyromonas gingivalis.

8 ood mononuclear cells (PBMCs) in response to Porphyromonas gingivalis.

9 rally infected with the periodontal pathogen Porphyromonas gingivalis.

10 m johnsoniae and the nonmotile oral pathogen Porphyromonas gingivalis.

11 isease that is triggered by bacteria such as Porphyromonas gingivalis.

12 . pylori as well as Streptococcus mutans and Porphyromonas gingivalis.

13 y Actinobacillus actinomycetemcomitans), and Porphyromonas gingivalis.

14 f LPS purified from the periodontal pathogen Porphyromonas gingivalis.

15 eriodontal disease is an anaerobic bacterium-Porphyromonas gingivalis.

16 Mice were orally infected with Porphyromonas gingivalis 15 times over 2 weeks.

17 er actinomycetemcomitans (MT4/MSP: 42%/36%), Porphyromonas gingivalis (78%/66%), Tannerella forsythia

18 Culture supernatants of Porphyromonas gingivalis, a bacterium that secretes prot

19 Porphyromonas gingivalis, a Gram-negative asaccharolytic

20 Porphyromonas gingivalis, a key periodontal pathogen, is

21 erase Chain Reaction (qPCR) for detection of Porphyromonas gingivalis, a keystone pathogen and to ass

22 The peritoneal infection model using Porphyromonas gingivalis, a keystone pathogen for period

23 important role in eliciting inflammation to Porphyromonas gingivalis, a keystone pathogen in periodo

24 rted between RA and periodontal disease, and Porphyromonas gingivalis, a known driver of periodontiti

25 Porphyromonas gingivalis, a major etiologic agent of per

26 In contrast, infection with Porphyromonas gingivalis, a principal pathogen in chroni

27 nalysed in mice after oral administration of Porphyromonas gingivalis, a representative periodontopat

28 n 9 times with 10(9) colony-forming units of Porphyromonas gingivalis A7436 through an oral gavage mo

29 he chronic periodontitis-associated pathogen Porphyromonas gingivalis activates a Toll-like receptor

30 operties against three periodontal bacteria: Porphyromonas gingivalis, Aggregatibacter actinomycetemc

31 Levels of Porphyromonas gingivalis, Aggregatibacter actinomycetemc

32 The susceptibility of Porphyromonas gingivalis, Aggregatibacter actinomycetemc

33 contamination with >/=3 specific pathogens (Porphyromonas gingivalis, Aggregatibacter actinomycetemc

34 issue-degrading enzymes in the oral pathogen Porphyromonas gingivalis Although a number of subunits o

35 re Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis amounts in saliva and their ant

36 lipid A structures in the outer membrane of Porphyromonas gingivalis, an agent of human periodontal

37 Porphyromonas gingivalis, an asaccharolytic member of th

38 eases are critical virulence determinants of Porphyromonas gingivalis, an emerging Alzheimer's diseas

39 Porphyromonas gingivalis,an anaerobic bacterium strongly

40 killing of periodontal pathogens, including Porphyromonas gingivalis and Aggregatibacter actinomycet

41 Porphyromonas gingivalis and Aggregatibacter actinomycet

42 he same biofilm plus the periodontopathogens Porphyromonas gingivalis and Aggregatibacter actinomycet

43 iovascular disease, such as the link between Porphyromonas gingivalis and atherosclerosis.

44 54, are produced by the periodontal pathogen Porphyromonas gingivalis and can be detected in lipid ex

45 Porphyromonas gingivalis and especially Prevotella inter

46 Porphyromonas gingivalis and Filifactor alocis are fasti

47 ring inhibition of the periodontal pathogens Porphyromonas gingivalis and Fusobacterium nucleatum gro

48 odontitis was induced by oral inoculation of Porphyromonas gingivalis and Fusobacterium nucleatum in

49 ) on the virulence of a mixed infection with Porphyromonas gingivalis and Fusobacterium nucleatum in

50 Although not statistically significant, Porphyromonas gingivalis and Fusobacterium nucleatum occ

51 Periodontal pathogens such as Porphyromonas gingivalis and Fusobacterium nucleatum pro

52 ribosomal RNA analysis of keystone pathogen Porphyromonas gingivalis and its consortium members Fuso

53 se in 2 animal models through inoculation of Porphyromonas gingivalis and ligature around the murine

54 Mice were orally infected with human Porphyromonas gingivalis and observed for 42 days.

55 mDC microbiome by 16S rDNA sequencing showed Porphyromonas gingivalis and other species, including (c

56 inulin and whether LS inhibits the growth of Porphyromonas gingivalis and Prevotella intermedia.

57 d oral inoculations of periodontal pathogens Porphyromonas gingivalis and Prevotella nigrescens induc

58 The Orange-Red cluster score (that included Porphyromonas gingivalis and Prevotella spp.) was positi

59 d Treponema denticola, and the prevalence of Porphyromonas gingivalis and T. denticola associated sig

60 ted with periodontal pathology and number of Porphyromonas gingivalis and Tannerella forsythia (forme

61 tus), two red-complex periodontal pathogens (Porphyromonas gingivalis and Tannerella forsythia), and

62 es that gram-negative oral bacteria, such as Porphyromonas gingivalis and Tannerella forsythia, use d

63 The keystone pathogen Porphyromonas gingivalis and the accessory pathogen Stre

64 The periodontal pathogen Porphyromonas gingivalis and the endodontic species Ente

65 modes of mucosal vaccination with whole-cell Porphyromonas gingivalis and to test the role of various

66 The recolonization of Porphyromonas gingivalis and Treponema denticola was red

67 how galactose-inhibitable coaggregation with Porphyromonas gingivalis and were defective in cell bind

68 presence of specific bacterial species (i.e. Porphyromonas gingivalis) and their effects in immune re

69 the proteases of Pseudomonas aeruginosa and Porphyromonas gingivalis, and enables its antimicrobial

70 uced the numbers of Streptococcus anginosus, Porphyromonas gingivalis, and Fusobacterium nucleatum, a

71 iodontitis is initiated by bacteria, such as Porphyromonas gingivalis, and is caused largely by host

72 ously Actinobacillus actinomycetemcomitans), Porphyromonas gingivalis, and Prevotella intermedia.

73 tory responses to a keystone oral bacterium, Porphyromonas gingivalis, and restrains periodontal infl

74 or more overtly pathogenic organisms such as Porphyromonas gingivalis, and the two species assemble i

75 Anti-Porphyromonas gingivalis antibody testing was performed

76 , FimA and Mfa1, of the periodontal pathogen Porphyromonas gingivalis are responsible for adherence t

77 rial manipulation of neutrophil responses by Porphyromonas gingivalis as a mechanism that contributes

78 early and late colonizer pathogens, such as Porphyromonas gingivalis, as the biofilm ages and period

79 gnaling in alveolar bone resorption, using a Porphyromonas gingivalis-associated ligature-induced per

80 , in particular, Fusobacterium nucleatum and Porphyromonas gingivalis, based on results from epidemio

81 gens (Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Campylobacter rectus, and Tann

82 However, the periodontal pathogen Porphyromonas gingivalis can control C5aR activation by

83 en" where low-abundance microbial pathogens (Porphyromonas gingivalis) can orchestrate inflammatory d

84 the periodontitis-associated oral bacterium Porphyromonas gingivalis, can subvert host immunity to r

85 rated that antibodies raised in mice against Porphyromonas gingivalis caused fetal loss in a mouse pr

86 The gram-negative anaerobe Porphyromonas gingivalis colonizes the gingival crevice

87 in substrates of a novel secretion system of Porphyromonas gingivalis contain a conserved C-terminal

88 Porphyromonas gingivalis contains lipid A with structura

89 A modest effect of DHA + aspirin on Porphyromonas gingivalis counts was associated with 14%

90 zation (red complex-KCNK1, p = 3.4 x 10(-7); Porphyromonas gingivalis-DAB2IP, p = 1.0 x 10(-6)).

91 echanism by which the opportunistic pathogen Porphyromonas gingivalis dampens innate immune responses

92 ved exaggerated proinflammatory responses to Porphyromonas gingivalis-derived lipopolysaccharide in c

93 ignificantly increased in the RA group, only Porphyromonas gingivalis displayed significant correlati

94 human gingival epithelial (HGEp) cells with Porphyromonas gingivalis disrupts barrier function by in

95 We show here that Porphyromonas gingivalis Dps (PgDps), previously describ

96 complex), produced by the keystone pathogen Porphyromonas gingivalis, dramatically increased their a

97 0068, Fusobacterium nucleatum ATCC10953, and Porphyromonas gingivalis DSM20709) were placed in a seri

98 periodontitis caused by oral infection with Porphyromonas gingivalis enhances articular bone loss.

99 that infection with the periodontal pathogen Porphyromonas gingivalis enhances the activity of Janus

100 ndard chow diet were given an oral gavage of Porphyromonas gingivalis for 15 wk.

101 ., Peptoniphilaceae [G-1] bacterium HMT 113, Porphyromonas gingivalis, Fretibacterium fastidiosum, Fi

102 taxa implicated in periodontitis, including Porphyromonas gingivalis, Fretibacterium fastidiosum, Fi

103 latter consisted of five microbial species (Porphyromonas gingivalis, Fusobacterium nucleatum, Actin

104 ntal pathogens such as Streptococcus mutans, Porphyromonas gingivalis, Fusobacterium nucleatum, and P

105 Single and multi-species biofilms of Porphyromonas gingivalis, Fusobacterium nucleatum, and S

106 , traditional periodontal pathogens, such as Porphyromonas gingivalis, Fusobacterium nucleatum, and T

107 Additionally, we compare a nearly complete Porphyromonas gingivalis genome to previously published

108 Encapsulation of Porphyromonas gingivalis has been demonstrated as respon

109 Porphyromonas gingivalis has been implicated in the etio

110 Porphyromonas gingivalis has orthologs for each of these

111 virulence factor of the periodontal pathogen Porphyromonas gingivalis, has been shown to induce prote

112 The oral microbiome, and specifically Porphyromonas gingivalis, has long been implicated.

113 of Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis have been shown to induce diffe

114 Studies with one of these bacteria, Porphyromonas gingivalis, have revealed that it contains

115 dy, we hypothesized that histatin 5 binds to Porphyromonas gingivalis hemagglutinin B (HagB) and atte

116 ed/attenuated the CXCL1 and TNF responses to Porphyromonas gingivalis hemagglutinin B (HagB).

117 of IL-6 by whole blood cells stimulated with Porphyromonas gingivalis HmuY protein.

118 the mechanism of action of ROS stimulated by Porphyromonas gingivalis in gingival epithelial cells.

119 ion, immunoglobulin (Ig)G subclasses against Porphyromonas gingivalis in individuals with pre-RA and

120 ly Actinobacillus actinomycetemcomitans) and Porphyromonas gingivalis in patients with chronic period

121 ne the impact of RvE1 on the phagocytosis of Porphyromonas gingivalis in T2D.

122 tory stimulus (lipopolysaccharide [LPS] from Porphyromonas gingivalis) in a manner consistent with th

123 Prevotella intermedia and Porphyromonas gingivalis increased in the control group.

124 acid (FA) levels on alveolar bone loss in a Porphyromonas gingivalis-induced model of periodontal di

125 wild-type (WT) controls in a murine model of Porphyromonas gingivalis-induced periodontitis and repor

126 s demonstrated in distinct models, including Porphyromonas gingivalis-induced periodontitis, ligature

127 se (DIO) mice and improves the resolution of Porphyromonas gingivalis-induced periodontitis.

128 etiological agent of chronic periodontitis, Porphyromonas gingivalis, infect blood myeloid dendritic

129 wild-type and Msx2 knock-in Swiss mice using Porphyromonas gingivalis infected ligatures.

130 ression was higher in periodontal tissues of Porphyromonas gingivalis-infected mice as compared with

131 Porphyromonas gingivalis is a Gram-negative obligate ana

132 Porphyromonas gingivalis is a Gram-negative obligately a

133 The oral bacterium Porphyromonas gingivalis is a key etiological agent of h

134 vity by the Gram-negative bacterial pathogen Porphyromonas gingivalis is a key event in the initiatio

135 Porphyromonas gingivalis is a keystone pathogen of perio

136 Porphyromonas gingivalis is a keystone pathogen that con

137 Porphyromonas gingivalis is a keystone pathogen, and its

138 Porphyromonas gingivalis is a keystone periodontal patho

139 Porphyromonas gingivalis is a major pathogen in periodon

140 Porphyromonas gingivalis is a member of the dysbiotic or

141 Porphyromonas gingivalis is a member of the human oral m

142 Porphyromonas gingivalis is a pathogen in severe periodo

143 Porphyromonas gingivalis is a peptide-fermenting asaccha

144 Porphyromonas gingivalis is a periodontal pathogen impli

145 Porphyromonas gingivalis is a predominant pathogen that

146 Porphyromonas gingivalis is a primary etiological agent

147 Porphyromonas gingivalis is able to invade and modulate

148 Porphyromonas gingivalis is an established pathogen in p

149 Overabundance of Porphyromonas gingivalis is associated with shorter surv

150 hereas the phylogenetically related pathogen Porphyromonas gingivalis is associated with the chronic

151 Porphyromonas gingivalis is considered a keystone pathog

152 Porphyromonas gingivalis is considered a keystone pathog

153 Porphyromonas gingivalis is intimately associated with p

154 rthermore, the mechanism of TLR induction by Porphyromonas gingivalis is investigated in human gingiv

155 Porphyromonas gingivalis is one of the oral microorganis

156 Porphyromonas gingivalis is strongly implicated in perio

157 By far, Porphyromonas gingivalis is the best-studied periodontal

158 Porphyromonas gingivalis is the major causative agent of

159 st response to lipopolysaccharide (LPS) from Porphyromonas gingivalis is unusual inasmuch as differen

160 The Gram-negative anaerobe, Porphyromonas gingivalis, is a key periodontal pathogen,

161 virulence of periodontal pathogens, such as Porphyromonas gingivalis, is expressed in the context of

162 umerous chronic infectious agents, including Porphyromonas gingivalis, is shown to drive-differentiat

163 In monoinfection, heat-killed Porphyromonas gingivalis led to augmented levels of IL-1

164 Porphyromonas gingivalis, like other bacteria belonging

165 Porphyromonas gingivalis lipid A heterogeneity modulates

166 olated from C57BL/6J mice were cultured with Porphyromonas gingivalis lipopolysaccharide (LPS) and cy

167 ation and inflammatory gene expression using Porphyromonas gingivalis lipopolysaccharide (LPS) as a p

168 with three consecutive palatal injections of Porphyromonas gingivalis lipopolysaccharide (LPS) at 48-

169 roducts (AGE) in the presence and absence of Porphyromonas gingivalis lipopolysaccharide (LPS) on IL-

170 erichia coli lipopolysaccharide (Ec-LPS) and Porphyromonas gingivalis lipopolysaccharide (Pg-LPS) sti

171 resence and absence of ultrapure or standard Porphyromonas gingivalis lipopolysaccharide (PgLPS), Pam

172 ived IFN-gamma in constitutively released or Porphyromonas gingivalis lipopolysaccharide (PgLPS)-stim

173 was induced via silk ligature placement with Porphyromonas gingivalis lipopolysaccharide injection in

174 ls upon stimulation by heat-killed wild-type Porphyromonas gingivalis, live P. gingivalis protease-de

175 a [TNF-alpha]), bacterial virulence factors (Porphyromonas gingivalis LPS) or a combination in a biom

176 In this study we report that Porphyromonas gingivalis LPS, a TLR2 agonist, fails to i

177 y be caused by periodontal bacteria, such as Porphyromonas gingivalis Mast cells are sentinels at muc

178 ain colonization by the periodontal pathogen Porphyromonas gingivalis may link these two inflammatory

179 We postulated that the periodontal pathogen, Porphyromonas gingivalis may suppress the inflammasome a

180 Recent reports indicate that Porphyromonas gingivalis mediates alveolar bone loss or

181 Although the periodontal pathogen Porphyromonas gingivalis must withstand high levels of n

182 Gram-negative, anaerobic periodontopathogen Porphyromonas gingivalis must withstand nitrosative stre

183 he present study is to analyze the effect of Porphyromonas gingivalis on differentiation of primary o

184 (LPS) purified from the periodontal pathogen Porphyromonas gingivalis on human macrophages.

185 e direct effects of the periodontal pathogen Porphyromonas gingivalis on osteoclast differentiation a

186 ly reduced in both groups at 3 and 6 months, Porphyromonas gingivalis only in the test group.

187 esponsiveness of whole blood stimulated with Porphyromonas gingivalis or Escherichia coli LPS were mo

188 experimental periodontitis induced by either Porphyromonas gingivalis or ligature, gamma-proteobacter

189 g a novel strain of the periodontal pathogen Porphyromonas gingivalis (P. gingivalis JCVI SC001) usin

190 nts with chronic periodontitis and intraoral Porphyromonas gingivalis (P. gingivalis) and Tannerella

191 analogs was synthesized and their effects on Porphyromonas gingivalis (P. gingivalis) elicited inflam

192 dontal health by investigating its effect on Porphyromonas gingivalis (P. gingivalis), lipopolysaccha

193 Tannerella forsythia (T.f.), 2.5 x 10(4) for Porphyromonas gingivalis (P.g.), 5.3 x 10(3) for Prevote

194 Porphyromonas gingivalis (P.g.)-induced TNF-alpha can be

195 tudy is to determine whether the presence of Porphyromonas gingivalis peptidylarginine deiminase (PPA

196 an and mouse macrophages exposed to LPS from Porphyromonas gingivalis (Pg LPS).

197 ucleic acids (DNA) of periodontal pathogens, Porphyromonas gingivalis (Pg) and Tannerella forsythia,

198 nis (Ss) and Sg/Fusobacterium nucleatum (Fn)/Porphyromonas gingivalis (Pg) biofilms elicited signific

199 Porphyromonas gingivalis (Pg) capsule enables evasion fr

200 LM group also presented greater reduction of Porphyromonas gingivalis (Pg) DNA counts at 6 months (P

201 This study compared the effect of Porphyromonas gingivalis (Pg) infection and experimental

202 Porphyromonas gingivalis (Pg) is a keystone pathogen in

203 Porphyromonas gingivalis (Pg) is a major periodontal pat

204 or TRAM, HGF and HPDLF were stimulated with Porphyromonas gingivalis (Pg) lipopolysaccharide (LPS) o

205 n vitro study examines the effect of EGCG on Porphyromonas gingivalis (Pg) lipopolysaccharide (LPS)-e

206 6 cells were used to determine the effect of Porphyromonas gingivalis (Pg) LPS on insulin secretion.

207 al administration of a periodontal pathogen, Porphyromonas gingivalis (Pg) to WT mice results in insu

208 The interaction of the periodontal pathogen Porphyromonas gingivalis (Pg) with commensal streptococc

209 Quantification of Tannerella forsythia (Tf), Porphyromonas gingivalis (Pg), Aggregatibacter actinomyc

210 Total amount of Porphyromonas gingivalis (Pg), Aggregatibacter actinomyc

211 Aggregatibacter actinomycetemcomitans (Aa), Porphyromonas gingivalis (Pg), Campylobacter rectus (Cr)

212 ates biofilms, consisting of species such as Porphyromonas gingivalis (Pg), in the etiology of peri-i

213 were used for analysis of bacterial DNA for Porphyromonas gingivalis (Pg), Prevotella intermedia (Pi

214 y associated with the submucosal presence of Porphyromonas gingivalis (Pg), Prevotella intermedia (Pi

215 factors from periodontal pathogens, such as Porphyromonas gingivalis (Pg), stimulate the respiratory

216 Quantitative analysis of Porphyromonas gingivalis (Pg), Tannerella forsythia (Tf)

217 on of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis (Pg), Tannerella forsythia (Tf)

218 study investigated the role of GRHL2 in the Porphyromonas gingivalis (Pg)-induced impairment of epit

219 tis (RA), specifically regarding the role of Porphyromonas gingivalis (Pg).

220 HP1 monocytic cells following infection with Porphyromonas gingivalis (Pg).

221 by ligature; 3) group G-Pg: oral gavage with Porphyromonas gingivalis (Pg); 4) group G-PgFn: oral gav

222 We documented earlier that Porphyromonas gingivalis(Pg), keystone oral-pathobiont,

223 AW264.7 and human monocyte THP-1 to LPS from Porphyromonas gingivalis (PgLPS), an oral microbe implic

224 of periodontitis and the anaerobic bacterium Porphyromonas gingivalis plays a key role in driving chr

225 SprF exhibits some similarity to Porphyromonas gingivalis PorP, which is required for sec

226 SprE is similar in sequence to Porphyromonas gingivalis PorW, which is required for sec

227 Porphyromonas gingivalis, Prevotella intermedia, and T.

228 owth inhibition of the periodontal pathogens Porphyromonas gingivalis, Prevotella intermedia, Fusobac

229 are anaerobic and include organisms such as Porphyromonas gingivalis, Prevotella intermedia, Fusobac

230 his adipokine and the presence and levels of Porphyromonas gingivalis, Prevotella intermedia, Prevote

231 is of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Prevotella intermedia, Tannere

232 cteroidetes sp., Porphyromonas endodontalis, Porphyromonas gingivalis, Prevotella spp., Tannerella fo

233 c periodontitis, the Gram-negative bacterium Porphyromonas gingivalis, produces a vast arsenal of vir

234 The Porphyromonas gingivalis recombinant VimA can interact w

235 by Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis, respectively.

236 show that intraoral inoculation of mice with Porphyromonas gingivalis resulted in infection, alveolar

237 Porphyromonas gingivalis secretes a serine phosphatase e

238 A key pathogen, Porphyromonas gingivalis, secretes gingipains, cysteine

239 Porphyromonas gingivalis showed the greatest reduction i

240 enotype was also observed following LPS from Porphyromonas gingivalis stimulation during osteogenic d

241 The Porphyromonas gingivalis strain ATCC 33277 (33277) and 3

242 sms of oral species Fusobacterium nucleatum, Porphyromonas gingivalis, Streptococcus mutans, and Camp

243 Bacterial loads of Porphyromonas gingivalis, T. forsythia, Parvimonas micra

244 of pathogens related to periodontal disease (Porphyromonas gingivalis, Tannerella forsythensis, Trepo

245 alysis identified twenty-one OTUs, including Porphyromonas gingivalis, Tannerella forsythia and Filif

246 Levels of Porphyromonas gingivalis, Tannerella forsythia, Aggregat

247 ding on probing and levels of microorganisms Porphyromonas gingivalis, Tannerella forsythia, and Camp

248 es were collected to determine the levels of Porphyromonas gingivalis, Tannerella forsythia, and Fuso

249 ls of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, and Fuso

250 ase) in GCF and subgingival plaque levels of Porphyromonas gingivalis, Tannerella forsythia, and Fuso

251 revealed that periodontal pathogens, such as Porphyromonas gingivalis, Tannerella forsythia, and Prev

252 lifactor alocis, Fretibacterium fastidiosum, Porphyromonas gingivalis, Tannerella forsythia, and Sele

253 on of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, and Trep

254 d for Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, and Trep

255 using immunofluorescence for the presence of Porphyromonas gingivalis, Tannerella forsythia, Fusobact

256 n for Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Fusobact

257 of bacterial DNA from Streptococcus mutans, Porphyromonas gingivalis, Tannerella forsythia, Prevotel

258 eria (Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Prevotel

259 T1 decreased the counts of Porphyromonas gingivalis, Tannerella forsythia, Prevotel

260 rial, Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Treponem

261 ts of Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Treponem

262 eria (Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Treponem

263 Eikenella corrodens, Campylobacter concisus, Porphyromonas gingivalis, Tannerella forsythia, Treponem

264 ctus, Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Tannerella forsythia, Treponem

265 ases and are more likely to be infected with Porphyromonas gingivalis than non-smokers.

266 ce factor secreted by the periodontopathogen Porphyromonas gingivalis that attacks host vasculature a

267 which activity was induced by infection with Porphyromonas gingivalis The expression of several miRNA

268 Porphyromonas gingivalis, the causative agent of adult p

269 In the periodontal pathogen Porphyromonas gingivalis, the CTD is cleaved off by PorU

270 biosis and preponderance of bacteria such as Porphyromonas gingivalis, the main etiological agent of

271 have evaluated the periopathogenic roles of Porphyromonas gingivalis, the oral microbiome, and mecha

272 ntly reported that the oral mucosal pathogen Porphyromonas gingivalis, through its 67-kDa Mfa1 (minor

273 gainst invader pathobiotic bacteria, such as Porphyromonas gingivalis, through small danger molecule

274 ptococcus gordonii SspB (AgI/II) is bound by Porphyromonas gingivalis, thus promoting oral colonizati

275 The adaptation of Porphyromonas gingivalis to H2O2-induced stress while in

276 biofilm formation promoted the tolerance of Porphyromonas gingivalis to oxidative stress under micro

277 SCs were seeded for 24 h and challenged with Porphyromonas gingivalis total protein extract (PgPE) (0

278 y, the abundance of the periodontal pathogen Porphyromonas gingivalis trended with higher risk of ESC

279 IgG levels to several species, including Porphyromonas gingivalis, Treponema denticola, and Campy

280 moderate evidence supporting association of Porphyromonas gingivalis, Treponema denticola, and Tanne

281 ed with polybacterial inoculum consisting of Porphyromonas gingivalis, Treponema denticola, and Tanne

282 For Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Treponema denticola, and Tanne

283 Porphyromonas gingivalis, Treponema denticola, and Tanne

284 drugs against red complex pathogens, namely, Porphyromonas gingivalis, Treponema denticola, and Tanne

285 ealing of abutments, rats were infected with Porphyromonas gingivalis, Treponema denticola, and Tanne

286 uding Aggregatibacter actinomycetemcomitans, Porphyromonas gingivalis, Treponema denticola, Tannerell

287 the ability of a polymicrobial consortium of Porphyromonas gingivalis, Treponema denticola, Tannerell

288 Porphyromonas gingivalis, Treponema denticola, Tannerell

289 periodontal pathogens Prevotella intermedia, Porphyromonas gingivalis, Treponema denticola, Tannerell

290 r 30 seconds and 1 minute, reducing, mainly, Porphyromonas gingivalis viability, with 2.78 and 1.7 lo

291 ic Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis viability.

292 The Porphyromonas gingivalis VimA protein has multifunctiona

293 Mice were infected with the oral pathogen Porphyromonas gingivalis W50 (P. gingivalis) in the maxi

294 Porphyromonas gingivalis W83 biofilm was cultured on tit

295 f F. alocis and its ability to interact with Porphyromonas gingivalis W83, several clinical isolates

296 An inferior frequency detection of Porphyromonas gingivalis was observed in the PDT protoco

297 nd the growth of Streptococcus sanguinis and Porphyromonas gingivalis was significantly reduced in th

298 ll-characterized human periodontal pathogen, Porphyromonas gingivalis We found that oral mucosal LCs

299 virulence factors of the periodontopathogen Porphyromonas gingivalis, which causes chronic periodont

300 ns Aggregatibacter actinomycetemcomitans and Porphyromonas gingivalis with extended NO-release kineti